ライフサイエンスコーパス: ethanolamine

1 histamine, glutamine, xanthurenic acid, and ethanolamine).

2 egulator gene was induced by the presence of ethanolamine.

3 anolamine, and the satiating factor N-oleoyl ethanolamine.

4 ay forming phosphatidylethanolamine from CDP-ethanolamine.

5 ctivase EutA protein under aerobic growth on ethanolamine.

6 (12) are necessary and sufficient to grow on ethanolamine.

7 nia lyase (EA-lyase), a catabolic enzyme for ethanolamine.

8 the metabolism of either 1,2-propanediol or ethanolamine.

9 the complex lipid, retinylidene-phosphatidyl-ethanolamine.

10 s-indacene-3-pentanoyl)-sn-glycero-3-phospho ethanolamine.

11 tment with the ceramidase inhibitor N-oleoyl ethanolamine.

12 elta strain can only grow in the presence of ethanolamine.

13 e and in the absence of exogenous choline or ethanolamine.

14 by phospholipids, particularly phosphatidyl ethanolamine.

15 uired for degradation of 1,2-propanediol and ethanolamine.

16 gomyelin and lysophosphatidylcholine, versus ethanolamine.

17 nhibit phosphatidylcholine biosynthesis from ethanolamine.

18 t of an aptamer-based assay for detection of ethanolamine.

19 mately 56%), which was partially restored by ethanolamine.

20 feedback regulation of FAAH activity by free ethanolamine.

21 zes these bioactive fatty acid conjugates of ethanolamine.

22 phosphatidylcholine or dioleoyl-phosphatidyl-ethanolamine.

23 hionate), which supports anaerobic growth on ethanolamine.

24 , filamentous growth, and is auxotrophic for ethanolamine.

25 es, phosphatidyl glycerols, and phosphatidyl ethanolamines.

26 AcsC together are able to condense citrate, ethanolamine, 2,4-diaminobutyrate, and alpha-ketoglutara

27 rmediates formed with the substrates, [1-13C]ethanolamine, [2-13C]ethanolamine, and unlabeled ethanol

28 he type 1 [choline: 3.4 +/- 1.5% (P < 0.01); ethanolamine: 5.9 +/- 2.5% (P < 0.05)] and type 2 [choli

29 in the control group (choline: 5.5 +/- 2.2%; ethanolamine: 7.5 +/- 2.5%).

30 In embryos and yolk sacs incubated with 3H-ethanolamine, 95% of recovered label was PtdEtn, but Ptd

31 Ethanolamine, a product of the breakdown of phosphatidyl

32 e bisretinoid compound diretinoid-pyridinium-ethanolamine (A2E) were increased in Rdh12-deficient mic

33 d with accumulation of diretinoid-pyridinium-ethanolamine (A2E), a condensation product of all-trans-

34 n products, including di-retinoid-pyridinium-ethanolamine (A2E), are thought to be transferred to RPE

35 dihydro-N-retinylidene-N-retinylphosphatidyl-ethanolamine (A2PE-H(2)), also accumulates in retinas of

36 s, phthalates, bisphenol A (BPA), triclosan, ethanolamines, alkylphenols, fragrances, glycol ethers,

37 Furthermore, in addition to PME synthesis, ethanolamine also contributes to the production of phosp

38 ata obtained support the conclusion that the ethanolamine ammnonia-lyase (EAL) enzyme (encoded by the

39 Ado-B12) is both the cofactor and inducer of ethanolamine ammonia lyase (EA-lyase), a catabolic enzym

40 netic field effect (MFE) investigations with ethanolamine ammonia lyase (EAL).

41 (9 microM) of the first degradative enzyme, ethanolamine ammonia lyase.

42 n coenzyme B12 (adenosylcobalamin)-dependent ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

43 dulation (ESEEM) spectroscopic properties of ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

44 (AdoCbl; coenzyme B(12)) in AdoCbl-dependent ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

45 eamination of S-2-aminopropanol catalyzed by ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

46 in the adenosylcobalamin (AdoCbl)-dependent ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

47 trate radical pair catalytic intermediate in ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

48 yzed by adenosylcobalamin (AdoCbl)-dependent ethanolamine ammonia-lyase (EAL).

