ライフサイエンスコーパス: exercise training

1 ypically observed in sedentary subjects with exercise training.

2 genesis, exercise tolerance, and response to exercise training.

3 muscle or whole-body aerobic adaptations to exercise training.

4 imals also performed low intensity treadmill exercise training.

5 quired for metabolic adaptation to endurance exercise training.

6 Cox models and explored an interaction with exercise training.

7 or the prediction of the VO2 max response to exercise training.

8 vements in insulin sensitivity after aerobic exercise training.

9 and no major adverse events were related to exercise training.

10 metabolism, and depot-specific responses to exercise training.

11 tolerate (and possibly even be improved by) exercise training.

12 ovements in glucose metabolism after aerobic exercise training.

13 raction </=35% to usual care with or without exercise training.

14 olic twist in an acute augmentation phase of exercise training.

15 LVCD may be possible with moderate lifelong exercise training.

16 g spatial neglect treatment with traditional exercise training.

17 of cardiac hypertrophy because of endurance exercise training.

18 enriched at endothelial cells, is induced by exercise training.

19 tially pathological heart rate adaptation to exercise training.

20 adaptation of subcutaneous adipose tissue to exercise training.

21 onse of individual proteins in humans during exercise training.

22 rowth under resting conditions and following exercise training.

23 unable to improve exercise capacity through exercise training.

24 ecruits before and after a period of intense exercise training.

25 tched control women but can be improved with exercise training.

26 ear whether statin use alters adaptations to exercise training.

27 scular dysfunction are reversed by late-life exercise training.

28 mitochondrial content in response to aerobic exercise training.

29 omen, and again in women with PCOS following exercise training.

30 1 +/- 6 kg m(-2)) then completed 16 weeks of exercise training.

31 ronal precursor cell proliferation following exercise training.

32 to 9, and also not in response to submaximal exercise training.

33 d related signalling pathways in response to exercise training.

34 r treating multiple defects at once, as with exercise training.

35 ny of the adaptations that accompany chronic exercise training.

36 ameliorated by correcting anemia and through exercise training.

37 s a key factor in the beneficial outcomes of exercise training.

38 muscle or whole-body aerobic adaptations to exercise training.

39 ecutive function; (2) Cognitive and physical exercise training; (3) Cognitive training combined with

40 non-invasive brain stimulation and physical exercise training; (4) Active control training in adapti

41 Chronic exercise training (6 weeks) increased soleus muscle PGC-

42 We show that muscles of WT mice subjected to exercise training activate the CaMKII signaling pathway

43 y sought to determine if simvastatin impairs exercise training adaptations.

44 alth benefits and potential harms of routine exercise training after solid organ transplantation are

45 mellitus, thyroid dysfunction, and endurance exercise training all cause structural remodeling.

46 s increased by 10% (p < 0.05) in response to exercise training alone, but was blunted by the addition

47 Exercise training also increased the insulin activation

48 Exercise training also increases expression of the brown

49 Chronic exercise training also promoted NRG1 cleavage, resulting

50 te autophagy; however, it is unknown whether exercise training alters basal levels of autophagy and w

51 Regular exercise training ameliorates age-related diastolic dysf

52 ural and functional adaptations to endurance exercise training among competitive male rowers (n=12; a

53 s controversial with the association between exercise training and collateral growth still unclear.

54 Before and after exercise training and detraining, 12 previously sedentar

55 Exercise training and energy restriction are first-line

56 ortance of lifestyle modification, including exercise training and energy restriction, in the regulat

57 Exercise training and home or housework were not associa

58 th SRVs in this study safely participated in exercise training and improved peak VO2.

59 vely evaluated the effects of a home aerobic exercise training and maintenance program (EX) on aerobi

60 e induces physiological responses that mimic exercise training and may underlie the beneficial effect

61 Moderate intensity exercise training and modest energy restriction provided

62 We also show that both exercise training and muscle-specific transgenic express

63 l arrhythmogenesis associated with endurance exercise training and occlusive coronary artery disease.

64 e benefits of weight loss, physical activity/exercise training, and increases in cardiorespiratory fi

65 computer-based cognitive training, physical exercise training, and non-invasive brain stimulation, a

66 pulations, screened for lifelong patterns of exercise training, and stratified into 4 groups: "sedent

67 s age-related microvascular dysfunction, and exercise training appears to be particularly effective i

68 es the current body of literature related to exercise training as a means of optimizing functional ca

69 We hypothesised that exercise training attenuates the changes in cutaneous va

70 Exercise training benefits many organ systems and offers

71 In additional experiments, voluntary exercise training by wheel running for only 11 days resu

72 rodent exercise studies have indicated that exercise training can alter circulating adipokine concen

73 aneous microvascular NO function and whether exercise training can ameliorate any impairment.

