ライフサイエンスコーパス: exhaust

1 compounds present in gasoline motor vehicle exhaust.

2 s as a result of in utero exposure to diesel exhaust.

3 d from the photochemical oxidation of diesel exhaust.

4 0 nm, near-spherical, single crystals in the exhaust.

5 ondary organic aerosol formation from diesel exhaust.

6 to minimize the amount of heat wasted in the exhaust.

7 remove NOx from gasoline-fueled diesel-like exhausts.

8 aling and were terminally differentiated and exhausted.

9 sed PD-1, indicating that they may have been exhausted.

10 to abandon the host cell when nutrients are exhausted.

11 cells that are persistently activated and/or exhausted.

12 nts in which starch reserves are prematurely exhausted.

13 e are subsequently consumed after sulfide is exhausted.

14 a final cell division as nutrients are being exhausted.

15 nt to galactose metabolism until glucose was exhausted.

16 ir use be restricted until other options are exhausted.

17 -specific CD8(+) T cells become functionally exhausted.

18 ment, 342; actual, 214), because funding was exhausted.

19 nd continuous activation, and are eventually exhausted.

20 T cells appear terminally differentiated and exhausted.

21 in a nonlinear fashion, so that reserves are exhausted 2 h prior to dawn.

22 st in a randomized, balanced order to diesel exhaust (200 mug/m(3) particulate matter with an aerodyn

23 roughly 30% of the observed particles in the exhaust, a new surface coating formed, approximately 2-5

24 nship with the uncovered surface area of the exhausted AC, which is, in turn, inversely proportional

25 Exhaust acids were largely removed by a fully warmed-up

26 an additional layer of dye molecules on the exhausted adsorbent through interlayer attractive forces

27 ated electrode-assisted deposition of diesel exhaust aerosol (DEA) on human lung epithelial cells (A5

28 he nanometer size range were observed in the exhaust aerosol with layer-structured aggregates formed

29 technologies from the viewpoint of engines, exhaust after-treatment systems (ATS) and fuels.

30 eased PD-1 and Lag-3 and became functionally exhausted after high-level T. cruzi systemic challenge.

31 heral blood, and they were also more rapidly exhausted after in vitro culture.

32 ects of driving cycles, fuel composition and exhaust aftertreatment devices.

33 The effect of emission controls (e.g., exhaust aftertreatment technologies, fuel formulation) o

34 regulations have spurred the development of exhaust aftertreatment technologies.

35 riving cycle) than from vehicles without any exhaust aftertreatment.

36 In utero exposure to diesel exhaust air pollution has been associated with increased

37 esulting in elaborated treatment systems for exhaust air.

38 We exhaust all Tier 1 options before proceeding to Tier 2,

39 h aggressive relapses and for those who have exhausted all available options, continued therapy until

40 med ductal pancreatic adenocarcinoma who had exhausted all other treatment options received (177)Lu-3

41 Exposure to allergen alone, diesel exhaust alone, or allergen and diesel exhaust together (

42 c myocytes after in utero exposure to diesel exhaust and found that the promoter for Mir133a-2 is dif

43 his way, adult neural precursor cell pool is exhausted and defective neurogenesis happens in AD brain

44 cell leukemia, which were also metabolically exhausted and had defective Akt/mTORC1 signaling, reduce

45 sistence) in TIL that are characteristically exhausted and senescent is not established.

46 consumed until all inorganic P minerals are exhausted and the final pH remains acidic.

47 leaky isolation valves, reciprocating engine exhaust, and equipment leaks were major sources, and sub

48 mbustion processes, are present in vehicular exhaust, and persist in the environment for weeks and bi

49 ndicated that industrial emission, vehicular exhaust, and solvent evaporation were three major pollut

50 With traditional search paradigms being exhausted, approaches that are altogether different from

51 tween particles in thermally treated vehicle exhaust as compared to in real-world vehicle plumes to w

52 primary particle formation in dilute engine exhaust as low quantities were collected on unimpregnate

53 and with minimum loss of material toward the exhausts as well as a sufficiently high plasma temperatu

54 epleted at pH 6.5, and all CaP minerals were exhausted at pH 5.5, showing that the CaP species presen

55 same lung was exposed to allergen and diesel exhaust but separated by approximately 4 weeks, signific

56 At the time that Acsl1(M-/-) mice were exhausted but control mice continued to run, liver and m

57 inhibited by Foxp3(+) cells, and are likely exhausted but that, nonetheless, subsets are likely func

58 r than emissions factor estimates for engine exhausts, but when combined with crankcase emissions, me

59 d the SOA yield from dilute gasoline vehicle exhaust by a factor of 8.

