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1 nantly express calretinin, thus appear to be excitatory.
2 citatory to inhibitory or from inhibitory to excitatory.
3  MOB where 5-HT2A receptors mediate a direct excitatory action.
4 ypes perform context-dependent modulation of excitatory activity, as well as regulate experience-depe
5 specifically manipulate CA1 pyramidal neuron excitatory activity, electrophysiology, hippocampus-sele
6         Although pharmacologic modulation of excitatory amino acid receptors is well studied, minimal
7 with FcgammaRs triggers coendocytosis of the excitatory amino acid transporter 2 (EAAT2).
8 f AMPH-dependent trafficking of the neuronal excitatory amino acid transporter 3 (EAAT3) blocks poten
9 y have enabled a deeper understanding of how excitatory amino acid transporters (EAATs) mediate chlor
10 centration occurs in part through astrocytic excitatory amino acid transporters (EAATs).
11  of neuronal diversity in the LTMR-RZ: seven excitatory and four inhibitory subtypes of interneurons
12 tal brain disorders arise from imbalances in excitatory and inhibitory (E/I) brain circuitry.
13 portant function for Robo2 in regulating the excitatory and inhibitory balance of the adult brain.
14 med unsupervised clustering on the resulting excitatory and inhibitory connection patterns.
15        The increases in excitability in both excitatory and inhibitory cortical neurons show that sel
16  of RTT-associated nuclear transcriptomes in excitatory and inhibitory cortical neurons.
17 itation and inhibition and tACS acts on both excitatory and inhibitory cortical neurons.
18             In neural networks with balanced excitatory and inhibitory engrams, the release of innate
19                             The CeA receives excitatory and inhibitory inputs from the basolateral nu
20 ere we report findings on the integration of excitatory and inhibitory inputs in healthy cortical cir
21  and stability relies on the coregulation of excitatory and inhibitory inputs onto principal neurons.
22            Using a ring network, we show how excitatory and inhibitory interactions can implement dif
23  of graft-derived cells by light resulted in excitatory and inhibitory junction potentials, the elect
24 here was a significant reduction in detected excitatory and inhibitory monosynaptic connections.
25 by splicing and RNA editing, localize to >20 excitatory and inhibitory neocortical neuron types defin
26 ous frequency reflect a common change in the excitatory and inhibitory neural activity that regulates
27 rf72 promoter activity is widespread in both excitatory and inhibitory neurons as well as in oligoden
28 ordings from primary visual cortex layer 2/3 excitatory and inhibitory neurons in awake mice during p
29 and establish synaptic transmission with BLA excitatory and inhibitory neurons in late infancy, event
30  guided whole-cell recordings from layer 2/3 excitatory and inhibitory neurons in the visual cortex o
31 erone in rodents) and is widely expressed in excitatory and inhibitory neurons, as well as in glial c
32 4 overexpression increases synapse number in excitatory and inhibitory neurons, suggesting an instruc
33 l cortex require the coordinated activity of excitatory and inhibitory neurons.
34  (R)-baclofen rescued the imbalances between excitatory and inhibitory neurotransmission evident in F
35 urons, such as mediating the balance between excitatory and inhibitory neurotransmission within the p
36 from imbalances in the relative strengths of excitatory and inhibitory neurotransmission.
37 ein, we use UV and visible radiation as both excitatory and inhibitory signals for the communication
38 ty-evoked responses appear normal while both excitatory and inhibitory spontaneous events exhibit red
39 cular mechanisms that control the balance of excitatory and inhibitory synapse function remain poorly
40 poorly understood; no proteins that regulate excitatory and inhibitory synapse strength in a coordina
41 napses, and chloride transporter function at excitatory and inhibitory synapses and facilitate inhibi
42  Ca(2+) dependence of spontaneous release at excitatory and inhibitory synapses and heterogeneity of
43 isk factor controls E/I ratios by regulating excitatory and inhibitory synapses in opposing direction
44 sufficiency also led to an imbalance between excitatory and inhibitory synapses in the cerebral corte
45 he values of the CB1 receptor-immunopositive excitatory and inhibitory synapses were Glu-CB1 -RS, 21.
46                         Select pairs promote excitatory and inhibitory synapses, with neuroligin 2 (N
47               As arbors mature, they acquire excitatory and inhibitory synapses, with the latter form
48 nderlying mechanisms of neurotransmission at excitatory and inhibitory synapses.
