ライフサイエンスコーパス: exchanger

1 eres (containing solvatochromic dyes and ion exchanger).

2 inositol (3,4,5)-triphosphate)-dependent Rac exchanger).

3 k1, l-type calcium channel, and Na(+)/Ca(2+) exchanger.

4 iculum calcium ATPase 2 and the Na(+)/Ca(2+) exchanger.

5 the major intestinal brush-border Na(+)/H(+) exchanger.

6 impairs the pore structure of the Na(+)/H(+) exchanger.

7 rs of the angiotensin receptor and the Na-Ca exchanger.

8 and enhancing the activities of Na(+)/Ca(2+) exchanger.

9 selective activation of the sodium/hydrogen exchanger.

10 inhibitor of the mitochondrial Na(+)/Ca(2+) exchanger.

11 SLDsp neurons by activating an Na(+)-Ca(2+) exchanger.

12 yrin-B, a binding partner for EHD3 and Na/Ca exchanger.

13 vesicular monovalent cation Na(+)(K(+))/H(+) exchanger.

14 siologic glucose concentrations inhibit this exchanger.

15 and by augmented levels of the Na(+)/Ca(2+) exchanger.

16 (Sc) sites in CLC-ec1, the Escherichia coli exchanger.

17 t that involved plasma membrane Na(+)/Ca(2+) exchanger.

18 sed by a loss of function of this Na(+)/H(+) exchanger.

19 l as the surface delivery of the AE1-4 anion exchanger.

20 ion of PaNhaP and other microbial Na(+)/H(+) exchangers.

21 systems, including optimization of the heat exchangers.

22 ge could not be explained by known glutamate exchangers.

23 ities (>5 W/m(2)) and with finite-sized heat exchangers.

24 ters do not function exclusively as obligate exchangers.

25 n electroneutral and electrogenic Na(+)/H(+) exchangers.

26 edically important eukaryotic NHE Na(+)/H(+) exchangers.

27 carrier family 26 member A6 (SLC26A6) anion exchangers.

28 Anion exchanger 1 (AE1) mediates Cl(-)/HCO3(-) exchange in ery

29 Anion exchanger 1 (AE1), also known as band 3 or SLC4A1, plays

30 on inside red blood cells, and band 3 [anion exchanger 1 (AE1)] provides a passage for HCO3(-) flux a

31 ajor interacting loop of the erythroid anion exchanger 1 (eAE1) with ankyrin-R, whereas the main bind

32 The sodium (Na(+))-calcium (Ca(2+)) exchanger 1 (NCX1) is an important regulator of intracel

33 Cardiac sodium (Na(+))-calcium (Ca(2+)) exchanger 1 (NCX1) is central to the maintenance of norm

34 The Na(+)-Ca(2+) exchanger 1 (NCX1) is reduced in stroke by the RE1-silen

35 sgenic (TG) mice expressing the Na(+)-Ca(2+) exchanger 1 (NCX1) to model its identified augmentation

36 We demonstrated earlier that Na(+)/Ca(2+) exchanger 1 (NCX1), a major calcium exporter in renal ep

37 se (PMCA) isoforms 2 and 3, and Na(+)/Ca(2+)-exchanger 1 (NCX1).

38 PtdIns(3,4,5)P3-dependent Rac exchanger 1 (PREX1) is a Rac-guanine nucleotide exchange

39 Likewise, the tomato CATION EXCHANGER 1 and TWO-PORE CHANNEL 1 (SlTPC1), key genes f

40 he renal ammonium transporter RhBG and anion exchanger 1 kAE1 colocalize in the basolateral domain of

41 1-induced phosphorylation of NHE1 (Na(+)/H(+)exchanger 1), which is involved in the regulation of int

42 and impaired activity of the sodium/hydrogen exchanger 1, a known regulator of skin pH.

