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2 C) ratio, and forced expiratory volume after exhaling 75% of vital capacity (FEF75), whereas those bo
3 ed by highly transmissible influenza viruses exhale a greater number of aerosol particles and more in
5 ted with fraction of exhaled nitric oxide in exhaled air (r = 0.48, P = .004), blood neutrophils (r =
6 frequency of breathing and peak flow rate of exhaled air are necessary parameters to detect chronic o
7 loxanes (D4 and D5) can be quantified in end-exhaled air at concentrations as low as background level
8 s developed for analysis of D4 and D5 in end-exhaled air by thermal desorption gas chromatography mas
9 el successful reproduced observed chloroform exhaled air concentrations resulting from an inhalation
10 The limit of quantification was 2.1 ng/L end-exhaled air for D4 and 1.4 ng/L end-exhaled air for D5.
14 e back of the oral cavity are transported by exhaled air through the nasal cavity to stimulate the ol
16 lso measured the fraction of nitric oxide in exhaled air, blood and sputum eosinophils, and airway hy
17 ires, measurement of exhaled nitric oxide in exhaled air, blood sampling for inflammatory biomarkers,
21 itor (PAI-1), and factor XIII (FXIII), NO in exhaled breath (FENO ), spirometry (FEV1 ) and eosinophi
28 electrospray ionization mass spectrometry of exhaled breath and UHPLC-HRMS/MS experiments using exhal
29 for detecting select biomarkers in simulated exhaled breath as a step toward enabling fast and inexpe
31 successfully applied to analyze PCs in human exhaled breath by using a simple and convenient collecti
32 y aimed to apply the metabolomic approach to exhaled breath condensate (breathomics) to discriminate
37 other readily accessible body fluids such as exhaled breath condensate (EBC), saliva, urine, and bloo
38 rlaps the generally accepted H2O2 content in exhaled breath condensate (EBC), with the sensitivity of
40 mentation of early screening methods such as exhaled breath condensate analysis and low dose computed
44 e aimed at verifying whether metabolomics of exhaled breath condensate from obese asthmatic (OA) pati
45 e aimed at verifying whether metabolomics of exhaled breath condensate from obese asthmatic (OA) pati
46 enals), biomarkers of lipid peroxidation, in exhaled breath condensate of three healthy subjects (N =
48 d oxidative stress (exhaled nitric oxide and exhaled breath condensate pH, malondialdehyde, and nitri
49 essed by plasma cytokines and intraoperative exhaled breath condensate pH; alveolar type 1 epithelial
50 tion, benzothiazoles were also determined in exhaled breath condensate samples by means of ultra high
54 the noninvasive detection of metabolites in exhaled breath could potentially help to address this pr
55 e informativeness of CO concentration in the exhaled breath during systemic infection and inflammatio
58 pplied for the quantification of VOCs in the exhaled breath from 3 groups of patients, viz., those wi
60 meliorates immunopathology, is measurable in exhaled breath in individuals with pulmonary tuberculosi
63 atio determination of (12)CO2 and (13)CO2 in exhaled breath is of critical importance in the field of
65 We describe the analysis workflow to profile exhaled breath metabolites and provide here a first libr
66 rger studies to elucidate the association of exhaled breath metabolites with gender-specific disease
67 rformed to test for associations between the exhaled breath metabolome and sonographic lung abnormali
68 weight, lung function, respiratory symptoms, exhaled breath nitric oxide [eNO], exhaled carbon monoxi
70 of planktonic or biofilm cultures or in the exhaled breath of adult cystic fibrosis patients with ch
71 enabled us to document baseline compounds in exhaled breath of healthy animals and to study changes i
74 tions of acetonitrile have been found in the exhaled breath of patients with cystic fibrosis1 and may
75 rk shows evidence for real-time detection in exhaled breath of the complete series of saturated linea
79 a and identify recognized pathogens of SRKW, exhaled breath samples were collected between 2006-2009
87 extraction platforms, kraft paper mills, and exhaled breath, but its determination at ppb levels rema
102 d peripheral airways and compare it with the exhaled bronchial and alveolar NO levels in patients wit
104 n-cabin UFPs by approximately 90%, passenger-exhaled carbon dioxide (CO2) can quickly accumulate insi
105 her attractive cues, including body heat and exhaled carbon dioxide (CO2), are common to all warm-blo
106 tion [VO(2)] and ventilatory equivalents for exhaled carbon dioxide [VE/VCO(2)] slope) were compared
107 nd trains) in Cape Town, South Africa, using exhaled carbon dioxide as a natural tracer gas to evalua
108 ey model of tuberculosis transmission, using exhaled carbon dioxide as a tracer gas, to describe tran
109 ial cues available to guide such navigation: exhaled carbon dioxide, a plethora of skin odors, the ho
110 symptoms, exhaled breath nitric oxide [eNO], exhaled carbon monoxide [eCO], and high-resolution compu
112 lf-reported smoking or if their preoperative exhaled carbon monoxide level was 10 ppm or higher.
