ライフサイエンスコーパス: explain

1 complex for the X chromosome has never been explained.

2 ties for two different shell thicknesses are explained.

3 This association is currently only partially explained.

4 This variant alone explained 14% of the variation in fetuin-A levels.

5 All associated loci explained 2.9%, 2.5%, and 3.1% of variation in systolic,

6 wer to identify new variants, which together explain 3.7% of IOP variation.

7 te of a quantitative trait locus on Ca5 that explained 59% of flowering time variation under short da

8 se I hypersensitivity sites were reported to explain 79% of SNP heritability; using our improved heri

9 A meta-regression including 5 variables explained 99.6% of between-study variability, revealing

10 This might explain a breakdown of the DMN in many neurological dise

11 and gaze indifference, are commonly cited to explain a diagnostic hallmark of autism: reduced attenti

12 These novel quasi-particles arguably explain a range of poorly understood experiments in gate

13 aspects of reward learning in humans can be explained algorithmically with RL in terms of an actor/c

14 nvironment that are not shared by twin pairs explained all remaining variance in drink preferences.

15 difiable factor in multiple disorders, could explain any of these associations.

16 ately 80% of the variance in translation and explains approximately 5% of the variance in protein exp

17 a(2+) -sensitive nature of SK channels could explain arrhythmic SAN pacemaker activity in the atrial-

18 al modeling confirmed that learning was best explained as a mixture of two mechanisms: a fast, capaci

19 the tight junction protein occludin (OCLN), explain, at least in part, HCV's limited ability to ente

20 actors than simply charismatic species might explain attractiveness of protected areas, and call for

21 edict the likelihood of stable remission and explain autoantigen gene regulation.

22 by Bayesian inference, which had a top-down explaining-away effect that enabled recognition of an in

23 Traffic explains between 60% and 96% (at +/-20%) of TEX levels w

24 This work provides a structural basis to explain biochemical and genetic data on the importance o

25 d neighborhood factors remains prevailing in explaining black-white differences in birth outcomes, th

26 is the key driving force that simultaneously explains both the equilibrium of supercooled liquid and

27 ion in the PDZ domain-ligand interaction was explained by 3D structural models, which showed a hormon

28 effect of PEG 20 on cold denaturation can be explained by a change in water activity, according to Pr

29 hat high HPV prevalence and diversity can be explained by a combination of a lack of homologous immun

30 e, variation in FF volume seems to be better explained by a combination of factors such as anatomical

31 Among the PI3K-Akt targets, 75% could be explained by a deactivation of adenylate-uridylate-rich

32 thermoelectric figure of merit, which can be explained by a dislocation scattering model.

33 We conclude that this behavior is best explained by a form of path integration in which the fli

34 noids in the PVAT-induced contraction can be explained by a greater release of thromboxane from PVAT

35 These results are quantitatively explained by a model in which partially dissociated boun

36 Outcomes are not completely explained by agricultural performance or the observed ch

37 -GREML found that the proportion of variance explained by all common single-nucleotide polymorphisms

38 Metabolite disruptions were not explained by altered expression of genes associated with

39 the growth parameters investigated has been explained by an analytical relationship between the effe

40 (4E-BP2) protein in [Formula: see text] are explained by an excluded volume effect, similar to that

41 olytic activity of Gly1629Glu can be readily explained by an increased availability of the ADAMTS13 c

42 l of adaptation in diverse streams cannot be explained by an increased recolonization of sensitive in

43 induced inflammatory responses, which may be explained by BCD-mediated attenuation of complement acti

44 pulation structure in A. ocellifera was best explained by both isolation by distance and isolation by

45 dicating that the reduction in CFU number is explained by cells entering into a Viable But Non-Cultur

46 rds of the decline in organic aerosol can be explained by changes in anthropogenic emissions, primari

47 These fear-inhibitory effects cannot be explained by changes in appetitive behavior.

48 much of plant morphological evolution can be explained by changes in gene expression, examining its e

49 bile salts in Fut2(-/-)(high) mice were not explained by cholestasis.