43 Anion exchanger-1 (AE1) mediates chloride-bicarbonate exchange

44 Na(+)/Ca(2+) exchanger-1 (NCX1) is a major calcium extrusion mechanis

45 as well as blocking Hyal2 and the Na(+)/H(+) exchanger-1 at the cell-surface prevented BMP7-driven HA

46 ession of the acid-loading transporter anion exchanger 2 (AE2) (SLC4A2 product) was detected in myofi

47 Anion exchanger 2 (AE2), the principal bicarbonate secretor in

48 To regulate pH, ameloblasts express anion exchanger 2 (Ae2a,b), chloride channel Cftr, and ameloge

49 ylinositol 3,4,5-trisphosphate-dependent Rac exchanger 2 (P-REX2).

50 tol 3,4,5-trisphosphate (PIP3)-dependent Rac exchanger 2 (PREX2) is a guanine nucleotide exchange fac

51 Enzyme 4a) > Enox2 (Ecto-NOX Disulfide-Thiol Exchanger 2) > Ube2d2 (Ubiquitin-conjugating enzyme E2D

52 basolateral Cl(-) /HCO3(-) exchanger (anion exchanger 2; AE2).

53 Ameloblasts were immunostained for anion exchanger-2 (Ae2), a transmembrane pH regulator sensitiv

54 ose uptake leads to activation of Na(+)-H(+) exchanger 3 (NHE3) in the intestine by a process that is

55 Levels of the Na+/H+ exchanger 3 (NHE3) were measured in human ileal biopsy s

56 Na(+)/H(+) exchanger 3 activity was similar in undifferentiated and

57 e and cortical expression of sodium/hydrogen exchanger 3 and attenuated the increased expression of u

58 ulator and partially dependent on Na(+)/H(+) exchanger 3 and Na(+)/K(+)/2Cl(-) cotransporter 1 inhibi

59 enapanor or other intestinal sodium-hydrogen exchanger 3 inhibitors increased fecal phosphorus, decre

60 a-hydroxylase, and the intestinal Na(+)/H(+) exchanger 3, agonists of components of the angiotensin-c

61 to other transporters, including Na(+)/H(+) exchanger 3, and to signaling pathways, such as the A-ki

62 inhibitor of the intestinal sodium-hydrogen exchanger 3, is being evaluated in clinical trials for i

63 s in abundance of proximal tubule Na(+)/H(+) exchanger 3, Na(+)/Pi co-transporter 2, phosphorylated N

64 mbranes, internalizes/inactivates Na(+)-H(+)-exchanger-3 and Na(+)/K(+)ATPase, prevents Na(+) retenti

65 ated major RPT Na(+) transporters Na(+)-H(+)-exchanger-3 and Na(+)/K(+)ATPase.

66 sion and internalized/inactivated Na(+)-H(+) exchanger-3 and Na(+)/K(+)ATPase.

67 primarily by the K(+)-dependent Na(+)/Ca(2+) exchanger 4 (NCKX4), and the removal of Ca(2+) leads to

68 c9a8 gene, resulting in mutant sodium/proton exchanger 8 (NHE8)-M120K protein.

69 elease, activating electrogenic Na(+)-Ca(2+) exchanger activity and triggering early afterdepolarizat

70 K(+)-2Cl(-) cotransporters (NKCC1) and anion exchangers (AE), the 2 primary basolateral Cl(-) uptake

71 ecretion is removed by the basolateral anion-exchanger AE1.

72 HCO3 (-) supplier of ductal Cl(-) -HCO3 (-) exchanger AE2, but not of NBCe1-B.

73 rks by activating the ductal Cl(-) -HCO3 (-) exchanger AE2.

74 transporter Nbce1, and the basolateral anion exchanger Ae2a,b in maturation ameloblasts suggests that

75 The anion exchanger AE3, expressed in hippocampal (HC) neurons but

76 ABSTRACT: The anion exchanger AE3, expressed in hippocampal (HC) neurons but

77 laries were coated with 65-nm-diameter anion exchanger (AEX) latex nanoparticles that attach electros

78 d an elevated expression of the Na(+)/Ca(2+) exchanger, although other excitation-contraction couplin

79 rbed layer of quaternary ammonium type anion exchanger and a phenolic azo type proton chromoionophore

80 orter genes, including vacuole cation/proton exchanger and inositol transporter, were considered to p

81 porate an ionic solvatochromic dye (SD), ion exchanger and ionophore.

