ライフサイエンスコーパス: excitatory synaptic transmission

1 ion leads to short- and long-term changes in excitatory synaptic transmission.

2 tral nervous system and play a major role in excitatory synaptic transmission.

3 synaptic states to maintain the strength of excitatory synaptic transmission.

4 , whereas HDAC2 overexpression (OE) reduced, excitatory synaptic transmission.

5 ve channels that mediate a slow component of excitatory synaptic transmission.

6 rm potentiation and long-term depression, of excitatory synaptic transmission.

7 endritic spines and functional plasticity of excitatory synaptic transmission.

8 t, neuronal or synaptic structures, or basal excitatory synaptic transmission.

9 ransporters have additional roles in shaping excitatory synaptic transmission.

10 ontaining, native receptors involved in fast excitatory synaptic transmission.

11 utamate receptors that mediate a majority of excitatory synaptic transmission.

12 eptors (AMPARs) at synapses is essential for excitatory synaptic transmission.

13 ent predominantly increased postsynaptically excitatory synaptic transmission.

14 the shift is expressed by a modification of excitatory synaptic transmission.

15 y been implicated in long-term plasticity of excitatory synaptic transmission.

16 om a shift of balance between inhibitory and excitatory synaptic transmission.

17 receptors mediate the majority of vertebrate excitatory synaptic transmission.

18 sease by removing dendritic spines, sites of excitatory synaptic transmission.

19 eased the spine density to markedly increase excitatory synaptic transmission.

20 d neuropathic pain facilitation by enhancing excitatory synaptic transmission.

21 tamate receptors (GluRs) play major roles in excitatory synaptic transmission.

22 receptors mediate the majority of vertebrate excitatory synaptic transmission.

23 sting that this SNARE complex is involved in excitatory synaptic transmission.

24 K signaling, AMPAR membrane trafficking, and excitatory synaptic transmission.

25 nc-sensitive signaling system that regulates excitatory synaptic transmission.

26 e of dendritic spines--postsynaptic sites of excitatory synaptic transmission.

27 o and from synapses controls the strength of excitatory synaptic transmission.

28 ssical mushroom-head morphology, and impairs excitatory synaptic transmission.

29 t neurons show normal morphologies and basal excitatory synaptic transmission.

30 enous neuromodulator adenosine and inhibited excitatory synaptic transmission.

31 , we report a role for opioids in modulating excitatory synaptic transmission.

32 tally with difficulty breathing and impaired excitatory synaptic transmission.

33 proteins or their binding partners regulates excitatory synaptic transmission.

34 ly, the light stimuli transiently facilitate excitatory synaptic transmission.

35 itical for the regulation of the efficacy of excitatory synaptic transmission.

36 creased to a level sufficient to depress the excitatory synaptic transmission.

37 tic increase in inhibitory and a decrease in excitatory synaptic transmission.

38 n activity in vivo occurs via suppression of excitatory synaptic transmission.

39 Rs and their auxiliary proteins control fast excitatory synaptic transmission.

40 ptors are responsible for fast inhibitory or excitatory synaptic transmission.

41 re ligand-gated cation channels that mediate excitatory synaptic transmission.

42 ns in dendritic spine density and diminished excitatory synaptic transmission.

43 PA receptor levels, and thus the strength of excitatory synaptic transmission.

44 injection of JWH133 into mice also increased excitatory synaptic transmission.

45 ted ion channels that play a crucial role in excitatory synaptic transmission.

46 uronal activity to fine-tune the strength of excitatory synaptic transmission.

47 d decreases both dendritic spine density and excitatory synaptic transmission.

48 s through FSI and not via a direct effect on excitatory synaptic transmission.

49 ribution of the glutamate-glutamine cycle to excitatory synaptic transmission.

50 in the brain where its activation depresses excitatory synaptic transmission.

51 ne's actions on depression-like behavior and excitatory synaptic transmission.

52 mediate a slow Ca(2+)-permeable component of excitatory synaptic transmission.

53 d by ambient glutamate to regulate levels of excitatory synaptic transmission.

54 y is usually correlated with the strength of excitatory synaptic transmission.

55 ate uptake in astrocytes and how this shapes excitatory synaptic transmission among neurons.

