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1                                     Finally, exercise induced a significant reduction in IkappaBalpha
2                           Furthermore, acute exercise induced a systemic anti-inflammatory environmen
3                                              Exercise-induced abnormalities during exercise treadmill
4  Conversely, enhanced Drp1 activity furthers exercise-induced acceleration of neuronal maturation.
5 a, cold shock, heat shock, oxidative stress, exercise-induced adaptation, caloric restriction, osmoti
6                   As a specific mechanism of exercise-induced adaptation, we identified various lysop
7                         To determine whether exercise-induced adaptations to adipose tissue contribut
8 -1alpha may mediate the initial phase of the exercise-induced adaptive increase in muscle mitochondri
9  as a key factor in the pathology of intense exercise-induced AF.
10             It remains unclear, however, how exercise-induced alterations in respiratory chemoreflex
11            In order to examine the effect of exercise-induced alterations in ventilatory chemoreflex
12                                              Exercise induced an increase in all strains in control s
13 s testing different exercise protocols, show exercise-induced analgesia involves activation of centra
14                                              Exercise-induced anaphylaxis (EIAn) describes a rare and
15 e result of a consensus on the mechanisms of exercise-induced anaphylaxis (EIAn), an unpredictable an
16 underwent tolerance tests, as food-dependent exercise-induced anaphylaxis (FDEIA) induced by citrus f
17                               Food-dependent exercise-induced anaphylaxis (FDEIAn) was suspected.
18 ter exercise was suspected as food-dependent exercise-induced anaphylaxis (FEIAn).
19                              Wheat-dependent exercise-induced anaphylaxis (WDEIA) is characterized by
20                              Wheat-dependent exercise-induced anaphylaxis (WDEIA) is often reported i
21 me disease entities such as 'wheat-dependent exercise-induced anaphylaxis' (WDEIA) are explicitly cha
22 cal exercise (dust mite ingestion-associated exercise-induced anaphylaxis).
23 ommon allergen implicated in food-dependent, exercise-induced anaphylaxis.
24  the source, biochemistry, and physiology of exercise-induced and disease-associated stress hyperlact
25                     The mechanisms governing exercise-induced angina and its alleviation by the most
26                  Low responders had impaired exercise-induced angiogenesis in muscle; however, mitoch
27 a powerful regulator of metabolism, mediates exercise-induced angiogenesis in skeletal muscle.
28  of a PGC-1alpha/ERRalpha/VEGF axis mediates exercise-induced angiogenesis in skeletal muscle.
29 estigate the role of caveolin-1 in treadmill-exercise-induced angiogenesis in the ischemic penumbra o
30 expression inhibited EC SMAD1 expression and exercise-induced angiogenesis.
31        We show here that PGC-1alpha mediates exercise-induced angiogenesis.
32 nd capillarization, but is not necessary for exercise-induced angiogenesis.
33 cerebellum, a region known to exhibit robust exercise-induced angiogenesis.
34  dorsomedial hypothalamus (DMH) of rats with exercise-induced anorexia, implying that central TTR may
35                                   Otherwise, exercise-induced arousal enhanced performance on tasks t
36 -blocker therapy alone for the prevention of exercise-induced arrhythmias in CPVT.
37 cal limits with the potential consequence of exercise-induced arrhythmias.
38 erns related to sustained VT can distinguish exercise-induced arrhythmogenic remodeling from ARVC and
39   This scar pattern may allow distinguishing exercise-induced arrhythmogenic remodeling from ARVC and
40                   Thus, the hypothesis of an exercise-induced arrhythmogenic right ventricular cardio
41       KEY POINTS: High work of breathing and exercise-induced arterial hypoxaemia (EIAH) can decrease
42 educing the work of breathing or eliminating exercise-induced arterial hypoxaemia (EIAH) during exerc
43 % O(2) substantially decreased or eliminated exercise-induced arteriovenous shunting in all subjects
44 ) of exercise-induced bronchoconstriction or exercise-induced asthma (which is no longer a preferred
45 ive exercise by elite athletes can result in exercise-induced asthma especially in elite swimmers and
46 , more severe bronchial hyperresponsiveness, exercise-induced asthma, and the late-phase allergic res
47                                              Exercise-induced AT lipolytic activity was significantly
48 t of the inflammatory cytokine, TNFalpha, in exercised-induced atrial remodelling.