82 The membrane is free of ion exchanger and is formulated with plasticized poly(vinyl

83 ivation of countertransporters (Na(+)/Ca(2+) exchanger and Na(+)/H(+) exchanger) coupled to the proto

84 y (Miptode 3), the optode containing the ion exchanger and the MIP particles (Miptode 2) showed impro

85 ally homologous transporters including anion exchangers and prestin.

86 niporter and 2) the evolutionarily conserved exchangers and solute carriers, which are also involved

87 Dinonylnaphthalene sulfonic acid (a cation exchanger) and chromoionophore I (a lipophilic optical p

88 channels, cardiac ryanodine receptors, Na/Ca-exchanger, and SR Ca-ATPase are involved in this regulat

89 polymerization, derivatized as strong anion exchangers, and used for lipoproteins enrichment.

90 e activity of the basolateral Cl(-) /HCO3(-) exchanger (anion exchanger 2; AE2).

91 The aspartate-glutamate exchanger Aralar/AGC1 (Slc25a12), a component of the mal

92 K(+)-dependent Na(+)/Ca(2+)-exchangers are broadly expressed in various tissues, and

93 Na(+)/H(+) exchangers are essential for regulation of intracellular

94 antial activity in conditions when wild-type exchangers are inactive) and do not promote cargo-depend

95 A suggest that both V-ATPases and Na(+)/H(+) exchangers are required for glycosomal pH regulation.

96 Our results indicate that Na(+) -H(+) exchangers are the principal source of synaptic cleft pr

97 we identify NHE9, an endosomal cation/proton exchanger, as a novel regulator of this system.

98 loaded onto the Fractogel EMD SO3 (M) cation exchanger at either pH 5 or pH 4.

99 Ion-exchanger based Donnan exclusion membranes (IEDEM) are s

100 suppressing the major interference of an ion-exchanger based membrane, the 80 muL desalinated sample

101 continuously operating microfluidic solvent exchanger based on the principle of free-flow electropho

102 K(+)-dependent Na(+)/Ca(2+) exchangers belong to the solute carrier 24 (SLC24A1-5) g

103 , which was recently identified as a PI4P-PS exchanger between the ER and PM, showed PI4P-dependent m

104 e suppressor consists of a solid polymer ion exchanger block.

105 odulation and was abolished by Na(+) /Ca(2+) exchanger blockade.

106 Na(+) /Ca(2+) exchanger, but not TRP channels, can also facilitate STO

107 and required Src family kinases and the Rap1 exchanger C3G.

108 O3 is that starch may act as a weak acid ion exchanger capable of exchanging alcoholic group protons

109 the generator eluate is trapped on a cation exchanger cartridge (100 mg, approximately 8 mm long and

110 Here, we identify a vertebrate Ca(2+)/H(+)exchanger (CAX) as part of a widespread family of homolo

111 sue, Melchionda et al. identify a Ca(2+)/H(+)exchanger (CAX) that is required for Ca(2+)uptake and ce

112 CN4, which encodes the chloride/hydrogen ion exchanger CIC-4 prominently expressed in brain, were rec

113 alkaline mine drainage water requires cation exchangers combining excellent sorption properties with

114 ably, the addition of monensin, a Na(+)/H(+) exchanger, completely halts the infection.

115 contributes to phase 1, whereas Na(+)-Ca(2+) exchanger contributes to phase 2.

