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1 ude of the NMDA component of a glutamatergic excitatory postsynaptic current.
2 ency and amplitude of alpha3*-nAChR-mediated excitatory postsynaptic currents.
3 hosphorylation restricts the potentiation of excitatory postsynaptic currents.
4 GluR1), and AMPA receptor-mediated miniature excitatory postsynaptic currents.
5  and peak amplitude of spontaneous miniature excitatory postsynaptic currents.
6 26E), as determined by analysis of miniature excitatory postsynaptic currents.
7 ecruit surface AMPA receptors and potentiate excitatory postsynaptic currents.
8 traction did not alter parallel fiber-evoked excitatory postsynaptic currents.
9 entiation and NMDA receptor (NMDAR)-mediated excitatory postsynaptic currents.
10 tory postsynaptic currents with no effect on excitatory postsynaptic currents.
11 ely contribute to the biexponential decay of excitatory postsynaptic currents.
12 a(2+) concentration ([Ca(2+)](i) spikes) and excitatory postsynaptic currents.
13 pontaneous action potentials and spontaneous excitatory postsynaptic currents.
14 rrents and increased the amplitude of evoked excitatory postsynaptic currents.
15  in the amplitude and frequency of miniature excitatory postsynaptic currents.
16 teral amygdala pyramidal neurons, generating excitatory postsynaptic currents.
17 ing the frequency of glutamatergic miniature excitatory postsynaptic currents.
18 e amplitude, but not frequency, of miniature excitatory postsynaptic currents.
19 glia induces a rapid increase of spontaneous excitatory postsynaptic currents.
20 kable depression of AMPAR-mediated miniature excitatory postsynaptic currents, a significant reductio
21    Evoked excitatory transmitter release and excitatory postsynaptic currents also were heightened at
22 ion and reduced decay time of AMPAR-mediated excitatory postsynaptic currents (AMPAR-EPSCs), enhanced
23 on elicits a long-lasting decrease in evoked excitatory postsynaptic current amplitude and a delayed,
24 rea, as well as an increase in the miniature excitatory postsynaptic current amplitude and frequency.
25  mediated partly by an increase in miniature excitatory postsynaptic current amplitude and partly by
26                                              Excitatory postsynaptic current amplitude was reduced to
27 ptic current frequencies, although miniature excitatory postsynaptic current amplitudes remained simi
28 ed hippocampal neurons showed that miniature excitatory postsynaptic current amplitudes were larger i
29 yptamine1b-type serotonin receptor to reduce excitatory postsynaptic current amplitudes, an effect pr
30 A-type glutamate receptor-mediated miniature excitatory postsynaptic currents, an effect dependent on
31 significant metabolic advantage over quantal excitatory postsynaptic currents--an advantage that may
32 a short-term plasticity model and cumulative excitatory postsynaptic current analysis to quantify the
33                       They contribute to the excitatory postsynaptic current and to the detection of
34 botropic glutamate receptor type 1)-mediated excitatory postsynaptic currents and a reduced sensitivi
35 or single cKO of NL1 impaired NMDAR-mediated excitatory postsynaptic currents and abolished NMDAR-dep
36 cally reduced AMPA receptor (AMPAR)-mediated excitatory postsynaptic currents and AMPAR surface expre
37 of vesicle exocytosis accompanied by loss of excitatory postsynaptic currents and commensurately pert
38 t inhibition of these channels reduces PF-PC excitatory postsynaptic currents and excitatory postsyna
39 al slices, the compound reversibly increased excitatory postsynaptic currents and field excitatory po
40 sured via electrophysiological recordings of excitatory postsynaptic currents and hippocampal long-te
41 ex of BC1 knock out (KO) mice display larger excitatory postsynaptic currents and increased spontaneo
42 d the amplitude and frequency of spontaneous excitatory postsynaptic currents and increased the ampli
43 red using electrophysiological recordings of excitatory postsynaptic currents and long-term potentiat
44  and NR2B expression but also NMDAR-mediated excitatory postsynaptic currents and potentials, without
45 s of CPT1C KO mice, AMPAR-mediated miniature excitatory postsynaptic currents and synaptic levels of
46 nnelrhodopsin-2 elicited both inhibitory and excitatory postsynaptic currents and triggered network b
47 s demonstrate that kainate-receptor-mediated excitatory postsynaptic currents are decreased by SUMOyl
48 mbrane potential of 0 mV where glutamatergic excitatory postsynaptic currents are near equilibrium.
