ライフサイエンスコーパス: exposure to allergen

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 , preventing allergic airway disease upon re-exposure to allergen.

2 d AHR and lung eosinophilia after subsequent exposure to allergen.

3 CS, inducing allergic asthma after postnatal exposure to allergens.

4 xpected dendritic cell (DC) maturation after exposure to allergens.

5 e tissue recruitment of leucocytes following exposure to allergens.

6 ms by which MCM is reduced despite continued exposures to allergen.

7 is crucial for reducing MCM during prolonged exposures to allergen.

8 lowed by four intranasal sensitizations, and exposure to allergen aerosol 3 hours per day, 3 days per

9 s viral and other respiratory infections and exposure to allergens, air pollutants, and active or pas

10 These observations suggest that following exposure to allergen, airway T cells are functionally bu

11 Exposure to allergen alone, diesel exhaust alone, or all

12 g evidence that early life events, including exposure to allergen and infections, are critical in pro

13 acerbations are associated with the level of exposure to allergens and LPS.

14 assed parental history of allergy, potential exposure to allergens and stress, known to be associated

15 tivated intrapulmonarily after infections or exposures to allergen and C5 inhibition has profound eff

16 is an inflammatory disorder caused by airway exposures to allergens and chemical irritants.

17 o personal characteristics and environmental exposures to allergens and endotoxin and to the developm

18 nes, and the chemokines that are released on exposure to allergens, and the IgE pathway.

19 Respiratory exposure to allergens can lead to airway tolerance.

20 T-cell tolerance, induced by respiratory exposure to allergen, can inhibit the development of air

21 chronic airway inflammatory disease in which exposure to allergens causes intermittent attacks of bre

22 Exposure to allergens depends on multiple ecological str

23 ion is induced in airway epithelial cells by exposure to allergen-derived proteases and that PAR-2 is

24 bling, the authors examined the relations of exposure to allergens (dust mite, cockroach, cat, and do

25 atment with dexamethasone during respiratory exposure to allergen eliminated the development of IL-10

26 We sought to determine whether exposure to allergen, exposure to diesel exhaust (DE), o

27 Furthermore, after prolonged exposure to allergen, hyperplastic epithelial cells pers

28 erresponsiveness (AHR) after repeated airway exposure to allergen in sensitized mice.

29 ver, other cutaneous consequences of chronic exposure to allergens in implanted devices are not well

30 Moreover, animal models demonstrate that exposure to allergens in strongly sensitized mothers inf

31 Respiratory exposure to allergen induces T cell tolerance and protec

32 Respiratory exposure to allergen induces T cell tolerance and protec

33 epithelial cells (AEC), and during prolonged exposure to allergen, mucous cell hyperplasia remained e

34 tifactorial: the disease can be triggered by exposure to allergens or drugs, but a genetic background

35 mmatory or epithelial repair functions after exposure to allergens, pathogens, or chemical irritants.

36 provide the first evidence that epicutaneous exposure to allergens potently primes for EE via a Th2-d

37 Exposure to allergens produced by the German cockroach (

38 ominant feature, in human asthma, an initial exposure to allergen results in T(H)2 cell-dependent sti

39 Exposure to allergens, such as house dust mite (HDM), th

40 inflammatory flares resulting from repeated exposure to allergen that are a debilitating feature of

41 Re-exposure to allergen then activates mast cells, which re

42 nt factors, including repeated or persistent exposure to allergens, which can lead to Th2-cell expans

43 allergic subjects, persistent or repetitive exposure to allergens, which typically are intrinsically

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。