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1 ulated thyroglobulin less than 2 ng/ml (95%, euthyroid; 96%, hypothyroid).
2 f thyroxine therapy, and all patients became euthyroid after treatment with bexarotene was stopped.
3 riteria of negative whole body scans (84% of euthyroid and 94% of hypothyroid patients) and stimulate
4         Values for thyroid uptake of 123I in euthyroid and hyperthyroid patients in Boston have remai
5 ganded TR, we deleted NCoR1 in the livers of euthyroid and hypothyroid mice and examined the effects
6  experimental platform that exploited paired euthyroid and severe hypothyroid serum samples from huma
7         The association of TSH levels in the euthyroid and subclinical hypothyroid range with inciden
8 hyroidism who were clinically and chemically euthyroid and were receiving levothyroxine sodium, 0.1 o
9        At the end of treatment, hypothyroid, euthyroid, and hyperthyroid status was confirmed.
10 BAT)-specific hypothyroidism in an otherwise euthyroid animal.
11 rked increase in message level in T3-treated euthyroid animals depends primarily on post-transcriptio
12 ificant, increase in HCN2 mRNA occurred when euthyroid animals were made hyperthyroid.
13    ABR thresholds develop rapidly in normal, euthyroid animals, decreasing as much as 80 dB between P
14 e in that they were obtained in systemically euthyroid animals, revealing that brain D2 plays a domin
15 nes the magnitude of the response to T(3) in euthyroid animals.
16 er (1.8-fold increase) but not in T3-treated euthyroid animals.
17  formation markers in a sequential manner in euthyroid but not hypothyroid mice, thus providing evide
18 ce had normal serum T3 and were systemically euthyroid, but exhibited an approximately 3-fold elevati
19 is study shows that hyperthyroid compared to euthyroid cats have higher serum concentrations for some
20 -99 ratios are 0.47 and 0.32 for healthy and euthyroid cats, respectively, which differs significantl
21 other areas showing high TR expression under euthyroid conditions.
22 ignificantly inhibited (> 80%) compared with euthyroid control animals.
23 ished urinary concentration as compared with euthyroid controls (CTL) and hypothyroid rats replaced w
24 ion of CYP4A2 mRNA was decreased relative to euthyroid controls, but DHEA was as effective an inducer
25 d rats and hypothyroid rats were compared to euthyroid controls.
26 we used FloxD2 mice to generate systemically euthyroid fat-specific (FAT), astrocyte-specific (ASTRO)
27 om the carotid artery and thyroid vein of 10 euthyroid goiter patients and one patient with a toxic s
28 AF was also longer, compared with 11% in the euthyroid group (all P<0.05).
29                                  Relative to euthyroid hamsters (EH), HH exhibited decreased D1 recep
30 t vary with states of photoresponsiveness in euthyroid hamsters, but blockade of thyroid function acc
31  women with normal functioning thyroids (ie, euthyroid) have been associated with several complicatio
32 f the hypothalamus-pituitary-thyroid axis to euthyroid hormone levels.
33 nd aged rats (26 months old) were studied at euthyroid, hyperthyroid and hypothyroid conditions.
34 er rats at 4, 12 and 24 months of age during euthyroid, hypothyroid and hyperthyroid states.
35 unoassays can be successfully eliminated for euthyroid individuals as well as for patients with thyro
36             Higher fT4 levels at baseline in euthyroid individuals were associated with increased AF
37                                           In euthyroid individuals, higher circulating fT4 levels, bu
38 ly(A) signal in hypothyroid hearts were 0.26 euthyroid levels (P < 0.05).
39  total mRNA beta 1 content decreased to 0.18 euthyroid levels, which was associated with a disproport
40                     The prerequisites for an euthyroid metabolic state include a normally developed t
41 hyroid WT mice exposed to VILI compared with euthyroid mice, indicating that the lungs were functiona
42 and ghrelin(+) cells compared to grafts from euthyroid mice.
43 thin the study group, 60 of these cats had a euthyroid (n = 23) or hyperthyroid (n = 37) status, all
44 gery were randomized into hypothyroid (N=9), euthyroid (N=9), and hyperthyroid (N=9) groups.
45                                              Euthyroid, nondiabetic, healthy, adult Pima Indians (n =
46 ent in the liver, deletion of SMRT in either euthyroid or hypothyroid animals had little effect on TH
47 s and also after restricting the analysis to euthyroid participants (defined by thyroid-stimulating h
48 he absolute 10-year risk of SCD increased in euthyroid participants from 1% to 4% with increasing FT4
49 significantly associated with incident AF in euthyroid participants or those with subclinical hypothy
50                                           In euthyroid participants, higher thyroid function was asso
51                                           In euthyroid participants, we additionally examined the ass
52 iated with an increased risk of SCD, even in euthyroid participants.
53 ts remained similar or became stronger among euthyroid participants.
54 ith US guidance and conscious sedation in 20 euthyroid patients (mean age, 44.5 years) with a benign
55 measurements using 123I were obtained in 671 euthyroid patients and 274 hyperthyroid patients, of whi
56 lasma T(4) concentrations in critically ill, euthyroid patients and suggest that targeting the imbala
57  thyroid function test results that occur in euthyroid patients who are receiving amiodarone.
58 er end of the normal range, as often seen in euthyroid patients who are receiving amiodarone.
59                                          For euthyroid patients, there were no significant difference
60                                           In euthyroid Pima Indians, lower free T(3) but not free T(4
61 urons in each nucleus were identical in both euthyroid rats and hypothyroid rats induced by 6-n-propy
62 ted effects were robust in the current work, euthyroid rats retain thyroid hormone sensitivity which
63  approximately 80% alpha-MHC/20% beta-MHC in euthyroid rats to 100% beta-MHC, without altering the ex
64                                  Compared to euthyroid rats, hyperthyroidism in 4-month-old rats was
65                                   As seen in euthyroid rats, T3 administration potently suppressed DH
66 ed the effects of similar microinjections in euthyroid rats.
67 ly 12-fold, respectively, in hypothyroid and euthyroid rats.
68 ffective an inducer of this mRNA as it is in euthyroid rats.
69                                           In euthyroid rodent kidney, which only expresses CYP4A2 und
70 ss; however, the mechanisms involved in the "euthyroid sick syndrome" remain poorly understood.
71 ent with this observation and reminiscent of euthyroid sick syndrome, a stress-associated clinical co
72                        Originally termed the euthyroid sick syndrome, this phenomenon is now more com
73                                          The euthyroid state was defined as thyroid-stimulating hormo
74 e of excess radioiodine from the body in the euthyroid state with Thyrogen stimulation has significan
75                           Remarkably, in the euthyroid state, expression of many T(3)-targets is also
76 lated in L-NCoRDeltaID mice in the hypo- and euthyroid state, there was little effect seen on negativ
77  hormone levels, thus creating a biochemical euthyroid state.
78 odalities necessary to restore patients to a euthyroid state.
79 ely on serum TSH measurements to confirm the euthyroid status of these patients.
80 n January 30, 2004, and June 20, 2007, of 50 euthyroid study participants aged 18 to 65 years who wer
81 HB mRNA was consistently detected in AT from euthyroid subjects, and positively associated with serum
82 nonthyroidal illness, also known as the sick euthyroid syndrome, is characterized by a low plasma T3
83 on, contributing to the etiology of the sick euthyroid syndrome.
84                                Data from 368 euthyroid volunteers who provided blood samples at basel
85 ter stopping CBZ treatment and were rendered euthyroid with either long-term CBZ (n=3) or radioiodine

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