49 bstrate, to study radical pair generation in ethanolamine ammonia-lyase from Salmonella typhimurium a

50 trate radical pair catalytic intermediate in ethanolamine ammonia-lyase from Salmonella typhimurium h

51 rrinoid adenosyltransferase and for the EutA ethanolamine ammonia-lyase reactivase.

52 s the sole mediator of hydrogen transfers in ethanolamine ammonia-lyase.

53 The model predicts that EutBC (ethanolamine-ammonia lyase) lies outside the compartment

54 The ethanolamine ammonialyase microcompartment of Escherichi

55 The endocannabinoid arachidonoyl ethanolamine (anandamide) is a lipid transmitter synthes

56 For example, N-arachidonoyl-ethanolamine and 2-arachidonoyl-glycerol can be metaboli

57 roarray assay relying on competition between ethanolamine and an oligonucleotide complementary to the

58 oxyl groups are chemically tagged with (15)N-ethanolamine and detected using a 2D heteronuclear corre

59 n the endoplasmic reticulum by fusion of CDP-ethanolamine and diacylglycerol.

60 Competitive binding of ethanolamine and fluorescently labeled complementary oli

61 cid amide hydrolase (FAAH) degrades NAE into ethanolamine and free fatty acid to terminate its signal

62 inner membrane [palmitoyloleoylphosphatidyl ethanolamine and palmitoyloleoylphosphatidylglycerol (PO

63 referential cleavage of the C-O bond between ethanolamine and phosphate, enabling the selective ident

64 itamin B(12) biosynthesis and degradation of ethanolamine and propanediol was apparently acquired by

65 uired during anoxic growth of S. enterica on ethanolamine and tetrathionate.

66 EPR spectra of samples prepared with [1-13C]ethanolamine and the absence of such splitting in spectr

67 e hydrolase (FAAH), which hydrolyzes NAEs to ethanolamine and their corresponding free fatty acids.

68 he related pigments, all-trans-retinal dimer-ethanolamine and unconjugated all-trans-retinal dimer, i

69 tic MWFs, as well as duration of exposure to ethanolamines and nitrosamines.

70 sed surface (PFP/protein G'/whole antibodies/ethanolamine) and one optimized Fab' fragment-based surf

71 hatidylethanolamine, all-trans-retinal dimer-ethanolamine, and all-trans-retinal dimer) increased wit

72 ay intermediates phosphoethanolamine and CDP-ethanolamine, and an increase in the methylated derivati

73 ite can acquire the lipid precursors serine, ethanolamine, and choline from its environment and use t

74 ble dithiophospholipids 2-10 having choline, ethanolamine, and l-serine headgroups were synthesized,

75 hanolamine generated all-trans-retinal dimer-ethanolamine, and protonation/deprotonation of the Schif

76 pyrolysis products include alanine, glycine, ethanolamine, and small dipeptides, and many of these, t

77 ide, the anti-inflammatory lipid N-palmitoyl ethanolamine, and the satiating factor N-oleoyl ethanola

78 the substrates, [1-13C]ethanolamine, [2-13C]ethanolamine, and unlabeled ethanolamine were acquired u

79 The established pathways from serine to ethanolamine are indirect and involve decarboxylation of

80 growth of an S. enterica cobA eutT strain on ethanolamine as a carbon and energy or nitrogen source.

81 Effective utilization of ethanolamine as a carbon and nitrogen source may provide

82 peron that allows Salmonella enterica to use ethanolamine as a sole source of nitrogen, carbon, and e

83 al functions needed by this bacterium to use ethanolamine as a source of carbon and energy.

84 sferase (SDPM) pathway using host serine and ethanolamine as precursors.

85 Here, the ability of E. faecalis to utilize ethanolamine as the sole carbon source is shown to be de

86 o survive on small organic molecules such as ethanolamine as the sole source for carbon and nitrogen.

87 These mutants fail to use ethanolamine at a low pH.

88 llowing chemical mutagenesis, we isolated an ethanolamine auxotroph that we designate pstA1-1.