50 increase in fatty acid oxidation, cannot be explained by decarboxylated osteocalcin changes, suggest

51 port suggested much of the Kok effect can be explained by declining chloroplastic CO2 concentration (

52 nstant across hospital quintiles and was not explained by differences in patients presenting with mul

53 The amount of variance explained by each feature space was then assessed using

54 strate acceptance and stereoselectivity were explained by extensive molecular modeling (MM) and molec

55 and that roughly half of this effect can be explained by functional annotations negatively correlate

56 ntial part of the variation in tooth loss is explained by genetic as well as environmental factors sh

57 ontributions to cancer and that which can be explained by genomic variants, both inherited and somati

58 Variability among studies may be explained by geography and patient factors.

59 hroughout their lifespan, which is not fully explained by hormonal or genetic factors.

60 This pattern cannot be explained by inbreeding avoidance or as a response to mo

61 Their formation can be explained by Lewis acid induced opening of the epoxy bri

62 nted by Pepper & Nettle cannot be adequately explained by life-history theory.

63 46% of mortality risk was explained by multivariable modelling with these variable

64 PCH, and the vast majority of PCH cases are explained by mutations in TSEN54, which encodes a subuni

65 ldren of overweight or obese mothers was not explained by obesity-related pregnancy or neonatal compl

66 patients with HOCM after surgery, which was explained by opposite changes in the septum (decrease) a

67 Homochirality is explained by origin of prebiotic life in one hemisphere

68 netic diversity within paleodrainages can be explained by past riverine properties (i.e., area and nu

69 es in time to acceptance for transplant were explained by patients' demographic, cultural, psychosoci

70 the developing brain, those effects could be explained by patterns of neural activity that do not rep

71 sion during intermittent aeration was mostly explained by periodic inhibition caused by free ammonia

72 eds in potential spawning habitat change was explained by predicted increases in mean annual flood si

73 This effect could be explained by productive substrate binding that protects

74 Variations in attrition may be explained by program director attitudes, although larger

75 that such negative kin discrimination can be explained by selection for unrelated targets to invest m

76 nderant prevalence of ASD might partially be explained by sex differences in clinical symptoms, etiol

77 Plant growth promotion was explained by slow release of nutrients, although a mecha

78 community-associated MRSA rates was largely explained by socioeconomic factors.

79 , most pronounced in the gamma band, and not explained by spatial movement or anxiety-like behavior.

80 This difference might be explained by structural preservation after crush of Ia a

81 These spatially extended patterns were not explained by task variables or temporally discrete senso

82 ands within their shell walls that cannot be explained by temperature alone.

83 ic size-frequency scaling of icebergs can be explained by the emergence of a dominant set of driving

84 order to estimate the variance in depression explained by the genetic vulnerability, the stressors an

85 n of WSOCs with increasing carbonization was explained by the hydrophobic carbonaceous matrix acting

86 These interesting outcomes are explained by the influences of societal learning or cult

87 model, the required 20% DMC can be entirely explained by the large differences between donor and rec

88 observed in the presence of NO and it can be explained by the radical consumption by NO as SOx and NO

89 ere we show that this variability is largely explained by the remote Atlantic multidecadal oscillatio

90 in the hole-doped region, which is naturally explained by the theoretically predicted crossover into

91 mpound heterozygous variants, which could be explained by variable disease expression and warrants ca

92 50% of genome-wide expression variability is explained by variation across individuals and identified

93 ic variability in ADHD is independent of, or explained by, risk factors may provide etiological insig

94 ific regulatory circuitry of DLX5 might help explain certain aspects of preeclampsia.

95 This mechanism does not explain certain manifestations of HIV disease, such as i

96 two orthogonal dimensions of social value to explain contempt: relational value, predicted by coopera

97 extent to which balanced network theory can explain correlated neural variability.

