ライフサイエンスコーパス: explain why

1 This may also explain why a comparable response of plant communities t

2 : Spontaneous fluctuations of brain activity explain why a faint sensory stimulus is sometimes percei

3 We hypothesized that osmotic responses could explain why a minority of type III cells (34%) had AI sa

4 A novel mechanism is unveiled to explain why a pro-mutagenic nucleotide lesion (oxidized

5 However, this did not explain why a rapid rise in temperature (a temperature j

6 We also attempt to explain why a scaffold receives more attention over time

7 s for GATA2 in the lymphatic vasculature and explain why a select catalogue of human GATA2 mutations

8 e present findings that, taken together, can explain why a stronger association leads to a reduced re

9 This versatility may explain why a variety of organisms have extensively expl

10 stricted to hematopoietic cells, potentially explaining why a mutation in ARPC1B has now been observe

11 It also explained why acceleration data from wild cheetahs pursu

12 especially bitter in bacterial chromosomes, explaining why actively transcribed genes are always co-

13 We develop theoretical models that explain why adaptation to a wide range of patterns of en

14 This finding may help explain why adaptive radiation is common on oceanic arch

15 The existence of these isoforms might explain why affected individuals with biallelic FLAD1 fr

16 We propose that this concept may explain why agonist.receptor complexes can be inactive a

17 This might explain why all known GAPPS-affected families carry prom

18 robial strategy for transit to new hosts and explain why an organism expresses a toxin that damages t

19 of modern societies is the key ingredient to explain why and how multiculturality emerges and thrives

20 This difference explains why antibodies are effective against glioblasto

21 The mechanism explains why apoE3 differs from apoE4 with respect to di

22 igh tundra soil Hg concentrations might also explain why Arctic rivers annually transport large amoun

23 This may in part explain why ATP is maintained in such high concentration

24 sive neurons explains these changes and also explains why attention decreased or increased correlatio

25 ctuations in photosynthetic productivity and explains why Australia was a major contributor not only

26 This might explain why autoimmunity is often tissue-specific, and i

27 Such subsets likely explain why B cell depletion can either ameliorate or ex

28 The results explain why B. subtilis with its Asn synthetase genes kn

29 ionally resistant SMO mutations at baseline, explaining why BCNS-BCCs lack intrinsic resistance to SM

30 This may explain why both the immediate effects and after-effects

31 ced modeled near-infrared (NIR) reflectance, explaining why both modeled and observed EVI, which is e

32 o those of women from Western populations to explain why breast cancer is more aggressive and present

33 nferred by their high toxicity might help to explain why calling has not yet disappeared, and that vi

34 residues 292, 296, 299, 302, and 303), which explains why CaM binds two molecules of ER-alpha in a 1:

35 Such differences in neoantigen quality might explain why cancer immunotherapy induces tumor regressio

36 neurons produces aversive effects that might explain why cannabinoid is not rewarding in rodents and

37 cross the lambdaphage and human genomes, and explain why Cas9's off-target activity can be so high.

38 te that mediates p27 and substrate affinity, explaining why Cdk-Spy1 is poorly inhibited by p27 and l

39 This likely explains why certain neurodevelopmental disorders are ch

40 These findings help explain why CF mice are protected from infection and nom

41 ing methods of historical metal threads, and explain why chemical information has to be connected to

42 oscillators in different spatial groups, to explain why chimera states of certain numbers of cluster

43 show that the principle of electroneutrality explains why chloride influx is essential for the develo

44 Taken together these findings help to explain why CNS neurons die after axotomy, strongly sugg

45 Taken together, our data may explain why colonization of superantigen-producing S. au

46 Temperature effects can also explain why colored crust failed to reproduce the platea

47 which is highly amenable to intervention and explains why combining economic empowerment of women and

48 We then used this decomposition to explain why covariation of correlations with firing rate

49 The proposed etiology can explain why CP presents with features distinct from thos

50 This explains why CtXyl5A is capable of hydrolyzing xylans th

51 These observations can explain why daughter centrioles have to pass through mit

52 mbined with a longer time in the region, can explain why Daviesia is far more species-rich there than