116 closilicate (ZS-9), a novel selective cation exchanger, could lower serum potassium levels in patient

117 rters (Na(+)/Ca(2+) exchanger and Na(+)/H(+) exchanger) coupled to the proton gradient, ultimately ma

118 pport a model in which the AE3 Cl(-)/HCO3(-) exchanger, coupled with parallel Cl(-) and H(+)-extrusio

119 We found that a dominance of inward Na-Ca exchanger current (I(NCX)) over Ca-dependent inactivatio

120 'funny current' (If ) and the sodium-calcium exchanger current (INCX ).

121 ico, L-type calcium current and Na(+)/Ca(2+) exchanger current determine RA human cell-to-cell differ

122 Large Na(+)/Ca(2+) exchanger current is the main driver in translating Ca(2

123 component was identified as an Na(+)-Ca(2+) exchanger current mediated by Ca(2+) released from the s

124 The decay of Na(+) /Ca(2+) exchanger current that followed a stimulation protocol w

125 VMs, whereas the density of the Na(+)/Ca(2+) exchanger current was not different between PCs and VMs.

126 rough SLC4A11 stably expressed in Na(+)/H(+) exchanger-deficient PS120 fibroblasts.

127 Varying concentration of added cation-exchanger demonstrates that it limits the ion transfer c

128 eading to changes in ionic channel, pump and exchanger densities and kinetics.

129 Our solvent exchanger device demonstrates the power of micro-free-fl

130 Loss of the Drosophila Na(+)-H(+) exchanger DNhe2 lowers pHi in differentiating cells, imp

131 rt the crystal structure of the band 3 anion exchanger domain (AE1(CTD)) at 3.5 angstroms.

132 for ion-transfer voltammetry between an ion-exchanger doped and plasticized poly(vinyl chloride) (PV

133 ch reduced expression of the Cl(-) / HCO3(-) exchanger DRA (SLC26A3), via direct binding to the promo

134 mixing in the membrane module, and high heat exchanger efficiencies.

135 that it applies generally to all Na(+)/H(+) exchangers, electrogenic as well as electroneutral, and

136 ase is under the control of the Na(+)/Ca(2+) exchanger, encoded by the NCLX gene, and of a H(+)/Ca(2+

137 amily 8, member 1 (SLC8A1), a sodium/calcium exchanger encoding NCX1, were validated in an independen

138 Pendrin (SLC26A4) is a Cl(-)/anion exchanger expressed in the epithelium of inflamed airway

139 Pendrin is a Cl(-)/HCO3(-) exchanger expressed in type B and non-A, non-B intercala

140 due to a deficiency in a sodium calcium ion exchanger expressed specifically in the heart.

141 rters" for the constellation of ion channels/exchangers expressed in each sensory neuron.

142 is the best studied member of the Na(+)/H(+) exchanger family and a model system for all related Na(+

143 l (3,4,5)-trisphosphate (PIP3)-dependent Rac exchanger) family (P-Rex1 and P-Rex2) of the Rho guanine

144 e nanospheres (containing the probes and ion exchanger) followed by measuring the pH and Na(+) respon

145 , the compatibility of the developed solvent exchanger for cell based downstream applications was pro

146 erits make it be a promising material as ion exchanger for the removal of radioactive cations from wa

147 ization suggests that SLC9A9, an Na(+) -H(+) exchanger found in endosomes, appears to influence the d

148 mesophases, exemplified by the bacterial ClC exchanger from Escherichia coli (EcClC) as a model ion t

149 gulation of the electrogenic NhaA Na(+)/H(+) exchanger from Escherichia coli (EcNhaA, shown in a prev

150 2+) transients, upregulation of Na(+)/Ca(2+) exchanger function, reduction of Ca(2+) uptake to sarcop

151 many lines of evidence indicate that the CLC exchangers function according to a non-conventional tran

152 ic tail derived from the chicken AE1-4 anion exchanger (G(AE)).

153 remodelling of the ionic channels, pumps and exchangers gives rise to changes in the Ca(2+) transient

154 Many ion channels, transporters, and exchangers have been identified in the ES luminal epithe

155 pport the existence of a countercurrent heat exchanger in the head of R. prolixus, which decreases th

156 Activation of the sodium-hydrogen exchanger in the heart and vasculature (NHE1 isoform) an

157 Here we study the role of these ion exchangers in mitochondrial Ca(2+) extrusion and in Ca(2

158 nd a model system for all related Na(+)/H(+) exchangers, including eukaryotic representatives.