56 physiological concentrations enhances basal excitatory synaptic transmission and ameliorates deficit

57 utamate receptors (AMPARs) mediate most fast excitatory synaptic transmission and are crucial for man

58 soxazole-propionate (AMPA) receptors mediate excitatory synaptic transmission and are dynamically reg

59 AMPA receptors mediate fast excitatory synaptic transmission and are essential for s

60 receptors mediate the majority of vertebrate excitatory synaptic transmission and are therapeutic tar

61 Postsynaptic expression of parkin dampens excitatory synaptic transmission and causes a marked los

62 t this early life stressor leads to enhanced excitatory synaptic transmission and decreased levels of

63 rotein phosphatase-1 and actin and modulates excitatory synaptic transmission and dendritic spine mor

64 1)- and actin-binding protein that modulates excitatory synaptic transmission and dendritic spine mor

65 prevailing view, AMPA receptors mediate fast excitatory synaptic transmission and effect short-term p

66 exes of NR1 and NR2A-D subunits that mediate excitatory synaptic transmission and have a role in neur

67 ionotropic glutamate receptors that mediate excitatory synaptic transmission and have been implicate

68 tors (mGluRs) can induce acute depression of excitatory synaptic transmission and long-term depressio

69 Excitatory synaptic transmission and plasticity are crit

70 These results suggest that IgSF11 regulates excitatory synaptic transmission and plasticity through

71 euroadaptive changes in hippocampal area CA1 excitatory synaptic transmission and plasticity.

72 mate receptors (AMPARs) play a major role in excitatory synaptic transmission and plasticity.

73 l recordings show that Aph1b is required for excitatory synaptic transmission and plasticity.

74 rtate (NMDA) receptors (NMDARs) mediate fast excitatory synaptic transmission and play a critical rol

75 e DRN through both presynaptic inhibition of excitatory synaptic transmission and postsynaptic activa

76 NMDA receptors mediate excitatory synaptic transmission and regulate synaptic p

77 ynchronous burst onset mediated by recurrent excitatory synaptic transmission and similar intrinsic s

78 150 (AKAP79/150) signaling complex regulates excitatory synaptic transmission and strength through te

79 enhancement of paired-pulse facilitation in excitatory synaptic transmission and stronger paired-pul

80 , these mediators have been shown to enhance excitatory synaptic transmission and suppress inhibitory

81 the AMPA receptor plays an important role in excitatory synaptic transmission and synaptic plasticity

82 S-R1a activation in the hippocampus enhances excitatory synaptic transmission and synaptic plasticity

83 e receptors (AMPARs) mediate the majority of excitatory synaptic transmission and their function impa

84 eceptors (mGluR) are important modulators of excitatory synaptic transmission and therefore potential

85 pines belies the elaborate role they play in excitatory synaptic transmission and ultimately complex

86 pines belies the elaborate role they play in excitatory synaptic transmission and ultimately complex

87 ltifunctional aspect of PSD-95 in regulating excitatory synaptic transmission and unveil a novel form

88 bitory synaptic transmission, an increase in excitatory synaptic transmission, and concomitant increa

89 ignaling improves episodic memory, increases excitatory synaptic transmission, and enhances long-term

90 ignaling pathway proteins, enhanced cortical excitatory synaptic transmission, and restored dendritic

91 AMPA receptors are important for excitatory synaptic transmission, and their antagonists

92 tate (NMDA) receptors, the main mediators of excitatory synaptic transmission, are heterotetrameric r

93 rs (AMPARs), the principal mediators of fast excitatory synaptic transmission, are specifically excha

94 the long-term potentiation and depression of excitatory synaptic transmission, are widespread phenome

95 particular, in autism and implicate reduced excitatory synaptic transmission as a potential mechanis

96 r before or after disease induction restores excitatory synaptic transmission as well as presynaptic

97 AMPA receptors play a central role in basal excitatory synaptic transmission as well as synaptic mat

98 e central nervous system and are involved in excitatory synaptic transmission as well as synaptic pla

99 hly expressed in the CNS and are involved in excitatory synaptic transmission, as well as synaptic pl

100 sms and in particular through degradation of excitatory synaptic transmission associated with impaire

101 rebellar cortex, activation of CB1R inhibits excitatory synaptic transmission at parallel fiber (PF)-

102 their activation has a capacity to modulate excitatory synaptic transmission at primary afferent syn

103 indicate that TRPV1 activation can modulate excitatory synaptic transmission at the first sensory sy

104 well as subsequent in vivo E2 treatment, on excitatory synaptic transmission at the hippocampal CA3-

105 ecifically to the postsynaptic modulation of excitatory synaptic transmission at the larval neuromusc