49 neuronal" dynamins, dynamin 1 and 3, smaller excitatory postsynaptic currents are observed due to an
50 ly coupled myenteric neurons, nicotinic fast excitatory postsynaptic currents are occluded during act
51 causes a significant decrease in spontaneous excitatory postsynaptic currents at both two and twenty
52  in the amplitude and frequency of miniature excitatory postsynaptic currents at neuromuscular synaps
53 f variability in the amplitudes of miniature excitatory postsynaptic currents at single synapses reve
54      In control animals, IL-6 did not affect excitatory postsynaptic currents but selectively and rev
55 naptically localized ASICs contribute to the excitatory postsynaptic current by responding to the tra
56   Moreover, the depression of AMPAR-mediated excitatory postsynaptic currents by SNRIs required p38 k
57  yielded slower kinetics in vitro and slower excitatory postsynaptic current decays in neurons.
58                   The frequency of miniature excitatory postsynaptic currents decreased, accompanied
59 ls was reduced, the frequency of spontaneous excitatory postsynaptic currents decreased, while the fr
60  recording techniques, evoked or spontaneous excitatory postsynaptic currents (eEPSCs or sEPSCs) were
61 0-50 nm) also produced an increase of evoked excitatory postsynaptic currents (eEPSCs) at mossy fibre
62 oM) significantly decreased the amplitude of excitatory postsynaptic currents (eEPSCs) evoked by stim
63  but had no significant effect on the evoked excitatory postsynaptic currents (eEPSCs) in 10 of these
64                                              Excitatory postsynaptic currents (eEPSCs) were evoked by
65 educed the amplitude of evoked NMDA-mediated excitatory postsynaptic currents (eEPSCs), without affec
66 leading to increased amplitudes of miniature excitatory postsynaptic currents, enhancement of LTP, an
67         Win 55212-2 and Delta9-THC inhibited excitatory postsynaptic current (EPSC) amplitude by 96 +
68     Two additional properties, Q and average excitatory postsynaptic current (EPSC) amplitude, were u
69 r, exclusion of these failures leads to mean excitatory postsynaptic current (EPSC) amplitudes that a
70 ransmitter release, as measured by miniature excitatory postsynaptic current (EPSC) analysis and FM 1
71 creased in normal mice, a glutamate-mediated excitatory postsynaptic current (EPSC) between mitral ce
72  measures: steady-state NMDA currents, NMDAR excitatory postsynaptic current (EPSC) decay kinetics, p
73 mice, as revealed by a decrease in miniature excitatory postsynaptic current (EPSC) frequency and in
74 tion of trigeminal afferent fibres evoked an excitatory postsynaptic current (EPSC) in trigeminal neu
75 gnificant enhancement of the voltage-clamped excitatory postsynaptic current (EPSC) occurs during DSI
76 own that long-term potentiation (LTP) of the excitatory postsynaptic current (EPSC) through glutamate
77 he trapezoid body (MNTB) neurones during 1 s excitatory postsynaptic current (EPSC) trains delivered
78                   Fast P2X receptor-mediated excitatory postsynaptic current (EPSC) was identified in
79 te (NMDA) receptor-mediated component of the excitatory postsynaptic current (EPSC), but did not affe
80 pontaneous, miniature and stimulation-evoked excitatory postsynaptic current (EPSC).
81 gs confirmed that this aberrant AP evoked an excitatory postsynaptic current (EPSC).
82  generates a slow, kainate-receptor-mediated excitatory postsynaptic current (EPSC).
83 but not paired-pulse ratio of NMDAR-mediated excitatory postsynaptic currents (EPSC) in PFC slices.