89 reticulum and Psd1p that relies on isolating ethanolamine auxotrophs in suitable (psd2Delta) genetic

90 respectively, methoxyethanol (solvent), and ethanolamine (base).

91 resent distinct binding modes to the choline/ethanolamine-binding site of P. falciparum choline kinas

92 human disorder arising due to defective CDP-ethanolamine biosynthesis and provide new insight into t

93 a mixture of ethanolaminium bicarbonate and ethanolamine bisulphide is also produced.

94 phosphoethanolamine, an intermediate in the ethanolamine branch of the Kennedy pathway of phosphatid

95 d the synthesis of phospholipids via the CDP-ethanolamine branch of the Kennedy pathway were controll

96 esis of phosphatidylethanolamine via the CDP-ethanolamine branch of the Kennedy pathway.

97 Regulation involves specific sensing of ethanolamine by a sensor histidine kinase (EutW), result

98 nvolving conjugation of arachidonic acid and ethanolamine by fatty-acid amide hydrolase.

99 tion in the synthesis of PC from choline and ethanolamine by the compound.

100 n of the resulting glycerophospho-N-modified ethanolamines by liquid chromatography-tandem mass spect

101 Ethanolamine capping was applied to avoid unspecific int

102 osome is needed to concentrate low levels of ethanolamine catabolic enzymes, to keep the level of tox

103 particular genes involved in propanediol and ethanolamine catabolism and cobalamin biosynthesis.

104 The role of GSH in ethanolamine catabolism is complex and requires further

105 osome is not involved in the biochemistry of ethanolamine catabolism.

106 on of 1,2-propanediol and the eut operon for ethanolamine catabolism.

107 ica, a microcompartment encloses enzymes for ethanolamine catabolism.

108 e steady-state radical in the deamination of ethanolamine catalyzed by adenosylcobalamin (AdoCbl)-dep

109 l cloning strategy, we here identified a CDP-ethanolamine:ceramide ethanolamine phosphotransferase as

110 e methyl ester complex (5), 1-boraadamantane.ethanolamine complex (8), and (S)-2-[(adamantane-1-carbo

111 At pH 7.0, the standard ethanolamine concentration (41 mM) provides enough Eth0

112 When a lowered pH and/or ethanolamine concentration reduced the Eth0 concentratio

113 ted in fluorescence intensities dependent on ethanolamine concentration with a limit of detection of

114 e show here that use of a specific nutrient (ethanolamine) confers a marked growth advantage on Salmo

115 yltransferase with prominent activity toward ethanolamine-containing lysophospholipids, which we term

116 4), Ethanolamine-containing phosphoglycerides are generally

117 -adenosylmethionine-dependent methylation of ethanolamine-containing phospholipids to produce the abu

118 s an important enzyme in the biosynthesis of ethanolamine-containing phospholipids, especially in bra

119 ant for sicariid predatory behavior, because ethanolamine-containing sphingolipids are common in inse

120 enzyme B(12)- (adenosylcobalamin-) dependent ethanolamine deaminase from Salmonella typhimurium have

121 radical in vitamin B(12) coenzyme-dependent ethanolamine deaminase from Salmonella typhimurium have

122 le mediator of radical pair recombination in ethanolamine deaminase.

123 d to phospholipid by the cytidinediphosphate-ethanolamine-dependent Kennedy pathway.

124 arboamination reaction between a substituted ethanolamine derivative and an aryl or alkenyl bromide.

125 exhibited much greater activity with N-acyl ethanolamines (e.g. anandamide) and N-acyl taurines.

126 The ability of bacteria to utilize ethanolamine (EA) as a carbon and nitrogen source may co

127 Salmonellae can use ethanolamine (EA) as a sole source of carbon and nitroge

128 Ethanolamine (EA) is an important metabolite for EHEC in

129 Ethanolamine (EA) metabolism is a trait associated with

130 following the pyrolysis of citric acid (CA)-ethanolamine (EA) precursor at different temperatures.