49  and they derive their selectivity both from excitatory and inhibitory synaptic inputs from other neu
50  plateau potentials interact with subsequent excitatory and inhibitory synaptic inputs remains unknow
51  Neurons perform computations by integrating excitatory and inhibitory synaptic inputs.
52 ation, retrieval engages PL BDNF to regulate excitatory and inhibitory synaptic proteins neuroligin 1
53 ty mechanisms to stabilize firing, including excitatory and inhibitory synaptic scaling and homeostat
54 coding, and is accompanied by alterations in excitatory and inhibitory synaptic transmission, and int
55 een MCs and TCs are caused in part by weaker excitatory and stronger inhibitory currents onto MCs tha
56 l direct current stimulation (tDCS) with the excitatory anode either over contralateral or ipsilatera
57                                         This excitatory ARC-->PVH satiety circuit, and its modulation
58 he amygdala complex, and local plasticity in excitatory basolateral amygdala principal neurons is con
59 human intracranial (iEEG) work we found that excitatory broadband high-frequency activity transients,
60             Knockdown of cadherin-10 reduces excitatory but increases inhibitory synapse size and str
61   Recent studies have suggested that the two excitatory cell classes of the mammalian olfactory bulb,
62 n-activated MnPO neurons consist of a single excitatory cell type.
63 mmonly encountered units have RFs with small excitatory centers, combined with very extensive inhibit
64                                  The loss of excitatory cholinergic modulation may play a critical ro
65  drive iris-sphincter-muscle contraction via excitatory cholinergic parasympathetic innervation [1, 2
66             We describe a direct orexin-->D2 excitatory circuit and show that D2 cell activity is nec
67          However, activity and plasticity of excitatory circuits are tightly controlled by local inhi
68                      Besides the rewiring of excitatory circuits, an abnormal depolarizing gamma-amin
69  regulate experience-dependent plasticity of excitatory circuits.
70 ociative synaptic potentiation in prefrontal excitatory circuits.
71  postsynaptic potentials (PSPs), with a fast excitatory component followed by a slower inhibitory com
72                                              Excitatory connections among neurons within a cluster we
73                           When these lateral excitatory connections become small enough the OPM disap
74 thalamic input is amplified by the recurrent excitatory connections of the L4 circuits.
75         An increase in the radius of lateral excitatory connections subsequently increases the size o
76 re SE form functional recurrent monosynaptic excitatory connections with other granule cells.
77 dings from brain slices to explore intrinsic excitatory connectivity of the claustrum.
78 rop in intrinsic excitability and pruning of excitatory corticostriatal glutamatergic synapses.
79 increased responses evoked by stimulation of excitatory corticothalamic and inhibitory tectothalamic
80                  We found that with stronger excitatory coupling, a positive relationship emerged bet
81         A subset of C-IN pairs had increased excitatory cross-correlations during hypoxic episodes (3
82       We attempted to disable this intrinsic excitatory current by artificially activating an inhibit
83 eased steeply, suggesting that the intrinsic excitatory current was probably responsible for the init
84                  Thus, increased activity of excitatory DH interneurons coupled with the reduced exci
85 euron dendrites, provide an unusually strong excitatory drive at the soma of CA2 pyramidal neurons, w
86                                              Excitatory drive from the FE dominated amblyopic visual
87 n in non-adapting cells, suggesting a higher excitatory drive in adapting neurons.
88                                OXT increased excitatory drive onto reward-specific VTA dopamine (DA)
89                                 In contrast, excitatory drive was boosted to the adapting-firing exci
90  forming new synapses resulting in increased excitatory drive.
91           We built a network rate model with excitatory (E) and inhibitory (I) populations exhibiting
92                                         Both excitatory (E) and inhibitory (I) spinal neurons are nec
93                                  Second, the excitatory edges have nearby suppressive edges with orth
94             Concomitantly, immunolabeling of excitatory ending markers revealed an increase in the im
95                                          The excitatory events that trigger myotonic action potential
96 rate that serotonergic afferents are largely excitatory for mitral cells (MCs) in the MOB where 5-HT2
97 x believed to be integral to ILD processing (excitatory from one ear, inhibitory from the other: EI c
98 g the activity threshold required to produce excitatory GABAergic signaling, weaker stimuli are able
99         Besides the involvement of the major excitatory (glutamate) and inhibitory (gamma aminobutyri
100 s indicate that chronic cocaine use enhances excitatory glutamatergic input to these neurons, making
101                                          The excitatory glutamatergic synapse is the principal site o
102 e examine the role of upstream inputs to PCs-excitatory granule cells (GCs) and inhibitory molecular
103 ity signals and novelty signals arising from excitatory habenula and dopaminergic ventral tegmentum i
104                                              Excitatory hilar mossy cells (MCs) in the dentate gyrus
105 ce, resulting in selective expression of the excitatory hM3 receptor or the inhibitory hM4 receptor i
106 hat nAChR-mediated regulation of hippocampal excitatory-inhibitory balance could be a promising thera
107 ed gamma oscillatory activity and a shift to excitatory-inhibitory balance in PFC neural activity.
