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1 -4-CLP36 complex in podocytes and provide an explanation as to how alpha-actinin-4 deficiency or muta
2 ace of the T3 channel, providing a plausible explanation as to how BCL9 co-factors binding to Pygo PH
3                    This study also offers an explanation as to how CK2alpha exclusion mutations (S120
4 ivity and might begin to provide a molecular explanation as to how E2 modulates dendritic spine plast
5                  Our results provide a novel explanation as to how ICH in newborns results in seizure
6 rogen bonding in the active site suggests an explanation as to how in the wild-type and H98N mutated
7         These findings offer a network-level explanation as to how inhibition regulates the experienc
8 s of intracellular Na+ and hence provides an explanation as to how KNa channel can control normal neu
9             This mechanism provides a likely explanation as to how Mn combats superoxide stress in ce
10         These findings provide a mechanistic explanation as to how Rb regulates cell division and apo
11          Furthermore, this study provides an explanation as to how Salmonella evades activation of au
12 experimental evidence for its presence or an explanation as to how such a polar molecule could exit t
13 methylation in cis and provide a mechanistic explanation as to how these loci impact upon OA suscepti
14 ing pathway, and provide a first mechanistic explanation as to how upregulation of CD44 may constitut
15           These findings provide a metabolic explanation as to how VPA-mediated inositol depletion ca
16                        There are no definite explanations as to the variable sensitivity to progeria
17       This finding also offers a mechanistic explanation as to why a tumor suppressor gene, such as R
18 dy to cause pathogenicity and may provide an explanation as to why apparently similar auto-antibodies
19  p53 to maintain hepatic CSCs and provide an explanation as to why autophagy is required to promote h
20 ipose tissue and also represents a plausible explanation as to why brown adipocytes ultimately specia
21      These observations suggest a structural explanation as to why certain serine lipases are resista
22                      Results herein offer an explanation as to why EGFR inhibitors failed TNBC patien
23                 Our results offer a possible explanation as to why Env immunogens have been ineffecti
24 ng the optic nerves, could be an alternative explanation as to why glaucoma develops in patients with
25                       Our results provide an explanation as to why GPER is not readily detected on th
26 W loci in (NZB x NZW)F1 mice and provides an explanation as to why H2(d/z) heterozygosity is required
27                        These data provide an explanation as to why high trkA levels are associated wi
28               This could serve as a possible explanation as to why HS-deficient cells showed signific
29                These results also provide an explanation as to why IFN-gamma may play both pathogenic
30 f Slc30a8 deletion, though the physiological explanation as to why impaired insulin secretion is not
31          Our data also provide an additional explanation as to why malignant melanocytes lose cKit ex
32 l expansion of this population, providing an explanation as to why Mir155 deletion impairs affinity m
33 ery common, but to date there is no complete explanation as to why one out of two people systematical
34             This scenario leads to a natural explanation as to why only oscillations from a single el
35 urthermore, our findings provide a plausible explanation as to why P[6] RV strains are more common in
36              The present findings provide an explanation as to why previous whole-cell Ca2+ currents
37          These results provide a mechanistic explanation as to why rpaA-null mutants die in the dark,
38            As of yet, there is no systematic explanation as to why some static images are likely to p
39 tractility, and ECM stiffness and provide an explanation as to why stiffer environments result in enh
40  of the R(61,2)C adduct provides a potential explanation as to why that adduct does not induce A -->
41 vivo, and they provide a potential molecular explanation as to why the developmental impact of these
42                     This provides a possible explanation as to why the Hb dynamics are robust under p
43 perimental regioselectivities and provide an explanation as to why the hydrogen is transferred from t
44                 These results may provide an explanation as to why the multiantennary structure is ub
45 y Hsp70/40 chaperones, providing a molecular explanation as to why the N domain is dispensable for pr
46                   This could offer a partial explanation as to why the T-cell response is impaired in
47                          It also provides an explanation as to why the unfolded protein response may
48 ural communication signals, but also a novel explanation as to why these preferentially occur on top
49 heir bacterial homologues, thus providing an explanation as to why this novel chloroplast SRP pathway
50 mine transporter, and provides an intriguing explanation as to why use of these compounds, unlike coc
51                                      Several explanations as to why converging processes may drive dr
52    These experimental results offer possible explanations as to why Juliette is perceived to have a s
53 g merlin, Falco columbarius, and alternative explanations as to why observed behaviours should differ
54                          We discuss possible explanations as to why sequence-profile and contact-pote
55 d the acquisition/loss of mutations, to give explanations as to why there is a discrepancy in pharmac

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