ライフサイエンスコーパス: executive

1 Each evaluation included motor, executive and behavioural scales and disease staging usi

2 le frontal gyrus) that was recruited by both executive and choice tasks.

3 nitive performance were observed on tests of executive and intellectual function; performance on thes

4 linergic circuits at center stage for normal executive and mnemonic functioning and provides compelli

5 e acetyltransferase (ChAT) in regions of the executive and motor loops of the basal ganglia of humans

6 rder mental functions (default mode, central executive and salience networks) and externally-driven,

7 and episodic and semantic memory, language, executive and visuospatial functions assessment.

8 Regression analysis showed the effect on executive attention is mostly independent of the effect

9 xtual information, perhaps because of better executive/attentional control over behavior, which requi

10 HO) Regional Director for Africa and the WHO Executive Board to ramp up routine immunization (RI) act

11 Another finding of note was that executive boards may not be permanently fixed as either

12 rthern and Yorkshire National Health Service Executive, British United Provident Association Foundati

13 anipulation did not involve a single central executive but rather involved two systems with distinct

14 lience (SN), default mode (DMN), and central executive (CEN) networks-three brain systems that play a

15 es behavioral avoidance responses related to executive choice, consistent with its expression in cent

16 Drawing on executive coaching literature, a 3-part framework was de

17 Executive coaching research suggests that effective coac

18 t not hypotension, correlated with decreased executive cognitive function at 12 months (P=0.03).

19 of neural regions and circuits important in executive cognitive function can bias behavioral choices

20 nd risky rewards, we tested whether training executive cognitive function could influence choice beha

21 rediction of reward outcomes, contributes to executive cognitive function deficits in alcoholic indiv

22 a), indicating that selection for changes in executive cognitive functions characterized both great a

23 Processing speed and executive cognitive functions were further evaluated wit

24 Executive cognitive functions, including working memory,

25 Standards were reviewed and approved by the Executive Committee of the ADA Board of Directors, which

26 ds of Care were reviewed and approved by the Executive Committee of the ADA Board of Directors, which

27 unication by physician members of the SCORE2 Executive Committee to sites upon each randomization wer

28 her the effects of CR depend on the semantic/executive components of the task remains unknown.

29 nce Third Edition (WPPSI-III) and the global executive composite score from the Behaviour Rating Inve

30 The mean BRIEF-P global executive composite score was 90.6 (26.2) in the iodine

31 r adults underwent an fMRI session during an executive contextual task in which task difficulty varie

32 ty (rsFC) with regions important in top-down executive control (dorsolateral prefrontal cortex [dlPFC

33 ized as two independent memory systems under executive control [1, 2].

34 be one among many drivers of adjustments in executive control and that the ACC might be just one com

35 by showing an increased connectivity in the executive control and the default mode networks in the b

36 hension, while laboratory switches recruited executive control areas, fully natural switching within

37 h a region known to be important in top-down executive control at rest (left dlPFC), which, in turn,

38 Strikingly, salience-executive control between-network cohesion peaked at mod

39 with venlafaxine selectively normalizes the executive control function of attention in addition to i

40 tal circuitry involved in limbic arousal and executive control in 36 individuals-18 cocaine-dependent

41 We focused on executive control in a stop signal task, which is known

42 Executive control involves the ability to flexibly inhib

43 de network (DMN), salience network (SN), and executive control network (ECN) by using seed-based anal

44 in the dorsal default mode network (DMN) and executive control network (ECN).

45 sal, while cohesion of this network with the executive control network peaks at moderate arousal.

46 ence network is optimally able to engage the executive control network to coordinate cognitive activi

47 sal attention network, salience network, and executive control network).

48 ludes reduced thalamic connectivity with the executive control network, and is related to cognitive i

49 , and cerebellar areas of the well-described executive control network.

50 nitive functions, including the salience and executive control networks.

51 ted network, and across the default mode and executive control networks.

52 ral circuit remodeling process important for executive control of reward and addiction.

53 Executive control of voluntary movements is a hallmark o

54 holinergic activity in STs degrades top-down executive control over behavior, producing a bias for bo

55 t obese adolescents have impaired prefrontal executive control responses to drinking glucose and fruc

56 , in the domain of social emotional actions, executive control shifts from subcortical to prefrontal

57 en engaged, cocaine users can mobilize their executive control system similar to controls, but that t

58 Attention control (or executive control) is a higher cognitive function involv

59 d with default behaviors (and the absence of executive control) than with emotion.

60 oice is commonly regarded as part of general executive control, engaging prefrontal and anterior cing

61 on give rise to frontal overload and disrupt executive control, fuelling and perpetuating post-trauma

62 t into orbital and striatal contributions to executive control.

