ライフサイエンスコーパス: explanation for

1 These data provide a neural explanation for a key behavioral signature of human cogn

2 ed for VWM storage, and it provides a common explanation for a plethora of behavioral studies in whic

3 These findings provide an explanation for a variety of modification patterns toler

4 ction and could suggest a pathophysiological explanation for acquired bleeding in some patients.

5 sterone physiology that provides 1 potential explanation for age-related cardiovascular risk.

6 Although there are some proposed explanations for aggregation-induced emission, a phenome

7 s mechanistic versatility provides a unified explanation for all extant data and a new foundation for

8 Penitentes have been suggested as an explanation for anomalous radar data on Europa, but unti

9 However, a purely neurotransmitter-based explanation for antidepressant drug action is challenged

10 , and these interactions provide a molecular explanation for BCAP-mediated down-regulation of inflamm

11 ecies are now being identified, providing an explanation for bias in gene expression between subgenom

12 imensionality provides a general theoretical explanation for biodiversity-more niches, defined by mor

13 Our results provide a mechanistic explanation for both the cellular effects and in vivo ph

14 To evaluate possible explanations for changes, these trends were compared to

15 This cancer stem cell model offers an explanation for chemotherapy resistance and disease rela

16 of Cin8 identified here provides a potential explanation for Cin8 clustering along microtubules and p

17 The results provide an explanation for classic laws of psychophysics and their

18 No alternative explanation for clinical hemolysis was found.

19 This provides a potential explanation for clinical manifestations of melioidosis t

20 switch events may have been neglected as an explanation for complex mutations because of biases in c

21 information processing and offer a unifying explanation for contextual phenomena in fields ranging f

22 and unlearning does not provide a compelling explanation for current data.

23 ich have been previously used as mechanistic explanations for decision-making processes.

24 viral innate immunity provides a long-sought explanation for delayed activities of respective genes.

25 ous and crystalline phases provides a direct explanation for different physical properties and unders

26 function, and provide a potential molecular explanation for DPM1-related disease.

27 A molecular spectroscopic explanation for dual fluorescing peaks in amino acids an

28 Our findings provide an explanation for EgWRI1 as an important gene contributing

29 The results indicate a unified explanation for environmental drivers of influenza that

30 However, alternative explanations, for example, that repetitive motor perform

31 n together, our findings offer a mechanistic explanation for exercise-dependent suppression of breast

32 iferation of smooth chorion CTBs, a possible explanation for expansion of this layer, which may parti

33 computational model provides a satisfactory explanation for experimental observations, clearly defin

34 pply of electron donor is the most plausible explanation for failure of dechlorination.

35 rom CIZ1-null animals, suggesting a possible explanation for female-specific lymphoproliferative diso

36 atitude when testing alternative mechanistic explanations for geographic variation in diversification

37 b CMF/USH1C PDZ complex provides mechanistic explanations for >20 deafness-causing mutations in Myo7a

38 genic mice, providing a valuable mechanistic explanation for HCV-induced diabetes in humans.

39 Our data provide an explanation for high levels of IFN production and immune

40 icle proposes the insurance hypothesis as an explanation for higher levels of obesity among food-inse

41 on-condensation dynamics creates mechanistic explanation for how cells properly respond to variable l

42 We believe that our data provide a molecular explanation for how circadian phases, such as wake-sleep

43 is finding is that it provides a mechanistic explanation for how different types (strains) of rhinovi

44 ia throughout the mouse lifespan provides an explanation for how microglial priming early in life can

45 Our model provides a mechanistic explanation for how nucleotides regulate the p97-ataxin3

46 Our findings provide an elegant molecular explanation for how PAS sequences are recognized for mRN

47 ses lamina dynamics, providing a mechanistic explanation for how PKC activity influences nuclear size

48 r protease recognition provides a compelling explanation for how the oxidation-induced conformational

49 These data provide a mechanistic explanation for how the SUMOylation status of Drp1 acts

50 , the 'ABC model' is a simple and satisfying explanation for how this conserved floral architecture i

51 neage specification, and suggest mechanistic explanations for how CIC mutations may impact the pathog

52 alterations in these mechanisms provide one explanation for human chronotype.

53 ression and DNM3OS amplification provides an explanation for increased DNM3OS levels.