106 ored the strength of pathologically weakened excitatory synaptic transmission at the stress-sensitive

107 The impaired inhibitory (as well as excitatory) synaptic transmission at frequencies associa

108 This defect was specific for excitatory synaptic transmission, because no change in i

109 of neurosteroid E2 is to acutely potentiate excitatory synaptic transmission, but the mechanism of t

110 The neurotransmitter glutamate mediates excitatory synaptic transmission by activating ionotropi

111 Here, we examine whether the modulation of excitatory synaptic transmission by DA in the NAc shell

112 postsynaptic membrane form the foundation of excitatory synaptic transmission by establishing the arc

113 Glutamate receptors mediate excitatory synaptic transmission by forming cation chann

114 The neurotransmitter glutamate mediates excitatory synaptic transmission by gating ionotropic gl

115 siological analysis demonstrates an enhanced excitatory synaptic transmission by increasing the relea

116 volatile anesthetics in particular, depress excitatory synaptic transmission by presynaptic mechanis

117 Excessive levels of Abeta disrupt excitatory synaptic transmission by promoting the remova

118 ts indicate that activation of Rac1 enhances excitatory synaptic transmission by recruiting AMPARs to

119 spinal cord slices with IFN-alpha suppressed excitatory synaptic transmission by reducing the frequen

120 o exist at presynaptic terminals and reshape excitatory synaptic transmission by regulating presynapt

121 The enhancement of excitatory synaptic transmission by TNFalpha is in fact

122 and suggest an important mechanism by which excitatory synaptic transmission can be dynamically modu

123 hese results suggest that the suppression of excitatory synaptic transmission can facilitate the appe

124 In the neocortex, fast excitatory synaptic transmission can typically be blocke

125 We conclude that excitatory synaptic transmission dependent on N-type and

126 In the mammalian brain, the specificity of excitatory synaptic transmission depends on rapid diffus

127 The strength of excitatory synaptic transmission depends partly on the n

128 1.5-fold increase in AMPA receptor-mediated excitatory synaptic transmission, dramatically altered t

129 act with neurons and blood vessels and shape excitatory synaptic transmission due to their abundant e

130 ned the effects of presynaptic inhibition on excitatory synaptic transmission during physiologically

131 altered cytokine network, facilitated basal excitatory synaptic transmission, enhanced intrinsic neu

132 sis that an imbalance between inhibitory and excitatory synaptic transmission exists during the criti

133 Kainate receptors mediate fast, excitatory synaptic transmission for a range of inner ne

134 In the striatum, balanced excitatory synaptic transmission from multiple sources o

135 y, demonstrating a functional segregation of excitatory synaptic transmission from neuronal morpholog

136 e we describe the impact of this mutation on excitatory synaptic transmission from parallel and climb

137 cultured mouse hippocampal neurons, recorded excitatory synaptic transmission from transfected cells,

138 Long-term potentiation (LTP) of excitatory synaptic transmission has long been considere

139 Alteration in the inhibitory and excitatory synaptic transmission (I/E) balance is a fund

140 caused by imbalances between inhibitory and excitatory synaptic transmission (I/E).

141 etwork activity by postsynaptically reducing excitatory synaptic transmission in acute and organotypi

142 xazolepropionic acid) receptors mediate fast excitatory synaptic transmission in brain and underlie a

143 Basal excitatory synaptic transmission in CA1 was depressed, l

144 , whereas both stargazin and gamma-4 rescued excitatory synaptic transmission in cerebellar granule c

145 A(-/-) mice, indicating that potentiation of excitatory synaptic transmission in DA neurons is not ne

146 of a mechanism that balances inhibitory and excitatory synaptic transmission in developing neural ci

147 uperficial DH neurons suppressed spontaneous excitatory synaptic transmission in diabetic rats in gre

148 europeptide release from sensory neurons and excitatory synaptic transmission in dorsal horn neurons,

149 docannabinoid signaling in the regulation of excitatory synaptic transmission in frontal neocortex, a

150 MPAR trafficking, silent synapse number, and excitatory synaptic transmission in hippocampal and cort

151 a galanin receptor 1-triggered depression of excitatory synaptic transmission in indirect pathway nuc

152 that alpha-BTX-sensitive nAChRs mediate fast excitatory synaptic transmission in Kenyon cells in the

153 onclude that attenuation of local horizontal excitatory synaptic transmission in layer V pyramidal ne

154 opic glutamate receptors, which mediate most excitatory synaptic transmission in mammalian brains.