63 ated in working memory, decision making, and executive control.

64 a Go/No-Go task, which engages PFC-mediated executive control.

65 tentional networks: alertness, orienting and executive control.

66 Warburg effect linked to cognitive-executive deficits in FMR1 premutation.

67 working memory is dependent on the degree of executive demand.

68 ) or higher (digit span backwards, Trails B) executive demands, and lower (figure copying) or higher

69 cognitive dysfunction experience subjective executive difficulties during menopause.

70 althy menopausal women with midlife onset of executive difficulties include modulation of insular and

71 , with main involvement of visuo-spatial and executive domains.

72 However, determining the specificity of executive dysfunction across these disorders is challeng

73 ntration of methotrexate was associated with executive dysfunction as well as with a thicker cortex a

74 lopment of tools for potentially normalizing executive dysfunction in patient populations.

75 Many women with no history of executive dysfunction report difficulties in this domain

76 sk by reducing cognitive reserve, increasing executive dysfunction, and disrupting normative emotion

77 ith cognitive decline, including significant executive dysfunction, for which the neurobiological und

78 combinations of socioaffective symptoms and executive dysfunction.

79 rary chemotherapy-only protocols demonstrate executive dysfunction.

80 lated with increased severity of sensory and executive dysfunctions (i.e. hypervigilance and impulse

81 ntly shown to correlate with the severity of executive dysfunctions across a wide range of diseases,

82 lts of a multiyear program to predict direct executive elections in a variety of countries from globa

83 tion adherence (beta=0.51; P=0.008), whereas executive function (beta=0.24; P=0.075) and attention we

84 telligence, anxiety/depressive symptoms, and executive function (differences of 0.6-0.7 SD; P=1.2x10(

85 The relationship of executive function (EF) and theory of mind (ToM) deficit

86 plain information processing speed (IPS) and executive function (EF) in multiple sclerosis (MS).

87 on tests of dysexecutive behaviour (DB) and executive function (EF) in patients with behavioural var

88 s was associated with accelerated decline in executive function (for all relationships combined, beta

89 g Speed (HR = 0.58 [0.36-0.95], P = .03) and Executive Function (HR = 0.52 [0.28-0.97], P = .040).

90 skills, and sCD164 remained associated with executive function (P < .05 for all comparisons).

91 d with worse global cognition (P < 0.02) and executive function (P < 0.01) at 3 and 12 months.

92 year in patients without; p = 0.006), and in executive function (p = 0.01).

93 n on information processing speed (p=0.002), executive function (p<0.0001), motor function (p<0.0001)

94 the spatial working memory strategy index of executive function (primary end point) was -0.21+/-2.62

95 ia was associated with increased risk of low executive function (RD, 0.05; 95% CI, 0.01 to 0.10 and R

96 the spatial working memory strategy index of executive function (scores range from 4 to 28, with lowe

97 ions with treatment: baseline assessments of executive function (set shifting measured by the Trail M

98 ested that those with SDB had slightly worse executive function (standard mean difference, -0.05; 95%

99 ard Test 4.2, -1.3 to 9.7), and attention or executive function (Stroop Interference Test -2.6, -7.4

100 stimulation on a working memory (n-back) and executive function (Stroop) task in 28 individuals with

101 ion, including assessments of the domains of Executive Function (Trail Making Test, Stroop, Digit Spa

102 sual Retention Test; score range, 0 to -26), executive function (Trail-Making Test B minus A; range,

103 etylcholine in the neocortex is critical for executive function [1-3].

104 of their child (Behavior Rating Inventory of Executive Function [BRIEF]).

105 ganglia was inversely correlated with their executive function ability, suggesting that bilinguals c

106 able creche attendance are likely to improve executive function and emotional-behavioural development

107 l Making Test (TMT) is a widely used test of executive function and has been thought to be strongly a

108 Executive function and memory scores over 8 years (2002-

109 t-TBI cognitive impairment primarily affects executive function and processing speed, which were not

110 y include disturbances in memory, attention, executive function and processing speed.

111 adulthood were associated with worse midlife executive function and processing speed.

112 were temporally coincident with deficits in executive function and reduced rates of fear extinction

113 rum disorder completed a battery of tests of executive function and underwent diffusion-weighted imag

114 with a dose-dependent increased risk of poor executive function and visual motor function, even if no

115 ot associated with worse global cognition or executive function at 3 or 12 months in models incorpora

116 These receptors are important for prefrontal executive function because pharmacological and genetic m

117 the spatial working memory strategy index of executive function between the patients who received evo

118 possible to immediately change components of executive function by directly manipulating neural activ

119 s for AC- participants; P = .04) and a lower executive function composite score (raw mean scores: 0.5

120 reas the cortical factor was associated with executive function decline in Abeta+ MCI participants an

121 drome is likely a surrogate marker for other executive function defects, we suggest that microglia-di

122 diet (HFD)-fed offspring have cognitive and executive function deficits as well as whole-genome DNA

123 xpression-to positive psychotic symptoms and executive function deficits in bipolar disorder.