108     After a daily dosing regimen, a shift to excitatory-inhibitory balance in single-unit activity an
109 s, suggesting different rules for functional excitatory-inhibitory interactions in non-murine species
110               This establishment of neuropil excitatory-inhibitory structure largely precedes dendrit
111 nied by presynaptic effects that disrupt the excitatory/inhibitory (E/I) balance.
112 ICANCE STATEMENT The correct balance between excitatory/inhibitory (E/I) is crucial for normal brain
113 uronal subtype-specific contributions to the excitatory/inhibitory balance and circuit maturation.
114 cellular "infrastructure" that maintains the excitatory/inhibitory balance of the nervous system.
115 neurodevelopmental disorder characterized by excitatory/inhibitory imbalance and dopamine (DA) dysfun
116 se in ACC than in LPFC, resulting in a lower excitatory: inhibitory ratio and a greater dynamic range
117                  Therefore, there is a lower excitatory: inhibitory synaptic ratio and a greater dyna
118 -MSN dendritic atrophy, reduced frequency of excitatory input and altered in vivo activity.
119  was also disrupted, dampening low-frequency excitatory input and potentiating high-frequency sustain
120 ndirectly retract the vibrissae receive only excitatory input from interpolaris cells that further pr
121 adult hippocampus develops concurrently, and excitatory input is reorganized by exercise.
122 labeled area, supporting abnormally elevated excitatory input numbers.
123 y is generated by the interplay of dendritic excitatory inputs and axonal inhibitory inputs.
124 en the spatiotemporal window for integrating excitatory inputs and promote spiking.
125 tially clustered and temporally synchronized excitatory inputs at the distal dendrites could trigger
126 r complex T4 inputs and reveal that putative excitatory inputs cluster at T4's dendrite shafts, while
127                                   Vestibular excitatory inputs in Group I motoneurons are mediated pr
128  calcium channel composition and efficacy of excitatory inputs in the presence of dopamine inhibition
129 show that, in vivo, short-term plasticity of excitatory inputs to CA3 pyramidal cells combines with r
130 o probe the excitability of distinct sets of excitatory inputs to corticospinal neurons during the wa
131 ation during adolescence requires pruning of excitatory inputs to PV interneurons.
132 Developmental pruning eliminates superfluous excitatory inputs, suggesting that working memory matura
133 ive neurons exhibit increased sensitivity to excitatory inputs, which can then trigger large dendriti
134 ning competing information from less salient excitatory inputs.
135  (0-100 spikes s(-1) ) and number (0-800) of excitatory inputs.
136 tentials in response to temporally dispersed excitatory inputs.
137 tly has no effect on electrically stimulated excitatory inputs.
138  our understanding of the role of inhibitory-excitatory interactions.
139 ent evidence for a unique, tonically active, excitatory interneuron in the songbird basal ganglia tha
140                                          The excitatory interneuron thus serves as one biophysical me
141                      Circuitoids of purified excitatory interneurons were sufficient to generate osci
142            Yet as >80% of synapses in V1 are excitatory, it remains unanswered whether the majority o
143 duced amplitude of both evoked and miniature excitatory junctional potentials.
144 ory drive was boosted to the adapting-firing excitatory lamina II interneurons while GABAergic and gl
145 l delivery of the inhibitory SNAG-mGluR2 and excitatory light-activated ionotropic glutamate receptor
146                                However, like excitatory maps, inhibitory sensory maps are sensitive t
147 n, by preventing inappropriate activation of excitatory memory engrams, inhibitory engrams can make m
148 e of experience-dependent plasticity between excitatory mitral cells (MCs) and inhibitory internal gr
149 respectively, in inhibitory interneurons and excitatory mitral projection neurons of the main olfacto
150            The current data characterize the excitatory nature of neural projections activated by cis
151  Methylation signatures identified a layer 6 excitatory neuron subtype and a unique human parvalbumin
152 ius of the astrocytes, the extent of lateral excitatory neuronal connections can be manipulated.