60 Exhausted CD39(+)CD8(+) T cells from mice hydrolyzed ext

61 Furthermore, exhausted CD39(+)CD8(+) T cells inhibited IFNgamma produ

62 Exhausted CD4 population expressed elevated levels of mu

63 s not due to the appearance of inhibitory or exhausted CD4 T cells or a strong induction of regulator

64 g Programmed Death-1 (PD-1) can reinvigorate exhausted CD8 T cells (TEX) and improve control of chron

65 T cell depletion effectively rescued deeply exhausted CD8 T cells and enhanced antiviral control dur

66 ngitis virus infection in mice, we show that exhausted CD8 T cells during the late stage of infection

67 vel strategy for effectively rescuing deeply exhausted CD8 T cells.

68 However, the regulation of metabolism in exhausted CD8(+) T (Tex) cells is poorly understood.

69 define the accessible chromatin landscape in exhausted CD8(+) T cells and show that it is distinct fr

70 Specific subsets such as exhausted CD8(+) T cells and Tregs are preferentially en

71 xhaustion severity and identifies terminally exhausted CD8(+) T cells both in mice and humans.

72 environment elicits a subset of functionally exhausted CD8(+) T cells by creating conditions that ind

73 Exhausted CD8(+) T cells in humans and a mouse model of

74 red NFAT1 overlapped with genes expressed in exhausted CD8(+) T cells in vivo.

75 infected donors, although the proportions of exhausted CD8(+) T cells increased after adoptive transf

76 Mechanistically, PSGL-1 ligation on exhausted CD8(+) T cells inhibited T cell receptor (TCR)

77 and also differentiating into the terminally exhausted CD8(+) T cells that were present in both lymph

78 at RLI was not able to reactivate terminally exhausted CD8(+) T cells.

79 that alter gene expression preferentially in exhausted CD8(+) T cells.

80 ove immunotherapies that restore function in exhausted CD8(+) T cells.

81 tory T cells, M2 (protumor) macrophages, and exhausted CD8(+) T cells.

82 PD-1 identifies "exhausted" CD8 T cells with impaired HIV-specific effect

83 thway is critical in regulating established "exhausted" CD8(+) T cells (TEX cells), it is unclear whe

84 n lead to the emergence of dysfunctional or 'exhausted' CD8(+) T cells, and the restoration of their

85 ructure that was absent from other acute and exhausted cells types and included key effector and prol

86 on confers a unique accessibility profile on exhausted cells.

87 phatics) were calculated to characterize the exhaust chemical composition at different engine operati

88 Exhaust chemical composition at idle was also affected b

89 sequence, designated Release 6, effectively exhausts clone-based technologies for mapping and sequen

90 244 expression revealed that TSCMs were less exhausted compared with other types of memory T cells.

91 ng of precursors), is a highly toxic vehicle exhaust component.

92 dy was to identify which of the major pellet exhaust components (including high nitrogen oxide (NOx),

93 formation by oxidizing dilute, ambient-level exhaust concentrations from a fleet of on-road gasoline

94 EFs were an order of magnitude lower due to exhaust conditioning.

95 hat still need to be addressed in automotive exhaust control catalysis.

96 t promoted the differentiation of terminally exhausted CTLs.

97 t changes in blood cell composition (notably exhausted cytotoxic T cells), it is less clear what expl

98 her exposure to allergen, exposure to diesel exhaust (DE), or coexposures modulate miRNA, gene expres

99 arly aromatics and aliphatics) of the engine exhaust decreased with increasing thrust while the fract

100 However, these cells are not exhausted, despite high PD-1 expression.

101 aerosol and ozone formation potential of the exhaust does not depend on engine technology.

102 residence could provide an ideal setting for exhausting donor-reactive T cells.

103 ESAT-6-specific T cells became functionally exhausted due to chronic antigenic stimulation.

104 non-renewable energy and will eventually be exhausted due to the high consumption rate.