74 These findings suggest that structured exercise training can ameliorate striatal D2/D3 receptor

75 sfunction; these data suggest that late-life exercise training can be implemented to improve coronary

76 Endurance exercise training can increase the ability to perform pr

77 to exercise intolerance, and conversely how exercise training can potentially modulate aging phenoty

78 may help to pinpoint the mechanisms by which exercise training can reduce the risk of brain diseases,

79 Exercise training causes adaptations to scWAT that elici

80 We examined whether exercise training causes hypothalamic neurogenesis and w

81 he change in body composition in response to exercise training combined with calorie restriction in o

82 Aerobic exercise training combined with fibre-enriched diet can

83 All subjects performed resistance exercise training combined with high-intensity interval

84 onounced within the patients with supervised exercise training compared with the patients on the home

85 ) and if endurance- and/or strength-oriented exercise training could rescue decrements in insulin act

86 Exercise training demonstrated benefits in muscular stre

87 acity was intact and increased normally with exercise training, demonstrating that mitochondria are n

88 est that performing daily moderate intensity exercise training during continuous hypoxic exposure doe

89 Furthermore, multi-component exercise training effectively reduces symptoms of anxiet

90 Exercise training enhances extracellular superoxide dism

91 Whether exercise training enhances or injures the SRV is unclear

92 Exercise training enhances physical performance and conf

93 We previously showed that long-term moderate exercise training (ET) improves functional capacity and

94 r improved exercise capacity after endurance exercise training (ET) in elderly patients with heart fa

95 Protective effects of exercise training (ET) on endothelial function have been

96 sought to evaluate the effects of endurance exercise training (ET) on endothelial-dependent flow-med

97 e value of high levels of physical activity, exercise training (ET), and overall cardiorespiratory fi

98 adaptations to 12 weeks of HFHS diet and/or exercise training (Ex) in rats.

99 om calorie restriction (CR) and/or endurance exercise training (EX) is cardioprotective.

100 lity and induces insulin resistance, whereas exercise training exerts positive effects on substrate h

101 Daily exercise training for 7 days increased hypothalamic cell

102 l events, but not a differential response to exercise training for clinical outcomes or changes in ex

103 osocial factors may influence adherence with exercise training for heart failure (HF) patients.

104 to 2.21) mL/kg/min among participants in the exercise training group and +0.08 (95% CI, -0.62 to 0.79

105 cardiovascular death or HF hospitalization, exercise training had a greater impact on patients with

106 Exercise training had little impact on LV stiffness (tra

107 ata analysis, patients with HFPEF undergoing exercise training had significantly improved CRF (mL/kg

108 Resistance exercise training had smaller but qualitatively similar

109 Exercise training has been proven to be effective on rec

110 Exercise training has been reported to ameliorate heart

111 Exercise training has been shown to be effective in impr

112 Exercise training has been shown to improve cardiorespir

113 Exercise training has long been known to promote mitocho

114 Regular physical activity and exercise training have long been known to cause adaptati

115 Both exposure to hypoxia and exercise training have the potential to modulate appetit

116 A Controlled Trial Investigating Outcomes of Exercise Training (HF-ACTION) study-a multicenter, rando

117 A Controlled Trial Investigating Outcomes of Exercise Training (HF-ACTION), 2279 (97.8%) completed su

118 A Controlled Trial Investigating Outcomes of Exercise Training [HF-ACTION]; NCT00047437).

119 Habitual physical activity and regular exercise training improve cardiovascular health and long

120 and arteriovenous oxygen difference, whereas exercise training improved cardiac output, citrate synth

121 Exercise training improved cardiorespiratory fitness by

122 While 4 weeks of exercise training improved whole-body insulin sensitivit

123 n abundance of literature demonstrating that exercise training improves aerobic capacity and muscular

124 suggesting a cross-organ mechanism by which exercise training improves cardiac function in diabetes

125 To determine whether moderate-intensity exercise training improves exercise capacity in adults w

126 Regular exercise training improves insulin action, and is a prim

127 Given that exercise training improves outcomes in HF patients, asse

128 Exercise training improves whole-body glucose homeostasi

129 To examine the effect of exercise training in a weight-loss program on asthma con

130 rlying atrial fibrillation (AF) promotion by exercise training in an animal model.

