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1 henol A (BPA) and the pharmaceutical 17alpha-ethinyl estradiol (EE) are synthetic chemicals with estr
2 ted the effectiveness of oral treatment with ethinyl estradiol (EE) on EAE and the mechanisms involve
4 from livers of bile duct ligation (BDL)- or ethinyl estradiol (EE)-injected rats, were viewed and re
6 amine the bioavailability and bioactivity of ethinyl estradiol (EE2) sorbed onto SWCNTs in a fish gas
11 o 1.7 with oral contraceptives that included ethinyl estradiol at a dose of 20 mug and by a factor of
13 ent use of oral contraceptives that included ethinyl estradiol at a dose of 30 to 40 mug was associat
14 actor of 1.3 to 2.3 with those that included ethinyl estradiol at a dose of 30 to 40 mug, with relati
15 eceive either oral contraceptives (triphasic ethinyl estradiol at a dose of 35 microg plus norethindr
16 Mice treated with 17beta-estradiol or 17a-ethinyl estradiol had a total uterine tumor incidence of
19 droxyestradiol, 17beta-estradiol, or 17alpha-ethinyl estradiol on days 1-5 of neonatal life (2 microg
21 nicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol without generating obviously toxic byp
22 and better tolerated than the Yuzpe regimen (ethinyl estradiol, 100 microg, and levonorgestrel, 0.5 m
23 glucuronidation activity toward lamotrigine, ethinyl estradiol, chenodeoxycholic acid, and lithocholi
24 with maximally efficacious doses of 17-alpha ethinyl estradiol, the benzothiophene SERM, raloxifene,
25 However, the synthetic estrogens, DES and ethinyl estradiol, were detected in various lots of PC-S
28 er feeding pregnant rats a high-fat (HF)- or ethinyl-oestradiol (EE2)-supplemented diet affects carci
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