117 abstinence was established and confirmed by exhaled carbon monoxide measurements at TQD and at inter
119 ng behavior, cotinine plasma concentrations, exhaled carbon monoxide, and the Fagerstrom Test (FTND).
122 sociated negatively with microbial richness, exhaled CH4, presence of methanogens, and enterotypes en
128 d with individuals in the lowest quartile of exhaled CO, those in the highest quartile were more like
136 bjects; however, limited studies focusing on exhaled human breath are available in the literature.
137 The identification of chemical compounds in exhaled human breath is promising in the search for new
143 re susceptible to spider-attack because they exhaled less nicotine because of lower hemolymph nicotin
144 , 1.1-2.3); being overweight (1.5, 1.0-2.3); exhaled nitric oxide >/= 20 ppb (1.9, 1.3-2.7); and tota
146 (0.65 vs 0.39, P = 0.021), higher Fractional exhaled nitric oxide (38 ppb vs 25 ppb, P = 0.021) and i
150 , response to salmeterol, degree of EIB, and exhaled nitric oxide (FE(NO)) at baseline were examined
151 ify a test for nonadherence using fractional exhaled nitric oxide (Fe(NO)) suppression after directly
152 hildren's phthalate exposures and fractional exhaled nitric oxide (Fe(NO)), a biomarker of airway inf
153 ssessing the predictive value of fraction of exhaled nitric oxide (FENO ) for persistence of wheezing
156 of airway inflammation including fractional exhaled nitric oxide (FeNO) and sputum eosinophils would
157 the association between baseline fractional exhaled nitric oxide (FeNO) and the response to inhaled
159 relation to the asthma biomarker fractional exhaled nitric oxide (FeNO) in 155 subjects with asthma
160 microarray technique (BioIC) and fraction of exhaled nitric oxide (FeNO) in a population sample of 13
164 e have previously described that fraction of exhaled nitric oxide (Feno) levels and blood eosinophil
165 nts undergoing airway challenge, fraction of exhaled nitric oxide (FENO) levels decrease after bronch
167 nophil counts, total IgE levels, fraction of exhaled nitric oxide (Feno) levels, or FEV1 percent pred
168 were able to perform acceptable fraction of exhaled nitric oxide (Feno) maneuvers and spirometry (wi
169 , measurement of fractional concentration of exhaled nitric oxide (Feno) provides an in vivo assessme
174 y accessible biomarkers included fraction of exhaled nitric oxide (Feno) values, blood eosinophil (bE
175 ween 25% and 75% [FEF25-75]) and fraction of exhaled nitric oxide (Feno) were made at 14 to 15 years
176 cohort, we assessed whether the fraction of exhaled nitric oxide (FeNO), a biomarker of airway infla
178 ction, bronchial responsiveness, fraction of exhaled nitric oxide (Feno), and allergic sensitization.
179 lf-reported eczema ever, measured fractional exhaled nitric oxide (FeNO), and interrupter resistance
180 th respiratory resistance (Rint), fractional exhaled nitric oxide (Feno), and risks of wheezing and a
181 airway inflammation, assessed by fraction of exhaled nitric oxide (FeNO), in a population-based study
182 at 18 years of age, with normal fraction of exhaled nitric oxide (Feno), low bronchial hyperresponsi
186 ionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smell test, and peak nasal
187 nts to measure total eosinophils, fractional exhaled nitric oxide (Feno), sputum eosinophils, urinary
193 ometry, methacholine and mannitol challenge, exhaled nitric oxide (FeNO); Asthma Control Questionnair
197 ion, including blood eosinophils (P = .001), exhaled nitric oxide (P = .003), and epithelial CLCA1 (P
198 ones, 3.0 [95% CI, 2.4-3.7]), and changes in exhaled nitric oxide (placebo, -3.48 ppb [95% CI, -5.99
200 nchodilator reversibility >/=12%, fractional exhaled nitric oxide [FeNO] >/=35 parts per billion, and
201 of inflammatory biomarkers (ie, fraction of exhaled nitric oxide [Feno], sputum eosinophil count, an
204 pulmonary inflammation and oxidative stress (exhaled nitric oxide and exhaled breath condensate pH, m
205 ssessment (Asthma Control Test), spirometry, exhaled nitric oxide and induced sputum evaluation.