105 cotinoid residues escaping in vacuum-planter exhaust during maize planting were measured in 25 fields

106 As progenitors exhaust during retinal development, expression of Hmga2

107 t work demonstrates that CD4 not only become exhausted during chronic toxoplasmosis but this dysfunct

108 However, T cells that become fully exhausted during prolonged antigen exposure remain refra

109 13-2014 using filter bags to collect planter exhaust dust and horizontal and vertical sticky traps to

110 ented sources were combined with AP-42-based exhaust emission factors (for operating reciprocating en

111 ailable regarding the effect of biodiesel on exhaust emission rates of two-stroke engines used in man

112 Neonicotinoid exhaust emission rates were 0.0036 and 0.1104 g/ha for 2

113 (16%) was higher than the contribution from exhaust emissions (12%).

114 ted vegetable oil (HVO) diesel blends on the exhaust emissions from a passenger car was examined.

115 is paper reports the chemical composition of exhaust emissions from the main engines of five ocean go

116 Finally, a comprehensive set of exhaust emissions including criteria pollutants, NO2, to

117 Exhaust emissions of 23 individual city buses at Euro II

118 ecently, attention has focussed primarily on exhaust emissions of particulate matter from traffic as

119 Air pollution resulting from exhaust emissions of vehicles has risen in the recent ye

120 This study reports high numbers of exhaust emissions particles during engine motoring.

121 ir pollution, primarily consisting of diesel exhaust emissions, has increased at a similar rate to al

122 tertreatment technologies may affect vehicle exhaust emissions.

123 served in laboratory studies of fresh diesel exhaust emissions.

124 NO concentration is preferred in the engine exhaust, especially during engine cold-start.

125 lly reduce SOA formation potential of diesel exhaust, except at low speed operations.

126 We developed a simulated vehicle exhaust exposure model in rats.

127 nvestigate the effect of allergen and diesel exhaust exposure on bronchial epithelial DNA methylation

128 With emerging evidence that diesel exhaust exposure poses distinct risks to human health, t

129 ental evidence on cigarette smoke and diesel exhaust exposure.

130 ry of seed-applied neonicotinoid residues by exhaust filter bags were 0.015 and 0.437% for 2013 and 2

131 ltration experiments were conducted using an exhaust filtration analysis (EFA) system for model valid

132 thousand to 8.5 per thousand for individual exhaust/flue samples that generally tended to be negativ

133 a spray over the North Atlantic on board the exhaust-free solar-powered vessel "PlanetSolar", and the

134 Filters were loaded with exhaust from a modern passenger car diesel engine on a d

135 ute the vast majority of the SOA formed from exhaust from on-road diesel vehicles.

136 air toxics (MSATs), were measured in vehicle exhaust from three light-duty spark ignition vehicles op

137 ti-tumor T cells that have become anergic or exhausted from ex vivo expansion or exposure to the intr

138 h are used for removing nitrogen oxides from exhaust fuels, poses a problem for their use in diesel e

139 en source and reduces the NOx content in the exhaust gas by 84%.

140 Thus, exhaust gas recirculation (EGR) has been used extensivel

141 er the combustion cycle and as a function of exhaust gas recirculation (EGR) was investigated in a mo

142 ar equipped with a three-way catalyst and an exhaust gas recirculation system were studied while the

143 Exhaust gas temperatures below 250 degrees C, for more t

144 tate reduced CH4 emissions by at most 15% at exhaust gas temperatures representative of transient con

145 rom (and treatment of) sheet-foam-production exhaust gas.

146 the most abundant carbonyl compounds in the exhaust gas.

147 vessel(s) also operate in IMO-defined ECAs, exhaust-gas treatment technologies may be cost-effective

148 ECA region could reduce compliance cost with exhaust-gas treatment technology (scrubbers).

149 t promise for the fabrication of sensors for exhaust gases and vapours due to high selectivity and se

150 minimize air pollution originating from the exhaust gases of internal combustion engines.

151 ze range together with measurements of other exhaust gases under different engine loads and different

152 Compared to port fuel injection (PFI) engine exhaust, gasoline direct injection (GDI) engine exhaust

153 t, but grows faster rather than slower after exhausting glucose, generating a reversed diauxic shift.