153 rther investigation in the inhibitory versus excitatory neuronal networks and microcircuit connectivi
154 iscriminated between putative inhibitory and excitatory neurons and found that the inhibitory st-LFP
155 signatures both for the regular-spiking (RS) excitatory neurons and the fast-spiking (FS) inhibitory
156 primarily affect inhibitory neurons, whereas excitatory neurons are more sensitive to stimulus specif
157 ted in obese mice, and induction of Cadm1 in excitatory neurons facilitated weight gain while exacerb
158                                     However, excitatory neurons from the Emx-Cre; Clock(flox/flox) mo
159 rojections to paraventricular thalamus (PVT) excitatory neurons immediately (in 2 to 3 seconds) evoke
160          Two-photon imaging showed that many excitatory neurons in auditory cortex were suppressed du
161 tor subtype in whole neuronal populations or excitatory neurons in mPFC facilitates the induction of
162                                  Layer (L) 4 excitatory neurons in the barrel cortex, the major targe
163              Adding an adaptation current to excitatory neurons leads to spontaneous alternations bet
164         Results show autaptic connections to excitatory neurons with high average controllability led
165 interneurons in clusters and the same nearby excitatory neurons.
166  target and directly activate inhibitory and excitatory neurons.
167 ine and movement-related activity in layer 4 excitatory neurons.
168 ed a mouse overexpressing Ube3a isoform 2 in excitatory neurons.
169  ionotropic glutamate receptors mediate fast excitatory neurotransmission and are implicated in numer
170             Recent reports indicate enhanced excitatory neurotransmission in cortical neurons express
171 -type ionotropic glutamate receptors mediate excitatory neurotransmission in the central nervous syst
172                                         Fast excitatory neurotransmission is mediated by AMPA-subtype
173  ionotropic glutamate receptors mediate fast excitatory neurotransmission throughout the central nerv
174 ntial vanilloid type 1-dependent increase of excitatory neurotransmission was reduced in liver-relate
175 mmetric postsynaptic densities, a feature of excitatory neurotransmission.
176  receptors and the role of AMPA receptors in excitatory neurotransmission.
177 , ligand-gated ion channels activated by the excitatory neurotransmitter glutamate and have well-char
178                    Glutamate is the dominant excitatory neurotransmitter in the brain, but under cond
179 sing ion channels (ASICs), a small family of excitatory neurotransmitter receptors implicated in pain
180 cation channel P2X7, allowing the release of excitatory neurotransmitters to sustain spreading depola
181 modified newborn mice that specifically lack excitatory NTS neurons, we show that they are both mute
182 l SNR, the amplitude of each current, either excitatory or disinhibitory, was proportional to its SNR
183 elative to monetary rewards, and by applying excitatory or inhibitory repetitive transcranial magneti
184  synapse numbers but differentially impaired excitatory or inhibitory synaptic functions in an isofor
185       The pOFC also sends a uniquely one-way excitatory pathway to the amygdalar inhibitory intercala
186  circuits in generating functionally diverse excitatory pathways and suggest that decorrelation of pa
187 e, MLD/pedal peptide, allatotropin, RNamide, excitatory peptide, and FVRIamide showed a broad localiz
188 -adolescent GAD67-GFP male mice, we examined excitatory plasticity in fluorescent VTA GABA cells.
189                                These include excitatory populations that transmit information about b
190 t for boosting the amplitude and duration of excitatory post synaptic currents near the action potent
191  taum and is also able to prolong artificial excitatory post synaptic currents.
192 ion process of important synaptic behaviors, excitatory post-synaptic current (EPSC), has been update
193 pre-synaptic HCN channels enhances miniature excitatory post-synaptic currents (mEPSCs) onto EC layer
194 tance, enhanced spiking, and slowed decay of excitatory post-synaptic potentials (EPSPs).
195 uronal dendrites that harbor the majority of excitatory postsynapses.
196 nd significant increases in both spontaneous excitatory postsynaptic current (spEPSC) amplitude and R
197 pro-apoptotic gene Bax in stem cells reduced excitatory postsynaptic currents (EPSCs) and spine densi
198 d the frequency of spontaneous and miniature excitatory postsynaptic currents (s/mEPSCs).