98 he Marburg and Ebola virus VP35 proteins may explain differences between them in polymerase function

99 ion, characterizes successful ageing and may explain differential ageing trajectories across cognitiv

100 rdingly, variation in initial conditions may explain divergent population responses to infection that

101 acterized by light-analyte interactions, are explained explicitly.

102 fit global patterns of isotope data and may explain features such as the DUPAL anomaly and long-stan

103 he expression of AE3 in neurons, which could explain how AE3 reduces seizure susceptibility.

104 ensitive, providing a potential mechanism to explain how ageing influences their amplitude and functi

105 We explain how alterations to their profiles and functions

106 Neighbourhood effects also help explain how and why the benefits of interventions vary b

107 In this issue Ohsaki et al. explain how breastfeeding can prevent the onset of food

108 Specifically, using a common framework, we explain how cells characterized by contact inhibition of

109 atures of this multiplexed mechanism help to explain how complex connectivity is encoded and robustly

110 pare competing trophic cascade hypotheses to explain how dingoes could influence shrub recruitment.

111 In this Quick Guide, Votier and Sherley explain how diverse seabirds play important roles in eco

112 urrent study provides a mechanistic basis to explain how matrix controls the antifungal effector func

113 The goal of this study is to explain how RNA can remain soluble and available for int

114 g an 'imperfect' photon counting machine, we explain how these constraints give rise to adaptive quan

115 l walls was the first proposed hypothesis to explain how this element reduced the severity of plant d

116 rmational changes induced by leukotriene C4, explaining how substrate binding primes the transporter

117 ers into higher-order oligomers, potentially explaining how Tie2 is differentially clustered followin

118 The model also naturally explains how KaiA, by acting as a nucleotide exchange fa

119 The structure of TcPINK1 explains how PD-linked mutations that lie within the kin

120 cells, no existing quantitative model fully explains how structural differences between kinesins alt

121 referred orientations is demonstrated, which explains how the conversion reaction occurs in alpha-MnO

122 ere, we present a theoretical framework that explains how these larger fluctuations in archaeal cell

123 Our observations can be explained if ACh and glutamate are released from common

124 co-exist within a single individual and may explain in part the heterogeneous fates of metastatic le

125 ongenital HCMV infection can be sufficiently explained in terms of IE2 action alone.

126 area and human population density, variation explained increased to 75.60%, with 74.30% accuracy.

127 ed univariate connectivity in its ability to explain independent variance in the responses of individ

128 species distributions and can substantially explain isoprene emission variability in tropical forest

129 microscopy method micro-electron diffraction explain its dominant influence on tau aggregation.

130 but also readily excised from RNA, which may explain its limited efficacy in vivo.

131 e and superoxide, but none of them can fully explain its toxicity.

132 mbrane domain insertase, a function that may explain its widely pleiotropic membrane-associated pheno

133 ich are typical tobamovirus characteristics, explained its complex disease cycle and its ability to s

134 the cell membrane than GA + UV-A treatment, explaining its higher effectiveness than GA + UV-A treat

135 ial growth factor antibody bevacizumab could explain lack of effect in diffuse intrinsic pontine glio

136 Three signals explained late-onset degradation: miR-430 seeds, AU-rich

137 nd thermal degradation, but they only partly explain litter decomposition under dry conditions, sugge

138 % by the age of 50; however, the known genes explain little of the heritability.

139 under oxidized conditions, which could help explain low concentrations of organics within some smect

140 Such an atrial cardiomyopathy may explain many cases of embolic stroke of undetermined sou

141 at secondary introgression, rather than ILS, explains most of the shared nuclear genomic variation be

142 porting the dynamic CNS damage hypothesis to explain MS heterogeneity.

143 n trees, the type of mycorrhizal association explained much of the variation in plant-soil feedbacks.

144 l of numerosity coding that has been used to explain neural responses in the adult IPS.

145 Structural constraints also explain one-third of allosteric inhibitors, a finding ra

146 ighly heritable, but known pigmentation loci explain only a small fraction of the variance.

147 tantly, genetic factors operating at 8 years explained only approximately 50% of genetic variation at

148 additional hypothetical mechanisms that may explain our paradoxical findings.