131 In2O3 film precursor solutions consisting of ethanolamine (EAA) and InCl3 in methoxyethanol.

132 In2O3 film precursor solutions consisting of ethanolamine (EAA) and InCl3 in methoxyethanol.

133 However ethanolamine (ETA) content was lower.

134 Supplemental ethanolamine (ETA), which can be converted to PE via the

135 (Eth0) does not enter cells, while uncharged ethanolamine (Eth0) diffuses freely across the membrane.

136 Evidence is presented that protonated ethanolamine (Eth0) does not enter cells, while uncharge

137 Plants are known to synthesize ethanolamine (Etn) moieties by decarboxylation of free s

138 athway has evolved to be the major route for ethanolamine (EtN) synthesis, as EtN supplementation com

139 Blocking ceramide degradation with N-oleoyl-ethanolamine exacerbated Abeta cytotoxicity; and additio

140 Amides of fatty acids with ethanolamine (FAE) are biologically active lipids that p

141 were sensitive to corticosterone, selenium, ethanolamine, fatty acids and/or antioxidants.

142 enzymes that synthesize CDP-choline and CDP-ethanolamine for phosphatidylcholine and phosphatidyleth

143 olic labeling revealed an increased usage of ethanolamine for PtdEtn synthesis by the mutant.

144 The second pigment, A2-dihydropyridine-ethanolamine, forms from phosphate hydrolysis of A2-DHP-

145 Alanine, choline, ethanolamine, glucose, lactate, myoinositol, phosphochol

146 Surface dilution of PtdSer by choline, ethanolamine, glycerol, and inositol phospholipids marke

147 ospholipid and phosphatidylinositol than for ethanolamine glycerophospholipid and phosphatidylserine

148 antial decreases occurred in the choline and ethanolamine glycerophospholipid pools in murine myocard

149 EGF receptor-containing rafts contained more ethanolamine glycerophospholipids and less sphingomyelin

150 Ethanolamine glycerophospholipids are ubiquitous cell me

151 Leishmania synthesize the majority of their ethanolamine glycerophospholipids as 1-O-alk-1'-enyl-2-a

152 species; 2) alterations in both choline and ethanolamine glycerophospholipids, including a decreased

153 re [1-(14)C]16:0 was targeted to choline and ethanolamine glycerophospholipids, whereas more [1-(14)C

154 is well-defined in the electron density, the ethanolamine group is poorly defined, suggesting structu

155 OPA reacts with the ethanolamine head group of PE in human cells to form pyr

156 sphate, indicating that either a glycerol or ethanolamine headgroup is the chemical determinant for s

157 e recently, nonenzymatic modification of the ethanolamine headgroup of phosphatidylethanolamine (PE)

158 utions correlated with the capability of the ethanolamine headgroups to engage in hydrogen bonding wi

159 forms the corrinoid-dependent degradation of ethanolamine if given vitamin B12, but it can make B12 f

160 tment is used to sequester the metabolism of ethanolamine in bacteria such as Escherichia coli and Sa

161 Original data on the habitual presence of ethanolamine in beers are presented.

162 the rate of PtdEtn synthesis from exogenous ethanolamine in hepatocytes.

163 esis of phosphatidylcholine from choline and ethanolamine in P. falciparum, and provide evidence for

164 es, such as propionate, 1,2-propanediol, and ethanolamine, in addition to melibiose and ascorbate, th

165 ospholipase C treatment, or labeled by [(3)H]ethanolamine, indicating a glycosylphosphatidylinositol

166 ain a glycosylphosphatidylinositol moiety or ethanolamine, indicating that phospholipolysis is not in

167 Ethanolamine induced a 15-fold increase in the rate of a

168 y the absence of phospholipids with choline, ethanolamine, inositol, and serine head groups.

169 Salmonella enterica catabolizes ethanolamine inside a compartment known as the metabolos

170 the enzymatic machinery needed to metabolize ethanolamine into acetyl coenzyme A (acetyl-CoA).

