ライフサイエンスコーパス: extracellular fluid

1 may open and connect the cytoplasm with the extracellular fluid.

2 acterial exotoxins, GPI-linked proteins, and extracellular fluid.

3 ed by competition between LNAAs within brain extracellular fluid.

4 ession of CL-11 in iPS-RPE cells, and in the extracellular fluid.

5 d epitope-tagged prey, are released into the extracellular fluid.

6 need for compensatory Ca(2+) uptake from the extracellular fluid.

7 anes and connecting the vesicle lumen to the extracellular fluid.

8 showed that OPN protein is secreted into the extracellular fluid.

9 d by neuromodulatory substances in the brain extracellular fluid.

10 ntrations at which it is normally present in extracellular fluid.

11 ible for the removal of glutamate from brain extracellular fluid.

12 ntaining low glutamate concentrations in the extracellular fluid.

13 istributed between rehydrating cells and the extracellular fluid.

14 h effects, as neurons are kept in artificial extracellular fluid.

15 pore that connects the vesicle lumen to the extracellular fluid.

16 ed peptides derived from soluble proteins in extracellular fluid.

17 down their concentration gradients into the extracellular fluid.

18 tility and the resultant directional flow of extracellular fluid.

19 ding endosomes were still connected with the extracellular fluid.

20 tion and the net entry of magnesium into the extracellular fluid.

21 conductivities and permittivities than brain extracellular fluid.

22 id bilayers and separated by 1-mum layers of extracellular fluid.

23 nt balance, rather than net energy value, in extracellular fluid.

24 th an associated reduction of sMSLN in tumor extracellular fluid.

25 ,4-dihydroxyacetic acid (DOPAC) in the brain extracellular fluid.

26 lular infectious particles with release into extracellular fluids.

27 physiologic concentrations in the blood and extracellular fluids.

28 luble at the physiological ionic strength of extracellular fluids.

29 xpressing avrRpt2 in culture or in the plant extracellular fluids.

30 ith the release of FN fragments into cardiac extracellular fluids.

31 tions comparable to those found in serum and extracellular fluids.

32 ariation of bisulfide or hydrogen sulfide in extracellular fluids.

33 e dependent upon phenylalanine levels in the extracellular fluids.

34 e minimal evidence of direct interactions in extracellular fluids.

35 e, free, ionized Ca(2+) concentration in the extracellular fluids.

36 cking its synthesis or hydrolyzing it in the extracellular fluids.

37 lateral VMH microinjections of 1) artificial extracellular fluid, 2) B2AR agonist, or 3) B2AR antagon

38 orders of magnitude higher than that in the extracellular fluid (25 nM), as quantified by fluorescen

39 affibody, 2) control affibody, 3) artificial extracellular fluid, 4) insulin (50 microU), 5) insulin

40 into the cells triggered increased influx of extracellular fluid, a marker of macropinocytosis, and t

41 consistent with the hypothesis of intra- or extracellular fluid accumulation along these cells.

42 ignaling resulting from cilia-driven flow of extracellular fluid across the node.

43 In vivo cortical microdialysis samples the extracellular fluid adjacent to neurons and axons.

44 in an active form and are relocalized to the extracellular fluid after induction of PCD by either vic

45 reeze proteins prevent ice crystal growth in extracellular fluids, allowing fish to function in cold

46 tions reside both at the cell surface and in extracellular fluids, allowing their selective and tissu

47 actor) that might control the body's pool of extracellular fluid, an important determinant in hyperte

48 wall/extracellular space as monitored using extracellular fluid analysis (low- or high-ionic-strengt

49 Cells take up L-cystine from the extracellular fluid and convert it to L-cysteine intrace

50 al horn of the spinal cord for collection of extracellular fluid and for administration of drugs to t

51 ic lipids, proteins, and peptides present in extracellular fluid and hence minimizes electrode foulin

52 highly conserved endocytic process by which extracellular fluid and its contents are internalized in

53 sis serves as an internalization pathway for extracellular fluid and its contents.

54 Macropinocytosis, the internalization of extracellular fluid and material by plasma membrane ruff

55 t negatively charged residues are exposed to extracellular fluid and not bound to S4 residues, all of

56 d flagellar beating essential to movement of extracellular fluids and of cells through fluid.

57 ndopeptidases, which degrade peptides in the extracellular fluid, and beta-arrestins, which interact

58 mined correlations between total body water, extracellular fluid, and body cell mass and the drug vol

59 eled all-thiol HMGB1 was dissolved in actual extracellular fluids, and the oxidation and clearance ki

60 n the high molar ratio of p15s to BPI in the extracellular fluid (approximately 50:1), which greatly

61 Typical concentration ranges in extracellular fluids are quite narrow for the electrolyt

62 mineral inhibitor pyrophosphate (PPi) in the extracellular fluid around MVs.