124 (e.g., during adolescence) could ameliorate executive function deficits observed in offspring that w

125 tellectual, attention, memory, language, and executive function development.

126 ive domains: attention, response inhibition, executive function during visuospatial navigation, cogni

127 amic anatomical connectivity and severity of executive function impairment was examined in patients.

128 Scholastic performance and aspects of executive function improved when both interventions were

129 at has improved cognitive function including executive function in depressed patients in randomised p

130 t orexin transmission is closely involved in executive function in normal and pathological conditions

131 irect current stimulation (tDCS) can improve executive function in older adults.

132 eficits and stress surfeits, and compromised executive function in three stages.

133 ic metabolites associated with impairment in executive function in two cohorts of patients receiving

134 structural network maturation that supports executive function in youth.

135 We examined whether the cortico-cerebellar executive function network is altered in children with A

136 MT genetic effects on the cortico-cerebellar executive function network.

137 video games that do not specifically target executive function or adapt the level of difficulty thro

138 The n-6:n-3 ratio and n-3 predicted executive function performance differently in children a

139 o) was previously shown to be a predictor of executive function performance in children aged 7-9 y.

140 The executive function performance of 78 children aged 7-12

141 y exploring the role of the n-6:n-3 ratio in executive function performance.

142 at 18 (P = 0.233) or 24 (P = 0.146) mo or in executive function score at 24 mo (P = 0.467).Prenatal L

143 Median executive function scores were also similar at 3 months

144 ments of motor, language, socioemotional, or executive function skills at 18 mo of age.

145 EF was measured with the Delis-Kaplan Executive Function System (DKEFS: performance based), an

146 Improved performance on the executive function task was associated with reduced acti

147 function tasks; right caudate mapped to both executive function tasks and music-related processes.

148 Left inferior gyrus mapped to executive function tasks; right caudate mapped to both e

149 Declines in Executive Function tended to be predictive of worse surv

150 g impact on serotonergic circuits underlying executive function that are unmasked by loss of estradio

151 Impaired executive function was defined as a score >/=2 SDs below

152 e) on these behavioral and neural markers of executive function was examined using repeated measures

153 between 4-hydroxyphenylacetate and impaired executive function was replicated in the second cohort (

154 Factors positively associated with executive function were home stimulation (aOR 1.36 [95%

155 Results of the Behavior Rating Inventory of Executive Function were not significantly different.

156 up, n=159; placebo group, n=156) for overall executive function with BRIEF-P.

157 We assessed executive function with the Trail Making Test Part B and

158 Assessment of Neuropsychological Status and executive function with the Trail Making Test, Part B.

159 lity, declarative memory, procedural memory, executive function, academic achievement, fine motor dex

160 ce on neuropsychological measures of memory, executive function, and attention and hyperactivity, wer

161 ssments of episodic memory, semantic memory, executive function, and global cognitive function with l

162 ey domains of cognitive functioning (memory, executive function, and processing speed), cognition was

163 d with worse verbal learning, verbal memory, executive function, and psychomotor speed (P < .05 for a

164 ssociated with slower cognitive decline (for executive function, betaperson-meanxtime-in-study = 0.00

165 itive domains: information processing speed, executive function, episodic memory, working memory, and

166 ing memory capacity, a critical component of executive function, expands developmentally from childho

167 Three domains-executive function, incentive salience, and negative emo

168 ard Test) and compound scores (eg, G factor, executive function, information processing speed, memory

169 sed cognitive measures of attention, memory, executive function, IQ, and processing speed.

170 ed with impairments in verbal IQ, attention, executive function, language and visuospatial memory on

171 ing speed, visuospatial function, attention, executive function, language, or behavior.

172 gnitive function was assessed with tests for executive function, language, verbal reasoning and conce

173 es associated with motor function, language, executive function, memory, verbal learning, perceptual

174 egression analyses tested whether attention, executive function, or memory predicted 21-day medicatio

175 standard deviation, 15), though measures of executive function, processing speed, and memory were le

176 e functions, including general intelligence, executive function, processing speed, memory, perceptual

177 ce and worse verbal learning, verbal memory, executive function, psychomotor speed, and fine motor sk

178 onally associated with regulating memory and executive function, respectively.