171 ated with a decrease in the incorporation of ethanolamine into CDP-ethanolamine pathway intermediates

172 ve C3-alkylation of indoles with N-protected ethanolamines involving the "borrowing hydrogen" strateg

173 Ethanolamine is an abundant compound in the human intest

174 The choline/ethanolamine kinase (CEK) family catalyzes the initial s

175 The level of ethanolamine kinase activity increased when zinc was dep

176 ingly, these compounds primarily inhibit the ethanolamine kinase activity of the P. falciparum cholin

177 leted cells, indicating that the increase in ethanolamine kinase activity was attributed to a transcr

178 strated that the zinc-mediated regulation of ethanolamine kinase and the synthesis of phospholipids v

179 Regulation of the EKI1-encoded ethanolamine kinase by inositol and choline was examined

180 The repression of ethanolamine kinase by inositol supplementation correlat

181 Regulation of the EKI1-encoded ethanolamine kinase by the essential nutrient zinc was e

182 Ethanolamine kinase catalyzes the committed step in the

183 Ethanolamine kinase catalyzes the first step in the CDP-

184 ne was confirmed by corresponding changes in ethanolamine kinase mRNA, protein, and activity levels.

185 The eas(+) gene encodes for the protein Ethanolamine Kinase, involved in phospholipid biosynthes

186 Delta mutant defective in choline kinase and ethanolamine kinase, we examined the consequences of a b

187 hosphatidylation of eggPC in the presence of ethanolamine), lyso-phosphatidylcholine (LPC), and lyso-

188 in the IL-4-treated macrophages suggest that ethanolamine lysophospholipid (LPE) is an sPLA2-V-derive

189 only tested for PCCC applications, including ethanolamine (MEA), methyldiethanolamine (MDEA), and pip

190 s 2-arachidonoyl-glycerol and N-arachidonoyl-ethanolamine mediate an array of pro- and anti-inflammat

191 Our data show that the ethanolamine metabolosome is not involved in the biochem

192 like structure (hereafter referred to as the ethanolamine metabolosome) is thought to contain the enz

193 ith the protein, independent of the state of ethanolamine methylation, with introduction of polyunsat

194 t drug moieties (an aromatic ring and a beta-ethanolamine moiety) were further screened for aerobic b

195 inked to the A2E molecule via its pyridinium ethanolamine moiety.

196 s along with 56-60% loss of C1 and C2 phenyl ethanolamine-N-methyltransferase (PNMT)-ir neurones.

197 H characterized to date belong to the N-acyl ethanolamine (NAE) class of fatty acid amides, including

198 Termination of the activity of the N-acyl ethanolamine (NAE) class of lipid-signaling molecules, i

199 tic pathway(s) for anandamide and its N-acyl ethanolamine (NAE) congeners remain enigmatic.

200 ntative fatty acid amides include the N-acyl ethanolamines (NAEs) anandamide, which serves as an endo

201 N-Acyl ethanolamines (NAEs) are a large class of signaling lipi

202 N-Acyl ethanolamines (NAEs) constitute a large and diverse clas

203 le cross-linking monomer N,O-bismethacryloyl ethanolamine (NOBE) along with template, initiator, and

204 (f) the effect of the AC inhibitor N-oleoyl-ethanolamine (NOE) on cytotoxicity and ceramide species.

205 ggests a role in transport, eutH mutants use ethanolamine normally under standard conditions (pH 7.0)

206 have previously selected ssDNA aptamers for ethanolamine, one of the smallest aptamer targets so far

207 ly optimized to hydrolyze phospholipids with ethanolamine or serine headgroups.

208 rius terrosus showed a strong preference for ethanolamine over choline, whereas two paralogous enzyme

209 rence for positively charged (choline and/or ethanolamine) over neutral (glycerol and serine) headgro

210 e kinase catalyzes the first step in the CDP-ethanolamine pathway for the formation of the major memb

211 n the incorporation of ethanolamine into CDP-ethanolamine pathway intermediates and into phosphatidyl

212 ulation correlated with increases in the CDP-ethanolamine pathway intermediates phosphoethanolamine a

213 ion of diacylglycerol utilization by the CDP-ethanolamine pathway led to a 10-fold increase in triacy

214 hesis of phosphatidylethanolamine by the CDP-ethanolamine pathway.

215 tearoylglycerol lipid (POD) and phosphatidyl ethanolamine (PE) has been studied using an ANTS/DPX lea

216 psulated in polyethylene glycol-phosphatidyl ethanolamine (PEG-PE) conjugated micelles.