155 naptically expressed long-term depression of excitatory synaptic transmission in medium spiny neurons

156 suggest that the drug-induced enhancement of excitatory synaptic transmission in midbrain DA neurons,

157 ds to investigate dopaminergic modulation of excitatory synaptic transmission in monkey prefrontal co

158 opic glutamate receptor that governs most of excitatory synaptic transmission in neurons.

159 principal glutamate receptors mediating fast excitatory synaptic transmission in neurons.

160 -type glutamate receptors (AMPARs) regulates excitatory synaptic transmission in neurons.

161 Excitatory synaptic transmission in PMDS neurons can be

162 on or potentiation of AMPA receptor-mediated excitatory synaptic transmission in prefrontal cortex py

163 at fear extinction decreases the efficacy of excitatory synaptic transmission in projections from the

164 PACAP enhanced, however, excitatory synaptic transmission in projections from the

165 rgely determines the strength of odor-evoked excitatory synaptic transmission in rat piriform cortica

166 neurons revealed no changes in inhibitory or excitatory synaptic transmission in response to PGE2 exp

167 modulation of TRPV1-mediated enhancement of excitatory synaptic transmission in response to PKC acti

168 ive forms of synaptic plasticity that reduce excitatory synaptic transmission in response to prolonge

169 ential, it is unknown whether toluene alters excitatory synaptic transmission in reward-sensitive dop

170 Furthermore, excitatory synaptic transmission in spinal cord slices a

171 frequency of action potentials, and enhanced excitatory synaptic transmission in spinal cord slices,

172 Furthermore, facilitated excitatory synaptic transmission in spinal dorsal horn n

173 gs revealed severely affected inhibitory and excitatory synaptic transmission in the amygdala, hippoc

174 and emotional behavior, has dual effects on excitatory synaptic transmission in the basolateral amyg

175 annels whose function is critical for normal excitatory synaptic transmission in the brain and whose

176 The majority of excitatory synaptic transmission in the brain occurs at

177 Glutamate receptors that mediate excitatory synaptic transmission in the brain show unusu

178 esis of dendritic spines, the major sites of excitatory synaptic transmission in the brain, is import

179 4 subunits that mediate the majority of fast excitatory synaptic transmission in the brain.

180 a key mechanism to determine the strength of excitatory synaptic transmission in the brain.

181 cid (AMPA) receptors mediate the majority of excitatory synaptic transmission in the brain.

182 utamate receptors (AMPARs) mediate most fast excitatory synaptic transmission in the brain.

183 Glutamate mediates most excitatory synaptic transmission in the brain.

184 nnels responsible for a majority of the fast excitatory synaptic transmission in the brain.

185 PARs) play a critical role in mediating fast excitatory synaptic transmission in the brain.

186 channels are membrane proteins that mediate excitatory synaptic transmission in the central nervous

187 -Methyl-D-aspartate (NMDA) receptors mediate excitatory synaptic transmission in the central nervous

188 mediate a slow, Ca2+-permeable component of excitatory synaptic transmission in the central nervous

189 Glutamate receptors mediate the majority of excitatory synaptic transmission in the central nervous

190 ic glutamate receptors (iGluRs) mediate fast excitatory synaptic transmission in the central nervous

191 Ion channels activated by glutamate mediate excitatory synaptic transmission in the central nervous

192 Ionotropic glutamate receptors mediate fast excitatory synaptic transmission in the central nervous

193 mate-gated cation channels that mediate fast excitatory synaptic transmission in the central nervous

194 Although, other forms of inhibitory and excitatory synaptic transmission in the circuit were unc

195 ) receptors (iGluRs) mediate the majority of excitatory synaptic transmission in the CNS and are esse

196 Fast excitatory synaptic transmission in the CNS is mediated

197 AMPA receptors mediate the majority of fast excitatory synaptic transmission in the CNS, and evidenc

198 Dendritic spines are the major sites of excitatory synaptic transmission in the CNS, and their s

199 mate receptors, which underlie a majority of excitatory synaptic transmission in the CNS, associate w

200 azolepropionic acid (AMPA) type mediate fast excitatory synaptic transmission in the CNS.

201 Glutamate receptors mediate the majority of excitatory synaptic transmission in the CNS.

202 AMPA receptors mediate the majority of rapid excitatory synaptic transmission in the CNS.

203 l membrane are the primary mediators of fast excitatory synaptic transmission in the CNS.