53 This finding might explain why deep-brain stimulation of the STN can impair

54 ssure working on this alternative frame, may explain why, despite having lost the E6 open reading fra

55 ting epidermal barrier repair genes can help explain why developmental activation of the Toll, ERK, o

56 The model was also able to explain why different multisensory products are often ob

57 utations that modulate aggregation and might explain why different research groups report different r

58 Furthermore, it explains why discontinuing immunotherapy may result in e

59 Explaining why DLK inhibition is only partially protecti

60 ends results in nucleotide loss or addition, explaining why DSBs repaired by NHEJ are rarely restored

61 This finding may help explain why, during REM sleep, we remain disconnected fr

62 This in part may explain why each cascade has very similar "initiating tr

63 uced shift from tethered to direct contacts, explaining why EGCG remodeling occurs without release of

64 However, this model fails to explain why electronic communication between triarylamin

65 t increases in oil production in the SJB may explain why emission of CH4 has not declined.

66 endence upon interaction parameters may help explain why experimental work on the temperature depende

67 ciated with the activating transition, which explains why extensive conformational sampling is necess

68 considered with regards to human use and may explain why fatigue, headaches and nervousness have been

69 Computation explains why Fe2+ can be a more potent cofactor than Mg2

70 direct role for RANK in lung cancer and may explain why female sex hormones accelerate lung cancer d

71 ance or a change in hearing threshold, helps explain why fish that experienced 12 weeks of impulsive

72 We explain why fits of subdiffusive mean-square displacemen

73 Our findings explain why G protein polarization is beneficial and ill

74 generation and recovery, which can, perhaps, explain why general immunosuppression in TBI patients is

75 G2 active and protected from oxidation might explain why generation of epitope 1-targeting plasma cel

76 The data may also help to explain why genetic insults to alpha4 subunits are assoc

77 ACh strengthens higher cognition and help to explain why genetic insults to the alpha4 subunit weaken

78 and related analogs by GDH in the L cell may explain why GLP-1 secretion, but not that of insulin, is

79 rom grain interiors to grain boundaries help explain why grain boundaries may be beneficial, detrimen

80 his surprising variability in outcomes helps explain why Hamiltonella infection frequencies are often

81 nt innate control of HBV infection; this may explain why HBV has adapted to specifically replicate in

82 dergo intramolecular head-head interactions, explaining why head activity switches off.

83 istribution of fault geometrical complexity, explaining why Hector Mine has a smoother slip distribut

84 These data may explain why hereditary neuropathies associated with decr

85 This can explain why high amounts of sedentary time produce disti

86 These findings may help explain why high intensity sensory experiences are appet

87 sion of rRNA at high SRTs, which may further explain why high SRTs promote higher rates of micropollu

88 ormula: see text] have significant mobility, explaining why high-resolution spectra are observed, but

89 t in the later stages of fermentation, which explains why higher amylolytic activity prolonged the pr

90 enberger of Loyola University Medical Center explains why his laboratory offers these assays without

91 susceptibility to HIV-1 infection, which may explain why HIV-1 acquisition is increased during coinfe

92 n in KSHV-induced tumorigenesis, which helps explain why HIV-infected patients, who frequently suffer

93 parison of our structure with that of HIV RT explains why HIV RT cannot interact with eRF1.

94 wever, this ion pair decomposes immediately, explaining why HMSA and water alone are not sufficient f

95 cations, including this account's ability to explain why human-level intelligence evolved specificall

96 nature and social settings, this finding can explain why humans and animals so commonly behave accord

97 he proposed synergistic mechanisms therefore explain why ideas, rumours or products can suddenly and

98 This observation may explain why IL-5 neutralization reduces but does not com

99 , FKBP65 does not interact with LH1 and LH3, explaining why in BS triple-helical hydroxylysines are n