84 y is used to reverse ion influxes generating excitatory postsynaptic currents (EPSCs) and action pote
85  At parallel fiber synapses, mGluR1-mediated excitatory postsynaptic currents (EPSCs) and associated
86  quiescent MS patients on glutamate-mediated excitatory postsynaptic currents (EPSCs) and excitotoxic
87 that presynaptic depression of glutamatergic excitatory postsynaptic currents (EPSCs) and GABAergic i
88 )-Baclofen depressed the amplitude of evoked excitatory postsynaptic currents (EPSCs) and inhibitory
89                               NMDAR-mediated excitatory postsynaptic currents (EPSCs) and puff NMDA-e
90 pro-apoptotic gene Bax in stem cells reduced excitatory postsynaptic currents (EPSCs) and spine densi
91                     THC (1 microM) inhibited excitatory postsynaptic currents (EPSCs) and whole-cell
92 MF-gc) synapses of mature cerebellum, evoked excitatory postsynaptic currents (EPSCs) are multiquanta
93 orsal root evoked non-NMDA receptor-mediated excitatory postsynaptic currents (EPSCs) at -60 mV that
94   AMPA-type glutamate receptors mediate most excitatory postsynaptic currents (EPSCs) at central syna
95 luzole nearly completely depresses glutamate excitatory postsynaptic currents (EPSCs) at concentratio
96  (5-HT) receptor signaling potently inhibits excitatory postsynaptic currents (EPSCs) between lamprey
97 d in a slowing of the decay time constant of excitatory postsynaptic currents (EPSCs) by approximatel
98 ective locus for the generation of increased excitatory postsynaptic currents (EPSCs) by serotonin (5
99 he effects of extracellular zinc on NR1/NR2A excitatory postsynaptic currents (EPSCs) by simulating t
100 rtion of glutamate uncaging sites from which excitatory postsynaptic currents (EPSCs) could be evoked
101 PCs showed increased duration of spontaneous excitatory postsynaptic currents (EPSCs) during the symp
102 sent study aims to explore corticogeniculate excitatory postsynaptic currents (EPSCs) evoked by brief
103 ly identified neurons were patch-clamped and excitatory postsynaptic currents (EPSCs) evoked by elect
104 es N-methyl-D-aspartate (NMDA) responses and excitatory postsynaptic currents (EPSCs) evoked by elect
105   CRF (30-300 nM) increased the amplitude of excitatory postsynaptic currents (EPSCs) evoked by stimu
106                                              Excitatory postsynaptic currents (EPSCs) evoked by stimu
107 cantly reduced the amplitude of monosynaptic excitatory postsynaptic currents (EPSCs) evoked from the
108                                              Excitatory postsynaptic currents (EPSCs) following focal
109 se in the postnatal rat cochlea by recording excitatory postsynaptic currents (EPSCs) from afferent b
110 es are changed during lactation, we recorded excitatory postsynaptic currents (EPSCs) from identified
111 on-like behaviors and associated deficits in excitatory postsynaptic currents (EPSCs) generated in ap
112 marked increase in glutamatergic spontaneous excitatory postsynaptic currents (EPSCs) in apical dendr
113               Therefore, we recorded unitary excitatory postsynaptic currents (EPSCs) in caudal raphe
114 apses to sensory representation by recording excitatory postsynaptic currents (EPSCs) in cerebellar g
115 dramatic reduction of AMPA receptor-mediated excitatory postsynaptic currents (EPSCs) in cortical neu
116 ncentration-dependent inhibition of autaptic excitatory postsynaptic currents (EPSCs) in cultured hip
117 ds and cones that in turn produced transient excitatory postsynaptic currents (EPSCs) in horizontal a
118 used an increase in frequency of spontaneous excitatory postsynaptic currents (EPSCs) in layer V pyra
119 gnificantly increased the amplitude of basal excitatory postsynaptic currents (EPSCs) in MAGL(-/-) mi
120                                              Excitatory postsynaptic currents (EPSCs) in monosynaptic
121                       ST stimulation elicits excitatory postsynaptic currents (EPSCs) in NTS neurons
122 as addressed here by examining glutamatergic excitatory postsynaptic currents (EPSCs) in rat autaptic
123 armacologically isolate KA receptor-mediated excitatory postsynaptic currents (EPSCs) in rat hippocam
124 ion of the STN evoked complex, long-duration excitatory postsynaptic currents (EPSCs) in SNR neurons.
