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1                                  Significant extracardiac abnormalities were noted.
2 complication of heterogeneous background and extracardiac activity adjacent to the heart, which cause
3                              Corrections for extracardiac activity and partial-volume errors were per
4 n correction, which depends on the amount of extracardiac activity and pattern of attenuation.
5 of attenuation correction in the presence of extracardiac activity can have complex effects on ML rec
6                                    Increased extracardiac activity confounds conventional cardiac SPE
7 improvement of correlation was observed with extracardiac activity correction (R(2) = 0.85, y = 1.54x
8 uce a heuristic method for the correction of extracardiac activity into SPECT quantification and vali
9 also result in reconstruction artifacts when extracardiac activity is present.
10 orrections for the myocardial tissue weight, extracardiac activity, and partial-volume errors are cru
11  improved uniformity only in the presence of extracardiac activity.
12 s diminished with the addition of increasing extracardiac activity.
13 d less likely be affected by the presence of extracardiac activity.
14 l approaches, in the absence and presence of extracardiac activity.
15 and not for increased myocardial counts from extracardiac activity.
16 ere well without evidence of intracardiac or extracardiac amyloid accumulation, and median overall su
17 ains difficult because of the high number of extracardiac anomalies and chromosome defects in this gr
18 t, gestational age, maternal race/ethnicity, extracardiac anomalies, sex, and maternal age and educat
19 es without a documented chromosomal or major extracardiac anomaly, 13 (48%) died.
20 e an atrial septal defect through the use of extracardiac application of histotripsy in an open-chest
21 o, or more vs. none, odds ratio 1.75, 7.98), extracardiac arteriopathy (odds ratio 2.63), preoperativ
22  data indicate significant remodeling of the extracardiac autonomic nerve activity and structures aft
23                                 Infants with extracardiac birth defects or genetic disorders were exc
24                     Compared with those with extracardiac cancers of similar histopathology, patients
25 vide evidence for the involvement of another extracardiac cell population, the proepicardial cells.
26 l new model system for examining the role of extracardiac cell populations in cardiac morphogenesis a
27 is independent of the further recruitment of extracardiac cells from the secondary heart field and pe
28                          The contribution of extracardiac cells to atrial septation has recently been
29                 However, the contribution of extracardiac cells to CoE is thought to be minor and non
30                                    In HFpEF, extracardiac comorbidities such as metabolic risk, arter
31                                   FALD is an extracardiac complication that may lead to substantial c
32 essment of this complex scenario to rule out extracardiac complications and possible neoplasms.
33 nifestations had a higher rate of IE-related extracardiac complications than patients without skin ma
34 s and should alert physicians to examine for extracardiac complications, notably with cerebral imagin
35 potent neural crest cells (NCCs) are a major extracardiac component of cardiovascular development.
36 eral tunnel (LT) was performed in 92% and an extracardiac conduit (EC) in 8%.
37 during this period, only 6 (33%) required an extracardiac conduit as part of their complete repair.
38                                The use of an extracardiac conduit can be avoided in the majority of p
39 the need for, and use of, fenestration of an extracardiac conduit Fontan.
40 , should not be performed routinely with the extracardiac conduit Fontan.
41 ith minimal intervention in patients with an extracardiac conduit Fontan.
42 ot necessary in most Fontan patients when an extracardiac conduit technique is performed as described
43 5), 271 lateral tunnels (1988-2006), and 532 extracardiac conduits (1997-2010).
44      Transcatheter procedures for those with extracardiac conduits (extracardiac-total cavopulmonary
45 ently predicted worse survival compared with extracardiac conduits (hazard ratio, 6.2; P<0.001; 95% C
46 opulmonary connections, 7 lateral tunnels, 2 extracardiac conduits).
47 th, transplantation, takedown, conversion to extracardiac conduits, New York Heart Association III/IV
48 erval [CI], 94%-99%) for lateral tunnels and extracardiac conduits.
