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2 physiologic inhibitor of NLRP3 inflammasome activation and explain why patients with XLA are prone to develop Crohn's di
3 ance of calcium in the cell reduces its contractibility and explain why SERCA gene therapy, a change in calcium handling
4 We discuss the optimal group size and explain why, given the highly infectious nature of the diseas
5 ement of Leishmania to survive in changing environments can explain why they encode multiple eIF4E (LeishIF4Es) and eIF4G
6 Such a local mechanism for CP regulation can explain why plasticity induced by the odorant geranyl acetate
8 Thus, the reliance of RDH expression on CDK11 could explain why CDK11 is essential for the growth of many cancers
9 n humans, the analogous clinical-radiological paradox could explain why individuals with similar injuries can respond dif
10 c nerve vulnerable to further metabolic stress, which could explain why local neurodegeneration does not remain confined,
11 everal are involved in plant-pathogen response, which could explain why the 20rDNA line is hyper-resistant to both bacter
12 esults not only call for experimental testing but also help explain why polarization in beliefs about human-caused climat
13 Identifying distinct AD phenotypes here could help explain why only a subset of AD patients typically respond to
14 ur interpersonal ties, and alterations in this map may help explain why lonely individuals endorse statements such as "pe
15 ces in EC coupling and beta-adrenergic sensitivity may help explain why therapies that work in HFrEF are ineffective in H
19 ess significant for growth than previously assumed, helping explain why alternative nitrogenase genes persist in diverse
21 or why an insulin granule is selected for secretion and may explain why newly synthesized insulin is preferentially secre
22 t distinct levels of the perceptual-inference hierarchy may explain why hallucinations and delusions tend to cluster toge
23 Such selection acting over human evolutionary history may explain why we cooperate readily with unrelated and unfamilia
24 existence of mutually exclusive competing interactions, may explain why some proteins are dynamic while others are rigid.
25 w estimated inhibition levels at median exposure levels may explain why no relationship between exposure to TTR-binding c
27 Here we propose that Gestalt theory may explain why rodent islet architecture has historically been s
32 and aid in stress adaptation, and this stress tolerance may explain why many cancers aberrantly express MAGEs Here, we pr
33 the P-TEFb subunit cyclin T1 than primary cells, which may explain why many LRAs are functional in model systems but rel
34 n important species difference in OCN regulation, which may explain why serum concentrations of OCN are higher in mouse t
35 press high levels of TLR9 and low levels of TLR7, which may explain why TLR9 dysregulation is particularly consequential
36 tline the history of the concept of empathy within nursing, explain why nurses are sometimes warry of adapting concepts f
40 inor amounts of O are present in the CO-poisoned layer that explain why, surprisingly, CO desorbs at stepped and flat Pt
41 Can the Second Law of Thermodynamics explain why ecosystems naturally organize into a complex stru
44 duction and increase in S-RNase activity by NaTrxh helps to explain why S-RNase alone could be insufficient for pollen re
45 works are fragile, convincing theories are still lacking to explain why natural evolution and human design have failed to
46 roteins with altered viral replication in macrophages or to explain why Vpr is carried in the virus particle.
48 Furthermore, by generalizing Marcus theory framework, we explain why charge-transfer-dynamic modulations can only be u