179 Neuropsychological measures of executive function, speed and information processing, at

180 ngly recognized to play a role in regulating executive function, the dysfunction of which is a factor

181 ative impact of prefrontal KYNA elevation on executive function, the mechanism underlying such a disr

182 s segregated with increased incidence of low executive function, tremors, below-average IQ, and FXTAS

183 003) in reduced-intensity HCT recipients for executive function, verbal fluency, and working memory.

184 P < .001) post-HCT scores than controls for executive function, verbal speed, processing speed, audi

185 Cognitive function, executive function, visual function, and motor function

186 o scales of the Behavior Rating Inventory of Executive Function-Preschool (BRIEF-P).

187 score from the Behaviour Rating Inventory of Executive Function-Preschool Version (BRIEF-P) in the ch

188 nctional MRI, and functional connectivity of executive function-related Crus I/II in the cerebellum w

189 al cortex (PFC), a brain region critical for executive function-we examined whether early life methyl

190 and a modified Stroop interference test for executive function.

191 ce-dependent rewiring of circuits underlying executive function.

192 nt, potentially allowing for improvements in executive function.

193 or frontal gyrus (IFG) a critical region for executive function.

194 ognitive impairment and a small worsening in executive function.

195 tellectual, language, attention, memory, and executive function.

196 brain regions, regions often associated with executive function.

197 change in verbal fluency, a simple index of executive function.

198 onto-cingulate dysfunction was driven by hot executive function.

199 hism has been reported to be associated with executive function.

200 loss of which is central in apathy-as a core executive function.

201 schizophrenia and 2) related to deficits in executive function.

202 : -0.064, -0.006)-was associated with higher executive function.

203 assessments of global cognition, memory, and executive function.

204 ing prominent deficits in working memory and executive function.

205 cognitive training on six adaptive tests of executive function; (2) Cognitive and physical exercise

206 of memory, information processing speed, and executive function; and adjudicated incident dementia ca

207 om control 1.2, 95% CI -0.3 to 2.7), 0.3 for executive functioning (0.18, -0.07 to 0.29), 0.5 for pre

208 attention, episodic and working memory, and executive functioning after TBI.

209 CR may relate to executive functioning and semantic knowledge, leading to

210 s by which LDX acts to improve self-reported executive functioning in healthy menopausal women with m

211 However, the mechanism by which LDX improves executive functioning in these women is not known.

212 decline accelerated slightly with age, with executive functioning showing the largest additional rat

213 and memory, processing speed, language, and executive functioning test scores using linear mixed mod

214 1A-G carriers) also had poorer self-reported executive functioning than SN, but both groups reported

215 e performance and mediate the improvement of executive functioning with age.

216 ts (Digit-Span, Spatial-Span), self-reported executive functioning, and functional magnetic resonance

217 viors, positive alcohol expectancies), worse executive functioning, and thinner cortices and less bra

218 Intelligence, attention, memory, executive functioning, and visuospatial processing were

219 e interplay of brain regions associated with executive functioning, incentive salience, and interocep

220 opsychological tests of the frontal lobe and executive functioning, the Trail Making Test, and the Ve

221 l role of the thalamus as a relay station in executive functioning.

222 Executive functions (EFs) are used to set goals, plan fo

223 SIGNIFICANCE STATEMENT: Executive functions are supported by the dynamic coordin

224 Here, we tested the hypothesis that human executive functions arise from the dynamic interplay of

225 and implicit learning, language skills, and executive functions as well as atypical patterns of cort

226 The results suggest executive functions can be rapidly up- or down-regulated

227 e-developing prefrontal cortex that supports executive functions competes with procedural learning me

228 ce in conventional paper and pencil tests of executive functions for patients with PD.

229 tal cortical dopamine regulates a variety of executive functions governed by the frontal lobes via ac

230 Innovative computer-training regimes for executive functions have made tremendous inroads, yet th

231 oth MCI and AD, but was only associated with executive functions in patients with MCI.

