1 dels of RSV infection of the nasopharynx and Eustachian tube.

2 ars dynamically by closing its normally open Eustachian tubes.

3 Malformation and abnormal positioning of the eustachian tube, accompanied by OM, were observed in all

4 genes like EYA4 that regulate middle ear and eustachian tube anatomy.

5  site, thus preventing it from ascending the eustachian tube and inducing otitis media in chinchillas

6  crystals in the middle ear cavity, enlarged Eustachian tube, and chronic otitis media with effusion

7  monitor infection within the nasopharynges, eustachian tubes, and middle ears of chinchillas after i

8                                              Eustachian tube closure was observed in the field during

9      Vulnerability to otitis media is due to eustachian tube dysfunction as well as other poorly unde

10 most frequently in children with auditory or eustachian tube dysfunction.

11 dies revealed abnormal middle ear cavity and eustachian tube dysmorphology; thus, Eya4 regulation is

12  because the more horizontal position of the Eustachian tube (ET) in infants limits or delays clearan

13 luated on the cell surface in the chinchilla eustachian tube (ET) lumen of a cohort challenged intran

14 ated at the round window niche (RWN) and the Eustachian tube (ET).

15 ication, cholesterol crystal deposition, and Eustachian tubes in the etiology of otitis media.

16 d the presence of a ciliated region near the Eustachian tube orifice at the ventral region of the mid

17  mice results in abnormal structuring of the eustachian tube, thus predisposing these animals to OME

18 cted in RNA isolated from trachea, lung, and Eustachian tube tissues.