ライフサイエンスコーパス: excystation

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1 ntially preserved as mRNA in preparation for excystation.

2 Calmodulin antagonists also blocked excystation.

3 he resumption of motility and cytokinesis in excystation.

4 te a central role for calcium homeostasis in excystation.

5 nophores, and thapsigargin) strongly inhibit excystation.

6 tative growth and the cellular activation of excystation.

7 ar late in encystation, and during and after excystation.

8 es that release their contents just prior to excystation.

9 uring development, including encystation and excystation.

10 of the life cycle throughout encystation and excystation.

11 Cryptosporidium parvum excystation and host cell invasion have been characteriz

12 scribes our understanding of Cryptosporidium excystation and the events leading to host cell invasion

13 ot after, stage II was sufficient to inhibit excystation, and inhibition could be partially reversed

14 Encystation and excystation are crucial processes for the survival and t

15 ogy, as high-grade regulated encystation and excystation are readily achievable.

16 or to wheat germ agglutinin (WGA) inhibited excystation by > 90%.

17 host cell invasion and egress, encystation, excystation, catabolism of host proteins, differentiatio

18 a(2+) responses increased with each stage of excystation, consistent with the kinetics of inhibition.

19 Microscopy and excystation experiments were conducted to support the di

20 tion of macrophages in supporting sporozoite excystation following oocyst internalisation.

21 station induced by bile and alkaline pH, and excystation in response to gastric pH followed by alkali

22 We have been able to reliably induce excystation in vitro by mimicking cyst passage through t

23 uired for survival outside the host, whereas excystation initiates infection.

24 Excystation is blocked by specific cysteine protease inh

25 Although excystation is crucial to the initiation of infection by

26 Antigenic variation during encystation-excystation may be a novel form of immune evasion that c

27 ands, which bind cyst wall epitopes, inhibit excystation, most likely by interfering with proteolysis

28 ite- and host-specific molecules involved in excystation, motility and host cell invasion have been p

29 endent of cellular stress and, except during excystation, occurs throughout the G. lamblia life cycle

30 late (NaTC) are routinely used to induce the excystation of Cryptosporidium oocysts.

31 Excystation of Giardia lamblia, which initiates infectio

32 in morphological modifications and decreased excystation rates of sporozoites.

33 he sporocyst cavity of the oocysts and after excystation, secretion was detected in culture supernata

34 ts, but is downregulated during the stage of excystation that models cyst arrival in the small intest

35 Immuno-pathogenesis after excystation was similarly well advanced by the use of a

36 est that protein kinase A signaling triggers excystation, whereas calcium signaling is mainly require

37 ylated PKA inhibitor, amide 14-22, inhibited excystation with an IC(50) of 3 microm, suggesting an im

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