232 Rescuing executive functions in people with neurological and neur

233 Executive functions including behavioral response inhibi

234 d to both the complexity of the construct of executive functions itself and/or the methodological dif

235 Impaired verbal memory, attention, and some executive functions may persist after prolonged abstinen

236 at the claustrum may preferentially subserve executive functions orchestrated by the cingulate cortex

237 between dynamic network reconfiguration and executive functions over short timescales and provide a

238 The assessment of executive functions poses researchers with several chall

239 Executive functions rely on coordinated information proc

240 The medial prefrontal cortex (mPFC) serves executive functions that are impaired in neuropsychiatri

241 ver, learning was negatively correlated with executive functions that rely on the DLPFC in the contro

242 frontal cortex and temporal pole, while cool executive functions were associated with temporal abnorm

243 , language, visuospatial, memory and frontal executive functions while presence of CSS was associated

244 the flexible modulation of recent memories (executive functions) as well as for the stable organizat

245 or) and laboratory settings (tests measuring executive functions), showing increased concentration of

246 nscious experience from related enabling and executive functions, (2) suggest how critically reconsid

247 at are independent of sensory processing and executive functions, and (4) show how animal studies can

248 cal defects in social interaction, cognitive/executive functions, and repetitive behaviors reflective

249 nce that visuomotor behaviors, a hallmark of executive functions, are mediated by the interplay of mu

250 hed role played by COMT genetic variation in executive functions, its impact on remote memory formati

251 ization of the posterior caudate nucleus for executive functions, often considered the exclusive doma

252 Different executive functions, sequential processing, and concept

253 across multiple cognitive domains, spanning executive functions, working memory, and planning and pr

254 challenge for the cognitive neuroscience of executive functions.

255 sensory information, reward, cognition, and executive functions.

256 , and strongly related to working memory and executive functions.

257 upport of the PIT 360 degrees for evaluating executive functions.

258 e may restore prefrontal networks related to executive functions.

259 erior temporal lobe and supramarginal gyrus; executive functions: bilateral frontoparietal regions; v

260 Finally, reduced connectivity between executive, homeostatic, and hedonic brain regions was ob

261 ed significant extrapyramidal signs, greater executive impairment, and severe striatal and frontal GM

262 -beta1-42 values, and proportionally greater executive impairment; and (iv) individuals with notably

263 Executive impairments occurred equally in both groups.

264 hat MICALL2 is a new susceptibility gene for executive inhibition deficiency related to hyperactive-i

265 a two-stage genome-wide association study of executive inhibition in ADHD in Han Chinese.

266 Impaired executive inhibition is a core deficit of attention defi

267 oop color-word interference test to evaluate executive inhibition.

268 orienting (mean difference [MD] = 14.63) and executive (MD = 21.00) attention networks.

269 tionally with M1 and with regions within the executive motor system.

270 hat multiple subsystems comprise the central executive needed to manipulate stored phonological input

271 rk (DMN) and between the DMN and the central executive network (CEN) in 111 individuals, aged 11-60 y

272 across multiple disorders, including central executive network (CEN), default mode network (DMN), and

273 e anterior cingulate cortex, and the central executive network relative to the MDD, PTSD, and HC grou

274 several task-positive networks (eg, central executive network, CEN), which are still developing duri

275 rch UK, North Thames National Health Service Executive, Northern and Yorkshire National Health Servic

276 ate empirical evidence on the experiences of executive nurses working at board level in England and W

277 ) had ALS with FTD, 103 (28.9%) had ALS with executive or nonexecutive impairment, and 19 (5.3%) had

278 January 27, 2017, President Trump signed an executive order banning the citizens of 7 countries from

279 ry safer, while others have argued that this executive order will result in the weakening of our coun

280 , I will stay away from the politics of this executive order; rather, I want to discuss the impact of

281 work efficiency are associated with enhanced executive performance and mediate the improvement of exe

282 moderate for the default mode, left central executive, primary and secondary visual, sensorimotor, c

283 ience network, associated with attention and executive processes involved in encoding and retrieval.

284 parieto-occipital regions commensurate with executive processing demands.

285 erlying cellular mechanisms of catatonia, an executive "psychomotor" syndrome that is observed across

286 crease in glutamate following exercise in an executive region, the anterior cingulate cortex.

287 ease of activity in attentional, sensory and executive regions, with its peak in the brain stem retic

288 reased activation and decreased glutamate in executive regions.

289 uts from the sensory modalities may underlie executive regulation of the communication between the cl

290 pression of a network of YAP1 effectors with executive roles in invasion, chemotaxis and adhesion dow

291 cluding components of the attention, central executive, salience, and default mode networks.

292 h (Logical Memory Test (LMT) delayed recall) executive/semantic components.

293 An Executive Summary of a National Heart, Lung, and Blood I

294 This brief report is an executive summary of the officially approved guidelines

295 and dissociable deficits within the brain's executive system are present in association with dimensi

296 uggest that listeners recruit domain general executive systems to maintain successful communication w

297 ion is apparent during the performance of an executive task.

298 participants who performed hard versions of executive tasks but not in control groups who performed

299 Precise information flow during mnemonic and executive tasks requires the coactivation of adult prefr

300 hat manipulation is carried out by a central executive while storage is performed by two interacting