54 As a mechanistic explanation for inhibition-dependent increases in freque

55 To pursue metabolic explanations for leaf RN variation, parallel metabolite

56 Our findings provide the first mechanistic explanation for left-hemispheric lateralization of human

57 try, and signaling; and provides a molecular explanation for LSD's actions at human serotonin recepto

58 These data provide a partial explanation for LV leakage defects and potentially other

59 herrerasaurids and theropods, and offers an explanation for many of the anatomical features previous

60 en forward and epi-generated SHG provides an explanation for many of the wavelength-dependency discre

61 Our model provides a simple mechanistic explanation for many previously enigmatic features of eu

62 assess whether recently proposed mechanistic explanations for microcephaly are supported by molecular

63 iated PP2A inhibition is a novel mechanistic explanation for multikinase inhibitor resistance in glio

64 We thereby provide an explanation for multiple HLA associations with cutaneous

65 These findings rule out alternative explanations for neural entrainment and provide evidence

66 This model provides a simple explanation for non-canonical methylation patterns at so

67 This refractory period provides a simple explanation for not only the spring-summer timing of his

68 In evaluating the insurance hypothesis as an explanation for obesity, we propose one missing piece of

69 spread of resistance alleles is the primary explanation for observations of reduced pesticide effica

70 findings provide a physiologically plausible explanation for observed changes in cognitive performanc

71 ATION: Racial assortativity is an inadequate explanation for observed disparities.

72 A third study ruled out an alternative explanation for our findings.

73 een PGRN and cathepsin D provides a possible explanation for overlapping NCL-like pathology observed

74 hogenic lineages and provides an alternative explanation for pathogen transmission patterns.

75 channel activation, providing a mechanistic explanation for phenotypic outcomes.

76 l populations, providing a novel alternative explanation for physiological differences and earlier br

77 ermine individual risk, to derive a coherent explanation for population patterns in cardiovascular di

78 roglia and neurons), we offer an alternative explanation for previous data, which assumedly support t

79 Our findings provide a mechanistic explanation for previous observations that dimerization-

80 of E2F1 in SmgGDS-depleted cells provides an explanation for previous reports that SmgGDS depletion c

81 T recycling mechanism and provide a unifying explanation for previous, seemingly disparate, DAT endoc

82 Explanations for primate brain expansion and the evoluti

83 represent a disease-driving mechanism and an explanation for RAG off-target cleavage observed in huma

84 variation of the target loci may be another explanation for read abundance differences between taxa,

85 alternative scenarios representing different explanations for recent declines in TB incidence.

86 es has often been invoked as the most likely explanation for reduced fitness, and most studies to dat

87 t model, providing a plausible atomic-detail explanation for seam-to-lateral contact transition that

88 m the skin or cornea, suggesting a potential explanation for selectivity to migraine headache, but no

89 cycle but also provide potential mechanical explanations for several well-established and recently d

90 rfere with enzyme function, and a compelling explanation for severe Hunter syndrome phenotypes.

91 Kin selection theory, as an explanation for social behavior, can benefit from much g

92 lifetime and could represent an alternative explanation for some forms of pathologic scarring that a

93 e DOCK8-deficient CD4(+) T cells provided an explanation for some of the clinical features of this di

94 s homeostasis of CPCs, providing mechanistic explanations for some nongenetic causes of CHD.

95 measured at baseline, were also examined as explanations for subsequent group disparities.

96 , outgassing) provide testable and plausible explanations for sustained deviations from the long-term

97 stages of invasion, suggesting a mechanistic explanation for synergy.

98 A mechanistic explanation for tDCS should capture these spatiotemporal

99 What is the biological explanation for the "imprecision?" First, Precision Onco

100 hemokine interaction with CCR5, providing an explanation for the ability of CCR5 to recognize diverse

101 Our results provide a direct molecular explanation for the ability of EGCG to disrupt P. aerugi

102 These findings provide a mechanistic explanation for the ability of lncRNAs to regulate the e

103 This study provides an explanation for the anti-infective activity of peppertre

104 These findings provide an explanation for the apparently contradictory observation

105 More broadly, the results provide a physical explanation for the ATP-gated behavior of SF2 RNA helica

106 These results provide a mechanistic explanation for the behavioural changes in hosts infecte

107 This provides a potential explanation for the capacity of this asexual snail to sp

108 yperplasia, we now provide a full structural explanation for the clinical severity of AME resulting f

109 dysregulation by MYD88(L265P) and provide an explanation for the co-occurrence of MYD88 and CD79B mut

110 geneity in duration of carriage is a partial explanation for the coexistence of sensitive and resista

111 anding protein penetration depth provides an explanation for the composition of the protein corona, a

112 s to accommodate LSD, providing a structural explanation for the conformational selectivity of LSD's

113 There appears to be no persuasive explanation for the considerable number of species with

114 This would provide a mechanistic explanation for the constraint between offspring quality

115 One explanation for the contrasting views among studies is t

116 In conclusion, we provide a biochemical explanation for the cooperation between p16 inactivation

117 we have found an electrophysiological-level explanation for the correlation among domain preference,

118 The insurance hypothesis is a reasonable explanation for the current obesity epidemic.

119 d at Charon's winter pole was proposed as an explanation for the dark coloration on the basis of an i

120 However, an alternative explanation for the data are that individuals vary in th

121 the RNA/DNA helix, providing an alternative explanation for the data of O'Brien et al.

