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1 s and osteological correlates of the primary expiratory muscle.
2 eases ventilation through the recruitment of expiratory muscles.
3 e lower thoracic spinal cord to activate the expiratory muscles.
5 distinguishing between the contributions of expiratory muscle activity and elastic recoil to intrins
6 nvestigated the characteristic signatures of expiratory muscle activity depicted by EIT and esophagea
7 showed that the rise in respiratory-related expiratory muscle activity during progressive intensity
8 he importance of the timing and magnitude of expiratory muscle activity in causing patient-ventilator
11 mechanical ventilation might be worsened by expiratory muscle activity, which reduces end-expiratory
13 piratory motor output and the onset of tonic expiratory muscle activity; furthermore, once EMG(di) wa
14 in the electrical activity of the abdominal expiratory muscles, both in hearing and deafened adult n
15 n motor neurons that innervate laryngeal and expiratory muscles, but the brain center that coordinate
17 thoracic pressure, eventually exacerbated by expiratory muscle contraction and dynamic hyperinflation
19 suggest that coactivation of inspiratory and expiratory muscles during behaviors such as emesis and s
20 ings indicate that somatosensory feedback to expiratory muscles elicits compensatory adjustments that
21 lved in phonation and activate laryngeal and expiratory muscles essential for phonation and volume co
22 d through the process of phonation, in which expiratory muscles force air through the tensed vocal fo
29 g a cough, a natural maneuver recruiting the expiratory muscles, might prove to be a useful additiona
30 cause of greater recruitment of rib cage and expiratory muscles (p = 0.004) and because clinical sign
31 Tidal volume during both inspiratory and expiratory muscle pacing and end-tidal PCO2 remained sta
32 delay resulting in alternate inspiratory and expiratory muscle pacing at a combined rate of 14 breath
33 gest that combined alternate inspiratory and expiratory muscle pacing may be a viable alternative met
34 lity of combined inspiratory intercostal and expiratory muscle pacing to provide complete ventilatory
36 espiratory tract infections as the result of expiratory muscle paralysis and consequent inability to
37 al lesions of the MRF affect inspiratory and expiratory muscle responses to activation of the vestibu
38 fine cut-off points for both inspiratory and expiratory muscle strength (MIP and MEP, respectively) f
39 t-term mortality was associated with reduced expiratory muscle strength and markedly elevated dead sp
42 inspiratory muscle strength, whereas, of the expiratory muscle tests, only Tw Pga was significantly l