125 1) receptor-mediated inhibition of glutamate excitatory postsynaptic currents (EPSCs) in the nucleus
126 SCs; retigabine had no significant effect on excitatory postsynaptic currents (EPSCs) mediated by act
127                             Here we examined excitatory postsynaptic currents (EPSCs) of dorsal horn
128  increase in the amplitude of NMDAR-mediated excitatory postsynaptic currents (EPSCs) of dorsal horn
129             Here, we found that evoked NMDAR-excitatory postsynaptic currents (EPSCs) of retrogradely
130 aptic stimulation at 1 Hz for 30 s, enhanced excitatory postsynaptic currents (EPSCs) on non-pyramida
131 methyl-D-aspartate receptor (NMDAR)-mediated excitatory postsynaptic currents (EPSCs) onto VTA dopami
132 urkinje cells by measuring the inhibition of excitatory postsynaptic currents (EPSCs) produced by a l
133                                          The excitatory postsynaptic currents (EPSCs) recorded in lam
134 celeration of AMPA receptor (AMPAR)-mediated excitatory postsynaptic currents (EPSCs) remains elusive
135                                Evaluation of excitatory postsynaptic currents (EPSCs) revealed that r
136                In second-order NTS neurones, excitatory postsynaptic currents (EPSCs) synaptically ev
137  firing has been shown to arise from complex excitatory postsynaptic currents (EPSCs) that are evoked
138       Trains with 10 stimuli at 25 Hz evoked excitatory postsynaptic currents (EPSCs) that grew in am
139 y stimulation produced bursts of mossy fibre excitatory postsynaptic currents (EPSCs) that summate to
140 n of nucleus tractus solitarii (NTS) induced excitatory postsynaptic currents (EPSCs) that were reduc
141  of N-methyl-d-aspartate (NMDA) component of excitatory postsynaptic currents (EPSCs) through a posts
142 MDA (N-methyl-d-aspartate) receptor-mediated excitatory postsynaptic currents (EPSCs) was decreased a
143                                 Monosynaptic excitatory postsynaptic currents (EPSCs) were evoked by
144       Whole-cell voltage clamp recordings of excitatory postsynaptic currents (EPSCs) were performed
145                                   Whole-cell excitatory postsynaptic currents (EPSCs) were recorded f
146                                              Excitatory postsynaptic currents (EPSCs) were recorded f
147                                              Excitatory postsynaptic currents (EPSCs) were recorded u
148                         Evoked glutamatergic excitatory postsynaptic currents (EPSCs) were reversibly
149                                       Evoked excitatory postsynaptic currents (EPSCs) were reversibly
150                                              Excitatory postsynaptic currents (EPSCs) were studied in
151 ous and miniature (m) AMPA receptor-mediated excitatory postsynaptic currents (EPSCs) were studied in
152 e enhanced the frequency of spontaneous fast excitatory postsynaptic currents (EPSCs) with no change
153  synapses, potentiated AMPA receptor (AMPAR) excitatory postsynaptic currents (EPSCs), and occluded L
154 -methyl-D-aspartate (NMDA) receptor-mediated excitatory postsynaptic currents (EPSCs), but not alpha-
155 on in the perforant path-evoked monosynaptic excitatory postsynaptic currents (EPSCs), or in the intr
156                                       Evoked excitatory postsynaptic currents (EPSCs), spontaneous EP
157 R1 and beta(2)AR, and AMPA receptor-mediated excitatory postsynaptic currents (EPSCs).
158 ors (NMDARs) and a slowing of NMDAR-mediated excitatory postsynaptic currents (EPSCs).
159 ed into CA1 pyramidal cells while monitoring excitatory postsynaptic currents (EPSCs).
160 ith little variability compared with quantal excitatory postsynaptic currents (EPSCs).
161 d +100 mmol/l fructose) reversibly depressed excitatory postsynaptic currents (EPSCs).
162  increase in the amplitude of impulse-evoked excitatory postsynaptic currents (EPSCs).
163  sites on the time course of Purkinje neuron excitatory postsynaptic currents (EPSCs).
164 ion and decreased the frequency of miniature excitatory postsynaptic currents (EPSCs).
165 t with this, PrRP increased the amplitude of excitatory postsynaptic currents (EPSCs, 154 +/- 33%, 12
166 neous and miniature excitatory transmission (excitatory postsynaptic currents, EPSCs).
167                 At 42 h after ischemia, AMPA excitatory postsynaptic currents exhibited pronounced in
168 b consisted of an initial fast glutamatergic excitatory postsynaptic current followed by a slow-risin
169 ic NAc core inputs, we recorded light-evoked excitatory postsynaptic currents following viral-mediate
170 erentiation, together with reduced miniature excitatory postsynaptic current frequencies and behavior
171 d, consequently, by a reduction of miniature excitatory postsynaptic current frequencies, although mi
172 nsity is associated with increased miniature excitatory postsynaptic current frequency and amplitude,
173                      Second, basal miniature excitatory postsynaptic current frequency in alpha(1A) (
174 ly, P2Y1 stimulation of astrocytes increased excitatory postsynaptic current frequency through a meta
175 nd DG GCs exhibit increased mEPSC (miniature excitatory postsynaptic current) frequency.
