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1 AG-containing membranes provides a molecular explanation for why 1) DAG alone is sufficient to activa
2 sponse and tandem duplication and provide an explanation for why a large proportion of the RLK/Pelle
3 ecognized by MAb 2158 and offer a structural explanation for why a mismatched mutation at position 18
5 oxyanion hole becomes smaller, providing one explanation for why acylation may be less efficient foll
7 the water pulse and provides an alternative explanation for why ALD growth rates for this system dec
8 are imposed on mate search, and provides an explanation for why Allee effects are often observed in
10 ncapsulation mechanism provides a convincing explanation for why almonds have a low metabolizable ene
12 idity models can be rejected or supported as explanations for why and how these three disorders overl
13 ogenic model discussed here also provides an explanation for why angioedema can occur at multiple sit
17 al malaria, and the results offer a possible explanation for why artemether provides less advantage t
19 ate our hypothesis, also suggest a potential explanation for why axonal microtubules deteriorate in n
20 nsing (DS), have been suggested as competing explanations for why bacterial cells use the local conce
21 l Niche Conservatism hypothesis is a leading explanation for why biodiversity increases towards the e
23 targets with >100 muM affinity, offering an explanation for why both MRG15 CD and Pf1 PHD1 domains a
29 are large aggregates, suggesting a possible explanation for why disease pathology does not always co
31 particularly in the short term, may offer an explanation for why domesticators and breeders have real
32 n and its antiapoptotic function provides an explanation for why eIF2alpha kinase deficiency in disea
33 er modeling of chromosomes, and suggests new explanations for why elucidating the functional signific
34 ted HePTP/PP2A activity provides a molecular explanation for why ERK activity is sensitive to membran
35 oms as adaptive fail to provide parsimonious explanations for why even mild depressive symptoms impai
36 transplanted animals, providing a potential explanation for why exogenous IL-7 did not increase GVHD
37 eakest Allee effects and may thus provide an explanation for why extinctions due to Allee effects are
38 NA damage induced by HPV16 E7, providing one explanation for why FA patients are predisposed to HPV-a
40 n levels in fluorosed enamel and provides an explanation for why fluorosed enamel has a higher than n
41 sm in gammadelta T cells but also provide an explanation for why gammadelta T cells are less dependen
42 ences in these contacts provide a structural explanation for why GluR2 L483Y and GluR3 L507Y are nond
44 m the target lysine and provide a structural explanation for why H3T3 phosphorylation and H3K9 acetyl
50 eptor after loss of sex steroids provides an explanation for why IL-6 is important for skeletal homeo
51 onses illustrate the need for a more unified explanation for why immunity is compromised in neonates.
53 existing literature and posits epidemiologic explanations for why investigators might have failed to
54 fication of gas5 as a 5'TOP gene provides an explanation for why it is a growth arrest specific trans
57 r initiation provide a potential mechanistic explanation for why long-term use of low doses of NSAIDs
58 hat of complete MMR deficiency, providing an explanation for why loss of heterozygosity is not requir
59 results suggest a physiologically plausible explanation for why LTD induction is experimentally diff
60 Our model offers a quantitatively robust explanation for why membrane bioenergetics are universal
61 ogen and its receptor on HBV thus provide an explanation for why men have a higher risk of HBV infect
62 nterkinetochore tension, thereby offering an explanation for why MI in mammals is so error-prone.