204 nucleoside transporters in the regulation of excitatory synaptic transmission in the dorsal horn, we

205 tion of microglia drives the facilitation of excitatory synaptic transmission in the dorsal horn, whi

206 more, Ih does not significantly modify basal excitatory synaptic transmission in the hippocampus, whe

207 r chaperone hsp90 (heat shock protein 90) in excitatory synaptic transmission in the hippocampus.

208 vestigate whether PDE5 and/or PDE6 regulates excitatory synaptic transmission in the hippocampus.

209 s (iGluRs) that mediate the majority of fast excitatory synaptic transmission in the mammalian brain.

210 re tetrameric ion channels that mediate fast excitatory synaptic transmission in the mammalian brain.

211 gated ion channel, mediates most of the fast excitatory synaptic transmission in the mammalian centra

212 receptors are the primary mediators of fast excitatory synaptic transmission in the mammalian CNS.

213 cocaine elicits a long-lasting depression of excitatory synaptic transmission in the NAc, a change th

214 pathway-specific presynaptic enhancement of excitatory synaptic transmission in the NAc.

215 Most fast excitatory synaptic transmission in the nervous system i

216 is a ligand-gated ion channel that mediates excitatory synaptic transmission in the nervous system.

217 Excitatory synaptic transmission in the nucleus accumben

218 We have examined how dopamine affects excitatory synaptic transmission in the PFC using whole-

219 -type glutamate receptors that underlie fast excitatory synaptic transmission in the SDH.

220 tioning blocked cocaine-evoked depression of excitatory synaptic transmission in the shell of the NAc

221 are involved in bi-directional regulation of excitatory synaptic transmission in the spinal cord SG r

222 R effects the expression of neuroligin 1 and excitatory synaptic transmission in the spinal cord, and

223 (2+) channels in primary sensory neurons and excitatory synaptic transmission in the spinal dorsal ho

224 o regional- and training-specific changes in excitatory synaptic transmission in the striatum.

225 have determined the actions of adenosine on excitatory synaptic transmission in the subiculum, the m

226 Dendritic spines are the primary sites of excitatory synaptic transmission in the vertebrate brain

227 ndritic spines, which are the major sites of excitatory synaptic transmission in the vertebrate brain

228 tamate receptors (iGluRs) that mediate rapid excitatory synaptic transmission in the vertebrate brain

229 s are ligand-gated ion channels that mediate excitatory synaptic transmission in the vertebrate brain

230 ant peptides in the LH to VTA projection, on excitatory synaptic transmission in the VTA and reward-s

231 ous study we showed that kisspeptin enhances excitatory synaptic transmission in these cells.

232 A, it is unknown whether leptin can modulate excitatory synaptic transmission in this brain region.

233 re we characterize the dynamic properties of excitatory synaptic transmission in two major intracorti

234 Here we report that nicotine potentiates excitatory synaptic transmission in ventral tegmental ar

235 We examined whether increased excitatory synaptic transmission in ventral tegmental ar

236 perience-dependent homeostatic plasticity of excitatory synaptic transmission in vivo.

237 hat disruption of LIS1 has direct effects on excitatory synaptic transmission independent of laminar

238 Excitatory synaptic transmission is accompanied by a loc

239 Excitatory synaptic transmission is altered during aging

240 ices of mouse somatosensory cortex show that excitatory synaptic transmission is markedly suppressed

241 Excitatory synaptic transmission is mediated by AMPA-typ

242 central nervous system, the majority of fast excitatory synaptic transmission is mediated by glutamat

243 Thus, excitatory synaptic transmission is subject to divergent

244 Excitatory synaptic transmission is typically studied un

245 gests the hypothesis that the suppression of excitatory synaptic transmission itself could facilitate

246 PGE2 (2 mum; 48 h) presynaptically increases excitatory synaptic transmission, leading to a hyperexci

247 Long-term depression (LTD) of excitatory synaptic transmission mediated by type 5 meta

248 d a decrease in the frequency of spontaneous excitatory synaptic transmission (mEPSCs) in neurons lac

249 In the brain, fast, excitatory synaptic transmission occurs primarily throug

250 michannels in resting states regulates basal excitatory synaptic transmission of hippocampal CA1 pyra

251 perience induces a persistent enhancement of excitatory synaptic transmission on NAc shell D1+ neuron

252 hol session produced enduring enhancement of excitatory synaptic transmission onto dopamine D1 recept

253 s effect is mediated by the strengthening of excitatory synaptic transmission onto dopamine neurons t

254 on of insulin into the VTA, which suppresses excitatory synaptic transmission onto dopamine neurons,

255 rd, we hypothesized that leptin can decrease excitatory synaptic transmission onto dopamine neurons.