131 affect the response to 3 weeks of endurance exercise training in CACs, muscle mitochondrial capacity

132 ll maintained during 3-month aerobic dynamic exercise training in children and young adults with repa

133 ng intensity and strategy for individualized exercise training in chronic obstructive pulmonary disea

134 This study examined whether outcomes with exercise training in HF vary according to AF status.

135 ON) study-a multicenter, randomized study of exercise training in HF.

136 the current study, we aimed to determine if exercise training in humans diminished P-CoA attenuation

137 However, the effects of exercise training in liver transplanted FAP patients hav

138 Here, we determined the metabolic impact of exercise training in obese mice with cardiac and skeleta

139 cle mitochondrial content when combined with exercise training in overweight or obese patients at ris

140 ACTION was a randomized, controlled trial of exercise training in patients with chronic heart failure

141 CE-I, reduced the peak work rate response to exercise training in patients with chronic obstructive p

142 Exercise training in patients with HFPEF is associated w

143 inical trials that evaluated the efficacy of exercise training in patients with HFPEF were included i

144 95% confidence interval, -0.01 to 0.16) with exercise training in patients with HFPEF.

145 e aim to evaluate the efficacy and safety of exercise training in patients with pulmonary hypertensio

146 es that evaluated the efficacy and safety of exercise training in patients with pulmonary hypertensio

147 Exercise training in patients with pulmonary hypertensio

148 There are several approaches to exercise training in the heart failure population, each

149 aimed to assess the haemodynamic effects of exercise training in transposition of the great arteries

150 skeletal muscle to prolonged resistance-type exercise training in younger and older populations.

151 In contrast, exercise training increased mitochondrial ADP sensitivit

152 stable HFPEF, caloric restriction or aerobic exercise training increased peak VO2, and the effects ma

153 Exercise training increased peroxisome proliferator-acti

154 Six weeks of endurance exercise training increased the transcriptional level of

155 Exercise training increased Vo2max (2.2 +/- 0.2 vs. 2.5

156 Six weeks of exercise training increased whole-body glucose homeostas

157 lve weeks of endurance- or strength-oriented exercise training increased whole-body insulin action an

158 In rodent models, exercise training increases mitochondrial biogenesis and

159 We show that endurance exercise training increases multiple CAC types, an adapt

160 Consistent with the increase in UCP1, exercise training increases the presence of brown-like a

161 In conclusion, endurance exercise training induced increases in multiple CAC type

162 We propose that NP could contribute to exercise training-induced improvement in skeletal muscle

163 These findings reveal that endurance exercise training-induced increases in basal autophagy,

164 O2 supply, whereas, in trained subjects, an exercise training-induced mitochondrial reserve results

165 e corresponding ionic current, If, underlies exercise training-induced sinus bradycardia in rodents.

166 ed basal autophagy is required for endurance exercise training-induced skeletal muscle adaptation and

167 the effects of non-specific antioxidants on exercise training-induced vascular adaptations remain el

168 However, the effects of antioxidants on exercise training-induced vascular adaptations remain el

169 These data suggest that regular exercise training induces significant non-neuronal cell

170 Endurance and resistance exercise training induces specific and profound changes

171 Endurance exercise training induces substantial adaptive cardiac m

172 In contrast, endurance exercise training (initiated 2 weeks after AAV9-R735X in

173 Exercise training, initiated at an advanced age, reverse

174 Importantly, aerobic exercise training, initiated even at an advanced age, re

175 The optimal exercise training intensity and strategy for individuali

176 The impact of an exercise training intervention on the physiological resp

177 behind these impairments and the benefits of exercise training interventions, our laboratory has rece

178 Exercise training is a promising but unproven interventi

179 ent in a sedentary aging population and that exercise training is an effective intervention for treat

180 Exercise training is an effective treatment for importan

181 Exercise training is effective in improving the cardiova

182 Previous studies have shown that short term exercise training is feasible in TGA patients, but its e

183 Although exercise training is the most potent intervention to add

184 After exercise training, IVRT, a measure of active ventricular

185 These data indicate that exercise training leads to parallel reductions in hot fl

186 of other laboratories suggest that endurance exercise training leads to similar changes in sedentary

187 Lifelong exercise training maintains a youthful compliance of the

188 , the profound changes to WAT in response to exercise training may be part of the mechanism by which

189 itions affecting the elderly, this "dose" of exercise training may have important implications for pr

190 ermine whether nutritional interventions and exercise training may preserve muscle area and thereby i

191 re we felt it essential to determine whether exercise training might injure a systemic right ventricl

192 y assigned to 16 weeks of moderate-intensity exercise training (n = 67) or usual activity (n = 69).