49               Detraining did not abolish the exercise-induced attenuation in MAP in hypertensive rats
50  which is driven, in part, by alterations in exercise-induced autonomic reflexes triggered by skeleta
51                       We evaluated levels of exercise-induced brain-derived neurotrophic factor (BDNF
52 ta2-Agonists are the treatment of choice for exercise-induced bronchoconstriction (EIB) and act throu
53                                              Exercise-induced bronchoconstriction (EIB) describes acu
54                                              Exercise-induced bronchoconstriction (EIB) is a prototyp
55 hildren underwent methacholine challenge and exercise-induced bronchoconstriction (EIB) test (n = 478
56                                              Exercise-induced bronchoconstriction (EIB) was assessed
57              The first practice parameter on exercise-induced bronchoconstriction (EIB) was published
58                              The evidence of exercise-induced bronchoconstriction (EIB) without asthm
59 mental feature of asthma that is manifest as exercise-induced bronchoconstriction (EIB).
60 elf-reported symptoms to make a diagnosis of exercise-induced bronchoconstriction (EIB).
61 oth pharmaceutical and nonpharmaceutical) of exercise-induced bronchoconstriction or exercise-induced
62 ard approved study included 13 patients with exercise-induced bronchoconstriction.
63 le- and multicenter studies of patients with exercise-induced bronchoconstriction.
64                                  Conversely, exercise induced browning of the SC Ing WAT.
65  rather than circulating irisin mediated the exercise-induced browning effect on this fat tissue.
66 sed on these novel findings, we propose that exercise-induced browning of the subcutaneous WAT provid
67 group III and IV muscle afferents to dynamic exercise induced by electrical stimulation of the mesenc
68  this ability can reduce or limit resistance exercise-induced, Ca(2+)-dependent damage to the fibre b
69 rolongation and severe disease expression of exercise-induced cardiac arrest in early childhood (</=3
70  (C16:1n7) as a new molecular co-mediator of exercise-induced cardiac hypertrophy by inducing nonprol
71 polysis directly promotes the development of exercise-induced cardiac hypertrophy involving the lipok
72 t to pressure overload-, isoproterenol-, and exercise-induced cardiac hypertrophy.
73 ipose tissue lipolysis on the development of exercise-induced cardiac hypertrophy.
74        Training duration is a determinant of exercise-induced cardiac remodeling and has implications
75 ll sample of competitive endurance athletes, exercise-induced cardiac remodeling follows a phasic res
76                Contemporary understanding of exercise-induced cardiac remodeling is based on cross-se
77                      Temporal progression of exercise-induced cardiac remodeling remains incompletely
78 age, which increase in number in response to exercise-induced cardiac stress, whereas exercised mice
79             PI3K(p110alpha) is essential for exercise-induced cardioprotection and delivery of caPI3K
80 trated that FSTL1 is a potential mediator of exercise-induced cardioprotection in post-MI rats.
81  (PI3K) p110alpha is essential for mediating exercise-induced cardioprotection, and if so, whether it
82  mice, suggesting that PI3K is necessary for exercise-induced cardioprotection.
83 possible roles as physiological mediators of exercise-induced cardiovascular adaptation.
84 n 3.5-fold above that of sedentary mice, and exercise-induced cell proliferation was maintained in di
85       Antioxidant supplementation may hamper exercise-induced cellular adaptations.
86                              Tissue-specific exercise-induced changes in body fat distribution in typ
87           Novel mechanisms appear to involve exercise-induced changes in CD14+CD16+ cell populations,
88 ve velocity (odds ratio, 1.35; P=0.047), and exercise-induced changes in indexed left atrial area (od
89 e purpose of the current study was to define exercise-induced changes in indices of left ventricular
90                                              Exercise-induced changes in intramuscular metabolites we
91 ed EF, there were significant differences in exercise-induced changes in LV systolic and diastolic pr
92                                         When exercise-induced changes in mean aortic pressure gradien
93  These central responses are associated with exercise-induced changes in peripheral signals related t
94                                 However this exercise-induced cognitive improvement was attenuated in
95                                              Exercise-induced cognitive improvements have traditional
96                              The syndrome of exercise-induced collapse (EIC) in this breed is manifes
97                                              Exercise-induced cortical plasticity is associated with
98 epeated measures of respiratory symptoms and exercise-induced coughing at 5 and 7 years of age, adjus
99 ere associated with respiratory symptoms and exercise-induced coughing in the previous 12 months at 5
100  mechanisms, and management of patients with exercise-induced cTn elevations.