149 itiation sequences alone does not adequately explain our quantitative expression data.

150 nsfer ratio for all of the patients combined explained over 90% of the variance in enlarging lesion v

151 that increasing childhood obesity rates may explain part of this increase, but, as T1D is rare, inte

152 This bias can only be explained partially by the presence of fewer GR recognit

153 ceeds acceptable levels and damages neurons, explaining pathology of human syndromes caused by betaII

154 hypotheses have emerged in the literature to explain phytoplankton blooms, but over time the basic te

155 e DIV S2 helix was found to be sufficient to explain Pre1a's potent and selective inhibitory effect o

156 The MOR-Gal1R heteromer can explain previous results showing antagonistic galanin-op

157 e persistent peroxides on the powder further explain previously observed prolonged oxidation capabili

158 ffect tertiary folding and appear to largely explain previously observed variations in hairpin ribozy

159 del for the ion translocation mechanism that explains previously controversial data for this antiport

160 ymptomatic infection are infectious it could explain re-emergences of Ebola virus disease (EVD) witho

161 lation was invoked over 40 years ago to help explain reactivity patterns in cytochrome P450, and subs

162 Although population differentiation rate explains relatively little of the variation in speciatio

163 We review current molecular models to explain ribosomopathies and attempt to reconcile the tis

164 etylated in its activation loop, which could explain self-acetylation as an important feedback mechan

165 the same enhancers/promoters, an effect not explained solely by foamy virus' modest insertional site

166 l contribution of D2R in motor functions and explains some of the side effects elicited by D2R blocke

167 The factors explaining such similarities are still debated, and the

168 ening reaction is completely silenced, which explains that the majority of the ring-opening reactions

169 NPs with established functional impact could explain the association with the most well-replicated SN

170 stage sound level processing could partially explain the auditory processing deficits in FXS.

171 To explain the basis of this defect, we tracked the fates o

172 inherently interdisciplinary in its quest to explain the brain.

173 odinium cells in hospite, they could help to explain the breakdown of the coral-Symbiodinium symbiosi

174 A central goal in biochemistry is to explain the causes of protein sequence, structure, and f

175 d colorectal stem cell dynamics, which might explain the chemopreventive action of this drug indicati

176 sexually dimorphic reinforcement systems can explain the coexistence of gregariousness and monogamy.

177 rtitioning of food resources hypothesized to explain the coexistence of multiple sympatric granivores

178 These results potentially explain the convergence of global deep water mass proper

179 This approach allows us to explain the counterintuitive pseudocontact shift behavio

180 djustment for family characteristics did not explain the cross-cohort differences.

181 Here we report results that may explain the decreased efficiency of HDAC inhibition in E

182 ergic synapses in the ventral pallidum could explain the differential regulation of VP activity.

183 us agents is poorly understood, but it might explain the differential responsiveness of patients to s

184 uggest that recessive mutations in FOXI1 can explain the disease in a subset of these patients.

185 , compared with TBS-516 and TQS, may help to explain the distinct functional effects of A-867744.

186 This model could explain the dynamic relocalization of NM2 filaments in v

187 nd carboranyl ligands on the metal carbonyl, explain the ease of the chemical processes and the stabi

188 esistance entitlements of herders, which may explain the elevated effects of political pressures on l

189 at differences in DNA methylation may partly explain the enantioselectivity of fipronil to zebrafish

190 er of dynamical patterns, a state that could explain the enormous memory capacity of the brain, and t

191 Our theoretical calculations allow us to explain the experimental observations and to give experi

192 aMnO3-type phase at the interface region and explain the experimentally observed exchange bias.

193 es performed with structurally close analogs explain the FD44 specificity and the mechanism of inhibi

194 f radical-pair magnetoreception is unable to explain the findings of the reported behavioral experime

195 We further use the insight gained to explain the formation of the unusual structure of zeolit

196 atom donation process is frequently used to explain the high antioxidant activity of polyphenolic co

197 compensatory character of some phenomena and explain the highly diverse and unpredictable magnitude o

198 rstanding of the factors that can be used to explain the improvements in MAOS and how computational c

199 spects of immunity, which at least partially explain the increased susceptibility to infection.