193 ized to a group that received 1 h supervised exercise training (n=10) or one that received equal-time

194 Intense aerobic exercise training negatively affects iron status; howeve

195 proximately 25% kcal reduction vs. O-SED) or exercise training (O-EX; treadmill running 20 m min(-1)

196 affect the response to 3 weeks of endurance exercise training on CD3(+) , CD3(+) /CD31(+) , CD14(+)

197 The restorative effect of exercise training on coronary microvascular function may

198 -analysis, we aim to evaluate the effects of exercise training on CRF, quality of life, and diastolic

199 Studies superimposing exercise training on hypoxic exposure demonstrate an inc

200 There was no interaction between AF and exercise training on measures of functional status or cl

201 tudies that have investigated the effects of exercise training on mitochondrial function, the "beigin

202 PSSS did not impact the effect of exercise training on outcomes.

203 ought to determine the beneficial effects of exercise training on oxidative stress and inflammation i

204 Effect of exercise training on quality of life (estimated using Mi

205 ations of endurance athletes are a result of exercise training or a genetically determined characteri

206 locity; PWV) were evaluated before and after exercise training or cage confinement.

207 were randomly assigned to either submaximal exercise training or no forced exercise (untrained).

208 Young and old rats underwent 10 weeks of exercise training or remained as sedentary, cage-control

209 +) number did not change in this region with exercise training or skeletal myofiber VEGF gene deletio

210 eria) were randomized to 12 weeks of aerobic exercise training or to exercise in combination with sim

211 HF patients with ejection fraction </=35% to exercise training or usual care.

212 eart Association class II to IV HF to either exercise training or usual care.

213 Hct normalization, exercise training, or the combination thereof significan

214 Endurance exercise training partially reversed these abnormalities

215 A total of 469 exercise-training participants enrolled in 16 separate t

216 In general, the benefits of the exercise training perdure even after a 24-week detrainin

217 tudy was to determine whether daily moderate exercise training performed during a 10-day exposure to

218 The study design included a 6-month aerobic exercise training period followed by a 2-week detraining

219 xercise training period), M2 (immediate post-exercise training period), and M3 (24 weeks after M2).

220 valuation periods were accomplished: M1 (pre-exercise training period), M2 (immediate post-exercise t

221 We hypothesised that a short exercise-training period would affect muscle and bone ma

222 ompleted a crossover study consisting of two exercise training phases at two hematocrit (Hct) values:

223 A 12-week standardized aerobic dynamic exercise training program (3 one-hour sessions per week)

224 y aimed to evaluate the effects of a 24-week exercise training program (supervised or home-based) on

225 The exercise training program improved body composition (lea

226 this study was to evaluate whether adding an exercise training program to an inpatient behavioral int

227 (Exercise Training Program to Improve Clinical Outcomes i

228 performed a pilot study to assess whether an exercise training program would result in adverse change

229 ckening increased from 2.2% to 10% after the exercise-training program.

230 with TGA and SRV were enrolled in a 12 week exercise training programme (moderate and high-intensity

231 letal muscle responses to a short controlled exercise-training programme.

232 f brain diseases, inform the optimization of exercise training programmes and assist with the identif

233 d (and may be improved) in TGA patients with exercise training programmes that are typical of recreat

234 l variation even in response to standardized exercise training programmes.

235 lled trials (RCTs) comparing the outcomes of exercise training programs in solid organ recipients aga

236 the health benefits and harms of supervised exercise training programs in solid organ recipients.

237 Conversely, exercise training promoted autophagy protein expression

238 High-level exercise training promotes AF, but the underlying mechan

239 Exercise training promotes EcSOD expression in skeletal

240 discuss the various mechanisms through which exercise training promotes mitochondrial quantity and qu

241 A Controlled Trial Investigating Outcomes of Exercise Training) randomized 2,331 ambulatory HF patien

242 Before exercise training, rats selected as low and high respond

243 in fitness that are mediated by 16 weeks of exercise training reduce the severity of physiological s

244 In this study, exercise training reduced magnetic resonance imaging-det

245 In the absence of P-CoA, exercise training reduced mitochondrial ADP sensitivity.

246 Exercise training reduced the self-reported severity of

247 A period of preoperative supervised exercise training reduces postoperative cardiac, respira

248 Exercise training reduces self-reported hot flush severi

249 nate manner in response to a sprint interval exercise training regimen in humans and to denervation o

250 muscle-specific autophagy on the benefits of exercise training remains incompletely understood.