66 ns' metabolic theory provides a paradigmatic explanation for why most large-effect mutations are rece
67 nctions at middle promoters, and provides an explanation for why MotA can form non-specific multimers
69 cale orientation map provides a parsimonious explanation for why multivariate pattern analysis method
70 rate of GP Ibalpha degradation, providing an explanation for why mutation of their genes leads to def
71 lap in skeletal muscle and CNS (providing an explanation for why mutation of ZEB-1 alone has little e
72 protection function of BRCA1, and provide an explanation for why mutations within the BRCA1 RING doma
73 mputational analyses also provide a possible explanation for why N-debenzoyl-2'-deoxy-PTX is inactive
75 d-selective region in vOT while providing an explanation for why other studies have found a lack of w
76 decay of the mRNA body, and this provides an explanation for why p38 MAPK regulates mRNA stability in
77 th VEGF-A upregulation offered a mechanistic explanation for why patients exhibit improved outcomes a
78 Hebbian synaptic plasticity and a functional explanation for why place cells become directionally sel
79 are localized in the chloroplast, a possible explanation for why plants have a larger immunophilin fa
80 In particular, the model provides a simple explanation for why PPC encodes reach targets in referen
81 ugh not without flaws, the model provides an explanation for why primary plastids have evolved so rar
83 IFN-gamma-induced apoptosis is not a viable explanation for why RB expression rescues DRB inducibili
85 nderlie viral evolution and provide a likely explanation for why relatively few types of plant DNA vi
88 asC and rivR mutations do not provide a full explanation for why serotype M3 strains are associated w
92 of the mechanism of dissociation provides an explanation for why small molecules that bind at the AB/
93 ocks Mrc1 loading onto origins, providing an explanation for why so many mutants in DNA replication s
94 salt, temperature or osmolarity, but we lack explanations for why so many antiporters are needed and
95 ghts from this study may provide a plausible explanation for why some biogenic carbonates and carbona
98 diated by hER-alpha36 provide an alternative explanation for why some human breast cancers are resist
99 uccess as a breeder, offering one compelling explanation for why some individuals delay reproduction
102 protein stability in cells and provide a new explanation for why stressed cells accumulate osmolytes.
104 e for G4DNA provides an additional molecular explanation for why such sequences are prevalent in the
107 for transcriptional activity, providing one explanation for why synergy between these factors is imp
108 large and fast movements provide a possible explanation for why the conduction pathways of a wide ra
109 ts microtubule shortening, thus providing an explanation for why the drug can counteract the phenotyp
110 lity to neutralizing antibody, suggesting an explanation for why the elicited neutralization was not
111 erlapping binding sites provides a potential explanation for why the G/C-rich Mad binding site consen
112 timulatory activity of chitosan, offering an explanation for why the largest particles were nearly de
113 ctural basis for LCR function and provide an explanation for why the LCR core regions are so extremel
114 pace than is the mature form and provides an explanation for why the leader peptide is removed from t
116 is fails to provide an ultimate evolutionary explanation for why the male germline would tolerate mor
119 Therefore, these results provide a simpler explanation for why the proTAME-induced mitotic arrest i
120 tors of neurogenesis and provides a possible explanation for why the Q/R editing process exists.
121 -term premorbid level, providing a potential explanation for why the risks and benefits of lowering b
123 Together, these studies provide a simple explanation for why the vast majority of BRCA2 mutants a
125 Results of the qualitative analysis suggests explanations for why the programme worked in some contex
126 cific T cells in Hu patients and provided an explanation for why their detection has been elusive.
127 n the primary tumor, providing a biochemical explanation for why their expression specifically suppre
128 The final section discusses several possible explanations for why there are often little or no conseq
129 acetylcholine receptor (nAChR) subunits, an explanation for why these currents get smaller is not av
130 ion by beta-Trcp, thus providing a molecular explanation for why these mutations cause beta-catenin a
131 quences in the human p53 gene, providing one explanation for why these sites are mutational hot spots
133 bow joints when attempting any movement, one explanation for why they were better at the 'reach up' t
135 ppressed in HPV-positive cells, providing an explanation for why this microRNA is targeted in HPV-pos
136 aerobic conditions and provides a reasonable explanation for why this mutant protein is functional un
139 growth factor receptor providing a possible explanation for why tumorigenesis was not altered after
141 rovenance of this complex fold and offers an explanation for why Wnts utilize both lipid- and protein
142 malian cardiomyocytes as well as a potential explanation for why zebrafish and newts, but not mammals
143 Zoep mutant phenotype, providing a plausible explanation for why Zoep genetically interacts with spt
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