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1 A1 inhibitory postsynaptic potential into an excitatory postsynaptic potential.
2 currents sum effectively and produce a large excitatory postsynaptic potential.
3 eceptors mediate the slower component of the excitatory postsynaptic potential.
4 ersibly increased the slope and amplitude of excitatory postsynaptic potentials.
5  inhibitory effect of ethanol on NMDAR field excitatory postsynaptic potentials.
6 ion of ventral CA1 fibers, and this broadens excitatory postsynaptic potentials.
7 ices during extracellular recording of field excitatory postsynaptic potentials.
8 tentials from basket interneurons (BAS) into excitatory postsynaptic potentials.
9 erm potentiation and long-term depression of excitatory postsynaptic potentials.
10 equency but not the amplitude of spontaneous excitatory postsynaptic potentials.
11 mplitude and frequency of glutamate-mediated excitatory postsynaptic potentials.
12 cted by a concomitant decrease in the evoked excitatory postsynaptic potentials.
13  spike, but not those that exhibit only fast excitatory postsynaptic potentials.
14 n net synaptic efficacy as measured by field excitatory postsynaptic potentials.
15  temporal summation of their thalamocortical excitatory postsynaptic potentials.
16 ng spatial and temporal summation of smaller excitatory postsynaptic potentials.
17 AC-induced depression of inhibitory, but not excitatory, postsynaptic potentials.
18 isted of two components: pEPSP1, (population excitatory postsynaptic potential 1) and pEPSP2.
19 ime-dependent manner, significantly reducing excitatory postsynaptic potentials after a >/=30-min app
20      A plot of impulse rates or amplitude of excitatory postsynaptic potentials against ITDs (ITD cur
21 pines compartmentalize voltage, specifically excitatory postsynaptic potentials, albeit critical, rem
22                                 We find that excitatory postsynaptic potential amplitudes are inverse
23               The slope and amplitude of the excitatory postsynaptic potential and the number of evok
24 ression of the simultaneously recorded field excitatory postsynaptic potential and was greatly reduce
25                              Uncaging-evoked excitatory postsynaptic potentials and Ca transients are
26 pagating action potentials, the amplitude of excitatory postsynaptic potentials and dendritic excitab
27 erm potentiation (LTP) of both intracellular excitatory postsynaptic potentials and evoked field pote
28 by tetrodotoxin, and all display spontaneous excitatory postsynaptic potentials and IPSPs that remain
29 simultaneously recording the uncaging-evoked excitatory postsynaptic potentials and local Ca2+ signal
30 on in FB-m1KO hippocampal slices resulted in excitatory postsynaptic potentials and long-term synapti
31                                 Granule cell excitatory postsynaptic potentials and mitral cell inhib
32                   ASIC null mice had reduced excitatory postsynaptic potentials and NMDA receptor act
33 ) using voltage-sensitive dye imaging, field excitatory postsynaptic potentials and whole cell patch
34  shape and duration of the vestibular-evoked excitatory postsynaptic potential, and the time of onset
35 y depends on the magnitude and timing of the excitatory postsynaptic potentials, and that blockade of
36  action potentials fully invade spines, that excitatory postsynaptic potentials are large in the spin
37 enhancement of the monosynaptic sensorimotor excitatory postsynaptic potential, as compared with the
38 owever, the initial increase in the slope of excitatory postsynaptic potentials, as well as the eleva
39     Consistent with that idea, PTP of evoked excitatory postsynaptic potentials at Aplysia sensory-mo
40 ow dynamic range (1 log unit intensity), and excitatory postsynaptic potentials at light on and light
41 ment of mGlu II-mediated depression of field excitatory postsynaptic potentials at mossy fiber-CA3 sy
42 oes the low input resistance make individual excitatory postsynaptic potentials brief so that they mu
43 s PF-PC excitatory postsynaptic currents and excitatory postsynaptic potentials by 15-20%.
