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1 COS-7 cells by measuring the uptake of [(3)H]estrone sulfate.
2 n to the large hydrophobic organic substrate estrone sulfate.
3 k (odds ratio doubling, 0.82 [SHBG] to 1.37 [estrone sulfate]).
4 est compounds (10 microM) in the presence of estrone sulfate (1 microM) as the only source of estroge
7 the short OATP2B1 variant toward substrates estrone sulfate and rosuvastatin are similar to the well
8 strates MK571, probenecid, taurocholic acid, estrone sulfate, and bromosulfophthalein and inhibited b
10 or probenecid was observed for taurocholate, estrone sulfate, and para-aminohippurate in renal slices
11 Estradiol, bioavailable estradiol, estrone, estrone sulfate, androstenedione, testosterone, dehydroe
12 ic anions such as p-aminohippurate (PAH) and estrone sulfate as well as the basic compound, cimetidin
13 lating estrone (E(1)), estradiol (E(2)), and estrone sulfate decreased to 11%, 22%, and 13% of baseli
14 parately, were able to take up taurocholate, estrone sulfate, digoxin, and prostaglandin E(2), but no
16 he suppressed levels of plasma estradiol and estrone sulfate in postmenopausal women with early ER-po
18 13, and r = 0.30; P = .035 for estradiol and estrone sulfate, respectively), it was not with anastroz
19 ort function for the non-bile acid substrate estrone sulfate, suggesting this position may be part of
21 ther inclusion of plasma estradiol, estrone, estrone sulfate, testosterone, dehydroepiandrosterone su
22 eceptor-positive disease (selected hormones: estrone sulfate, testosterone, prolactin, and SHBG; chan
23 breast cancer cells may be the conversion of estrone sulfate to estrone by the enzyme estrone sulfata
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