63 Leftward flow of extracellular fluid at the node (nodal flow) is critical

64 The ideal would be to measure them in the extracellular fluid at the site of production.

65 ng the cylinder symmetry of a single axon in extracellular fluid based on the Poisson-Nernst-Planck e

66 in turn surrounded by the well-stirred bulk extracellular fluid (BECF) that represents an infinite r

67 We utilized microdialysis to sample the extracellular fluid before, during and after TENS and an

68 ls secrete enzymatically active PSA into the extracellular fluid but that once in the blood, PSA is n

69 in CORT may inhibit 5-HT transport from the extracellular fluid by acting on corticosterone-sensitiv

70 the hypothesis that CCL2 is removed from the extracellular fluid by CCR2-positive migrating cells as

71 Extracellular fluid collected from rat organotypic hippo

72 e of uncertainties in protein binding in the extracellular fluid compartment space.

73 f OCTs affect the clearance of 5-HT from the extracellular fluid compartment within the medial hypoth

74 ough most magnesium is stored outside of the extracellular fluid compartment, the regulated value is

75 rations are associated with increases in the extracellular fluid concentrations of the peptide and (i

76 cy and side effects, may change in different extracellular fluid conditions.

77 mply that exosomes present in circulation or extracellular fluids constitute an alternative source of

78 eins present on cell surfaces and located in extracellular fluids contain cysteine and methionine res

79 stigating the behavior of proteins in actual extracellular fluids containing an enormous number of di

80 Clearance of extracellular fluid did not prevent the spread of depres

81 ptic, we suggest that the composition of the extracellular fluid directly modulates extrasynaptic rel

82 Cerebral edema includes accumulation of extracellular fluid due to leakage of the brain's microv

83 toplasmic protein, it is also found in brain extracellular fluids, e.g., CSF.

84 ic feedback regulation mediated by a rise in extracellular fluid (ECF) [K(+)], there is evidence for

85 inase inhibitor, increases brain parenchymal extracellular fluid (ECF) accumulation of topotecan, a s

86 used on topotecan penetration into the brain extracellular fluid (ECF) and ventricular cerebrospinal

87 ercise, pathology and the composition of the extracellular fluid (ECF) can alter t-system volume (t-v

88 brospinal fluid (vCSF) and brain parenchymal extracellular fluid (ECF) compartments, we performed int

89 patial memory task, during which hippocampal extracellular fluid (ECF) glucose and lactate were measu

90 ip (20 +/- 1% below baseline) in hippocampal extracellular fluid (ECF) glucose seen in control animal

91 lications, the detection of ethanol in brain extracellular fluid (ECF) is of great importance.

92 vivo concentration changes in male rat brain extracellular fluid (ECF) of endogenous interleukin-1bet

93 s to measure drug concentration in the brain extracellular fluid (ECF) of the cerebral cortex during

94 tence and altered abundance of miRNAs in the extracellular fluid (ECF) or cerebrospinal fluid (CSF) m

95 sweat sodium chloride losses which leads to extracellular fluid (ECF) volume contraction and chlorid

96 elop in the presence of excess 5-HT in brain extracellular fluid (ECF).

97 l tissue and GABA concentrations in striatal extracellular fluid (ECF).

98 d catecholamine levels in adipose and muscle extracellular fluid (ECF).

99 over more than once each day by cycling, via extracellular fluids (ECF), between neurons and cataboli

100 ,000 micromol/L but only 0.5-2 micromol/L in extracellular fluids (ECFs).

101 n of EC-SOD from lung and vascular tissue to extracellular fluid (eg, bronchoalveolar lavage fluid an

102 ascorbate and when Asc(*-) concentrations in extracellular fluid exceeded 100 nM.

103 ence of an increase in total body sodium and extracellular fluid expansion.

104 lature is essential for the recirculation of extracellular fluid, fat absorption, and immune function

105 cond cell cleavage; however, models based on extracellular fluid flow at the node predict correct de

106 ed marrow (assuming a plasmacrit of 0.58, an extracellular fluid fraction of 0.40, and equilibration

107 Extracellular fluid from the CA1-CA3 regions of the hipp

108 We also collected and analyzed the extracellular fluid from this organ, in which the symbio

109 transiently, when the mutant was cultured in extracellular fluids from a WT culture, and biofilm form

110 issue and additional compartments, including extracellular fluids, further engineered tissue and livi

111 ver, rather than removing glutamate from the extracellular fluid, glioma cells release large amounts

112 ier separates the circulating blood from the extracellular fluid in the central nervous system and th

113 s of ethanol inhibits clearance of 5-HT from extracellular fluid in the mouse hippocampus.