101 , mechanism(s), and clinical significance of exercise-induced cTn release remains incompletely unders
102  wild-type levels, and protected muscle from exercised-induced damage.
103 sed TpTe is associated with both resting and exercise-induced DD.
104 RT1 deacetylase activity is not required for exercise-induced deacetylation of PGC-1alpha or mitochon
105 increased fatigability manifest early, rapid exercise-induced declines in SM high-energy phosphates a
106                                              Exercise-induced decreases in glycolytic activity stimul
107 e patients who had stable COPD with moderate exercise-induced desaturation (during the 6-minute walk
108           In patients with COPD experiencing exercise-induced desaturation, we evaluated improvements
109 ic dyskinesia or choreoathetosis, paroxysmal exercise-induced dyskinesia, and paroxysmal non-kinesige
110 ted pAS160 as a potential determinant of the exercise-induced elevation in insulin-stimulated GU for
111                                     However, exercise-induced energy expenditure may further improve
112 ete's paradox and illustrates a mechanism of exercise-induced enhancement of muscle insulin sensitivi
113  dynamic exercise, the specific mediators of exercise-induced eNOS phosphorylation and activation in
114 O) were measured as potential mechanisms for exercise-induced EPCs mobilization.
115  Overall, the results are consistent with an exercise-induced expansion of the cortical pools of glut
116                                    Moreover, exercise-induced expression of total BDNF, BDNF splice v
117 nditions are characterized by an exaggerated exercise-induced fatigue response that is disproportiona
118 aled that mdx((5)cv) mice showed more severe exercise-induced fatigue, Rotarod performance deficits,
119                                  The maximum exercise-induced FEV1 fall was 27.9 +/- 1.4% during the
120                                              Exercise-induced glucagon levels were approximately 2-fo
121 anisms governing cutaneous blood flow during exercise-induced heat stress and provide direction for f
122 ercise and may be an important surrogate for exercise-induced hemodynamic impairment in HF patients.
123 lity exhibited significantly faster rates of exercise-induced high-energy phosphate decline than did
124 n the central and peripheral contributors to exercise-induced hyperaemia is unclear.
125 y to be responsible for the 30% reduction in exercise-induced hyperaemia with age.
126 unctional disruption of the Glp1r results in exercise-induced hyperglycaemia associated with an exces
127                      Glp1r-/- mice displayed exercise-induced hyperglycaemia due to an excessive incr
128                    Thus, we hypothesize that exercise-induced hyperglycaemia in Glp1r-/- mice is due
129                            This implies that exercise-induced hyperglycaemia in Glp1r-/- mice results
130 9-39) (Ex9) in Glp1r+/+ mice would result in exercise-induced hyperglycaemia.
131                                              Exercise-induced hyperinsulinism (EIHI) is an autosomal
132 bited (56 +/- 3%CVCmax) sites in response to exercise-induced hyperthermia.
133 n for 2 weeks and, more remarkably, enhanced exercise-induced hypertrophy and sudden death.
134 /Ang II infusion, but showed no reduction in exercise-induced hypertrophy.
135 cy is also evident in patients with diet- or exercise-induced hypothalamic amenorrhea and lipoatrophy
136 ercise is an important factor in attenuating exercise-induced IL-6 by maintaining muscle glycogen.
137 hianina cattle congenital pseudomyotonia, an exercise-induced impairment of muscle relaxation.
138 echin may be a suitable compound to maintain exercise-induced improved capillarity and mitochondrial
139  of the signal transduction pathways mediate exercise induced improvement in cognition are elucidated
140                                              Exercise-induced improvement in metabolic syndrome featu
141     This effect appears to be mediated by an exercise-induced improvement in nutrient-stimulated vaso
142     Additionally, detraining did not reverse exercise-induced improvement in PICs in the PVN of hyper
143               This effect was accompanied by exercise-induced improvements in insulin action and redu
144 neurogenesis and whether this contributes to exercise-induced improvements in insulin action.