159 evant cellular component (e.g., ion channel, exchanger) independently.

160 The Na(+) /Ca(2+) exchanger inextricably couples the cycling of Ca(2+) and

161 rtially inhibited by monocarboxylate/HCO3(-) exchanger inhibition without affecting GLP-2 release, im

162 colleagues reveal that the cystine/glutamate exchanger is an important source of excitotoxic glutamat

163 KX4-deficient mouse cones revealed that this exchanger is essential for the wide operating range and

164 The activity of sodium-hydrogen exchanger is markedly increased in patients with heart f

165 es had greater phosphorylation of Na(+)/H(+) exchanger isoform 3 (NHE3), distribution of NHE3 at the

166 ll molecule inhibitor of the sodium/hydrogen exchanger isoform 3 that functions in the gut to reduce

167 nique relative to other cells in which these exchanger isoforms have been characterized and it has th

168 Therefore, the sodium-hydrogen exchanger may play a central role in the interplay of di

169 In placenta, these exchangers mediate placental uptake of substrates for oe

170 tes with the solute carrier family 26 (anion exchanger)-member 9 (SLC26A9) cRNA, promoted WNK4 autoph

171 le and nonpolarizable Donnan exclusion anion-exchanger membrane reference/counter electrode.

172 we introduced previously ionophore-free ion exchanger membranes doped with a well controlled ratio o

173 hibition of the mitochondrial sodium-calcium exchanger (mNCE) resulted in a rise in [Ca(2+) ]m at bas

174 ion could be completely retained on a cation-exchanger Nafion membrane, constituting a colorimetric s

175 d lipid-binding properties of the Na(+)/H(+) exchanger NapA from Thermus thermophilus and compare thi

176 hannels and extrusion via Na(+)/Ca(2+), K(+) exchangers (NCKX1).

177 e-specific isoform of the Na(+)/Ca(2+), K(+) exchanger, NCKX2, is responsible for removing Ca(2+) fro

178 Here, we show that the Na(+)/Ca(2+), K(+) exchanger NCKX4 is expressed in zebrafish, mouse, and pr

179 mice lacking the K(+)-dependent Na(+)/Ca(2+)-exchanger, NCKX4 (gene slc24a4 or Nckx4), display a rema

180 in activating the mitochondrial Na(+)/Ca(2+) exchanger (NCLX) causing enhanced mitochondrial Na(+) up

181 lc8b1 encodes the mitochondrial Na(+)/Ca(2+) exchanger (NCLX), which is proposed to be the primary me

182 at accomplish this task are the Na(+)/Ca(2+) exchanger (NCX) and the Na(+)/K(+) pump (NKA).

183 The Na(+)-Ca(2+) exchanger (NCX) appears to play an important role in the

184 lts implicate activation of the Na(+)-Ca(2+) exchanger (NCX) as an important player in the generation

185 icate T-type CaV channels and sodium-calcium exchanger (NCX) as predominant mechanisms of calcium inf

186 function by activation of electrogenic Na/Ca exchanger (NCX) during diastole.

187 The Na(+)/Ca(2+) exchanger (NCX) has been implied to cause arrhythmias.

188 Ca transport by the sodium-calcium exchanger (NCX) is determined by Vm as well as Na and Ca

189 advantage of atrial-specific sodium-calcium exchanger (NCX) knockout (KO) SAN cells to study the inf

190 In atrial-specific sodium-calcium exchanger (NCX) knockout (KO) SAN cells, where the Ca(2+

191 In the atrial-specific Na(+) /Ca(2+) exchanger (NCX) knockout mouse, cellular Ca(2+) accumula

192 The electrogenic Na/Ca exchanger (NCX) mediates bidirectional Ca movements that

193 The electrogenic sodium/calcium exchanger (NCX) mediates bidirectional calcium transport

194 +) mediated by the plasmalemmal Na(+)/Ca(2+) exchanger (NCX) operating in the reverse mode.