200 This provides a molecular basis to explain the induction of RAS-RAF association by RAF inhi

201 A mechanical model is developed to explain the interaction of the three components.

202 Lipid metabolites may partially explain the inverse association between the Mediterranea

203 regarding optoelectronic properties and help explain the long carrier lifetime in halide perovskite.

204 erization have yielded formation models that explain the mass distribution, chemical abundances and p

205 ed dendritic compartments, which may help to explain the morphology of neocortical pyramidal neurons.

206 eet dynamics nor CO2 change in isolation can explain the MPT.

207 ich random sensorimotor noise is required to explain the non-linear remnant.

208 Each mechanism is deemed inadequate to explain the observations of the eddy-covariance systems,

209 plankton, combined with sediment mixing, can explain the observations.

210 9 and 12 of diferulic acids key compounds to explain the observed AC.

211 Sexual behavior may partly explain the observed association between penile and oral

212 To explain the observed differences in oligogalactolipid bi

213 mixing between water masses is sufficient to explain the observed distribution of DOS, concluding tha

214 owering, these two effects are sufficient to explain the observed emission current distribution.

215 transferase DNMT3b and deacetylase SIRT1 may explain the observed p66(Shc)-related epigenetic changes

216 n could contribute to but may not completely explain the pathology detected in Pabpn1+/A17 mice.

217 wth to regions of high mechanical strain can explain the plastic response of cells to bending(4) and

218 d signal through alpha4beta7, which may help explain the preferential infection of gut CD4(+) T cells

219 Our results can explain the present distribution of many clays on Mars,

220 oiesis, this new immunomodulatory role could explain the protection conferred by THP during AKI.

221 de and lithium tert-butoxide are not, and we explain the relative reactivities of different (hetero)a

222 y miR-H2 has been a major hypothesis to help explain the repression of lytic gene expression during l

223 Further studies are needed to explain the residual part of the spatial variability of

224 underlying pathophysiological mechanisms may explain the risk of developing epilepsy following incide

225 rmalities or metabolic conditions that could explain the seizures.

226 the epitopes in the EDII fusion loop likely explain the sequence and antigenic conservation of these

227 onetheless, both sufficient and necessary to explain the speed of pCPI-17 dephosphorylation and the c

228 We derive a mechanistic framework to explain the stepwise spacer integration process and the

229 This update aims to confirm and explain the superiority of hyperfractionated radiotherap

230 These collective results explain the unique features of the aerobic C-H imidoylat

231 mer structure provides insights that help to explain the unusual CC dimer structure and potential sou

232 Inclusion in the IR was not sufficient to explain the variation of nucleotide substitution rates.

233 /Thr difference between the two species that explained the 30 Da mass difference.

234 In nonfarm children, Moraxella colonization explained the association between bacterial diversity an

235 havior of a small-resistance GaSb sample and explained the discontinuous change of resistivity after

236 Body mass index explained the largest proportion of AF risk.

237 differences (i.e., day versus night) better-explained the short-term variability in water quality pa

238 the mechanism of triplet energy transfer and explained the unusual regiocontrol.

239 n tomography retention pattern significantly explained the variance in cognitive performance and clin

240 ACP occupies the hydrophobic core of ISD11, explaining the basis of ACP stabilization.

241 or polarization transfer which is capable of explaining the experimental observations.

242 adients toward grain boundaries, potentially explaining the extended photocarrier lifetimes and diffu

243 viral and antifungal immunity, respectively, explaining the infectious susceptibility of DOCK8-defici

244 ively transplacental, although a causal link explaining the interval delay between maternal symptoms

245 Explaining the maintenance of adaptive diversity within

246 pothesis provides a conceptual framework for explaining the maintenance of tree diversity in tropical

247 s heat sensitive to a certain degree, partly explaining the mechanism of action of local hyperthermia

248 ical and climatic features are important for explaining the observed patterns of genetic variation ac

249 vagal tone, did not appear to have a role in explaining the protection against depression.