122 a higher staining intensity are a plausible explanation for the difference in allergic symptom sever

123 eatures of the strong binding and offered an explanation for the differences in inhibitory potency be

124 in pollinators than in herbivores may be an explanation for the differences in learning seen between

125 busulfan disposition provided no mechanistic explanation for the differences in mortality.

126 ty were consistent with biodegradation as an explanation for the differences.

127 make several predictions, and to propose an explanation for the different behavior of the transition

128 pared with fibroblasts, offering a potential explanation for the different calcium content in tumor c

129 We propose a novel explanation for the discrepancy between isotope ratios o

130 Haploinsufficiency is a popular explanation for the disease, in part because most of the

131 These results offer a molecular explanation for the distinctive ability of spiperone and

132 iferromagnetic order, and provides a natural explanation for the distinctive hourglass shape of the m

133 This observation offers an explanation for the dominant effects of DYNC1H1 mutation

134 C assembly and function provides a plausible explanation for the efficient replication of EEEV and ma

135 These findings provide an image-based explanation for the emergence of higher-level representa

136 rthermore, our results provide a mechanistic explanation for the empirical finding that glucocorticoi

137 These findings offer an explanation for the enhanced virulence of CDT-expressing

138 This finding provides a potential explanation for the ephemeral nature of clinical desensi

139 In addition to providing a possible explanation for the evolution of crosstalk, our work ind

140 cultural intelligence hypothesis is the best explanation for the evolution of general intelligence.

141 The new data present a plausible explanation for the exceptionally high replication rates

142 Our results provide a simple explanation for the existence of salt-and-pepper arrange

143 The model also provides a quantitative explanation for the experimental finding that a mutant a

144 An often-cited explanation for the failure of anti-Abeta mAb trials is

145 Our findings provide a possible explanation for the failures of anti-EGFR therapy in GBM

146 ain positive symptoms and provides a unified explanation for the following experimental findings in s

147 lain negative symptoms and provide a unified explanation for the following experimental findings in s

148 d anterograde forces on the MT, providing an explanation for the frequent pauses of short MTs and the

149 Here, we provide a molecular explanation for the functional divergence of structurall

150 The classical explanation for the gating compliance is that the confor

151 metabolism and detoxification and a possible explanation for the high Km values that have been noted

152 This result provides a mechanistic explanation for the hypomorphic phenotype of the CTR-del

153 more, this phenomenon suggests a mechanistic explanation for the idea that early tuning of the immune

154 r to that of control rats, which provides an explanation for the improved BSEP activity.

155 ce insulin-resistance and provide a possible explanation for the improvement of insulin resistance/di

156 immune and antiviral defenses and provide an explanation for the incidence of influenza infections ev

157 Our results provide a potential explanation for the inconsistent associations between sm

158 s severely impaired, providing a mechanistic explanation for the increased mutation rate in the POLD1

159 bands in B6O, provide a plausible atomistic explanation for the influence of nanotwins on the deform

160 nds, our results also provide a parsimonious explanation for the influence of wounds on leaf dorsoven

161 These kinetic studies suggest a biochemical explanation for the interplay between PRMT5- and PRMT7-m

162 xpression and provide a putative mechanistic explanation for the Koebner phenomenon in psoriasis.

163 rs, previously predicted in silico, offer an explanation for the lack of antiviral activity of compou

164 One explanation for the lack of movement is that motor corti

165 agen has an inhibitory role that provides an explanation for the lack of thrombosis in patients with

166 The results provide an explanation for the large number of cases of meningococc

167 Our results offer an explanation for the limited convergence observed between

168 eveal a novel function of RUNX1 and offer an explanation for the link between RUNX1 mutations and che

169 y cross-seeding Abeta, providing a molecular explanation for the link between these diseases.

170 on of the Igh locus and provides a plausible explanation for the link with 53BP1 enforcement of delet

171 ved in heart tissues and provided a possible explanation for the low level of RNA replication observe

172 Our results provide an explanation for the lysosomal origin of AA amyloidosis.

173 region and their properties would provide an explanation for the many enigmatic seismic features that

174 upus with other CNS diseases and provides an explanation for the neurological symptoms observed in so

175 ve significant effects, perhaps providing an explanation for the numerous changes seen in cancer.