100 TF and chromatin dynamics in primary MK and explain why individual TF losses cause thrombopocytopeni

101 Here, we explain why industry is essential to the success of thes

102 in the pathogenesis of SIV/HIV and begin to explain why infants are more prone to rapid disease prog

103 better than the G1m3,-1 mAb cetuximab, which explains why infliximab is a better competitor for endog

104 A key challenge for any model of CSW is to explain why IOR is not always observed.

105 The Stoner criterion explains why iron is ferromagnetic but manganese, for ex

106 al, highly dispersed organization, which may explain why it does not trigger X chromosome inactivatio

107 lineages of CD8(+) T cells, and potentially explain why it is challenging to elicit memory CD8(+) T

108 pression pattern over generations, which may explain why it is prevalent among natural populations.

109 he replication process of HIV-1 virus, which explains why it has been the main target for design of d

110 This explains why it has previously been difficult to demonst

111 U gene within the sonic hedgehog pathway may explain why its loss is associated with relatively well-

112 inds to the LAP* but not to the LAP isoform, explaining why its inhibitory activity is selective for

113 This explains why JAK2V617F is associated with polycythemia v

114 eyed to kin by post-reproductive females can explain why killer whales have evolved the longest post-

115 To explain why klotho preferentially targets lipid rafts we

116 ories of language must confront: the need to explain why language has the particular properties that

117 tures common with warm-active trypsins would explain why LAT acts at high temperatures, unlike cold a

118 This could explain why LIG3 is less prone to abortive ligation than

119 ical environment of the nasopharynx, thereby explaining why lineage structure is so stable within SP

120 Collectively, its functions explain why lipoic acid is required for cell growth, mit

121 How can we explain why major evolutionary transitions have or haven

122 Their capacity to adapt may explain why male fetuses were significantly less growth

123 xual dimorphism in cerebral hemodynamics may explain why males are more vulnerable to perinatal brain

124 As a male-specific risk factor, LOY might explain why males on average live shorter lives than fem

125 Overall, our results could explain why many ground-breaking studies in science end

126 localization of DPP4 in alveolar regions may explain why MERS is characterized by lower respiratory t

127 ese mutations are present in MERS-CoV spike, explaining why MERS-CoV infects human cells.

128 e assemblies are far from perfect, partially explaining why metagenome assemblies are not used for th

129 Evolutionary theory explains why metazoan species are largely protected agai

130 ional inactivation, our findings may help to explain why microbial interdependencies are common in na

131 This paradigm may help explain why microvascular dysfunction is such a powerful

132 These insights may explain why modeled viral fusion occurs preferentially n

133 luted this early Neolithic farmer component, explaining why modern-day Sardinian populations, instead

134 These findings help explain why modes of symbiont transmission and reproduct

135 These findings may explain why more advanced cancers are more likely to res

136 ng for cationic, amphipathic AMPs, which may explain why most AMPs require micromolar concentrations

137 el role for TamB-like proteins, but also may explain why most diderms harbor a TamB-like protein whil

138 complaints from company outsiders, which may explain why most off-label promotion rulings relate to p

139 Several criteria have been formulated to explain why most zeolites are unfeasible synthesis targe

140 e underlying pathophysiological mechanism(s) explaining why motor memory consolidation in older adult

141 he hexameric form, and the structure finally explains why Mpa is unable to stimulate robust protein d

142 on of MTAs sequesters them in the cytoplasm, explaining why MTA/DDA combinations are common anticance

143 our data provide a mechanistic framework to explain why multiple CMV exposures are typically require

144 d by bone marrow exposure to contrast, which explains why multiple exposures correlate with severity.

145 ysis and structural modelling do not readily explain why NasA, NarK1 and NarK2, as well as other tran

146 These observations might explain why nature has evolved the principle of antipara

147 and the neuronal action potential threshold explains why NaV1.9 mutations that evoke small degrees o

148 capsule to combat antimicrobial peptides may explain why nearly all clinical isolates of pneumococci

149 These age-dependent associations might explain why negative selection has not removed variants

150 This explains why net erosion has occurred in some areas of t

151 ng neutrophils ex vivo and in vitro and help explain why neutrophils become easily activated after se

152 me of the van der Waals interaction probably explains why NMR efforts to detect alkane hydration shel

153 erimentally measured values of ring tension, explains why nodes move bidirectionally, and shows that

154 uired for Nun to establish its interactions, explaining why Nun acts on paused TECs.