250 on waterhemp and the results are critical in explaining the rapid dispersal of GR common waterhemp in

251 man cells together with HsRAD51, potentially explaining the synergistic effect of attenuating the exp

252 Importantly, this phenomenon explains the aberrant expression patterns of some cancer

253 Our picture explains the formation of the species observed experimen

254 a phenomenological theory that successfully explains the magnetoelectric coupling.

255 am-type phase lens dominates the imaging and explains the measured focal length of about one millimet

256 s in the periphery of the Tarim Basin, which explains the presence of the normal faults interpreted i

257 tension and whether reducing the risk of LVH explains the reported cardiovascular disease (CVD) benef

258 d compounds is strongly required in order to explain their behavior in solution and in biological env

259 ations by memantine and ketamine and help to explain their differential clinical effects.

260 y ordering the disordered phase, which could explain their role in cell membranes.

261 in the presence of ATP are much too small to explain their substantial differences in light scatterin

262 ty in parietal cortex of individual subjects explained their movement extent variability.

263 Drift alone could not explain these changes.

264 To explain these effects, we used behavioural responses to

265 To explain these findings, we propose a mean-field theory f

266 We explain these observations using an analytically solvabl

267 To explain these results, we explored modified Langmuir-Hin

268 Explaining these observations, enhancers show TF-depende

269 Explaining these results, we have found that MR-409 exer

270 A model explains these codon-context effects by suggesting that

271 Several hypotheses have been proposed to explain this genomic variability, but the fundamental dr

272 urces of bias and offer potential reasons to explain this observation, as well as propose a recommend

273 We explain this pattern by topographic and ecological chang

274 protein content or PLIN5+ LDs increased the explained variance in GIR (74.7% and 80.7% for PLIN5 pro

275 ult to identify consistent host factors that explain variation in microbiomes across hosts, despite l

276 In explaining variation in violent aggression across popula

277 ntal economic games in ways that are hard to explain via evolutionary mating motives.

278 ent and low concentrations in water could be explained via a hypothetical dynamic emission scenario c

279 nferred by their high toxicity might help to explain why calling has not yet disappeared, and that vi

280 neurons produces aversive effects that might explain why cannabinoid is not rewarding in rodents and

281 Taken together these findings help to explain why CNS neurons die after axotomy, strongly sugg

282 direct role for RANK in lung cancer and may explain why female sex hormones accelerate lung cancer d

283 and related analogs by GDH in the L cell may explain why GLP-1 secretion, but not that of insulin, is

284 To explain why klotho preferentially targets lipid rafts we

285 of Schepaschenko et al was not used, and we explain why our uncertainty assessment is complete and h

286 r proposes a cultural evolutionary theory to explain why shamanism consistently develops, and in part

287 portant intracellular location of CD24; they explain why surCD24(-) cells can remain aggressive, and

288 ld theory (DFDMFT) scheme to comprehensively explain why tetragonal FeS shows both semiconducting and

289 These findings might help to explain why TNF blockade improves lung function in only

290 igns that might achieve better outcomes, and explain why we need open and international discussions c

291 especially bitter in bacterial chromosomes, explaining why actively transcribed genes are always co-

292 Explaining why DLK inhibition is only partially protecti

293 signaling, and endoplasmic reticulum stress, explaining why SRp55 depletion triggers beta-cell apopto

294 This difference explains why antibodies are effective against glioblasto

295 This likely explains why certain neurodevelopmental disorders are ch

296 t in the later stages of fermentation, which explains why higher amylolytic activity prolonged the pr

297 s encode features along specific axes, which explains why they were previously found to respond to ap

298 rns of atypical development are difficult to explain with existing models that emphasize stress pathw

299 structure results from different methods is explained with respect to their difference in measurable

300 sured resonant elastic X-ray response can be explained with the "site-selective" Mott scenario withou