154 and France, we demonstrate that road traffic exhaust has a far greater impact on concentrations of ni

155 aust, gasoline direct injection (GDI) engine exhaust has higher emissions of black carbon (BC), a cli

156 te to the shaping of a broader repertoire of exhausted HSV-specific CD8(+)T cells in latently infecte

157 response by shaping a broader repertoire of exhausted HSV-specific CD8(+)T cells within the latency/

158 ficantly larger repertoire of dysfunctional (exhausted) HSV-specific CD8(+)T cells were found in the

159 cessary for mAb activity can be saturated or exhausted if tumor burdens are high, thus substantially

160 mprised a broad population of suppressive or exhausted immune cells, such as CD4(+)Foxp3(+) or PD1(+)

161 The exhausted immune response subclass expressed many genes

162 by markers of an adaptive T-cell response or exhausted immune response.

163 d 2 subclasses, characterized by adaptive or exhausted immune responses.

164 confirmed that short-term exposure to diesel exhaust in healthy subjects is associated with acute vas

165 rolled exposure study to allergen and diesel exhaust in humans, and measured single-site (CpG) resolu

166 istent with previous studies measuring plume exhaust in the ambient air.

167 hearts from mice that were exposed to diesel exhaust in utero and that have subsequently undergone tr

168 V infection but may become dysfunctional and exhausted in chronic disease.

169 al autoimmune encephalitis, platelets became exhausted in their ability to produce proinflammatory fa

170 rolonged exposure to pro-oxidants in vehicle exhaust increased anxiety-and depression-like behavior a

171 No association was found between diesel exhaust inhalation and flow-mediated dilation.

172 Diesel exhaust inhalation, which is the model traffic-related a

173 Exhausted intrahepatic CD8(+) T-cell targeting-conserved

174 Gasoline-exhaust is an important PM source with largely unknown h

175 h concentration of oxygen in the diesel-like exhausts, leading to zero pollution.

176 the expansion of specific tumor-infiltrating exhausted-like CD8 T cell subsets.

177 in addition to engaging specific subsets of exhausted-like CD8 T cells.

178 ticles which are partially carbonized in the exhaust line.

179 nitropyrene (1-NP), a highly specific diesel exhaust marker, at the neighborhood scale.

180 Exposure to diesel exhaust may play a role in the development and progressi

181 ch is known about the features of resting or exhausted memory T cells (Tmem), little is known about t

182 Exhaust methane emissions were lower than emissions fact

183 using more appropriate emission factors for exhaust methane from reciprocating engines under Subpart

184 inaccurate emission factors for leakers and exhaust methane, and various exclusions.

185 hat comprised of pro-oxidant constituents of exhaust, namely, carbon dioxide (13%), carbon monoxide (

186 boratory experiments, the emission factor of exhaust NCA varied from a relatively low value of 1.6.10

187 capable of clearing Borrelia spirochetes and exhausted neutrophils; however, the role of macrophage p

188 Sulfur driven diesel exhaust nucleation particle formation processes were stu

189 The exhaust of a burning candle was introduced in the chambe

190 nonvolatile particulate matter (nvPM) in the exhaust of a gas turbine engine for a conventional Jet A

191 ions from research aircraft that sampled the exhaust of engines onboard a NASA DC-8 aircraft as they

192 and the higher quality heat generated in the exhaust of natural gas fuel cycles are expected to incre

193 Such particles were observed in the exhaust of two heavy duty vehicles with no diesel partic

194 the position of the sampler orifice and the exhausts of the torch are provided.

195 rmation process of soot and on the impact of exhausts on the environment.

196 waste sites, and mobile sources (automobile exhaust); paints, paint products, and thinners; and seco

197 5-Aza-2'-deoxycytidine and diesel exhaust particle exposure in human bronchial epithelial

198 espect to its impact on both solid and total exhaust particle number and size distribution, downstrea

199 Exhaust particle number concentration, size distribution

200 IPSD method was evaluated using TSI's engine exhaust particle sizer (EEPS, 5.6-560 nm) and gravimetri

201 Here we examine gasoline-exhaust particle toxicity from a Euro-5 passenger car in

202 Diesel exhaust particles (DEPs) are a major component of partic

203 ion particulate matter, predominantly diesel exhaust particles (DEPs), increases the risk of asthma a

204 to environmental pollutants, such as diesel exhaust particles (DEPs).

