ライフサイエンスコーパス: explain why

1 gene drive designs that might achieve better outcomes, and explain why we need open and international discussions concer

2 n tracking gilding methods of historical metal threads, and explain why chemical information has to be connected to 3D te

3 ing returns in nature and social settings, this finding can explain why humans and animals so commonly behave according t

4 amical mean-field theory (DFDMFT) scheme to comprehensively explain why tetragonal FeS shows both semiconducting and meta

5 asting effects of climate change and land use changes could explain why the predicted enriching effects from climate warm

6 genetic mutations that increase susceptibility to IA, could explain why only certain patients develop the disease.

7 icrodiversity hinders the metagenomic assembly, which could explain why their genomes have not been identified before.

8 The data explain why rigid collagen fibrils potentiate PI3K activation

9 These findings help explain why modes of symbiont transmission and reproduction a

10 nments in nature are inherently complex these findings help explain why sex is maintained in natural populations.

11 This finding may help explain why adaptive radiation is common on oceanic archipela

12 gnificant implications for microvascular flows and may help explain why the effects of physiological RBC aggregation are

13 ance of these factors will be context dependent and so help explain why the pattern of bias is sometimes variable.

14 This surprising variability in outcomes helps explain why Hamiltonella infection frequencies are often inte

15 Such subsets likely explain why B cell depletion can either ameliorate or exacerb

16 rain should be considered with regards to human use and may explain why fatigue, headaches and nervousness have been repo

17 data uncover a direct role for RANK in lung cancer and may explain why female sex hormones accelerate lung cancer develo

18 m of glutamine and related analogs by GDH in the L cell may explain why GLP-1 secretion, but not that of insulin, is acti

19 Taken together, our data may explain why colonization of superantigen-producing S. aureus

20 These data may explain why hereditary neuropathies associated with decreased

21 These differences may explain why pediatric cancer patients have a higher risk of d

22 Our findings may explain why osteogenesis imperfecta-causing mutations in both

23 These insights may explain why modeled viral fusion occurs preferentially near t

24 ities and suggest a potential underlying mechanism that may explain why variability quenching occurs.

25 This may explain why TBI patients are more vulnerable to cognitive dys

26 This versatility may explain why a variety of organisms have extensively explored

27 adopts an unusual, highly dispersed organization, which may explain why it does not trigger X chromosome inactivation at

28 hanism of killing for cationic, amphipathic AMPs, which may explain why most AMPs require micromolar concentrations for a

29 cape may often be driven by conserved mechanisms, which may explain why T-cell-based immunotherapy can provide durable be

30 These divergent mechanisms explain why the combination of maternal obesity and offspring

31 sing glutamate neurons produces aversive effects that might explain why cannabinoid is not rewarding in rodents and might

32 Altogether, these factors could not explain why participants from Russia had higher CVD mortality

33 on, but neither sequence features nor functions, may partly explain why the two cell lines differ in translational compen

34 y sequence analysis and structural modelling do not readily explain why NasA, NarK1 and NarK2, as well as other transport

35 These results explain why stalk formation in Dictyostelia always initiates

36 These studies explain why pyridylhydrazines are poorly reactive in Fischer

37 functionally important intracellular location of CD24; they explain why surCD24(-) cells can remain aggressive, and they

38 To explain why klotho preferentially targets lipid rafts we show

39 To explain why parasite clearance rates were not faster with spl

40 The model was also able to explain why different multisensory products are often observe

41 and macrophages in the pathogenesis of SIV/HIV and begin to explain why infants are more prone to rapid disease progressi

42 We then used this decomposition to explain why covariation of correlations with firing rate-a re

43 during infancy, our data provide a mechanistic framework to explain why multiple CMV exposures are typically required bef

44 st predators conferred by their high toxicity might help to explain why calling has not yet disappeared, and that visual

45 Taken together these findings help to explain why CNS neurons die after axotomy, strongly suggest t

46 These findings might help to explain why TNF blockade improves lung function in only some

47 luence typical of modern societies is the key ingredient to explain why and how multiculturality emerges and thrives in o

48 This paper proposes a cultural evolutionary theory to explain why shamanism consistently develops, and in particula

49 sed on this invariance and balanced resource allocation, we explain why the total cell size is the sum of all unit cells.

50 fy why the work of Schepaschenko et al was not used, and we explain why our uncertainty assessment is complete and how it