155 These observations might in part explain why obese melanoma patients have a poorer progno

156 ffer and less fatigue resistant, potentially explaining why older tendons become more injury-prone.

157 th high- and low-affinity IgE receptors, and explains why omalizumab selectively binds free IgE.

158 rophilic moieties at C3 for binding, and may explain why only 3beta-hydroxylated bufadienolides are p

159 s found in certain motor proteins, which may explain why only a subset of DNA translocases can carry

160 ns that increase susceptibility to IA, could explain why only certain patients develop the disease.

161 Our findings may explain why osteogenesis imperfecta-causing mutations in

162 w mechanisms involving light scattering that explain why other plaque components can masquerade as TC

163 of Schepaschenko et al was not used, and we explain why our uncertainty assessment is complete and h

164 changes in human societies, but has trouble explaining why our ancestors, rather than any other grea

165 This in part explains why P450cam has such a strict requirement for P

166 These changes in the material properties explain why paler shade yellow colors of this pigment ar

167 To explain why parasite clearance rates were not faster wit

168 Altogether, these factors could not explain why participants from Russia had higher CVD mort

169 This might explain why patients with asthma, in particular those wi

170 Our results also explain why patients with NFU1 gene deletions exhibit ph

171 These differences may explain why pediatric cancer patients have a higher risk

172 nghaus et al. offer a comprehensive model to explain why people pursue darker emotions in art, but we

173 site, providing evidence for a mechanism to explain why phylogenetically rare species are more likel

174 side chains also tend to be "buried" inside, explaining why polyQ domains are insoluble on their own.

175 ients, but we lack a consistent mechanism to explain why predation risk should vary in this manner.

176 Our findings help explain why previous targeting of PKM2 in cancer cells t

177 els than similar-sized nonprimate V1s, which explains why primates have better visual acuity.

178 lar properties that it does, and the need to explain why processing pressures are manifested in the p

179 ver-reliance on cranial shape characters may explain why published parsimony studies have diverged an

180 This provides a mechanism to explain why PV IRES-mediated translation is stimulated b

181 ent of CTL at the trailing edge, which could explain why Pyk2 is important for chemotactic and migrat

182 These studies explain why pyridylhydrazines are poorly reactive in Fis

183 f fishing effort and bioeconomic simulations explain why quantifying effects of hypoxia on fisheries

184 into R-gaps and then to double-strand breaks-explaining why R-tracts do not accumulate in RNase H-def

185 contribute to the poor patient prognosis and explain why R179H disrupts even visceral smooth muscle c

186 drift is stronger for rare words, which may explain why rare forms are more prone to replacement tha

187 on the substrate flux becomes rate-limiting, explaining why reaction rates in vivo can be independent

188 This explains why recent experiments that attempt to ablate p

189 This explains why relA-spoT-deficient cells are sensitive to

190 The data explain why rigid collagen fibrils potentiate PI3K activ

191 This mechanism explains why S. lugdunensis causes more-aggressive infec

192 Here we explain why science advocacy is important and provide st

193 hizospheric phosphorus and iron mobilization explains why seagrasses are widely distributed in oligot

194 in preventing bacterial resistance and could explain why sequence-function relationships in AMPs rema

195 e are inherently complex these findings help explain why sex is maintained in natural populations.