49 HD) patients have an increased prevalence of extracardiac congenital anomalies (CAs) and risk of neur
50     There is a functional, ischemia-reducing extracardiac coronary artery supply via ipsilateral but
51                              The presence of extracardiac defects increased the adjusted risk of deat
52 y CCHD type and study site, and infants with extracardiac defects were significantly less likely to h
53 e the functions of Csx/Nkx2.5 in cardiac and extracardiac development in the vertebrate, we have gene
54 ee consecutive patients with a biopsy-proven extracardiac diagnosis of systemic sarcoidosis (21 men;
55 r and have worse survival than patients with extracardiac disease of similar histopathology (P<0.001)
56 zed sham-controlled study to investigate the extracardiac effects of EECP on peripheral artery flow-m
57 pmental abnormalities may be attributable to extracardiac effects of GRK2 ablation.
58 oth conferred an increased risk for nonfatal extracardiac events (hazard ratio of 1.52 per 1 SD [P <
59 al transgene expression and pose the risk of extracardiac expression.
60                                              Extracardiac factors (e.g., obesity and diabetes) influe
61 y present, increasing evidence suggests that extracardiac factors such as renal dysfunction and enhan
62                      These data suggest that extracardiac factors, via volume overload, may contribut
63 t in a similar cardiac phenotype but lack in extracardiac features of the syndrome, suggesting that a
64        None of the 3 patients presented with extracardiac features suggestive of AFC.
65 nd of operation, intracardiac Fontan (IF) or extracardiac Fontan (EF), days with chest tube output pe
66  innovations, such as the lateral tunnel and extracardiac Fontan modifications, and fenestration, as
67             The utility of fenestration with extracardiac Fontan operation has not been determined.
68  connection to a total cavopulmonary artery, extracardiac Fontan; arrhythmia surgery, typically with
69 eptal defects were successfully created with extracardiac histotripsy in a live canine model.
70                                              Extracardiac imaging is highlighted as an increasingly i
71 fication of the Fontan procedure in which an extracardiac inferior cavopulmonary conduit is used in c
72  cardiac performance, cardiac pathology, and extracardiac intrathoracic abnormalities.
73 mmary artery device closure seems to augment extracardiac ipsilateral coronary supply to the effect o
74          There were 1 early death (1%) and 4 extracardiac late deaths.
75 surgical procedures were required to correct extracardiac lesions in 18 patients (75%).
76                                 Detection of extracardiac lesions is an essential component of the ma
77  42 pregnancies that continued, 15 had major extracardiac malformations and/or chromosomal abnormalit
78 f these patients with those of patients with extracardiac malignancies of similar histopathology.
79 lly not be discerned from the cardiac and/or extracardiac manifestations and requires molecular genet
80                 With some genetic mutations, extracardiac manifestations are likely to be present.
81                                         Such extracardiac manifestations, in addition to other late c
82 um, which influences its relationship to the extracardiac mediastinal mesoderm.
83 dge (the spina vestibuli) did not accumulate extracardiac mesoderm, nor did it undergo the pronounced
84  of the coronary vessels arise from a unique extracardiac mesothelial cell population, the proepicard
85 indings demonstrate multiphasic responses in extracardiac metabolism to pathogenic cardiac stress, wi
86 ar mortality and non-SCD but neither SCD nor extracardiac mortality in heart failure across the conti
87 plasia syndrome characterized by cardiac and extracardiac myxomas in the setting of spotty skin pigme
88 associated with cutaneous hyperpigmentation, extracardiac myxomas, and nonmyxomatous tumors.
89          Whether MI results in remodeling of extracardiac nerve activity remains unclear.
90                                           An extracardiac nerve trunk innervated the bulbus arteriosu
91 ine the electrophysiological consequences of extracardiac neuronal remodeling in humans.
92 h PD+OH or PAF have neuroimaging evidence of extracardiac noradrenergic denervation.
93                    Aortic homograft type and extracardiac operative technique predicted homograft fai
94 f aortic homograft in the older patient, and extracardiac operative technique.
95 ated; and whether there may be intracardiac, extracardiac, or intracranial neuropathological conditio
96 n circulatory properties are associated with extracardiac organ dysfunction, numerous gaps in our und
97 rophic cardiomyopathy because of the lack of extracardiac organ involvement.