176 ise generated by these synapses, we recorded excitatory postsynaptic currents from mammalian retinal
177                     The decay time course of excitatory postsynaptic currents generated by slow gluta
178 ency led to reduced frequencies of miniature excitatory postsynaptic currents, i.e., to defects in sy
179 hospholipase D in the generation of the slow excitatory postsynaptic current in cerebellar Purkinje c
180 recordings revealed a concomitant nonquantal excitatory postsynaptic current in the calyx terminal th
181 0%) attenuation of electrically evoked local excitatory postsynaptic current in the saline and ShA gr
182 ses the frequency of AMPA-mediated miniature excitatory postsynaptic currents in CA1 pyramidal neuron
183        Stimulation of the vagus nerve evoked excitatory postsynaptic currents in CVNs that were rever
184 m, we show that the frequency of spontaneous excitatory postsynaptic currents in dentate gyrus granul
185 equency, but not the amplitude, of miniature excitatory postsynaptic currents in dorsal horn neurons
186 uced calcium influx, and increased miniature excitatory postsynaptic currents in hippocampal neurons.
187 endrites and decrements in apically targeted excitatory postsynaptic currents in layer V pyramidal ce
188 cleus accumbens measuring glutamate-mediated excitatory postsynaptic currents in medium spiny neurons
189 d synaptic spine density/diameter, increased excitatory postsynaptic currents in mPFC layer V pyramid
190 n hippocampal slices and autaptically evoked excitatory postsynaptic currents in neuronal cultures wi
191                                              Excitatory postsynaptic currents in post-ischemic CA1 ex
192 mulation of superficial layer neurons evoked excitatory postsynaptic currents in premotor cells.
193 ently, the rate of spontaneous and miniature excitatory postsynaptic currents in pyramidal neurons an
194 (2+) responses despite a marked reduction in excitatory postsynaptic currents in response to whisker
195 nd causes increased frequency of spontaneous excitatory postsynaptic currents in spinal cord nocicept
196 e effectively block NMDAR-mediated miniature excitatory postsynaptic currents in the absence of Mg(2+
197 ritic density and the frequency of miniature excitatory postsynaptic currents in the mPFC were preven
198 s serotonin-induced increases in spontaneous excitatory postsynaptic currents in the mPFC, mimicking
199 as cocaine produced comparable inhibition of excitatory postsynaptic currents in the nucleus accumben
200 ge clamp to record spontaneous and miniature excitatory postsynaptic currents in the presence of caly
201 nist at GABA(A) receptors, strongly enhanced excitatory postsynaptic currents in young rats but had l
202 xcitatory synaptic transmission (spontaneous excitatory postsynaptic currents) in spinal lamina IIo n
203 oid subtype-1 (TRPV1)- and TNF-alpha-induced excitatory postsynaptic current increases and TNF-alpha-
204                      Specifically, miniature excitatory postsynaptic currents, input resistance, hipp
205 notypic slices, NMDAR-dependent LTD of AMPAR excitatory postsynaptic currents is abolished in neurons
206           Indeed, the amplitude of miniature excitatory postsynaptic currents is enhanced in both Vac
207                            By regulating KAR excitatory postsynaptic current kinetics, Neto1 can cont
208 ipolar cell output by recording light-evoked excitatory postsynaptic currents (L-EPSCs) from postsyna
209 rent study, the light-evoked and spontaneous excitatory postsynaptic currents (light-evoked EPSCs and
210 ptors in the cerebellum also leads to a slow excitatory postsynaptic current mediated by nonselective
211  for AMPAR clustering at synapses, miniature excitatory postsynaptic currents mediated by TARPless AM
212 nic acid receptor (AMPAR)-mediated miniature excitatory postsynaptic current (mEPSC) amplitudes due t
213 he inhibitory synapses assessed by miniature excitatory postsynaptic current (mEPSC) and electron mic
214  subunit, reduces the frequency of miniature excitatory postsynaptic current (mEPSC), and reduces AMP
215 de and frequency of AMPAR-mediated miniature excitatory postsynaptic current (mEPSC), while expressin