195 eration of the plasma membrane Na(+) /Ca(2+) exchanger (NCX) plays a key role in Ca(2+) influx for tr

196 Na(+)/Ca(2+) exchanger (NCX) proteins operate through the alternating

197 eration of the plasma membrane Na(+) /Ca(2+) exchanger (NCX) rather than presynaptic depolarization o

198 Inhibition of Na(+)-Ca(2+) exchanger (NCX) slowed [Ca(2+)]SR decline during rest by

199 cellular Ca(2+) implicating the Na(+)/Ca(2+) exchanger (NCX), a major Ca(2+) extrusion mechanism acti

200 in Ca(2+)-extruding system, the Na(+)/Ca(2+) exchanger (NCX), in NGF-induced differentiation remains

201 mibefradil) and by blocking the Na(+)/Ca(2+) exchanger (NCX), suggesting an important contribution of

202 reased the Na+ level sensed by the Na+, Ca2+ exchanger (NCX).

203 repressor ID2 and the cardiac sodium-calcium exchanger NCX1 in the proximal CCS, where GATA6 transact

204 The Na(+)/Ca(2+) exchanger NCX1 was found to interact with Ano6 in a two-

205 ) oscillations induced by the sodium/calcium exchanger NCX1/3 working in its reverse mode.

206 by sequential expression of the Na(+)-Ca(2+) exchanger (NCX1) and L-type Ca(2+) channel (LTCC).

207 elopments regarding the cardiac Na(+)/Ca(2+) exchanger (NCX1) and Na(+)/K(+) pump and the controversi

208 erized cardiac promoters, the sodium calcium exchanger (Ncx1) and the brain natriuretic peptide (Bnp)

209 Isoform 3 of the Na(+)-Ca(2+) exchanger (NCX3) is crucial for maintaining intracellula

210 Na(+)/Ca(2+) exchangers (NCXs) are ubiquitous membrane transporters w

211 andem with the Na(+)-dependent Cl(-)/HCO3(-) exchanger (NDCBE) encoded by Slc4a8 to mediate NaCl abso

212 pendrin and the Na(+)-driven Cl(-)/2HCO3(-) exchanger (NDCBE) in beta-intercalated cells of the coll

213 he exchange activity of the human Na(+)/H(+) exchanger NHA2 (SLC9B2) is electroneutral, despite harbo

214 nvestigation of an electroneutral Na(+)/H(+) exchanger, NhaP1 from Methanocaldococcus jannaschii (MjN

215 th epithelial Na(+) channels (ENaC) and Na-H exchangers (NHE), inhibition of NHE-mediated Na(+) absor

216 In human melanoma, the Na(+)/H(+) exchanger NHE1 is an important modifier of the tumour na

217 The Na+/H+ exchanger NHE1 regulates proximal tubule cell survival t

218 and impaired activity of the sodium/hydrogen exchanger NHE1, a known regulator of skin pH.

219 We found that the expression of Na+/H+ exchanger NHE3 and several scaffold proteins, including

220 The epithelial brush-border Na(+)/H(+) exchanger NHE3 is acutely inhibited by cGKII/cGMP, but h

221 Basal activity of the BB Na(+)/H(+) exchanger NHE3 requires multiprotein complexes that form

222 We show that the neuron-enriched Na(+)/H(+) exchanger NHE5 is abundantly expressed in C6 glioma cell

223 The Na(+)/H(+) exchanger NHE6 provides a leak pathway for protons, limi

224 the predominantly Golgi-resident Na(+)/H(+) exchanger NHE8 localizes to the developing acrosome of s

225 s a leak pathway for protons, the Na(+)/H(+) exchanger NHE9 limits luminal acidification to circumven

226 Multiple Na(+)/H(+) exchangers (NHEs) are implicated in maintenance of neura

227 utations in human endosomal Na(+)(K(+))/H(+) exchangers (NHEs) NHE6 and NHE9 are implicated in neurol

228 Mammalian Na(+)/H(+) exchangers (NHEs) regulate numerous physiological proces

229 acidic synaptic vesicles and (3) Na(+) /H(+) exchangers (NHEs).