199 stsynaptic currents (sIPSCs) and spontaneous excitatory postsynaptic currents (sEPSCs) three- and two
200 ex of BC1 knock out (KO) mice display larger excitatory postsynaptic currents and increased spontaneo
201 ritic density and the frequency of miniature excitatory postsynaptic currents in the mPFC were preven
202 ve only subtle consequences for nerve-evoked excitatory postsynaptic currents: vesicle heterogeneity,
203 tion of these synapses in vitro evoked large excitatory postsynaptic responses in the majority of pyr
204                We show that changes occur in excitatory postsynaptic structure and function in the so
205 ctions from the motor cortex, generate rapid excitatory potentials followed by slower inhibitory pote
206 ound that one-third of mice lacking Nalcn in excitatory preBotC neurons died soon after birth; surviv
207 nd that cocaine place conditioning increases excitatory presynaptic and postsynaptic transmission in
208 cated on ascending inhibitory and descending excitatory presynaptic inputs onto MGB neurons, likely i
209                        The number of opposed excitatory presynaptic terminals was sharply reduced upo
210 genic mouse to label and analyze proteins in excitatory principal neurons and Purkinje neurons in vit
211 tal stages with enrichment of glutamatergic (excitatory) processes laterally and glycinergic (inhibit
212                            This hypothalamic excitatory projection predominates over LHb neurons mono
213            These findings may be related via excitatory projections from the DLPFC to the ventral mes
214 nsporter 2 (VGLUT2), providing evidence that excitatory projections mediate cisplatin-induced energy
215         The basolateral amygdala (BLA) sends excitatory projections to the nucleus accumbens (NAc) an
216    An integrate-and-fire model incorporating excitatory pyramidal and inhibitory interneurons indicat
217 rimates involves the coordinated activity of excitatory pyramidal neurons and a specific population o
218  Effects of conditional Lis1 inactivation in excitatory pyramidal neurons, starting in juvenile mouse
219 translates this into compensatory changes in excitatory quantal amplitude.
220                                     Putative excitatory regular-spiking (RS) and inhibitory fast-spik
221 ystems create and use inhibitory replicas of excitatory representations for important cognitive funct
222 projecting vCA1 neurons induced monosynaptic excitatory responses in both the mPFC and basal amygdala
223 reotyped 3-5 Hz Vm oscillations that disrupt excitatory responsiveness to visual stimuli.
224                      Other units have larger excitatory RF centers, but these always cover a coherent
225 where inhibitory amacrine cells modulate the excitatory signal of bipolar and ganglion cells.
226 acellular calcium homeostasis in response to excitatory signaling.
227 , we report on the modulation of the gain of excitatory signals from the AE by signals from its domin
228  demonstrate a functional distribution among excitatory SNc afferent nuclei in response to cocaine, a
229 al alters the balance between inhibitory and excitatory spiking activity directly.
230                   We find that assemblies of excitatory spinal interneurons are recruited by sensory
231               The theta-phase preference and excitatory strength of the afferent CA3 and entorhinal i
232 MMP-dependent shedding of NgR1 in regulating excitatory synapse development.SIGNIFICANCE STATEMENT In
233 todomain of NgR1 is sufficient to accelerate excitatory synapse formation in dissociated cortical neu
234                   However, how OGT regulates excitatory synapse function is largely unknown.
235 , PSD-95, and SAP102 differentially regulate excitatory synapse properties in the NAc.
236                                              Excitatory synapses and fast-spiking properties matured
237  is a form of synaptic plasticity induced at excitatory synapses by metabotropic glutamate receptors
238 reted glypican 4 induces formation of active excitatory synapses by recruiting AMPA glutamate recepto
239                 The postsynaptic proteome of excitatory synapses comprises 1,000 highly conserved pr
240 esent findings supporting a special role for excitatory synapses connecting pyramidal neurons of the
241 age clamp is completely ineffective for most excitatory synapses due to spine electrical compartmenta
242 , and two-photon Ca(2+) imaging, we analyzed excitatory synapses formed by climbing fibers on Purkinj
243                         Astrocytes depressed excitatory synapses from basolateral amygdala via A1 ade
244 ease, to be necessary for the development of excitatory synapses in cortical neurons.
245 mediate neurotransmission at the majority of excitatory synapses in the brain.
246                         Synaptic strength at excitatory synapses is determined by the presence of glu
247 hat neuroligin-3 specifies the properties of excitatory synapses on parvalbumin-containing interneuro
248                                              Excitatory synapses on PV interneurons are dependent on
249                  KCC2 is highly localized to excitatory synapses where it regulates spine morphogenes
250 ed with changes in the strength or number of excitatory synapses with MCs but was instead associated
251 have been identified as the Ca(2+) sensor at excitatory synapses, but the Ca(2+) sensor(s) at inhibit
252                            Microglia control excitatory synapses, but their role in inhibitory neurot
253 nd chaperone levels, maintenance of striatal excitatory synapses, clearance of Htt aggregates and pre
254 orn shortly after SE did not form functional excitatory synapses, despite robust sprouting.