256 that GluN2D-containing NMDARs participate in excitatory synaptic transmission onto hippocampal intern

257 In turn, Abeta selectively depresses excitatory synaptic transmission onto neurons that overe

258 Notably, fear conditioning potentiates excitatory synaptic transmission onto these long-range p

259 Notably, we found that excitatory synaptic transmission onto these neurons was

260 t persistent long-term potentiation (LTP) of excitatory synaptic transmission onto ventral tegmental

261 Thus, enhanced CRF-induced potentiation of excitatory synaptic transmission onto VTA dopamine neuro

262 motes drug-seeking behaviors and potentiates excitatory synaptic transmission onto VTA dopamine neuro

263 Finally, oxA/hcrt-1-mediated excitatory synaptic transmission onto VTA neurons was no

264 ial dorsal horn neurons either by increasing excitatory synaptic transmission or by decreasing inhibi

265 c Ca2+ fluxes affects spontaneous and evoked excitatory synaptic transmission or synaptic plasticity.

266 Postsynaptic kainate receptors mediate excitatory synaptic transmission over a broad range of t

267 important consequences for the regulation of excitatory synaptic transmission, plasticity, epileptoge

268 Long-term potentiation (LTP) of excitatory synaptic transmission plays a major role in m

269 la pyramidal neurons shows an attenuation of excitatory synaptic transmission, presumably because of

270 nf transcription impaired inhibitory but not excitatory synaptic transmission recorded from layer V p

271 ic glutamate receptors (iGluRs) that mediate excitatory synaptic transmission, regulate neurotransmit

272 ioral function; however, the degree to which excitatory synaptic transmission relies on the normal op

273 hermore, nerve injury persistently increased excitatory synaptic transmission (spontaneous excitatory

274 loss of synaptic AMPA receptors and reduced excitatory synaptic transmission that corresponds with i

275 ependent translation of proteins involved in excitatory synaptic transmission that in turn drives the

276 ogical studies revealed impairments in basal excitatory synaptic transmission that involved both AMPA

277 Fast excitatory synaptic transmission that is contingent upon

278 Among diverse factors regulating excitatory synaptic transmission, the abundance of posts

279 As a key element in excitatory synaptic transmission, the receptor regulates

280 GABA release from A17s, regulate the flow of excitatory synaptic transmission through the rod pathway

281 uRs in the dorsal BNST induces depression of excitatory synaptic transmission through two distinct me

282 ligand-gated ion channels that mediate fast excitatory synaptic transmission throughout the central

283 utamate-gated ion channels that mediate fast excitatory synaptic transmission throughout the nervous

284 However, actions of serotonin on the excitatory synaptic transmission to LHb neurons have not

285 nipulating neurogenesis in adult mice alters excitatory synaptic transmission to mature dentate neuro

286 d peripheral nerve injury-induced changes in excitatory synaptic transmission to neurones in lamina I

287 vestigated the effects of glucocorticoids on excitatory synaptic transmission to putative DR 5-HT neu

288 ABSTRACT: During excitatory synaptic transmission, various structurally u

289 ence suggests that anaesthetics also inhibit excitatory synaptic transmission via a presynaptic mecha

290 After blockade of ionotropic excitatory synaptic transmission, voltage-clamp recordin

291 Retinogeniculate excitatory synaptic transmission was also suppressed by

292 epileptiform activity appeared only when the excitatory synaptic transmission was depressed by furthe

293 Similar results were obtained when excitatory synaptic transmission was eliminated in a low

294 This study shows that excitatory synaptic transmission was enhanced in the bed

295 protein kinase II (CaMKII) in regulation of excitatory synaptic transmission was proposed two decade

296 ved on dendritic shafts, and the strength of excitatory synaptic transmission was unaffected, showing

297 f NL3 in WT or NL1 KO neurons did not affect excitatory synaptic transmission, whereas P0 knockdown o

298 s in the hippocampus for 7-10 days increases excitatory synaptic transmission, whereas short-term act

299 how that the protein is important for normal excitatory synaptic transmission, while its dysfunction

300 m presynaptic excitatory terminals regulated excitatory synaptic transmission within hippocampal CA3.