208 , RV, TLC, DLCO, and KCO) and measurement of exhaled nitric oxide before HSCT and 3, 6, and 12 months
210 eline bronchodilator response and fractional exhaled nitric oxide had good sensitivity and specificit
212 a, YKL-40 levels correlated with fraction of exhaled nitric oxide in exhaled air (r = 0.48, P = .004)
213 ocol included questionnaires, measurement of exhaled nitric oxide in exhaled air, blood sampling for
214 d value or >/=15% increase), (3) fraction of exhaled nitric oxide levels (<24 ppb), and (4) sputum eo
215 l polyposis (54% vs 27%, P </= .001), higher exhaled nitric oxide levels (38 vs 27 ppb, P = .02) and
216 onchodilator response (P = 0.03), and higher exhaled nitric oxide levels (P = 0.04) compared with the
218 here was no farm effect on lung function and exhaled nitric oxide levels in the general study populat
220 iomarkers sputum eosinophilia and fractional exhaled nitric oxide levels, along with oral corticoster
221 ital capacity percentage values, fraction of exhaled nitric oxide levels, and bronchodilator reversib
222 rgest cluster, had normal lung function, low exhaled nitric oxide levels, and lower inhaled steroid r
223 evels, sputum eosinophil counts, fraction of exhaled nitric oxide levels, and serum periostin levels,
225 osal eosinophils, as well as high fractional exhaled nitric oxide levels, exacerbation rates, and ora
229 trolled asthma had worse FEV(1), fraction of exhaled nitric oxide measured at 200 mL/s (Feno), Scond,
230 nitric oxide sensor, and the performance of exhaled nitric oxide measurement was in good agreement w
231 aires, atopy and pulmonary function testing, exhaled nitric oxide measurement, and blood collection.
237 n response, 28 (52%) of 54 had a fraction of exhaled nitric oxide response, and 29 (54%) of 54 had a
238 V) was built and integrated into a hand-held exhaled nitric oxide sensor, and the performance of exha
239 sputum eosinophils) or baseline fraction of exhaled nitric oxide to stratify patients by eosinophili
240 points for IgE levels (268 IU), fraction of exhaled nitric oxide values (14.5 ppb), and blood eosino
242 ls, blood eosinophil counts, and fraction of exhaled nitric oxide values in relationship to sputum eo
243 cts performed spirometry and had fraction of exhaled nitric oxide values measured twice during the sc
245 factors, allergic sensitization, fraction of exhaled nitric oxide values, spirometric measurements, a
247 ssment-based adjustment and biomarker-based (exhaled nitric oxide) adjustment, the dose of inhaled co
249 tors for allergic rhinitis were 25% for high exhaled nitric oxide, 22% for allergic sensitization to
250 rometry, plethysmography, sputum cell count, exhaled nitric oxide, airway hyperresponsiveness to mann
251 and important risk factors include elevated exhaled nitric oxide, allergic sensitization to common h
253 impulse oscillometry, alveolar and bronchial exhaled nitric oxide, and a methacholine provocation.
254 clinical outcome measures (FEV1, fraction of exhaled nitric oxide, and blood eosinophils) were assess
255 pes of asthma by using blood, bronchoscopic, exhaled nitric oxide, and clinical data from the Severe
256 -gal had normal lung function, low levels of exhaled nitric oxide, and low prevalence of asthma sympt
257 cs such as atopy, lung function, fraction of exhaled nitric oxide, and medication use were calculated
258 uiet natural sleep included tidal breathing, exhaled nitric oxide, and multiple breath washout measur
259 objective markers, such as lung function and exhaled nitric oxide, and their interrelation with atopy
260 nation of blood eosinophil count, fractional exhaled nitric oxide, Asthma Control Questionnaire, medi
261 level, sputum eosinophil count, fraction of exhaled nitric oxide, asthma duration, and body mass ind
262 ge, body mass index, FEV1, PC20, fraction of exhaled nitric oxide, blood eosinophil counts, and inhal
263 Skin prick testing, lung function tests, exhaled nitric oxide, hematimetry, and total serum IgE w
264 rkers from exhaled breath include fractional exhaled nitric oxide, measurement of which can help iden
266 escue therapy, pulmonary function), based on exhaled nitric oxide, or on a day-to-day basis guided by
267 ma, or acute urticaria underwent spirometry, exhaled nitric oxide, questionnaires, and serum IgE anti
268 2 airway inflammation, including fractional exhaled nitric oxide, serum IgE, periostin, and blood an
269 er clinical biomarkers for asthma, including exhaled nitric oxide, total serum IgE and pulmonary func
272 [101 completed], n = 115 to biomarker-based [exhaled nitric oxide] adjustment [92 completed], and n =
273 nvestigated relationships between fractional exhale NO (FENO) and initial pulmonary tuberculosis seve
276 asthma severity or by levels of fraction of exhaled NO (FENO), a biomarker of airway inflammation.
277 ST2 predicts asthma and asthma with elevated exhaled NO (FeNO), compared to the commonly used Asthma
284 typic traits, sputum and blood eosinophilia, exhaled NO, serum cytokines and chemokines, total serum
289 idgut into hemolymph, from which nicotine is exhaled through the spiracles as an antispider signal.
290 tive and negative likelihood ratios (LR) for exhaled VOC profiles were calculated; and publication bi
293 esults from the revised studies suggest that exhaled VOCs are promising biomarkers for asthma diagnos
294 matic search for published studies regarding exhaled VOCs in asthma diagnosis was conducted based on
298 assess the value and classification rate of exhaled volatile organic compounds (VOCs) in asthma diag
299 Biomarkers in exhaled breath condensate, exhaled volatile organic compounds (VOCs), gene expressi
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