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1 s and civilian trauma victims with traumatic exsanguination.
2 a now do so as a result of causes other than exsanguination.
3 later period, as did the rate of deaths from exsanguination (9% to 1%), multiple organ failure (12% t
5 s appear to be in reducing early deaths from exsanguination and late deaths from multiple organ failu
6 ar to those of today, rapidly preventing any exsanguination and the breakdown of osmoregulation of th
8 the 1980s in dog outcome models of prolonged exsanguination cardiac arrest has culminated in brain an
9 ation (EPR) of trauma victims who experience exsanguination cardiac arrest may allow survival from ot
11 have developed and used novel dog models of exsanguination cardiac arrest to explore suspended anima
13 ats showing no neurologic injury, despite an exsanguination cardiac arrest, followed by 20 mins of EP
14 f cold saline immediately after the start of exsanguination cardiac arrest-which rarely can be resusc
17 decreased to 40 mm Hg during 60 minutes with exsanguination from the 30th to the 60th minute to a mea
26 ontraumatic (e.g., ruptured aortic aneurysm) exsanguination, without severe brain trauma, by enabling
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