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1 regulating fluid secretion in the sublingual exocrine gland.
2 ent of salivary glands and potentially other exocrine glands.
3 secretion is utilized by epithelial cells of exocrine glands.
4  by a progressive chronic attack against the exocrine glands.
5 e (SjS), an autoimmune disease targeting the exocrine glands.
6 ic alterations in secretory cells of several exocrine glands.
7 e control of fluid and enzyme secretion from exocrine glands.
8 eaf clipping, and application of fluids from exocrine glands.
9  epithelial barriers of mucous membranes and exocrine glands.
10 ized by diminished secretory function of the exocrine glands.
11 nvolved in the generation and maintenance of exocrine gland acinar cells have not yet been establishe
12                                              Exocrine glands also were obtained from rats after RCMV
13 mplex class II gene expression as well as in exocrine gland and skeletal development.
14                         The morphology of 21 exocrine glands and 13 supposedly exocrine structures re
15 helial mononuclear cell infiltrates, both in exocrine glands and in other parenchymal organs (kidney,
16  the apical membranes of epithelial cells in exocrine glands and trachea.
17 -1 immunoreactivity was also detected in the exocrine glands and was stronger than DAX-1 in the inner
18 borate cuticular crypts, supported by unique exocrine glands, and that these structures have been hig
19  Although IgG autoantibodies against various exocrine gland antigens have been described in IgG4-RD,
20      We demonstrate that the vacuoles in the exocrine glands are enlarged autolysosomes containing un
21 l of the intestinal epithelium as well as in exocrine glands associated with the gastrointestinal tra
22 s similar to CpMRP and assume therefore that exocrine gland-based defensive strategies, evolved by th
23 , exhibited extensive vacuolization of their exocrine gland cells, while the liver, brain, and muscle
24 ion, our data report a novel role of Irf6 in exocrine gland development and support a rationale for p
25 viated the need for surgical revision of the exocrine gland drainage and thus eliminated the morbidit
26 ver 3 wk post-AdV infection, consistent with exocrine gland dysfunction as a consequence of the infla
27          Some of these findings suggest that exocrine gland dysfunction may precede autoimmunity or r
28 rtile, yet experience testicular atrophy and exocrine gland dysplasia.
29  lymphocytic infiltration and destruction of exocrine glands, followed by systemic organ damage and B
30 show a requirement for the MC5-R in multiple exocrine glands for the production of numerous products,
31 ve of a coordinated system for regulation of exocrine gland function by melanocortin peptides.
32 d diabetes in these animals, we investigated exocrine gland function in NOD.B10.H2b mice, which have
33  in acinar cells and are required for proper exocrine gland function.
34 ammation, sexual function, pigmentation, and exocrine gland function.
35 idogenesis, obesity, energy homeostasis, and exocrine gland function.
36 oidogenesis, obesity, energy homeostasis and exocrine gland function.
37 likely calcium-dependent chloride channel of exocrine glands has led to a more detailed understanding
38 HAT-L4 was expressed in epithelial cells and exocrine glands in tissues including skin, esophagus, tr
39 sorder of unknown etiology, affects multiple exocrine glands including the salivary and lacrimal glan
40 ic mice results in widespread dysfunction of exocrine glands, including a marked decrease in sebum pr
41                             Morphogenesis of exocrine glands is a complex stepwise process of epithel
42 thelial cells that line the airway, gut, and exocrine glands; it is well established that CFTR plays
43 nifesting as lymphocytic infiltration of the exocrine glands leading to chronically impaired lacrimal
44               NOD.B10.H2b mice exhibited the exocrine gland lymphocytic infiltration typical of the S
45 characterized by lymphocytic infiltration of exocrine glands, mainly salivary and lacrimal, resulting
46 c autoimmune disorder that typically affects exocrine glands--mainly labial and lacrimal--leading to
47  IL-1beta, as they occur in Sjogren syndrome exocrine glands, may impair the secretory function of th
48 thological findings were observed in several exocrine glands of patients with mucolipidosis II.
49 Nonobese diabetic (NOD) mice develop an anti-exocrine gland pathology similar to human Sjogren syndro
50 they exhibited leukocyte infiltration of the exocrine glands, produced anti-nuclear autoantibodies, a
51 se activity, appearance of autoantibodies to exocrine gland proteins, and glandular lymphocytic focal
52  defined circuit by which a single compound, exocrine gland-secreted peptide-1 (ESP1), enhances repro
53 roduced by young mice before puberty, termed exocrine-gland secreting peptide 22 (ESP22).
54 lling, smooth muscle contraction, airway and exocrine gland secretion, and rhythmic movements of the
55 S. invicta venom, N. fulva applies abdominal exocrine gland secretions to its cuticle.
56  identify the role of IRF6 in development of exocrine glands, specifically the major salivary glands.
57  Characterizations of the myriad neuropteran exocrine gland systems, including prothoracic, metathora
58 oimmune disease involving salivary and other exocrine glands that leads to progressive lymphocytic in
59 ch as the secretions of airway epithelia and exocrine glands, the contraction of smooth muscles, and
60 lytic enzyme activity contributes to loss of exocrine gland tolerance by generating abnormally proces
61 rome is a chronic autoimmune disorder of the exocrine glands with associated lymphocytic infiltrates

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