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1 uate sleep quantity, poor sleep quality, and excessive daytime sleepiness.
2 exhibit insomnia, delayed sleep habits, and excessive daytime sleepiness.
3 ) and without (n=144) complaints of frequent excessive daytime sleepiness.
4 ty to predict important consequences such as excessive daytime sleepiness.
5 rousals, could prove useful in prediction of excessive daytime sleepiness.
6 sturbance may reduce sleep fragmentation and excessive daytime sleepiness.
7 during sleep are a well recognized cause of excessive daytime sleepiness.
8 atients showed high risk for OSAS, 46.3% had excessive daytime sleepiness, 41.5% were positive for bo
10 chronic neurologic disorder characterized by excessive daytime sleepiness and abnormal manifestations
11 ye movement (REM) sleep, is characterized by excessive daytime sleepiness and cataplexy, a loss of mu
16 0 x 10(-13)) and between increased levels of excessive daytime sleepiness and increased measures for
17 n SWS, reproducing, respectively, narcolepsy excessive daytime sleepiness and poor sleep quality.
19 way resistance syndrome (UARS) is defined by excessive daytime sleepiness and tiredness, and is assoc
21 vements during sleep are not associated with excessive daytime sleepiness, and therefore appear unlik
23 central nervous system disorders, including excessive daytime sleepiness, attention deficit hyperact
25 arcolepsy, a sleep disorder characterized by excessive daytime sleepiness, cataplexy, and other patho
26 is a neurological disorder characterized by excessive daytime sleepiness, cataplexy, hypnagonic hall
29 erred for polysomnography with complaints of excessive daytime sleepiness (EDS) and clinically suspec
30 is was tested by exploring the links between excessive daytime sleepiness (EDS) and vulnerability to
31 lness for the treatment of disorders such as excessive daytime sleepiness (EDS) as well as other slee
32 o determine the safety and efficacy of LT on excessive daytime sleepiness (EDS) associated with PD.
35 obesity and snoring and between obesity and excessive daytime sleepiness (EDS), although for the mos
36 excessive daytime sleepiness, patients with excessive daytime sleepiness (Epworth Sleepiness Scale s
37 ple, 26% were habitual snorers, 18% reported excessive daytime sleepiness (ESS > or = 11), and 29% we
38 leep disorder narcolepsy is characterized by excessive daytime sleepiness, fragmentation of nighttime
41 al study assessing multiple risk factors for excessive daytime sleepiness in older subjects (mean age
42 servoventilation produces an improvement in excessive daytime sleepiness in patients with Cheyne-Sto
43 sleep fragmentation (SF) appear to underlie excessive daytime sleepiness in patients with sleep apne
45 ported sleep duration, insomnia symptoms and excessive daytime sleepiness in the UK Biobank (n = 112,
46 the most common medical disorder that causes excessive daytime sleepiness, increasing the risk for dr
47 ular, eczema associated with atopy, fatigue, excessive daytime sleepiness, insomnia, and only 0 to 3
48 at often are seen by neurologists, including excessive daytime sleepiness, insomnia, narcolepsy, rapi
50 L1 and TGFBI in females and WDR27 in males), excessive daytime sleepiness (near AR-OPHN1) and a compo
51 95% CI, 1.11-2.10) (P = .009, I2 = 74%), and excessive daytime sleepiness (OR, 2.27; 95% CI, 1.54-3.3
52 ion (adjusted OR, 2.65; P=0.023), those with excessive daytime sleepiness (OR, 2.51; P=0.037), and th
53 95% CI, 1.14-1.88) (P = .003, I2 = 76%), and excessive daytime sleepiness (OR, 2.72; 95% CI, 1.32-5.6
54 porter availability and fatigue, depression, excessive daytime sleepiness, or rapid eye movement slee
56 uate sleep quantity, poor sleep quality, and excessive daytime sleepiness, studied according to an a
58 patic encephalopathy might be unable, due to excessive daytime sleepiness, to accumulate the need/abi
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