205 fic-related particulate matter (e.g., diesel exhaust particles [DEPs]) is associated with acute infla

206 ophilic pollutants benzo[a]pyrene and diesel exhaust particles impact on the activation of lipid-spec

207 istic doses of atmospherically-aged gasoline-exhaust particles impairs epithelial key-defence mechani

208 ound that as much as 20-30% of the number of exhaust particles larger than 3 nm may be formed during

209 Similarly, diesel exhaust particles showed a marginal inhibitory effect.

210 Examples of UFPs are diesel exhaust particles, products of cooking, heating, and woo

211 In utero exposure to diesel exhaust particulates is associated with an altered cardi

212 hibitory molecules-double-positive, severely exhausted PD-1(+)CD8(+) T cells, leading to reduced tumo

213 ollers have a high proportion of potentially exhausted (PD1(+)CD160(+)2B4(+)) HIV-specific CD8(+) T c

214 acteristics: T cells with a regulatory or an exhausted phenotype and macrophages with an M2 phenotype

215 HIV-specific CD8(+) T cells demonstrate an exhausted phenotype associated with increased expression

216 ferentiation and acquisition of a chronic or exhausted phenotype depend critically on the frequency o

217 These cells exhibited an exhausted phenotype with impaired production of IFNgamma

218 ose from naive macaques, is indicative of an exhausted phenotype.

219 ons or in tumors, CD8(+) T cells acquire an "exhausted" phenotype.

220 ntify pharmacodynamic changes in circulating exhausted-phenotype CD8 T cells (Tex cells).

221 Acquisition of effector, memory, or exhausted phenotypes was associated with stable changes

222 despite the reduction of their emissions at exhaust pipe over the last two decades.

223 and size distributions were measured in the exhaust plumes of approximately 1400 drayage trucks.

224 wind speed, suggesting that buoyant aircraft exhaust plumes were the likely source.

225 wed that their distribution patterns in ship-exhaust PM2.5 were very similar to the PM2.5 emitted by

226 th, the need for fine-scale models of diesel exhaust pollutants is growing.

227 modeling was successful in predicting diesel exhaust pollution at a very fine scale and identifying c

228 ) to identify fine-scale gradients in diesel exhaust pollution in two Seattle, WA neighborhoods.

229 ed frequency of T-bet(high) cells within the exhausted population.

230 roliferative hierarchy necessary to maintain exhausted populations long term.

231 rack [2] and control [3] prey, as well as to exhaust prey by causing involuntary fatigue through remo

232 g with the strong accumulation of terminally exhausted programmed cell death-1 (PD-1)(high) T cell Ig

233 Diesel exhaust-related vasoconstriction was primarily observed

234 treatment with antioxidants augmented diesel exhaust-related vasoconstriction with a mean change in B

235 Methane slip from exhausts represented 44% of the total emissions.

236 mpared with filtered air, exposure to diesel exhaust resulted in a significant reduction in BAd (mean

237 Experiments consisted of 60 exhaust samples for seven engine types (used in commerci

238 ns have been regulated and estimated through exhaust smoke visibility (smoke number).

239 ns, human immune cells are known to adopt an exhausted state in which their effector functions are lo

240 aling drives T lymphocytes to a functionally exhausted state.

241 mechanisms by which these ligands affect the exhausted states of immune cells, however, are largely u

242 increase fuel economy, was captured from the exhaust stream of a diesel engine and was characterized

243 al conditions because moisture in combustion exhaust streams blocks oxidation sites, decreasing NO co

244 as a low-cost component for low temperature exhaust streams that are expected to be a characteristic

245 PGM catalysts for CO oxidation in simulated exhaust streams while showing no signs of inhibition by

246 dard for control of pollutants in automotive exhaust streams.

247 rees C) due to inhibition by hydrocarbons in exhaust streams.

248 In on-road exhaust studies with a heavy duty diesel vehicle and in

249 analysis of these cells revealed a partially exhausted T cell phenotype.