196 The results explain why sfRNA production is evolutionarily conserved

197 r proposes a cultural evolutionary theory to explain why shamanism consistently develops, and in part

198 Our findings may help explain why shift work increases hypertension, inflammat

199 This explains why SiH2OO could not be characterized in recent

200 , namely increased cortical synchrony, could explain why sleep slow-wave activity (SWA) is higher aft

201 nstructed nature of autonomous selfhood, and explaining why social clustering has occurred and been a

202 With these insights, we explain why softer spheres skip more easily than stiffer

203 This negative result explains why solvent additives mixtures are not necessar

204 e vs the baseline level of ADHD symptoms and explain why some children remit from ADHD, whereas other

205 This decoupling may explain why some forms of sexual violence have been larg

206 diabetic retinopathy and nephropathy and may explain why some individuals with type 1 diabetes are le

207 This result may explain why some metastatic cells have been observed to

208 We also advance a hypothesis to explain why some morbidities-and their root inflammatory

209 Further, the 2D model can explain why some nonspherical particles engulf fastest (

210 These data may help to explain why some P[II] RVs, such as P[6] and P[19], comm

211 f salt bridges on the folding kinetics might explain why some proteins contain salt bridges that do n

212 This enabled us to explain why some subjects performed more variable moveme

213 However, social scientists have not explained why some advocacy organizations produce social

214 ur study uncovers the first genetic evidence explaining why some people look older for their age and

215 Further, an ecological framework explains why some older adults are resilient whilst othe

216 signaling, and endoplasmic reticulum stress, explaining why SRp55 depletion triggers beta-cell apopto

217 These results explain why stalk formation in Dictyostelia always initi

218 ohydrates from cold roots to warm canopy and explained why starch levels surged in canopies of orchar

219 he inability to standardize sugar colourants explained why starch results from these methods could no

220 Stx2a-mediated toxicity, a finding that may explain why STEC that produce only Stx2a are associated

221 in BG physiology and pathophysiology and may explain why STN is such an effective site for invasive t

222 discuss how conflicting foraging rules could explain why sunbirds do not use sweet taste to inform th

223 portant intracellular location of CD24; they explain why surCD24(-) cells can remain aggressive, and

224 be driven by conserved mechanisms, which may explain why T-cell-based immunotherapy can provide durab

225 This observation may explain why T2D patients are susceptible to developing P

226 Our model explains why targeting protein synthesis, or repairing t

227 and the seeding threshold we identified may explain why tau pathology develops very slowly in neurod

228 This may explain why TBI patients are more vulnerable to cognitiv

229 , adenine, inhibited the enzyme, which might explain why TbMTAP-mediated protection is less efficient

230 ld theory (DFDMFT) scheme to comprehensively explain why tetragonal FeS shows both semiconducting and

231 onomic gambles, researchers typically try to explain why that behaviour is biased.

232 These differences potentially explain why the activity of different mutations appears

233 These findings explain why the addition of FH5-7 to plasma leads to exc

234 The present study can partly explain why the beta-alanine supplementation protocol is

235 The findings may also explain why the Cmah(-/-) state alters severity in mouse

236 From first-principles calculations, we explain why the Co chalcogenides are ferromagnets instea

237 These divergent mechanisms explain why the combination of maternal obesity and offs

238 In our response, we explain why the differences we report result from a diff

239 cations for microvascular flows and may help explain why the effects of physiological RBC aggregation

240 Our findings also serve to explain why the elastic properties of organic layers in

241 iffness to crystallographic orientation help explain why the existing methods of dendrite suppression

242 tington disease and those of R6/2 mice might explain why the former but not the latter cause neuronal

243 enic KRas4B-but not by HRas or NRas-may help explain why the KRas4B isoform is especially highly popu

244 actors will be context dependent and so help explain why the pattern of bias is sometimes variable.