217 classes, viz., phosphatidyl-cholines (PCs), -ethanolamines (PEs), -serines (PSs), -inositoles (PIs),

218 DC1, acts as a phosphodiesterase removing an ethanolamine phosphate (EtN-P) from mannose 2 of the gly

219 the conversion of sphingosine-1-phosphate to ethanolamine phosphate and a fatty aldehyde.

220 competent for transfer to protein, (ii) the ethanolamine phosphate group on the third mannose residu

221 tical for GPI recognition by GPIT, (iii) the ethanolamine phosphate residue linked to the first manno

222 ll four of the mannosyltransferases, and the ethanolamine phosphate transferase.

223 enzyme that modifies the second mannose with ethanolamine phosphate, which is removed soon after GPI

224 Results of in vivo studies also show that ethanolamine-phosphate (EA-P) is a substrate of CbiB, bu

225 enzyme of the Kennedy pathway, the cytosolic ethanolamine-phosphate cytidylyltransferase (TbECT).

226 trates, initiated by the methylation of free ethanolamine-phosphate.

227 and CPG pools, some going to the serine and ethanolamine phosphoglyceride (SPG and EPG) pools.

228 cerides and red blood cell (RBC) choline and ethanolamine phosphoglyceride FAs were assessed.

229 s than of the control subjects, and cord RBC ethanolamine phosphoglycerides were lower in DHA (P < 0.

230 ion promoted uptake of exogenous choline and ethanolamine phospholipids.

231 nd TbSLS4, a bifunctional sphingomyelin (SM)/ethanolamine phosphorylceramide (EPC) synthase, were ina

232 am-stage parasites contain sphingomyelin and ethanolamine phosphorylceramide (EPC), but no detectable

233 reviously to be a bifunctional sphingomyelin/ethanolamine phosphorylceramide synthase, whereas functi

234 inositol phosphorylceramide, TbSLS2 produces ethanolamine phosphorylceramide, and TbSLS3 is bifunctio

235 nia major through the characterization of an ethanolamine phosphotransferase (EPT) mutant.

236 mic glucose metabolism, we perturbed choline/ethanolamine phosphotransferase 1 (CEPT1), the terminal

237 here identified a CDP-ethanolamine:ceramide ethanolamine phosphotransferase as the enzyme responsibl

238 sferase, diacylglycerol acyltransferase, and ethanolamine phosphotransferase were not affected by Scd

239 ificant decrease in the level of the choline/ethanolamine-phosphotransferase (PfCEPT), a key enzyme i

240 of A2E (adduct of two vitamin A aldehyde and ethanolamine) photodegradation products, and in a zymogr

241 reported effect of FABP on plasmalogen mass, ethanolamine plasmalogen mass was reduced 30% in gene-ab

242 subtype of ether phospholipids also known as ethanolamine plasmalogen whose functions are not well ch

243 not enriched in arachidonic acid-containing ethanolamine plasmalogens.

244 A and treated with di-palmitoyl-phosphatidyl-ethanolamine polyethylene glycol (DPPE-PEG), a CD1d-bind

245 yl)-1,2-dihexadecanoyl-sn-glycero-3 -phospho-ethanolamine present in bilayer vesicles.

246 these isoquinolinediones with methylamine or ethanolamine produced the isoquinolinedione alkaloids ca

247 Together, these findings suggest that ethanolamine production is likely required for Leishmani

248 The deuterium-labeled ethanolamine reaction product ([(2)H(4)]EA) was analyzed

249 turally occurring amide of palmitic acid and ethanolamine, reduces pain and inflammation through an a

250 Breakdown of ethanolamine requires adenosylcobalamin (AdoCbl) as a co

251 Therefore, aerobic growth on ethanolamine requires import of Ado-B12 or a precursor (

252 with large and small headgroups (choline and ethanolamine, respectively), and of the removal of a lip

253 To grow aerobically on ethanolamine, Salmonella enterica must be provided with

254 During growth on ethanolamine, Salmonella enterica synthesizes a multimol

255 Here we show that niclosamide ethanolamine salt (NEN) uncouples mammalian mitochondria

256 ve, direct acting S1P1 agonists utilizing an ethanolamine scaffold containing a terminal carboxylic a