251 lume reserve during exercise correlates with exercise training response in our TGA patients, identify

252 tem, we have discovered key pathways for low exercise training response that may represent novel targ

253 ingle acute bout of exercise and (2) chronic exercise training resulting from 3, 5, 7, 14 and 28 days

254 Long-term endurance exercise training results in a reduction in the rates of

255 Exercise training results in significant improvements on

256 s of fully supervised, whole-body resistance exercise training (RET) (72.8 +/- 1.4 years; BMI 26.3 +/

257 Resistance exercise training (RET) can rejuvenate limb blood flow r

258 Resistance exercise training (RET) has a beneficial effect on muscl

259 Resistance exercise training (RET) is one of the most effective str

260 Resistance exercise training (RET) is widely used to increase muscl

261 mpaired hypertrophic responses to resistance exercise training (RET).

262 Exercise training reversed obesity-related mitochondrial

263 In old rats, exercise training reversed the reduction in E/A, reduced

264 Exercise training reverses age-induced declines in diast

265 dysfunction, and (2) initiation of late-life exercise training reverses age-related diastolic and mic

266 h oxidative and glycolytic muscle, late-life exercise training reverses age-related microvascular dys

267 nor high-intensity intervals, nor short-term exercise training seem to injure the systemic right vent

268 Also independent of MitoQ supplementation, exercise training significantly increased quadriceps mus

269 Exercise training significantly reduced both MRI-detecte

270 odiesterase 5 inhibitors, muscle weakness by exercise training, sodium retention by diuretics and mon

271 f the left ventricle (LV), whereas a year of exercise training started later in life fails to reverse

272 w through hippocampal regions independent of exercise training state.

273 ovascular blood flow, protein metabolism and exercise training status in older men.

274 hysiological system, regardless of endurance exercise training status.

275 stressors during acute bouts of exercise or exercise training stimulate enhancement of cellular stre

276 ngs by adjusting performance in response to "exercise" training stimuli.

277 A Controlled Trial Investigating Outcomes of Exercise Training) study, which evaluated exercise train

278 care such as adherence to medical treatment, exercise training, symptom monitoring and symptom manage

279 itate individualized approaches to implement exercise training therapy in clinical practice.

280 skeletal muscle function and is improved by exercise training through both mitochondrial biogenesis

281 ulin resistance and highlight the ability of exercise training to diminish P-CoA attenuation in mitoc

282 ations of an erythroid-stimulating agent and exercise training to examine if and where limitation to

283 Specificity is a core principle of exercise training to promote the desired adaptations for

284 A Controlled Trial Investigating Outcomes of Exercise Training) trial to advance precepts of CR are c

285 A Controlled Trial Investigating Outcomes of exercise traiNing) trial, 10 CPX test variables measured

286 A Controlled Trial Investigating Outcomes of Exercise Training) trial.

287 st the hypothesis that resistance to aerobic exercise training underlies metabolic disease risk, we u

288 of Exercise Training) study, which evaluated exercise training versus usual care in chronic systolic

289 tolic HF patients who had been randomized to exercise training versus usual care.

290 In the pooled analysis, exercise training was associated with significant improv

291 n, 16 weeks of supervised moderate intensity exercise training was found to improve cardiorespiratory

292 Exercise training was performed three times per week for

293 Multi-component exercise training was the only intervention subgroup ass

294 Furthermore, exercise training was well tolerated with a low dropout

295 ic and metabolic adaptations of the heart to exercise training, we subjected mice with cardiomyocyte-

296 he chronic impacts of interventions, such as exercise training, which affect both sympathetic activit

297 A Controlled Trial Investigating Outcomes of Exercise Training, which randomized 2331 patients with e

298 ns attended, completion of CR, and change in exercise training workload while in CR.

299 marked energy deficit in addition to intense exercise training would affect changes in body compositi

300 othesis that, similar to humans and rodents, exercise training would enhance mitochondrial (Mt) bioge