44 measured as the change in layer II/III field excitatory postsynaptic potentials by a multielectrode a
45 ow that reliably timed action potentials (or excitatory postsynaptic potentials) can be observed up t
46 nterneurons to the extent that even a single excitatory postsynaptic potential could initiate spiking
47 other neurons in the ARC and that all evoked excitatory postsynaptic potentials could be blocked by t
48 ion to neurotransmitter release, feedforward excitatory postsynaptic potentials could spread through
49 uency sensitivity of NMDA receptor-dependent excitatory postsynaptic potentials differed significantl
50 quency voltage fluctuations (presumed "field excitatory postsynaptic potentials") during 89% of chron
51  CA1 are associated with an increase in both excitatory postsynaptic potential (EPSP) amplitude and a
52 an excitatory action while also reducing the excitatory postsynaptic potential (EPSP) amplitude throu
53 llaterals depressed the initial slope of the excitatory postsynaptic potential (EPSP) in aged but not
54  a small amplitude, short latency population excitatory postsynaptic potential (EPSP) in the PRh.
55 cal pyramidal neurons, we observed an evoked excitatory postsynaptic potential (EPSP) or current (EPS
56 c population spike (PS) and minor effects on excitatory postsynaptic potential (EPSP) slope amplitude
57 mplitude was by 72+/-17% of control, and the excitatory postsynaptic potential (EPSP) slope was decre
58  IPSP, the amplitude of the cholinergic fast excitatory postsynaptic potential (EPSP) was partially i
59 tic potential (IPSP) is abolished before the excitatory postsynaptic potential (EPSP) when the extrac
60 postsynaptic potential (IPSP) followed by an excitatory postsynaptic potential (EPSP) whereas CSS onl
61 increase in stimulus intensity evoked a slow excitatory postsynaptic potential (EPSP) which was assoc
62 rimary afferent fibers-mediated monosynaptic excitatory postsynaptic potential (EPSP), and long-lasti
63 edback loop within the spine head; during an excitatory postsynaptic potential (EPSP), Ca(2+) influx
64 eta more potently blocks the potentiation of excitatory postsynaptic potential (EPSP)-spike coupling
65 arization of summating the NMDA component of excitatory postsynaptic potential (EPSP).
66 solitarii (TS) stimulation with a monophasic excitatory postsynaptic potential (EPSP).
67  depression reported for the I a-motoneurone excitatory postsynaptic potential (EPSP).
68 ribute to hippocampal CA(1) stratum radiatum excitatory postsynaptic potentials (EPSP) is a matter of
69 -methyl-D-aspartate (NMDA) receptor-mediated excitatory postsynaptic potentials (EPSPAs and EPSPNs, r
70  facilitation' of the NMDA receptor-mediated excitatory postsynaptic potential (EPSPN).
71 at activation of K(v)1 channels by dendritic excitatory postsynaptic potentials (EPSPs) accelerated m
72     As a result, subthreshold parallel fiber excitatory postsynaptic potentials (EPSPs) activate Cav3
73 fects Ca(2+) elevations evoked by coincident excitatory postsynaptic potentials (EPSPs) and back-prop
74 h that their activity modulates the shape of excitatory postsynaptic potentials (EPSPs) and increases
75 d temperature-insensitive neurones displayed excitatory postsynaptic potentials (EPSPs) and inhibitor
76 0 nM) increased the frequency of spontaneous excitatory postsynaptic potentials (EPSPs) and spontaneo
77 ked compound glutamatergic receptor-mediated excitatory postsynaptic potentials (EPSPs) and spontaneo
78 unisensory and multisensory stimulation with excitatory postsynaptic potentials (EPSPs) and/or action
79 uch a model: it is reversible, anti-hebbian (excitatory postsynaptic potentials (EPSPs) are depressed
80                                              Excitatory postsynaptic potentials (EPSPs) are greatly p
81 ification (increase in duration and area) of excitatory postsynaptic potentials (EPSPs) at depolarize
82                      After the depression of excitatory postsynaptic potentials (EPSPs) by 60 min of
83                      The depolarization from excitatory postsynaptic potentials (EPSPs) can inactivat
84 N-methyl-D-aspartate (NMDA) and compared the excitatory postsynaptic potentials (EPSPs) elicited by N
85  the observed EPSC changes, we also compared excitatory postsynaptic potentials (EPSPs) elicited by P
86                                              Excitatory postsynaptic potentials (EPSPs) elicited by s
