ライフサイエンスコーパス: external genitalia

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1 lved in the normal development of gonads and external genitalia.

2 sinus, and clitoral hypertrophy or ambiguous external genitalia.

3 he developmental and evolutionary origins of external genitalia.

4 ation, outgrowth or normal patterning of the external genitalia.

5 es showed strong contrast enhancement of the external genitalia.

6 normal development of the male internal and external genitalia.

7 es, except that males and females had female external genitalia.

8 eam targets of the androgen receptor (AR) in external genitalia.

9 as 6.7% in the oral cavity and 16.9% for the external genitalia.

10 nd dominate them in social encounters; their external genitalia also are highly masculinized.

11 ions for the evolutionary diversification of external genitalia and for the association between exter

12 Shh knockout mice lack external genitalia and have a persistent cloaca.

13 tissue layers and cell types within the LUT, external genitalia and lower reproductive structures.

14 (Ihh) as a novel downstream target of AR in external genitalia and show that conditional deletion of

15 e at 9 weeks of age displayed underdeveloped external genitalia and uteri.

16 ng, urorectal septation, and modeling of the external genitalia; and internally, the emergence of bas

17 Male and female external genitalia appear identical early in gestation.

18 Malformations of the external genitalia are among the most common congenital

19 External genitalia are body appendages specialized for i

20 The external genitalia are some of the most rapidly evolving

21 support a limb-like developmental origin of external genitalia as the ancestral condition.

22 gene cause incomplete masculinization of the external genitalia by disrupting AR function in males wi

23 In mammalian embryos, male and female external genitalia develop from the genital tubercle.

24 humans, the genetic pathways governing early external genitalia development and urethra formation are

25 s study, we show that inactivation of Shh in external genitalia extends the cell cycle from 8.5 to 14

26 ere we show that the developmental origin of external genitalia has shifted through evolution, and in

27 Development of external genitalia in mammalian embryos requires tight c

28 as led to an altered developmental route for external genitalia in mammals, while preserving parts of

29 egulated during embryonic development of the external genitalia in mice, and that this regulation is

30 gnaling, during embryonic development of the external genitalia in mice.

31 The size of external genitalia increased in both sexes, but they rem

32 We propose that induction of external genitalia involves an epithelial-epithelial int

33 Development of the mammalian external genitalia is controlled by a network of signali

34 wever, the origin of cells that give rise to external genitalia is unknown.

35 Although congenital external genitalia malformations represent the second mo

36 Circulating levels of testosterone, external genitalia, or fertility were not altered in pes

37 Hypospadias is a congenital defect of the external genitalia that results from failure of urethral

38 In the early phase, the embryonic anlage of external genitalia, the genital tubercle (GT), is morpho

39 limbs and an altered Hoxd gene regulation in external genitalia, the limb-associated bimodal HoxD chr

40 nal cord conveying information from the male external genitalia to MRF, and (2) ascending bilateral p

41 Serial MR imaging of the external genitalia was performed in 12 healthy sexually

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