257 sporter, EutH contributed to the toxicity of ethanolamine seen under some conditions; furthermore, fu

258 g in spectra of samples prepared with [2-13C]ethanolamine show that the unpaired electron is localize

259 Of the five different head groups, only ethanolamine showed appreciable activity.

260 In(4)Sn(4)O(15), grown from 2-methoxyethanol/ethanolamine solutions, were used to fabricate thin-film

261 NA was established as a soluble protein with ethanolamine-specific kinase activity that was most high

262 residues are "masked" by positively charged ethanolamine substituents, leading to an overall zero ne

263 In the anaerobic environment of the gut, ethanolamine supports little or no growth by fermentatio

264 osome membranes, is synthesized de novo from ethanolamine through the Kennedy pathway.

265 Two central enzymes convert ethanolamine to acetaldehyde (EutBC) and then to acetyl-

266 sion to imine functionality by reaction with ethanolamine to give ZIF-91 and ZIF-92, respectively.

267 the introduction of a high concentration of ethanolamine to the solution.

268 her GSH nor EutA was needed during growth on ethanolamine under reduced-oxygen conditions.

269 found that the N-terminal sequences from the ethanolamine utilization (Eut) and glycyl radical-genera

270 The ethanolamine utilization (eut) locus of Enterococcus fae

271 shell constituents of a functionally complex ethanolamine utilization (Eut) microcompartment.

272 ng specific functions encoded by the 17-gene ethanolamine utilization (eut) operon established the mi

273 The ethanolamine utilization (eut) operon of this bacterium

274 EHEC encodes the ethanolamine utilization (eut) operon that allows EHEC t

275 rase (EutT) is encoded within the operon for ethanolamine utilization (eut).

276 nt expression of the Salmonella enterica LT2 ethanolamine utilization bacterial microcompartment shel

277 tory strategies that influence expression of ethanolamine utilization genes (eut) in Enterococcus, Cl

278 ultiple regulatory strategies for control of ethanolamine utilization genes.

279 sing EA and regulating the expression of the ethanolamine utilization genes.

280 c metabolic process (e.g. 1,2-propanediol or ethanolamine utilization).

281 serovar Typhimurium include those coding for ethanolamine utilization, a universal stress protein, a

282 ORFs for ethanolamine utilization, thermostable carboxypeptidase,

283 by the eut operon proved to be essential for ethanolamine utilization, when subjected to sufficiently

284 Lack of CobA and EutT blocked ethanolamine utilization.

285 somes for CO2-fixation, and propanediol- and ethanolamine-utilizing microcompartments that contain B1

286 s interfacial anchors: acetyl-GWW(LA)(6)LWWA-ethanolamine (WALP19).

287 th of E. faecalis in a synthetic medium with ethanolamine was abolished in the response regulator RR1

288 iff base nitrogen of all-trans-retinal dimer-ethanolamine was pH-dependent.

289 nolamine, [2-13C]ethanolamine, and unlabeled ethanolamine were acquired using RMFQ trapping methods f

290 in, as N-arachidoyl glycine and N-arachidoyl ethanolamine, which did not inhibit the Ca(v)3.3 current

291 ability to use an abundant simple substrate, ethanolamine, which is provided by the host.

292 med gut is because of its ability to respire ethanolamine, which is released from host tissue, but is

293 Supplementation with ethanolamine, which led to increased PE synthesis, or wi

294 ) synthase/decarboxylase are auxotrophic for ethanolamine, which must be transported into the cell an

295 noid receptors, and N-oleoyl and N-palmitoyl ethanolamine, which produce satiety and anti-inflammator

296 ublimation produced DL-alanine, glycine, and ethanolamine, while in the presence of hydrogen sulfide,

297 Replacement of ethanolamine with ethylene diamine or 1,3-diaminopropane

298 A direct condensation of N-benzyl ethanolamine with glyoxylic acid yielded a 2-hydroxy-1,4

299 For the series of ethanolamines with different levels of headgroup methyla

300 utants isolated for their ability to degrade ethanolamine without added DMB converted Cbi to pseudo-B