87 cally connected cells were used to study the excitatory postsynaptic potentials (EPSPs) elicited in b
88 tion of oxytocin (1 and 10 microM) inhibited excitatory postsynaptic potentials (EPSPs) evoked by dor
89 upling determined the degree of summation of excitatory postsynaptic potentials (EPSPs) evoked by eac
90 ave performed a detailed quantal analysis of excitatory postsynaptic potentials (EPSPs) evoked by min
91                                              Excitatory postsynaptic potentials (EPSPs) evoked by pre
92 Specifically, we sought to determine whether excitatory postsynaptic potentials (EPSPs) evoked by sti
93             The width, area and rise time of excitatory postsynaptic potentials (EPSPs) evoked by sti
94 ibly decreased the amplitude of monosynaptic excitatory postsynaptic potentials (EPSPs) evoked in BA1
95 ne, but resembled asynchronous glutamatergic excitatory postsynaptic potentials (EPSPs) evoked in the
96 of the L-MAN and the HVc fiber tracts evoked excitatory postsynaptic potentials (EPSPs) from >70% of
97       Peripheral noxious stimulation induced excitatory postsynaptic potentials (EPSPs) in CA1 pyrami
98 vin-binding protein ApVAP33 inhibited evoked excitatory postsynaptic potentials (EPSPs) in cultured c
99 ge-gated A-type Kv4 channels shape dendritic excitatory postsynaptic potentials (EPSPs) in hippocampa
100 nels on dendritic processing of subthreshold excitatory postsynaptic potentials (EPSPs) in mouse CA1
101 on evoked faster rising and shorter duration excitatory postsynaptic potentials (EPSPs) in MSNs (n =
102  channels) modulate excitability and curtail excitatory postsynaptic potentials (EPSPs) in neuronal d
103  evoked abrupt increases in the frequency of excitatory postsynaptic potentials (EPSPs) in two thirds
104                            LTP occurred when excitatory postsynaptic potentials (EPSPs) led single po
105 lls, spikes evoked 5 ms after parallel-fiber excitatory postsynaptic potentials (EPSPs) led to long-t
106                                  We recorded excitatory postsynaptic potentials (EPSPs) of regular (n
107 te or pyramidal cells) (n = 6) produce large excitatory postsynaptic potentials (EPSPs) of up to 4 mV
108 ormed to investigate the interaction between excitatory postsynaptic potentials (EPSPs) or currents (
109 th the mean peak amplitudes of the resulting excitatory postsynaptic potentials (EPSPs) ranging betwe
110                          The visually evoked excitatory postsynaptic potentials (EPSPs) recorded duri
111           Here, by monitoring spine size and excitatory postsynaptic potentials (EPSPs) simultaneousl
112 rong stimulus by increasing the frequency of excitatory postsynaptic potentials (EPSPs) to TC cells,
113   In CA3 neurons, the amplitude and slope of excitatory postsynaptic potentials (EPSPs) transiently d
114                       Whether single unitary excitatory postsynaptic potentials (EPSPs) trigger spike
115                               Simulations of excitatory postsynaptic potentials (EPSPs) were analysed
116                                              Excitatory postsynaptic potentials (EPSPs) were elicited
117                       Short-latency (< 5 ms) excitatory postsynaptic potentials (EPSPs) were evoked i
118                                              Excitatory postsynaptic potentials (EPSPs) were evoked u
119 4) rats, TBS failed to induce LTP unless the excitatory postsynaptic potentials (EPSPs) were of suffi
120                           When low-amplitude excitatory postsynaptic potentials (EPSPs) were paired w
121                      Pairing of subthreshold excitatory postsynaptic potentials (EPSPs) with back-pro
122 d excitatory postsynaptic currents and field excitatory postsynaptic potentials (EPSPs) with threshol
123 es (paired-pulse facilitation, small initial excitatory postsynaptic potentials (EPSPs), a graded act
124 the endogenous release of glutamate, mediate excitatory postsynaptic potentials (EPSPs), alter presyn
125 hose temporal periods, and time constants of excitatory postsynaptic potentials (EPSPs), both increas
126 ential discharges to previously subthreshold excitatory postsynaptic potentials (EPSPs), even though
127 ulation of forward input evoked monosynaptic excitatory postsynaptic potentials (EPSPs), followed by
128 uctances have previously been shown to boost excitatory postsynaptic potentials (EPSPs).