245 of climate change and land use changes could explain why the predicted enriching effects from climate

246 ariance and balanced resource allocation, we explain why the total cell size is the sum of all unit c

247 sequence features nor functions, may partly explain why the two cell lines differ in translational c

248 sed 5 of the 8 recommended disclosure items: explained why the event happened (55 of 60 surveys [92%]

249 similar in TRAF6[L74H] and wild-type cells, explaining why the bone structure and teeth of the TRAF6

250 erization, providing mechanistic insight and explaining why the chirality of the catalyst is able to

251 -2* subsite can also bind to Xylp and Arap, explaining why the enzyme can utilize xylose and arabino

252 left by E1 and E on HM14c10 largely overlap, explaining why the formation of binary complexes is mutu

253 allele A bound A-specific hot spots, perhaps explaining why the former is dominant in A/C heterozygot

254 " should be stronger in short-lived species, explaining why the generation time has a major influence

255 to confer high avidity for importin alpha7, explaining why the virus efficiently replicates in the r

256 nisms to evade the immune response, possibly explaining why the virus is never eliminated during the

257 gly by increasing sigma-donation by X, which explains why the few documented examples of H-NH2 oxidat

258 This effect also explains why the highest value of delta(iso) is observed

259 This commentary explains why the meta-analytic approach, frequently used

260 This likely explains why the N1 TMDs have become more polar and sugg

261 calculations of transformation pathways and explains why the new phase is found in an annular region

262 The model also explains why the poxvirus resolvases are more promiscuou

263 ower rates of Me-1a compared to 1a, and also explains why the reaction rate then drops with increasin

264 This bipartite character explains why the SP1 spacer is a critical element of HIV

265 inders the metagenomic assembly, which could explain why their genomes have not been identified befor

266 and cultivars constrain crop yield, possibly explaining why their mutualism has remained limited in s

267 UK10K and the combined reference panels may explain why there was less of an increase in imputation

268 E from degradation by helminth proteases may explain why these common and normally non-disease causin

269 Our model and kinetic data explain why these large steps and high velocities can on

270 ular redundancy of CspC and CspE that likely explains why these proteins have evaded selection in pre

271 ediate between the eye and hand, and further explains why these reference frames shift during movemen

272 forms (gl) bind Env inefficiently, which may explain why they are not elicited by HIV-1 Env-immunizat

273 This explained why they overwhelmingly preferred the under-va

274 *)](+) ions are formed by the MALDI process, explaining why they were not observed with ESI.

275 This probably explains why they have not previously been considered as

276 s encode features along specific axes, which explains why they were previously found to respond to ap

277 These findings satisfactorily explain why thiobases exhibit the fastest intersystem cr

278 essional antigen-presenting cells, which may explain why this LAV is so immunogenic and efficacious.

279 associated with programmed cell death could explain why this pathway is essential in most, but not a

280 and tolerogenic environment of the liver may explain why this potentially inflammatory "meeting" ofte

281 or epoxidations of electron-rich olefins and explain why Ti-based catalysts have been identified as t

282 mutant stem cells, this hypothesis does not explain why tissues without defined stem cell population

283 tern; thus, inappropriate trafficking likely explains why TLR9(471-1032) is nonfunctional.

284 These findings might help to explain why TNF blockade improves lung function in only

285 dvanced stage of the incubation period, thus explaining why tonsil specimens were not reliable for de

286 functional analyses with RNA sequencing, we explain why truncations in the A-band domain of TTN caus

287 Longer and larger studies are needed to explain why tumor necrosis factor-alpha blockade appears

288 logical processes that are regulated by MYC, explaining why tumor-specific MYC levels induce specific

289 ptional and functional analyses, may help to explain why type III strains are associated with PMH.

290 addressing limitations in reaction scope and explaining why unexpected variations in enantioselectivi

291 Whether such a role helps to explain why, unique among the large family of RhoGAPs, p

292 st a potential underlying mechanism that may explain why variability quenching occurs.

293 This model may explain why vimentin expressing metastatic lung cancer c

294 We use it to explain why viscous fluids are observed to perform worse

295 In our response, we explain why we chose an existing and internationally rec

296 impaired translation and growth, which helps explain why we detected few G-quadruplex-forming regions

297 igns that might achieve better outcomes, and explain why we need open and international discussions c

298 Two theories that attempt to explain why we sometimes reverse a decision shortly afte

299 nism for amplification of sour taste and may explain why weak acids that produce intracellular acidif

300 ing that weaker CO maturation efficiency may explain why younger men have a higher risk of fathering