216 fects were seen with glutamatergic miniature excitatory postsynaptic currents (mEPSCs) and GABAergic
217                                The miniature excitatory postsynaptic currents (mEPSCs) and miniature
218 tic scaling up of the amplitude of miniature excitatory postsynaptic currents (mEPSCs) and of synapti
219 uced increases in the frequency of miniature excitatory postsynaptic currents (mEPSCs) and the number
220 ic events were larger than quantal miniature excitatory postsynaptic currents (mEPSCs) but had the sa
221 ic density (PSD) and contribute to miniature excitatory postsynaptic currents (mEPSCs) elicited by si
222                                    Miniature excitatory postsynaptic currents (mEPSCs) from embryonic
223 ers, octopus and T stellate cells, miniature excitatory postsynaptic currents (mEPSCs) had similar sh
224 ad no effect on glutamate-mediated miniature excitatory postsynaptic currents (mEPSCs) in 12 of 15 ne
225 napses, allowing us to record NMDA-miniature excitatory postsynaptic currents (mEPSCs) in addition to
226 mplitude of the NMDAR component of miniature excitatory postsynaptic currents (mEPSCs) in CA1 interne
227  in the frequency of glutamatergic miniature excitatory postsynaptic currents (mEPSCs) in cardiac vag
228 uency and amplitude of spontaneous miniature excitatory postsynaptic currents (mEPSCs) in cultured mo
229                   The frequency of miniature excitatory postsynaptic currents (mEPSCs) in principal n
230 sed a decrease in the frequency of miniature excitatory postsynaptic currents (mEPSCs) in SON neurone
231 frequency but not the amplitude of miniature excitatory postsynaptic currents (mEPSCs) mediated by al
232 ole cell patch clamp recordings of miniature excitatory postsynaptic currents (mEPSCs) revealed that
233                                    Miniature excitatory postsynaptic currents (mEPSCs) were recorded
234 hibition were blocked, spontaneous miniature excitatory postsynaptic currents (mEPSCs) were recorded.
235 e amplitude and charge transfer of miniature excitatory postsynaptic currents (mEPSCs) were significa
236 ctivity increased the amplitude of miniature excitatory postsynaptic currents (mEPSCs) without changi
237 on increased the amplitude of AMPA miniature excitatory postsynaptic currents (mEPSCs), and shRNA-med
238 ith electron microscopy, and giant miniature excitatory postsynaptic currents (mEPSCs), reflecting mo
239 rapidly decreases the amplitude of miniature excitatory postsynaptic currents (mEPSCs), suggesting th
240 rfold increase in the frequency of miniature excitatory postsynaptic currents (mEPSCs).
241  in the amplitude and frequency of miniature excitatory postsynaptic currents (mEPSCs).
242 ollowed by reduction in evoked and miniature excitatory postsynaptic currents (mEPSCs).
243          Moreover, analysis of the miniature excitatory postsynaptic currents (mEPSCs)/inhibitory pos
244 quency or amplitude of spontaneous miniature excitatory postsynaptic currents (mEPSCs); however, syna
245 porters and mGluRs by evoking mGluR-mediated excitatory postsynaptic currents (mGluR EPSCs) in slices
246 n addition to reduced frequency of miniature excitatory postsynaptic currents (mini-EPSCs) and smalle
247 ed the frequency of miniature inhibitory and excitatory postsynaptic currents (mIPSCs and mEPSCs, res
248 ly isolated miniature NMDA receptor-mediated excitatory postsynaptic currents (mN-EPSCs) were recorde
249 AA IPSCs) and reduces NMDA receptor-mediated excitatory postsynaptic currents (NMDA EPSCs) in a conce
250          We recorded NMDA and AMPA miniature excitatory postsynaptic currents (NMDA- and AMPA-mESPCs)
251 RG neurons and the amplitude of monosynaptic excitatory postsynaptic currents of dorsal horn neurons
252 rrents in small DRG neurons and monosynaptic excitatory postsynaptic currents of spinal dorsal horn n
253 nt with experimental recordings of miniature excitatory postsynaptic currents only when ectopic trans
254 s do not associate with changes in miniature excitatory postsynaptic currents or paired-pulse facilit
255 ermeability and a change in the amplitude of excitatory postsynaptic currents, owing to the incorpora