176 ectively our study offers possible molecular explanation for the observable phenotypic differences be

177 They provide a mechanistic explanation for the observation of nonsynonymous SNPs in

178 Our findings also provide a molecular-level explanation for the observation that synaptotagmin and o

179 catalyzed kinetics, provides a thermodynamic explanation for the observed departure and the convergen

180 n (influence of mood on intake) as potential explanation for the observed linkage.

181 Our study reveals a possible explanation for the observed pattern and highlights the

182 Our results offer a novel explanation for the observed preponderance of dominant s

183 A more plausible explanation for the observed significant change in senso

184 These findings provide a molecular explanation for the observed structural heterogeneity of

185 However, their simple model lacks an explanation for the origin of sulfur-depleted organic ma

186 A convincing explanation for the origin of the unusually old zircons

187 cancer cells, our work offers a mechanistic explanation for the paradoxical antitumorigenic roles of

188 ions, the present study provides a potential explanation for the paradoxical effects of CBZ on epilep

189 Although these factors provide an explanation for the pattern of neuronal loss in PD, they

190 This provides a potential explanation for the phenomenon that social insect coloni

191 drug-binding pocket, providing a mechanistic explanation for the polyspecificity of P-gp in substrate

192 n the CNS, and stress the need for no better explanation for the presentation.

193 te for allosteric inhibitors and a molecular explanation for the previously baffling substrate-inhibi

194 Thus, our findings provide mechanistic explanation for the protumorigenic potentials of FTY720

195 refrontal cortex appears to be one important explanation for the puzzling question of why addicted in

196 to human mast cells and offer a mechanistic explanation for the reduced anaphylactic activity.

197 Together, the data provide a new explanation for the reliance of neurotransmitter recepto

198 our analysis, the mathematically most likely explanation for the renewed growth in atmospheric methan

199 Our observations provide an explanation for the reported high rates of sequence dive

200 pression profiling studies offer a molecular explanation for the resolution of palatal defects, showi

201 (IDH) is well-known in ecology providing an explanation for the role of disturbance in the coexisten

202 ndent protein kinase A allow for a molecular explanation for the role of protein conformational entro

203 This finding provides an explanation for the seemingly very different peptide bin

204 ur model for T5 also provides an algorithmic explanation for the selectivity of T5 for moving dark ed

205 This model may finally offer a mechanistic explanation for the striking mass-independent fractionat

206 tive (ts) phenotype, providing a most likely explanation for the stronger attenuation observed.

207 s unique binding mode provides a mechanistic explanation for the structure-activity relationship data

208 and IBS symptomatology, and might provide an explanation for the success of LFM diet in IBS patients.

209 A possible explanation for the sustained neuronal injury is that th

210 e a locus of interest and provide a succinct explanation for the transmission of the trait locus.

211 rrounding environment, providing a potential explanation for the variation in biofilm matrix secretio

212 es in zika tropism, which offers a potential explanation for the variety of zika-associated symptoms.

213 hing OFF of the thick filament, providing an explanation for the very low rate of ATP utilization dur

214 s of study there is a lack of a quantitative explanation for the Warburg effect in cancer.

215 gene, which will in turn enable alternative explanations for the association between incompatibility

216 e trans-eQTLs point toward novel mechanistic explanations for the association of the SNP with the GWA

217 evolutionary and taste-based and stereotype explanations for the beauty premium, have too many confo

218 Analysis of the WGS data provided explanations for the discordant results between TB&S and

219 This study provides explanations for the discrepancies to clarify the relati

220 Proposed explanations for the discrepancy include ocean-atmospher

221 ntific effort to find the pathophysiological explanations for the disease, the neurobiological links

222 ow the highest rates among blacks, where the explanations for the disparity are not as obvious and ar

223 evidence, whereas others propose alternative explanations for the evidence we reviewed and suggest wa

224 t article falls short of offering compelling explanations for the evolution of intelligence.

225 Explanations for the improved motor function during RBD

226 Moreover, the biological mechanistic explanations for the observed differences are poorly und

227 In this Personal View, we discuss possible explanations for the observed reduced effectiveness of t

228 nded as a biophysical discipline that sought explanations for the origins of life from chemical and p

229 as are not this low, arguing for alternative explanations for the oxygenation of the atmosphere.

230 etween adaptive models of storing energy and explanations for the pathological metabolic state of obe

231 ucted follow-up analyses to test alternative explanations for the pay gap, examining earnings for nur

232 In this review, we consider explanations for the remarkable stability of the latent

233 icular dysfunction and investigate potential explanations for the superiority of edoxaban-ie, differe

234 Quantitative analysis ruled out a posteriori explanations for the vesicles with low release probabili

235 tudies and provide an overarching conceptual explanation for their findings.