145 maging techniques, studies have demonstrated exercise-induced improvements in lipoprotein subfraction
146 le capillary density (CD) also contribute to exercise-induced improvements in whole-body insulin sens
147               Finally, our data suggest that exercise-induced inactivity correlates with loss of sarc
148 nt gave an additional benefit, and truncated exercise induced increase at 2 d (30% reduction; P < 0.0
149                      Finally, we observed an exercise-induced increase in both JNK phosphorylation an
150 .17 to 0.04); p = 0.002]), with reduction in exercise-induced increase in E/e' (-3.0 [95% CI: -3.9 to
151 ion in skeletal muscle may contribute to the exercise-induced increase in IL-6 expression through alt
152                                          The exercise-induced increase in intramuscular inorganic pho
153                            Identification of exercise-induced increase in LV filling pressure in pati
154 letal muscle during exercise may mediate the exercise-induced increase in mitochondria.
155 n PGC-1alpha protein expression mediates the exercise-induced increase in mitochondrial biogenesis.
156               Importantly, prevention of the exercise-induced increase in MnSOD activity via antisens
157                      We hypothesized that an exercise-induced increase in MnSOD would provide cardiop
158 ars it has been widely accepted that (i) the exercise-induced increase in muscle fatty acid oxidation
159                 Our results revealed that an exercise-induced increase in neural precursor cell activ
160                  Recent studies suggest that exercise-induced increase in PAP to a mean higher than 3
161                                              Exercise-induced increase in peroxisome proliferator-act
162 ne in the subset of patients with HFpEF with exercise-induced increase in ratio between early mitral
163                                           An exercise-induced increase in secondary MR, however, is a
164 , p = 0.001), with simultaneous reduction in exercise-induced increase in the ratio of peak early dia
165 (PEI) following handgrip partially maintains exercise-induced increases in arterial blood pressure (B
166 and tetralogy of Fallot patients had blunted exercise-induced increases in arteriovenous oxygen conte
167                             However, blunted exercise-induced increases in chronotropy, contractility
168                                    Moreover, exercise-induced increases in dentate gyrus CBV were fou
169   These findings support the hypothesis that exercise-induced increases in endogenous opioid peptides
170                           Data indicate that exercise-induced increases in eNOS and EC SOD seen in yo
171 or antioxidant supplementation in mitigating exercise-induced increases in IL-6.
172 ulmonary baroreflex reduces the magnitude of exercise-induced increases in MAP and CBR resetting.
173 o blunt the homeostatic threats generated by exercise-induced increases in muscle energy and oxygen d
174 duced basal PGC-1 gene expression but normal exercise-induced increases in PGC-1 expression.
175 ed the molecular effects of exercise in that exercise-induced increases of BDNF, synapsin I and CREB
176 gnature of TGFbeta activation in response to exercise-induced injury in Sgcd null flies, finding that
177 tameres as well as protection of muscle from exercise-induced injury.
178 it is not known whether TRPC1 is involved in exercise-induced insulin sensitivity.
179            The major new findings are (1) an exercise-induced intensity-dependent metabolic attenuati
180                        In untrained, greater exercise-induced interstitial VEGF protein during exerci
181                                              Exercise-induced interstitial VEGF protein was lower in
182         This regulatory mechanism limits the exercise-induced intramuscular metabolic perturbation, p
183 ' to working locomotor muscle and limits the exercise-induced intramuscular metabolic perturbation.
184 scade of pathophysiological responses during exercise-induced ischemia and reperfusion at rest that a
185 egment depression that became more marked as exercise-induced ischemia became more pronounced, associ
186  augment the effects of GM-CSF in PAD, since exercise-induced ischemia enhances progenitor cell relea
187  stress-induced ischemia is more common than exercise-induced ischemia in patients with clinically st
188 induced ischemia occurred in 43.45%, whereas exercise-induced ischemia occurred in 33.79% (p = 0.002)
189                                     Rates of exercise-induced ischemia were slightly lower at 6 weeks
190  Patients with echocardiographic evidence of exercise-induced ischemia, ejection fractions lower than
191  and to increase supply, thereby attenuating exercise-induced ischemia.
192 s no statistically significant difference in exercise-induced ischemia.
193                                  The role of exercise-induced lactate in mediating these effects, pot
194 ness suggests a possible mechanistic role of exercise-induced left ventricular hypertrophy as the bas
195 of CPVT (n = 110) or an initial diagnosis of exercise-induced long QT syndrome but with QTc <480 ms a
196 les compared to sedentary controls; however, exercise-induced Mdm2 phosphorylation was significantly
197                                 Furthermore, exercise-induced metabolic adaptation requires Ulk1.
198 V muscle afferents in limiting the endurance exercise-induced metabolic perturbation assayed in muscl
199 flammatory and repair phases associated with exercise-induced microtrauma. kg(-1) . d(-1)) after musc
200 r illnesses in their differential diagnosis, exercise-induced miRNA patterns in cerebrospinal fluid i
201 tion and reveals a significant impairment in exercise-induced miRNA/mRNA regulation with aging.