129 n of cholinergic axons evoked nicotinic fast excitatory postsynaptic potentials (EPSPs).
130  the auditory nerve excited Golgi cells with excitatory postsynaptic potentials (EPSPs).
131            Each event was supported by giant excitatory postsynaptic potentials (EPSPs).
132 reatment had any significant effect on field excitatory postsynaptic potentials (EPSPs).
133 olarizations than do dendritic shafts during excitatory postsynaptic potentials (EPSPs).
134  evoked bAPs, some of which were paired with excitatory postsynaptic potentials (EPSPs).
135 A) caused a dose-related reduction in evoked excitatory postsynaptic potentials (EPSPs).
136 gically isolated N-methyl-d-aspartate (NMDA) excitatory postsynaptic potentials (EPSPs).
137 mplitude of the second of two rapidly evoked excitatory postsynaptic potentials (EPSPs).
138 rate at which each source was bombarded with excitatory postsynaptic potentials (EPSPs); and (3) the
139 ort-term depression, where successive evoked excitatory postsynaptic potentials (EPSPs; >5 Hz) usuall
140 anism depends on a passively conducted giant excitatory postsynaptic potential evoked by a mossy fibe
141 indle afferent population was estimated from excitatory postsynaptic potentials evoked by muscle stre
142                            Analysis of field excitatory postsynaptic potentials evoked by stimulation
143 tocol altered both the [Zn2+]t and the field excitatory postsynaptic potential (fEPSP) coordinately,
144 mined the cornu ammonis region 1 (CA1) field excitatory postsynaptic potential (fEPSP) response to co
145 tion had different actions on both the field excitatory postsynaptic potentials (fEPSPS) and LTP in t
146 s of isoflurane were similar on evoked field excitatory postsynaptic potentials (fEPSPs) and paired p
147 P) acting at P2 receptors mediates some fast excitatory postsynaptic potentials (fEPSPs) in myenteric
148      Intracellular recordings of evoked fast excitatory postsynaptic potentials (fEPSPs) in myenteric
149  acts at P2X receptors to contribute to fast excitatory postsynaptic potentials (fEPSPs) in myenteric
150 ically isolated NMDA-receptor-mediated field excitatory postsynaptic potentials (fEPSPs) in the CA1 r
151                             Spontaneous fast excitatory postsynaptic potentials (FEPSPs) occurred in
152                                        Field excitatory postsynaptic potentials (fEPSPs) or populatio
153 red oxygen and the amplitude of evoked field excitatory postsynaptic potentials (fEPSPs) recorded fro
154 ted one or two populations of nicotinic fast excitatory postsynaptic potentials (fEPSPs) that were gr
155                                        Field excitatory postsynaptic potentials (fEPSPs) were recorde
156                                        Field excitatory postsynaptic potentials (fEPSPs) were recorde
157                                        Field excitatory postsynaptic potentials (fEPSPs) were recorde
158  while in one AH-cell, some spontaneous fast excitatory postsynaptic potentials (FEPSPs) were recorde
159 perpolarizing pulse and (v) spontaneous fast excitatory postsynaptic potentials (FEPSPs).
160 oved the recovery of Schaffer collateral-CA1 excitatory postsynaptic potentials following a 15 min hy
161 potential, as compared with the sensorimotor excitatory postsynaptic potential in preparations that r
162 gulation of a prominent thalamocortical NMDA excitatory postsynaptic potential in stellate cells regu
163                         The S cell-triggered excitatory postsynaptic potential in the R cell diminish
164 n fact, both NMDAR- and AMPAR-mediated field excitatory postsynaptic potentials in CA1 decrease with
165 rlier reversal to polarizing currents of ATD excitatory postsynaptic potentials in comparison to thos
166 y correlated with both the onset of compound excitatory postsynaptic potentials in fast-spiking inter
167 d the size (amplitude and duration) of field excitatory postsynaptic potentials in hippocampal slices
168              Stimulation of the pTRG induced excitatory postsynaptic potentials in ipsi- and contrala
169    Excitatory stimuli drove trains of single excitatory postsynaptic potentials in relay cells, but g
170                             The light-evoked excitatory postsynaptic potentials in some types were re
171 A1 pyramidal neuron selectively inhibits the excitatory postsynaptic potentials in that cell.
172  optic tract stimulation routinely evoked an excitatory postsynaptic potential/inhibitory postsynapti
173                                              Excitatory postsynaptic potential-like voltage commands
174 e course of NMDA receptor-mediated miniature excitatory postsynaptic potentials (mEPSPs).