236 Fritz et al. provide a potential explanation for these clinical observations by examining

237 One possible explanation for these deficits is loss of myelin, creati

238 A likely explanation for these findings is that preterm birth imp

239 ng internal models provides a novel unifying explanation for these findings.

240 An explanation for these monodominant forests remains elusi

241 his perspective offers the most parsimonious explanation for these mysterious diseases.

242 The possible explanation for these observations is that papaya pectin

243 he possible genetic and ancestral hereditary explanations for these associations are confounded by so

244 ften provide simple physical and geometrical explanations for these associations in regards to substr

245 The explanations for these effects are elusive.

246 Potential explanations for these elevated rates and the heterogene

247 rdiovascular risk factors are the prevailing explanations for these findings, there are data showing

248 While this pattern is robust, theoretical explanations for these large differences in aggression a

249 Explanations for these persistent disparities in cardiov

250 Informative explanations for these potentially contradictory associa

251 e history theory (LHT) framework provided an explanation for this association.

252 However, the mechanistic explanation for this correlation remains elusive, and an

253 One explanation for this difference is recycling of material

254 Studies reported here provide a mechanistic explanation for this differential response by identifyin

255 In this study, we provide a theoretical explanation for this discrepancy between previous theore

256 of PQ on gametocyte sex ratio as a possible explanation for this early sterilizing effect.

257 The canonical explanation for this effect is that frames differentiall

258 affect stem cell state, but the mechanistic explanation for this effect is unknown.

259 Although we could not provide a conclusive explanation for this excess of significant findings, our

260 ange, P < .001) which provides a mechanistic explanation for this feature.

261 The most likely explanation for this finding is the persistence of HRP2

262 ession in neovasculature provides a possible explanation for this finding; however, other contributin

263 Here, we test one possible explanation for this gap, motivated by developmental res

264 A possible explanation for this is formation of a supramolecular sy

265 oacoustic hypothesis provides an alternative explanation for this longstanding mystery about generati

266 We provide an explanation for this observation by demonstrating that d

267 One explanation for this paradox is that current therapies a

268 in the electrolyte; however, a satisfactory explanation for this phenomenon has not been developed.

269 Our explanation for this phenomenon is that organic foulants

270 bic environment, the Warburg effect, but the explanation for this preference is not clear.

271 ical simulation provide a plausible physical explanation for this result and support a picture in whi

272 One explanation for this size bias in migration is that larg

273 We suggest a novel explanation for this trade-off.

274 Two classes of explanation for this unusual requirement are (1) recycli

275 The explanation for this wide difference in activity against

276 ature and phenology, and we examine possible explanations for this countergradient pattern, including

277 Possible explanations for this discrepancy could include influenc

278 ot predicted by most previously hypothesised explanations for this gradient.

279 Possible explanations for this patient's condition, include immun

280 nt species Canarina eminii to explore causal explanations for this pattern.

281 al modeling has sought ultimate evolutionary explanations for this seemingly altruistic behavior.

282 constants of dsDNA and dsRNA and provide an explanation for three striking differences in the mechan

283 Many explanations for TLD have been advanced, with recent eff

284 in the absence of Hh signaling, providing an explanation for tumor development in CJS.

285 ciated with the menstrual cycle are possible explanations for variable infection outcomes.

286 Our work provides a general explanation for variation in observed diversity-ecosyste

287 population provides a potential mechanistic explanation for vision compromise in RPD compared with o

288 One possible explanation for what seems to be mutually exclusive find

289 are imposed on mate search, and provides an explanation for why Allee effects are often observed in

290 We provide a physical explanation for why allostery is related to dihedral com

291 ogenic model discussed here also provides an explanation for why angioedema can occur at multiple sit

292 Our model provides a molecular explanation for why certain mutations of Mettl3 and Mett

293 eakest Allee effects and may thus provide an explanation for why extinctions due to Allee effects are

294 Our suggested explanation for why individuals report food insecurity i

295 This independence provides an explanation for why reaction times are usually so sluggi

296 Our results provide a more mechanistic explanation for why sexual selection appears to drive ea

297 protein stability in cells and provide a new explanation for why stressed cells accumulate osmolytes.

298 It also provides a plausible general explanation for why swimming cells tend to have strong a

299 ppressed in HPV-positive cells, providing an explanation for why this microRNA is targeted in HPV-pos

300 Mechanistic explanations for why antibiotics differ in their optimal