230 ard cells, the tonoplast-localized K(+)/H(+) exchangers NHX1 and NHX2 are pivotal in the vacuolar acc

231 A study of the sodium/calcium exchanger now suggests that secondary structure may hold

232 Sodium/proton exchangers of the SLC9 family mediate the transport of p

233 comparison to the optode containing the ion exchanger only (Miptode 3), the optode containing the io

234 onophores in addition to a lipophilic cation-exchanger, overlaid on an electropolymerized poly-3-octy

235 the parallel operation of the Cl(-)/HCO3(-) exchanger pendrin and the Na(+)-driven Cl(-)/2HCO3(-) ex

236 arizes the contribution of the Cl(-)/HCO3(-) exchanger pendrin in distal nephron function.

237 trin, L-type calcium channel, sodium-calcium exchanger, phospholamban, calcineurin, and calcium/calmo

238 nding of the scaffolding proteins Na(+)/H(+) exchanger regulatory factor (NHERF) 1 and 2.

239 NHE3 directly binds Na(+)/H(+) exchanger regulatory factor (NHERF) family scaffolding p

240 In this study, Na(+)/H(+) exchanger regulatory factor (NHERF)-1 loss-of-function a

241 o interact with the PDZ protein, Na(+) /H(+) exchanger regulatory factor 1 (NHERF-1/EBP50).

242 ther the scaffolding protein sodium-hydrogen exchanger regulatory factor 1 (NHERF1) interacts with th

243 The scaffolding protein Na(+)/H(+) exchanger regulatory factor 2 (NHERF2) bridges iNOS with

244 in (MAGI)-1, MAGI-2, and MAGI-3], Na(+)/H(+) exchanger regulatory factor proteins (NHERFs) (NHERF1, N

245 The pancreatic expression of Na/H exchanger regulatory factor-1 and cystic fibrosis transm

246 Importantly, deletion of Na/H exchanger regulatory factor-1 had no deleterious effect

247 Wild-type and Na/H exchanger regulatory factor-1 knockout mice.

248 Deletion of Na/H exchanger regulatory factor-1 resulted in gross mislocal

249 se models and mice with deletion of the Na/H exchanger regulatory factor-1 that have selectively impa

250 Deletion of Na/H exchanger regulatory factor-1, which specifically impair

251 dinonylnaphthalene sulfonate (TDMA/DNNS) ion-exchanger salt.

252 Na(+) compartmentalization (i.e. Na(+)/H(+) EXCHANGERs, SALT OVERLY SENSITIVE, HIGH-AFFINITY K+ TRAN

253 voltage-gated chloride flux through the ion exchanger SLC26A11, active KCC2-mediated chloride extrus

254 Loss of the AE3 Cl(-)/HCO3(-) exchanger (Slc4a3) in mice causes an impaired cardiac fo

255 re quantified by immunoblots and the leucine exchanger (SLC7A5) and glutamine transporter (SLC38A2),

256 Band 3 (also known as the anion exchanger, SLCA1, AE1) constitutes the major attachment

257 This mechanism may work through a Na(+)/H(+) exchanger (sNHE) and/or a bicarbonate transporter, which

258 In addition, the sperm-specific Na(+)/H(+) exchanger (sNHE) possess a putative voltage sensor that

259 transporter (NBC1) and apical Cl(-) /HCO3(-) exchanger (solute carrier family 26 member A6; SLC26A6),

260 These values are greater than current anion exchangers such as the resins Amberlite IRA-400 (249 mg/

261 A mutation eliminating the Na(+)/Ca(2+) exchanger suppresses the gap-crossing phenotype of trpga

262 ability to serve as a highly localized lipid exchanger that ensures that PtdIns synthesis is matched