256       We found that in leaner PCs, PF-evoked excitatory postsynaptic currents (PF-EPSCs) are approxim
257 AMPARs, generating rapid, lasting changes in excitatory postsynaptic current properties.
258   These mice exhibit a decrease in NMDA/AMPA excitatory postsynaptic current ratio in area CA1 of the
259 d the frequency of spontaneous glutamatergic excitatory postsynaptic currents recorded from these neu
260                          Quantal analysis of excitatory postsynaptic currents, recorded at neuromuscu
261 on responded more actively with an increased excitatory postsynaptic current response upon the applic
262 d the frequency of spontaneous and miniature excitatory postsynaptic currents (s/mEPSCs).
263 holding current, but facilitated spontaneous excitatory postsynaptic current (sEPSC) frequency in 41%
264                                  Spontaneous excitatory postsynaptic currents (sEPSC) recorded in cNT
265 ion by reducing the frequency of spontaneous excitatory postsynaptic current (sEPSCs).
266                 Spontaneous and light-evoked excitatory postsynaptic currents (sEPSCs and leEPSCs) in
267 y of glutamatergic spontaneous and miniature excitatory postsynaptic currents (sEPSCs and mEPSCs, res
268 n frequency but not amplitude of spontaneous excitatory postsynaptic currents (sEPSCs) and an increas
269 nctionally affected, we examined spontaneous excitatory postsynaptic currents (sEPSCs) and dopamine (
270  195 % and amplitude to 118 % of spontaneous excitatory postsynaptic currents (sEPSCs) during expirat
271                      We recorded spontaneous excitatory postsynaptic currents (sEPSCs) from lamina II
272 eased the frequency of spontaneous glutamate excitatory postsynaptic currents (sEPSCs) in >90% of NTS
273 crt-2 increased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) in a few neuro
274 0.6%) decreased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) only in Ndufs4
275 stsynaptic currents (sIPSCs) and spontaneous excitatory postsynaptic currents (sEPSCs) three- and two
276  in adults where the increase in spontaneous excitatory postsynaptic currents (sEPSCs) was mediated s
277                                  Spontaneous excitatory postsynaptic currents (sEPSCs) were recorded
278 antly increased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) without affect
279  the frequency of spontaneous inhibitory and excitatory postsynaptic currents (sIPSCs and sEPSCs), wh
280 nd significant increases in both spontaneous excitatory postsynaptic current (spEPSC) amplitude and R
281 , the amplitude and frequency of spontaneous excitatory postsynaptic currents (spEPSCs) increased dur
282                           Virtually all fast excitatory postsynaptic currents studied so far in the c
283    As NR2a-containing NMDARs mediate shorter excitatory postsynaptic currents than those containing N
284 ed an increase in the frequency of miniature excitatory postsynaptic currents that could be rapidly a
285 pal neurons, trains of depolarizations evoke excitatory postsynaptic currents that show facilitation
286  increasing Dalpha7-nAChRs boosted miniature excitatory postsynaptic currents, the ensuing increase i
287                          First, two distinct excitatory postsynaptic currents transmit signals on dif
288 quencies, indicating that the rapid burst of excitatory postsynaptic currents underlying the sensory-
289 ve only subtle consequences for nerve-evoked excitatory postsynaptic currents: vesicle heterogeneity,
290  and the amplitude of AMPA receptor-mediated excitatory postsynaptic currents was also observed.
291 soxazolepropionic acid receptor ratio of the excitatory postsynaptic currents was significantly incre
292                                              Excitatory postsynaptic currents were dramatically reduc
293                 In control hippocampus, AMPA excitatory postsynaptic currents were electrically linea
294                                              Excitatory postsynaptic currents were large enough to ev
295 d unitary conductance of channels underlying excitatory postsynaptic currents were matched by those o
296                  Both evoked and spontaneous excitatory postsynaptic currents were predominantly glut
297 ihydroxy-7-niroquiinoxaline (CNQX)-sensitive excitatory postsynaptic currents were recorded from cell
298                                              Excitatory postsynaptic currents were recorded in respon
299 iated with a decrease in the size of quantal excitatory postsynaptic currents, whereas for mGluR-LTD
300 induced action potential firing and enhanced excitatory postsynaptic currents, whereas muscarinic ago

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