ライフサイエンスコーパス: ethyl methanesulfonate

1 affect their probability to be alkylated by ethyl methanesulfonate.

2 o ricin was prepared by mutagenesis by using ethyl methanesulfonate.

3 been added, whereas the other was induced by ethyl methanesulfonate.

4 ntrachromosomal recombination events against ethyl methanesulfonate and acridine mutagen agents.

5 To that end, we treated strain F62 with ethyl methanesulfonate and grew approximately 175,000 co

6 Use of ethyl methanesulfonate and site-directed mutagenesis has

7 Using a combination of both ethyl methanesulfonate and site-directed mutagenesis, we

8 DNA-alkylating agents such as ethyl methanesulfonate and the chemotherapeutic drug mel

9 , human sarcoma HT1080 cells were exposed to ethyl methanesulfonate and Thymitaq selection.

10 traviolet irradiation, 5-fluorodeoxyuridine, ethyl methanesulfonate, and methyl methanesulfonate.

11 that specify C(4) leaf anatomy, we generated ethyl methanesulfonate- and gamma-ray-mutagenized popula

12 ve stronger antimutagenic properties against ethyl methanesulfonate compared to the acridine mutagen

13 was affected in tomato RNA interference and ethyl methanesulfonate-cus1 mutants.

14 Interestingly, this ethyl methanesulfonate-derived mutant shows unusual chro

15 The ethyl methanesulfonate-derived vte3-1 allele alters toco

16 inogenic compounds 4-nitroquinoline N-oxide, ethyl methanesulfonate, diethylstilbestrol, and 2-aminoa

17 y transformed luciferase reporter lines were ethyl methanesulfonate (EMS) mutagenized, and stable M(2

18 Ethyl methanesulfonate (EMS) mutants of tomato have alre

19 h to examine Scp160p function, we applied an ethyl methanesulfonate (EMS) screen for loci synthetical

20 gregating for autosomes heavily treated with ethyl methanesulfonate (EMS), approximately 12,000 lines

21 y treatment with either 1 mM octanol, 0.5 mM ethyl methanesulfonate (EMS), or cytoplasmic acidificati

22 N Genomes) method combines the efficiency of ethyl methanesulfonate (EMS)-induced mutagenesis with th

23 comprehensive analysis of approximately 1900 ethyl methanesulfonate (EMS)-induced mutations in 192 Ar

24 ble for a specific phenotype, observed in an ethyl methanesulfonate (EMS)-mutagenized Caenorhabditis

25 ression is evident in more than one-third of ethyl methanesulfonate (EMS)-treated bal M(1) plants.

26 nogaster males by treating them with 21.2 mm ethyl methanesulfonate (EMS).

27 and internally applied 2-(trimethylammonium)ethyl methanesulfonate failed to cause desensitization (

28 rabidopsis (Arabidopsis thaliana), including ethyl methanesulfonate, fast-neutron and defined T-DNA m

29 h as UV light (generating UV photoproducts), ethyl methanesulfonate (generating alkylation damage), m

30 After mutagenesis with ethyl methanesulfonate, growing cells were selectively k

31 pression, we identified three closely linked ethyl methanesulfonate-induced changes as putative candi

32 In the ethyl methanesulfonate-induced mutant hcf222-1, the accu

33 the frequency and spectra of spontaneous and ethyl methanesulfonate-induced mutations across the Sacc

34 Moreover, ethyl methanesulfonate-induced mutations that change ami

35 Ethyl methanesulfonate-induced mutations within the seco

36 We conducted a screen for ethyl methanesulfonate-induced suppressors of Arabidopsi

37 Neither function is affected in ethyl-methanesulfonate-induced Pl1-Rhoades derivatives t

38 OB3 allele (sob3-4) was generated through an ethyl methanesulfonate intragenic suppressor screen of s

39 In cells treated with ethyl methanesulfonate, intragenic deletions were detect

40 ing were inducible by the carcinogen x-rays, ethyl methanesulfonate, methyl methanesulfonate, ethyl n

41 PvGal biosynthesis was investigated by ethyl methanesulfonate mutagenesis of S. pombe, followed

42 Ethyl methanesulfonate mutagenesis of the model legume M

43 C. trachomatis was subjected to low-level ethyl methanesulfonate mutagenesis to generate chlamydia

44 e obtained a petal-closed flower mutation by ethyl methanesulfonate mutagenesis.

45 lines, TG01 and TG10, were generated through ethyl methanesulfonate mutagenesis.

46 ccessful symbiotic infection, we screened an ethyl methanesulfonate mutagenized population of Lotus j

47 A subset of an ethyl methanesulfonate mutagenized population of soybean

48 h medium was carried out for a population of ethyl methanesulfonate-mutagenized Arabidopsis (Arabidop

49 We screened 4,960 ethyl methanesulfonate-mutagenized colonies for defects

50 leaf11 (spl11) mutant was identified from an ethyl methanesulfonate-mutagenized indica cultivar IR68

51 Here, we report the identification of eight ethyl methanesulfonate-mutagenized loss-of-function bin2

52 ow-like (vyl) phenotype was identified in an ethyl methanesulfonate-mutagenized population derived fr

53 olved in seed Fe homeostasis, we screened an ethyl methanesulfonate-mutagenized population of nramp3n

54 By screening ethyl methanesulfonate-mutagenized populations of Arabid

55 We screened cotyledons from ethyl methanesulfonate-mutagenized seeds of Arabidopsis

56 ) progeny and a half million F(3) progeny of ethyl-methanesulfonate-mutagenized animals were treated

57 An ethyl methanesulfonate mutant (jar1) with decreased sens

58 htness and cuticle in tomato, we screened an ethyl methanesulfonate mutant collection in the miniatur

59 tified an Arabidopsis (Arabidopsis thaliana) ethyl methanesulfonate mutant, modified vacuole phenotyp

60 In parallel, three independent ethyl methanesulfonate mutants in the S. lycopersicum cu

61 We have isolated several ethyl methanesulfonate mutants of salt cress that have r

62 ting agents including N-ethyl-N-nitrosourea, ethyl methanesulfonate, N-propyl-N-nitrosourea and N-but

63 of M. xanthus dsp were mutagenized either by ethyl methanesulfonate or by Tn5 insertions.

64 in insight into GDU1's role, we searched for ethyl-methanesulfonate suppressor mutants and performed

65 ANT1 (BZR1) and BRASSINOSTEROID INSENSITIVE1-ETHYL METHANESULFONATE-SUPPRESSOR1 (BES1)/BZR2, blocks t

66 A BRASSINOSTEROID INSENSITIVE1-ETHYL METHANESULFONATE-SUPPRESSOR1-yellow fluorescent pr

67 onjugation properties, were mutagenized with ethyl methanesulfonate to obtain variants that were indu

68 From a collection of 12,326 strains carrying ethyl-methanesulfonate-treated, homozygous viable second

69 nsitivity of UV40 to mitomycin C, cisplatin, ethyl methanesulfonate, UV, and gamma-radiation.

70 inoside and luteolin-7-O-glucuronide against ethyl methanesulfonate were 57.6%, 58.3% and 62.5%, resp