ライフサイエンスコーパス: ethosuximide

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1 e cortex and thalamus that were inhibited by ethosuximide.

2 ice were evaluated by EEG and sensitivity to ethosuximide.

3 hism on channel physiology in the absence of ethosuximide.

4 gs, phenytoin, phenobarbital, valproate, and ethosuximide.

5 on T-type calcium channel responsiveness to ethosuximide.

6 panied by behavioral arrest and inhibited by ethosuximide.

7 pectively; odds ratio with valproic acid vs. ethosuximide, 1.26; 95% confidence interval [CI], 0.80 t

8 who were randomly assigned to treatment with ethosuximide (156), lamotrigine (149), or valproic acid

9 Ethosuximide, a T-type calcium channel blocker, eliminat

10 The results demonstrate that both ethosuximide and the active metabolite of methsuximide,

11 Ethosuximide and valproic acid are more effective than l

12 therapy, the freedom-from-failure rates for ethosuximide and valproic acid were similar (53% and 58%

13 d with behavioral arrest, were suppressed by ethosuximide, and were strongest in the cerebral cortex

14 e of the clinically used antiepileptic drugs ethosuximide (ED50 = 161 mg/kg), phenobarbital (ED50 = 2

15 The in vivo P640L variant's ethosuximide effect was confirmed by in vitro electrophy

16 Ethosuximide (ETH), an anticonvulsant drug is used for t

17 ferentially blocked the sustained phase, but ethosuximide had no effect.

18 s current was especially sensitive to block (ethosuximide IC(50) = 0.6 mM).

19 was more common with valproic acid than with ethosuximide (in 49% of the children vs. 33%; odds ratio

20 Ethosuximide is associated with fewer adverse attentiona

21 safety profile than clinically relevant AEDs ethosuximide, lacosamide, or valproic acid.

22 ildren in a randomized double-blind trial of ethosuximide, lamotrigine, and valproate had short-term

23 ell-known antiepileptic drugs (AEDs) such as ethosuximide, levetiracetam, and lacosamide.

24 ts of well-known antiepileptic drugs such as ethosuximide, levetiracetam, and lacosamide.

25 o normal by either the absence epilepsy drug ethosuximide or a novel T-channel blocker, TTA-P2.

26 Ethosuximide's effect on rate of decay of CaV 3.2 was si

27 Treatment with ethosuximide significantly blocks seizures in both genot

28 Treatment with ethosuximide significantly blocks seizures in both stg/s

29 In ethosuximide subjects, 2 polymorphisms (CACNA1H rs617344

30 Here, we report that ethosuximide, trimethadione, and 3,3-diethyl-2-pyrrolidi

31 erability, and neuropsychological effects of ethosuximide, valproic acid, and lamotrigine in children

32 c epilepsy syndrome, is usually treated with ethosuximide, valproic acid, or lamotrigine.

33 e rate for lamotrigine (29%; odds ratio with ethosuximide vs. lamotrigine, 2.66; 95% CI, 1.65 to 4.28

34 ult of a spinal cord injury and suggest that ethosuximide will relieve human CPS by restoring normal

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