202         Additionally, the magnitude of these exercise-induced mitochondrial adaptations is currently
203 1 (SIRT1), is a proposed master regulator of exercise-induced mitochondrial biogenesis in skeletal mu
204 indings support the hypothesis of resistance exercise-induced mitochondrial gene-shifting in muscle c
205    These findings provide direct evidence of exercise-induced mitophagy and demonstrate the importanc
206                    The mechanisms underlying exercise-induced mitophagy have not been fully elucidate
207  In patients with stable COPD and resting or exercise-induced moderate desaturation, the prescription
208 tive pulmonary disease (COPD) and resting or exercise-induced moderate desaturation.
209 s significantly advance our understanding of exercise-induced muscle fatigue and its role in muscle d
210 l AAV therapy successfully prevented chronic exercise-induced muscle force drop.
211 e insulin-like growth factor (IGF) system in exercise-induced muscle hypertrophy in the context of RA
212 elected body regions and measure wasting and exercise-induced muscle hypertrophy.
213 ose of the current study was to determine if exercise-induced muscle pain is modulated by central neu
214 ure and MSNA do not appear to modulate acute exercise-induced muscle pain.
215 ut past studies mainly studied patients with exercise-induced myocardial ischemia.
216 , during the regenerative phase that follows exercise-induced myoinjury, and concomitant with myoblas
217 e gyrus CBV provides an imaging correlate of exercise-induced neurogenesis and that exercise differen
218                         The understanding of exercise-induced neurogenesis might offer preventive but
219 relationship between serotonin signaling and exercise-induced neurogenesis.
220  of Parkinson's disease is beginning to show exercise-induced neuroplastic effects at the level of sy
221 ization, suggesting a possible mechanism for exercise-induced neuroprotection in aging.
222                        The data suggest that exercise-induced NK1-R internalization results in a redu
223 al predictors and prognostic significance of exercise-induced nonsustained ventricular tachycardia (N
224     After adjustment for clinical variables, exercise-induced NSVT did not independently increase the
225                                              Exercise-induced NSVT occurred in nearly 4% of this asym
226 ent for age, sex, and coronary risk factors, exercise-induced NSVT was not significantly associated w
227                             Mechanistically, exercise-induced nuclear transcription factor FOXO3 bind
228                Heart failure symptoms can be exercise-induced or persistent at rest.
229 briefly summarize the history of research in exercise-induced oxidative stress and will discuss the m
230        The invasive hemodynamic phenotype of exercise-induced PAH (n=78) was compared with resting PA
231                                              Exercise-induced PAH is an early, mild, and clinically r
232                                           An exercise-induced PAH plateau (n=32) was associated with
233 onary exercise testing, we hypothesized that exercise-induced PAH represents a symptomatic stage of P
234 ) was lowest in resting PAH, intermediate in exercise-induced PAH, and highest in normals, whereas me
235           We describe a 61-year-old man with exercise-induced pain from a young age and a 3-year hist
236 glycogenosis that classically manifests with exercise-induced pain from childhood.
237                             Animal models of exercise-induced pain have been developed and point to c
238 nt evidence on central mechanisms underlying exercised-induced pain and analgesia.
239 ere classified into 2 groups according to an exercise-induced PASP increase above or below the median
240                                              Exercise-induced PASP increase is of high clinical and p
241    Of 124 patients, 66 were below the median exercise-induced PASP increase of 30 mm Hg (low PASP), a
242          On the basis of the assumption that exercise-induced PASP is a measure of right ventricular
243 redictions from this hypothesis, we measured exercise-induced peak metabolic rates (PMR(E)) in 45 spe
244                                              Exercise-induced peripheral quadriceps fatigue was asses
245 test whether D2-generated T3 plays a role in exercise-induced PGC-1a expression, male rats and mice w
246 ene in skeletal muscle fibres impaired acute exercise-induced PGC-1a expression.
247 se out of proportion PH (eoPH) group, and an exercise-induced PH (ePH) group.
248  There are limited data on the prevalence of exercise-induced PH determined by right heart catheteriz
249   Understanding the molecular basis for such exercise-induced phenomena is thus of considerable inter
250 loss of intracellular levels of NAD, but not exercise-induced physiologic hypertrophy.