98     However, the AAV genomes can be found in extracardiac organs after intramyocardial injection.
99 ine can visualize sympathetic innervation in extracardiac organs and, if so, whether patients with PD
100 inent in the heart but is also detectable in extracardiac organs.
101 nt studies have identified cardiomyocytes of extracardiac origin in transplanted human hearts, but th
102 tic areas containing fibroblasts of putative extracardiac origin.
103 uses multiple acknowledged SCD substrates of extracardiac origin: diabetes mellitus, hypercholesterol
104 ings highlight the importance of considering extracardiac pathogenesis when investigating arrhythmoge
105                  The presence of cardiac and extracardiac pathologic indicators was also determined.
106                              It helps detect extracardiac pathology leading to hypoxemia and may be u
107 y calcification were found to have important extracardiac pathology requiring additional work-up.
108 Of 1326 patients, 103 (7.8%) had significant extracardiac pathology requiring clinical or imaging fol
109                                              Extracardiac progenitor cells are capable of repopulatin
110                                              Extracardiac progenitor cells are capable of repopulatin
111  first time human bone marrow as a source of extracardiac progenitor cells capable of de novo cardiom
112 1-mm(2) "hot spots." Thus, adult humans have extracardiac progenitor cells capable of migrating to an
113  and further degradation of cardiac SPECT by extracardiac radioactivity and partial-volume effect.
114 ntified simultaneously with incorporation of extracardiac radioactivity correction, gaussian fitting,
115 xical embolization then may link cardiac and extracardiac right-to-left shunts to migraine aura.
116 oronary vessels and epicardium arise from an extracardiac rudiment called the proepicardium.
117 vascular magnetic resonance in patients with extracardiac sarcoidosis and preserved left ventricular
118 -one consecutive patients with biopsy-proven extracardiac sarcoidosis were prospectively recruited fo
119  left ventricular ejection fraction >50% and extracardiac sarcoidosis who underwent cardiovascular ma
120  coronary vascular development, we show that extracardiac septum transversum/proepicardium (ST/PE)-de
121 ization defects that appear to be induced by extracardiac signals.
122 r anomalous migration of epicardial cells to extracardiac sites.
123 her derived intracardially or immigrate from extracardiac sources.
124 ree of chimerism, averaging 24.3+/-8.2% from extracardiac sources.
125 , however, have suggested the presence of an extracardiac stem cell population, perhaps in bone marro
126 ic conditions, with little contribution from extracardiac stem cell sources.
127 ired reoperation to treat lead dislodgement, extracardiac stimulation, or infection during follow-up.
128 te and the common coexistence of cardiac and extracardiac stroke risk factors suggest benefits from g
129 CMRI allows clear delineation of cardiac and extracardiac structures as well as accurate and reproduc
130 detail its relationship to other cardiac and extracardiac structures.
131                          A patient-specific, extracardiac TCPC with 85% DS was studied in its origina
132 monstrated the formation of intracardiac and extracardiac teratomas.
133  and rAAV2/8 demonstrate similar behavior in extracardiac tissue.
134  exerts proinflammatory effects, at least in extracardiac tissues.
135 al databases were explored for patients with extracardiac-total cavopulmonary connection and postoper
136 ubstantial proportion of patients undergoing extracardiac-total cavopulmonary connection in childhood
137 edures for those with extracardiac conduits (extracardiac-total cavopulmonary connection) are perhaps
138                                 In addition, extracardiac tumor manifestations were detected in 4 pat
139 (+/-) mice exhibited a marked propensity for extracardiac tumorigenesis.
140              Algorithm failure was caused by extracardiac uptake (10/24, or 41.7%) or inaccurate iden
141  In 24 of 51 patients with IE, we also found extracardiac uptake, indicating septic embolism in 21 of
142 r prevalence of congestive heart failure and extracardiac vascular disease.
143 death, nonfatal cardiac events, and nonfatal extracardiac vascular events over a mean period of 7.8 y
144  expression in non pro-valve endocardium and extracardiac vasculature.

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