175 rkinje cell TRPC3 channels underlie the slow excitatory postsynaptic potential observed after paralle
176 uced a similar biphasic modulatory action on excitatory postsynaptic potentials or currents (EPSPs/EP
177 rated predominantly by synchronised cortical excitatory postsynaptic potentials oscillating at freque
178 of the rising phase of the evoked population excitatory postsynaptic potential (pEPSP).
179 pecific dissociation of synaptic [population excitatory postsynaptic potential (pEPSP)] and cellular
180 omega-conotoxin GVIA (CTX) on the population excitatory postsynaptic potentials (pEPSP) in stratum ra
181          Extracellularly-recorded population excitatory postsynaptic potentials (pEPSPs) in stratum r
182                                         Fast excitatory postsynaptic potentials recorded from S neuro
183  contributions are reduced because the early excitatory postsynaptic potential retards the opening of
184               Repetitive stimuli evoked slow excitatory postsynaptic potentials (SEPSPs) in some toni
185 urone excitability and/or contribute to slow excitatory postsynaptic potentials (sEPSPs).
186 nule cells NMDA receptors are increased, and excitatory postsynaptic potentials should be strongly NM
187 onstrated that ethanol inhibited NMDAR field excitatory postsynaptic potential slope and amplitude to
188 from control mice and (ii) the plot of field excitatory postsynaptic potential slope versus the popul
189 l recording was used to study a type of slow excitatory postsynaptic potential (slow EPSP) that was m
190 hippocampal area CA3 sum distal and proximal excitatory postsynaptic potentials sublinearly and activ
191 oping nicotine-induced potentiation of field excitatory postsynaptic potential that appeared to be de
192 r and nonlinear mechanisms were caused by an excitatory postsynaptic potential that reversed near 0 m
193 lectrical stimulation generated monosynaptic excitatory postsynaptic potentials that were indistingui
194 d under minimal synaptic stimulation and the excitatory postsynaptic potentials they generate.
195 , and slow-wave sleep, produce a large field excitatory postsynaptic potential throughout stratum rad
196                       NMDA receptors mediate excitatory postsynaptic potentials throughout the brain
197            Simultaneously, we recorded field excitatory postsynaptic potentials to monitor changes in
198                   Furthermore, recordings of excitatory postsynaptic potential-to-spike coupling (E-S
199       This inhibitory postsynaptic potential-excitatory postsynaptic potential transformation was pre
200 dual spines while monitoring uncaging-evoked excitatory postsynaptic potentials (uEPSPs) and Ca trans
201 nstant for STP of the AMPA receptor-mediated excitatory postsynaptic potential was approximately 6 mi
202  and ethanol's inhibition of the NMDAR field excitatory postsynaptic potential was attenuated by a br
203 nstant for STP of the NMDA receptor-mediated excitatory postsynaptic potential was only 1 min.
204               The SK channel contribution to excitatory postsynaptic potentials was absent in SK2-S o
205    High-frequency summation of AMPA-mediated excitatory postsynaptic potentials was smaller in OT neu
206  their activation during simulated dendritic excitatory postsynaptic potential waveforms.
207       Spontaneous depolarizations resembling excitatory postsynaptic potentials were observed at E12.
208 lateral pathway was stimulated and the field excitatory postsynaptic potentials were recorded in the
209 in post-tetanization the slopes of the field excitatory postsynaptic potentials were significantly di
210 red synaptic inputs as the amplitude of fast excitatory postsynaptic potentials were significantly la
211 ion that increase the size of NMDAR-mediated excitatory postsynaptic potentials, whereas at high conc
212 acellular spike-triggered averaging revealed excitatory postsynaptic potentials, which confirmed thes
213 m-CPP enhances the occurrence of spontaneous excitatory postsynaptic potentials with no alteration in

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