263 ClC-4 is an intracellular Cl(-)/H(+) exchanger that is highly expressed in the brain and whos

264 Pendrin is a Na(+)-independent Cl(-)/HCO3(-) exchanger that localizes to type B and non-A, non-B inte

265 This transporter is a Ca(2+)-H(+) exchanger that raises cell surface pH.

266 conium cyclosilicate is a nonabsorbed cation exchanger that selectively binds potassium in the intest

267 lude that SlCAT9 is a tonoplast Glu/Asp/GABA exchanger that strongly influences the accumulation of t

268 NCLX is a Na(+)/Ca(2+) exchanger that uses energy stored in the transmembrane s

269 However, exchangers that cannot be palmitoylated do not inactivat

270 characterized of all pH-regulated Na(+)/H(+) exchangers that control cellular Na(+) and H(+) homeosta

271 ier (Slc) family 26A encodes different anion exchangers that exchange Cl(-)/HCO3 (-), including Slc26

272 ngly retained than the analyte ion on an ion exchanger, the analyte peak is fronting.

273 ction of several classes of ion channels and exchangers, they could act as functional "reporters" for

274 The latter could be used as an ion exchanger through manipulation of transport proteins in

275 action, employing an organic polymeric anion exchanger through the enrichment of lipoproteins/peptide

276 altering the equilibrium of the Na(+)-Ca(2+) exchanger to impair forward-mode (Ca(2+) extrusion), and

277 Raising [Na(+)]i activates the Na(+)/Ca(2+) exchanger to operate in a reverse mode leading to Ca(2+)

278 The membrane condenser is used as a heat exchanger to recover the latent heat of the exiting vapo

279 he surface provide dual-functionality as ion-exchangers to the hybrid material, firstly used for the

280 of TGF-beta, microRNA-21, and sodium calcium-exchanger type 1 were upregulated, whereas the levels of

281 releases increase forward mode Na(+)/Ca(2+) exchanger type 1, thereby slowing repolarization and ena

282 rough increased forward mode of Na(+)/Ca(2+) exchanger type 1.

283 aminoindanes as inhibitors of the Na(+)/H(+) exchanger type 3 (NHE3) are described based on a hit fro

284 Chicken Na(+)/H(+) exchanger type I (chNHE1), a multispan transmembrane pro

285 rafenib treatment and activate an Na(+)/H(+) exchanger type-1 (NHE1), may underlie tyrosine kinase in

286 successful application as industrial cation exchangers under near neutral conditions, their performa

287 ified SlCAT9 as a candidate protein for this exchanger using quantitative proteomics of a tonoplast-e

288 A tubular thermal exchanger was used for a rapid cooling treatment (CT) of

289 ally verified a kinetic model for Na(+)/H(+) exchangers, where a single binding site is alternatively

290 tions (avoiding excess of ionophore over ion-exchanger), which is purposely different from common pra

291 NCX1 is a Na(+)/Ca(2+) exchanger, which is believed to provide a key route for

292 ctionally interacts with the sodium-hydrogen exchanger, which is responsible for the majority of sodi

293 Acute inhibition of the sodium-calcium exchanger, which targets the downstream effects of enhan

294 lites represent a family of inorganic cation exchangers, which naturally occur in hyper alkaline cond

295 PNs, thus, represent an immobilized ion exchanger with ion sorting properties high enough to par

296 ins are enriched by the interaction of anion exchanger with the phosphate groups and eluted at the pH

297 enetic evidence linking endosomal Na(+)/H(+) exchangers with Alzheimer disease, suggest that proton l

298 oss through a series of counter-current heat exchangers within its gills.

299 n quantifying changes in channels, pumps and exchangers without quantitatively linking these changes

300 , due to reduced extrusion through the Na/Ca exchanger, without affecting resting Na(+) and Ca(2+) co