255 ntaining NMDA receptors (NMDARs) at immature excitatory synapses, via a transcription-dependent mecha
256 eptor (R) synaptic transmission, or silenced excitatory synapses, whereas more prolonged (24 hr) firi
257 pocampal CA1 neurons without affecting their excitatory synapses.
258 T is enriched in the postsynaptic density of excitatory synapses.
259 d with an increase in an inward current from excitatory synapses.
260 apses and neuroligin 1 (NLGN1) dominating at excitatory synapses.
261 on of double-projecting vCA1 neurons induces excitatory synaptic activity in both the mPFC and amygda
262 rocircuits are interconnected with recurrent excitatory synaptic connections that are thought to ampl
263    We observe that with weak gap junction or excitatory synaptic coupling, network heterogeneity and
264 emichannel activity reduced the LTP of these excitatory synaptic currents triggered by high-frequency
265        Our results demonstrate a key role of excitatory synaptic drive in shaping the function of mot
266                    Consistent with a role of excitatory synaptic drive in the maturation of PV(+) neu
267 ice lacking PRMT8 reveal multiple defects in excitatory synaptic function and plasticity.
268 insight into the role of presenilins (PS) in excitatory synaptic function, we address the relevance o
269 ry cortex, and a deficit in the formation of excitatory synaptic inputs on to these neurons in neonat
270 f intrinsic ion conductances, inhibitory and excitatory synaptic inputs that differ among this cell p
271     This result shows that the regulation of excitatory synaptic plasticity is fundamentally altered
272 ion specific as no differences were found in excitatory synaptic properties in the NAc shell.
273 ype IV (CaMKIV) is a key sensory/effector in excitatory synaptic scaling that senses perturbations in
274 Previous studies have shown that PE enhances excitatory synaptic strength by facilitating an anti-Heb
275  GLP-1 receptor (GLP-1R) activation augments excitatory synaptic strength in PVN corticotropin-releas
276 ion risk which could be mediated by enhanced excitatory synaptic strength in ventral tegmental area (
277 nhanced LTP and the maintenance of augmented excitatory synaptic strength in VTA DA neurons and incre
278  could result in the maintenance of enhanced excitatory synaptic strength in VTA DA neurons, which in
279 e, mPFC-BLA circuits are further modified by excitatory synaptic strengthening as well as a transient
280 ate uptake in astrocytes and how this shapes excitatory synaptic transmission among neurons.
281 t this early life stressor leads to enhanced excitatory synaptic transmission and decreased levels of
282 e receptors (AMPARs) mediate the majority of excitatory synaptic transmission and their function impa
283            Excessive levels of Abeta disrupt excitatory synaptic transmission by promoting the remova
284 act with neurons and blood vessels and shape excitatory synaptic transmission due to their abundant e
285 -type glutamate receptors that underlie fast excitatory synaptic transmission in the SDH.
286 d decreases both dendritic spine density and excitatory synaptic transmission.
287 ion leads to short- and long-term changes in excitatory synaptic transmission.
288 om inhibitory thalamic reticular nucleus and excitatory tectothalamic terminals.
289  local field potentials evoked in the OFC by excitatory thalamic afferent stimulation, and this was p
290 with an abrupt shift from glutamate-dominant excitatory to GABA-dominant inhibitory processing in ear
291 euronal apoptosis, and an increased ratio of excitatory to inhibitory (E/I) neurotransmission.
292 pears to change the sign of the synapse from excitatory to inhibitory or from inhibitory to excitator
293 n hits a key neurodevelopmental process, the excitatory-to-inhibitory gamma-aminobutyric acid switch;
294 nerating a functional diversity that sculpts excitatory transmission and behavioral function.
295                     A persistent increase in excitatory transmission can also be triggered by exogeno
296  mGlu3 induces long-term depression (LTD) of excitatory transmission in the PFC at inputs from the ba
297 e important for the bidirectional scaling of excitatory transmission; however, whether this subunit p
298                                              Excitatory V2a interneurons in particular are an integra
299              We localized a subpopulation of excitatory vasopressin neurons in the anterior hypothala
300  reveal that ATM associates exclusively with excitatory (VGLUT1(+)) vesicles, while ATR associates on

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