251 his process is a potential mediator of rapid exercise-induced plasticity.
252 emote ischemic preconditioning [IPC]) reduce exercise-induced platelet reactivity.
253 RyR2 that result in leaky channels and cause exercise induced polymorphic ventricular tachycardia in
254 netic and functional determinants underlying exercise-induced polymorphic ventricular arrhythmia pres
255  A large 4-generation family presenting with exercise-induced polymorphic ventricular arrhythmia, whi
256 nction of Nav1.5 may cause familial forms of exercise-induced polymorphic ventricular arrhythmias.
257                                              Exercise induced postural tachycardia in one third of GW
258 diac dystrophin protein, which prevented the exercised induced progression of cardiomyopathy, normali
259               However, a master regulator of exercise-induced protection has yet to be identified.
260                    The clinical relevance of exercise-induced pulmonary arterial hypertension (PAH) i
261 y sought to analyze a new approach to assess exercise-induced pulmonary artery systolic pressure (PAS
262                                              Exercise-induced pulmonary hypertension (ePH) may repres
263                                              Exercise-induced pulmonary hypertension (PH) may represe
264                                  The role of exercise-induced pulmonary hypertension in decision maki
265                         We reasoned that the exercise-induced pulmonary hypertension response could n
266 failure with preserved ejection fraction and exercise-induced pulmonary hypertension.
267 entified during exercise: a normal group, an exercise-induced pulmonary venous hypertension (ePVH) gr
268  modulation of brain regions by PYY, and the exercise-induced PYY response.
269 IAH) can decrease O2 delivery and exacerbate exercise-induced quadriceps fatigue in healthy men.
270      However, the mechanism(s) that underlie exercise-induced reductions in fatty liver are unclear.
271 iver fat depletion, whereas relatively large exercise-induced reductions in IHTG content (20-40%) can
272                                    Voluntary exercise induced robust angiogenesis in mouse skeletal m
273                                              Exercise-induced RV dysfunction provides important incre
274 n of ARVC and left ventricular fibrosis with exercise-induced SCD reinforces the need for early detec
275 her promising recent research has focused on exercise-induced signalling pathways governing glucose m
276 e to treatment strategies aimed at improving exercise-induced signalling.
277                         We hypothesized that exercise-induced skeletal muscle angiogenesis would be a
278 urine double minute (Mdm)-2 is essential for exercise-induced skeletal muscle angiogenesis.
279                                 Irisin is an exercise-induced skeletal muscle hormone that induces WA
280 glycan null muscle was also found to exhibit exercise-induced SMAD signaling.
281                                              Exercise-induced ST-segment depression was present in al
282                                         Such exercise-induced structural and functional change has be
283        Mutations in RyR2 have been linked to exercise-induced sudden cardiac death (catecholaminergic
284 morphic ventricular tachycardia is a form of exercise-induced sudden cardiac death that has been link
285 d the same leaky channels in the heart cause exercise-induced sudden cardiac death.
286 ng proteins in the heart have been linked to exercise-induced sudden cardiac death.
287 dentified in patients with a genetic form of exercise-induced sudden death (catecholaminergic polymor
288 low-up, 2 asymptomatic relatives experienced exercise-induced syncope.
289  abnormalities; RV outflow tract ectopy; and exercise-induced T-wave pseudonormalization.
290 s presented combining allometric scaling and exercise-induced variations in oxygen consumption and bl
291                                              Exercise-induced vascular endothelial adaptations in the
292 y (k coefficient, P<0.0001), in keeping with exercise-induced vasoconstriction of stenosed epicardial
293              (4) In 1 patient with sustained exercise induced ventricular tachycardia, ECM accurately
294                 The prognostic importance of exercise-induced ventricular arrhythmia (EIVA) may be co
295  have reported variable risk associated with exercise-induced ventricular arrhythmia.
296 inical and genetic disorders associated with exercise-induced ventricular arrhythmias in patients wit
297 rred in the absence of cardiac arrhythmias), exercise-induced ventricular arrhythmias, and sudden car
298 drive pacing may be a therapy for preventing exercise-induced ventricular tachycardia in treatment-re
299 tion persisted after adjusting for age, QTc, exercise-induced wall motion abnormalities, and left ven
300 OR, 1.77; 95% CI, 1.14-2.75; P = 0.011), and exercise-induced wheeze (OR, 2.29; 95% CI, 1.37-3.85; P

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