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1 o three intravascular fluid protocol groups: euvolemic (3 mLkg/hour isotonic crystalloid), hypervolem
3 or on minimal exertion, who were clinically euvolemic, and who had an ejection fraction of less than
4 sfusion of stored leukodepleted red cells to euvolemic, anemic, critically ill patients has no clinic
5 F activity, essentially at normal levels for euvolemic animals, persists during AT1 receptor blockade
7 e in modulation of renal hemodynamics in the euvolemic diabetic rat, in the absence of KKS stimulatio
14 merular feedback (TGF) activity in 7-wk-old, euvolemic spontaneously hypertensive rats (SHR) and in W
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