250 tch off molecular programs for quiescence in exhausted T cell populations, but at the expense of losi

251 ligand 1 (PD-L1) Ab can restore functions to exhausted T cell populations, recovery is often incomple

252 ncreased PD-1 and TIM3 expression similar to exhausted T cells and that T cells from leukemic hosts b

253 on of CD8(+) effector T cells into memory or exhausted T cells during acute and chronic viral infecti

254 oint-blockade (ICB)-mediated rejuvenation of exhausted T cells has emerged as a promising approach fo

255 Exhausted T cells in cancer and chronic viral infection

256 Exhausted T cells in chronic infections and cancer have

257 owever, the regulation of gene expression in exhausted T cells is poorly understood.

258 tion of the PD-1 promoter DNA is observed in exhausted T cells isolated from chronically infected ind

259 ogen clearance following an acute infection, exhausted T cells secrete lower levels of effector cytok

260 kinetics of PD-1-positive (PD-1(+)) LAG-3(+) exhausted T cells were monitored in A. marginale-challen

261 sembles that of terminally differentiated or exhausted T cells, lack of KLRG1, high eomesodermin, and

262 a tumour environment, CEACAM1 and TIM-3 mark exhausted T cells.

263 he infection, it is important to salvage the exhausted T cells.

264 the absence of NFAT1 compared with wild-type exhausted T cells.

265 a dysfunctional immune system, as shown by "exhausted" T cells, defective immunologic synapse format

266 The 'non-exhausted' T-cell state driven by CD2-induced co-stimula

267 ngines were significantly higher despite the exhaust temperature being high enough for proper SCR fun

268 At the exhaust temperature of 340 degrees C NOx reduction was 8

269 hydrocarbons (HCs)) and PM emissions at low exhaust temperatures was improved from the combustion of

270 ot reduce SOA production at idle loads where exhaust temperatures were low enough to limit removal of

271 a modern aftertreatment system (ATS) at low exhaust temperatures.

272 he model used a simulated mixture of vehicle exhaust that comprised of pro-oxidant constituents of ex

273 ctable quantities of Ce(III), whereas in the exhaust the ceria particles had additional electron diff

274 prolongs bacterial infection sufficiently to exhaust the immune response directed toward invariant su

275 lysis of delta(15)N-NOx emitted from vehicle exhaust, the largest source of anthropogenic NOx.

276 profiling revealed that the transgenic lines exhausted their carbohydrates during the dark period to

277 axies were extremely compact and had already exhausted their fuel for star formation.

278 st stars become white dwarfs after they have exhausted their nuclear fuel (the Sun will be one such).

279 culties, which raise concerns that GWAS have exhausted their potential, particularly for complex trai

280 Once the eels were exhausted, they could be safely collected.

281 diesel exhaust alone, or allergen and diesel exhaust together (coexposure) led to significant changes

282 the impact of aftertreatment on PAH-derived exhaust toxicity.

283 em of potential interest for low-temperature exhaust treatment catalysts.

284 e) emissions for CNG buses, a <5% change for exhaust treatment scenarios, and a 13% (90% confidence i

285 es that are 100 degrees C lower than current exhaust-treatment catalysts.

286 est that the relative abundance of partially exhausted tumor-infiltrating CD8+ T cells predicts respo

287 lpha-pinene in the presence of pellet boiler exhaust (under high-NOx conditions) was 18.7% and in the

288 as 18.7% and in the absence of pellet boiler exhaust (under low-NOx conditions) was 34.1%.

289 ration of formaldehyde was determined in car exhaust using a portable GC/MS.

290 ration is mainly due to an expansion of the "exhausted," virus-specific B cells, i.e., activated memo

291 clude that high proportions of senescent and exhausted VZV-specific T cells in the older adults contr

292 nths, 90 guinea pigs breathed only untreated exhaust ward air, and another 90 guinea pigs breathed on

293 he mass-weighted delta(15)N-NOx from vehicle exhaust was constructed on the basis of average commute

294 The effective SOA yield of diesel exhaust was similar to that of unburned diesel fuel.

295 very rates of seed-applied neonicotinoids in exhaust were 0.014 and 0.365% in 2013 and 2014, respecti

296 unknown downstream particles in the process exhaust were collected and characterized via aerosol and

297 etical Ni sources (industrial and automobile exhausts) were evaluated, demonstrating the health benef

298 Glucose was exhausted while fructose residues remained at the end of

299 control cannot effectively treat diesel-like exhausts with high NOx concentrations.

300 Ignoring secondary chemistry from diesel exhaust would lead to underestimates of both organic aer