114 Intracellular and extracellular fluid in the scraped endothelial cell mass

115 e sugar, UDP-glucose, which is released into extracellular fluids in response to stress, mediates HSP

116 other amino acids accumulate transiently in extracellular fluids in the perihematoma region during t

117 affects ACh levels in striatum and striatal extracellular fluid, in aged and young rats.

118 concentrations of 50-100 microM in blood and extracellular fluid increased to peaks of >8 mM.

119 increases the appearance of VEGF protein in extracellular fluids; induction of VEGF is suppressed by

120 How the brain extracellular fluid influences the activity of GABAergic

121 is a potentially powerful method for pulling extracellular fluid into a fused-silica capillary in con

122 he wound, allowing direct calcium entry from extracellular fluid into damaged cells.

123 t the uptake of glutamate by islets from the extracellular fluid is insignificant and that glutamate

124 The composition of brain extracellular fluid is shaped by a continuous exchange o

125 oids has never been assessed, although their extracellular fluid is under-saturated with respect to t

126 om the lung and pulmonary circulation to the extracellular fluids is beneficial in alveolar lung dise

127 Extracellular fluid levels of glutamate and aspartate in

128 precipitated withdrawal signs and increased extracellular fluid levels of glutamate in the LC of mor

129 the K(+) concentration and osmolality of the extracellular fluid may have profound effects on the cer

130 onally, L. monocytogenes grew more slowly in extracellular fluid (mouse serum) than within tissue cul

131 jury, strong correlations were found between extracellular fluid neurofilament heavy chain levels and

132 mpact traumatic brain injury, microdialysate extracellular fluid neurofilament heavy chain levels wer

133 Finally, extracellular fluid neurofilament heavy chain levels wer

134 rane the two proteolytic breakdown products, extracellular fluid neurofilament heavy chains NfH(476-9

135 Extracellular polyamines were measured in extracellular fluid obtained by microdialysis.

136 Cilia-driven leftward flow of extracellular fluids occurs in mammalian, fish and amphi

137 eping the glutamate concentration low in the extracellular fluid of brain.

138 following the real time changes of DA in the extracellular fluid of intact tissue with fast scan cycl

139 Endogenous peptides from brain extracellular fluid of live rats were analyzed using cap

140 leases intracellular potassium ions into the extracellular fluid of mouse and human tumours, causing

141 Such conditions do not exist in the extracellular fluid of the brain where the pH is neutral

142 ility, we measured the levels of PGE2 in the extracellular fluid of the POA (collected by microdialys

143 The extracellular fluid of the striatum contains a high leve

144 matrix of multicellular organisms and in the extracellular fluid of unicellular organisms, has been s

145 these HTLV-I-infected cell lines, and in the extracellular fluids of ATL patients.

146 of monoamine neurotransmitter levels in the extracellular fluids of the brain is critical in maintai

147 amine the neuromodulatory influence of brain extracellular fluid on GABAergic activity, we recorded f

148 Pathways for presenting proteins from the extracellular fluids on MHC class I molecules have been

149 We used microdialysis to deliver artificial extracellular fluid or L-lactate into the VMH and sample

150 In contrast, urchins maintained extracellular fluid pH under OA by accumulating bicarbon

151 the plasma pool but less than the estimated extracellular fluid pool.

152 ncentrations of neuromodulators in the brain extracellular fluid powerfully influence the excitabilit

153 ch is in the expected concentration range in extracellular fluid, preferentially inhibits the degradi

154 Endolymph is the specialised extracellular fluid present inside the inner ear.

155 y changes in the chemical composition of the extracellular fluid produced by glial or neuronal stimul

156 on that HCO3- entry into duct cells from the extracellular fluid requires Na+ but is not dependent on

157 Extracellular fluid S1P thus is required for optimal act

158 sgow Coma Score of 9, providing 640 cortical extracellular fluid samples for longitudinal data analys

159 Extracellular fluid samples were collected from the peri

160 When challenged with extracellular fluid shear stress, vascular endothelial c

161 OX) are influenced by protein binding in the extracellular fluid, since NSAIDs are bound to circulati

162 The clinical condition, assessment of extracellular fluid space volume status, measurement of

163 lls secrete DPPI that is active at the pH of extracellular fluids, suggesting that MC-DPPI may act ou

164 DPPIV is a serine protease present in extracellular fluids that cleaves peptides with a prolin

165 the rats were microinjected with artificial extracellular fluid, the GABA(A) receptor agonist muscim

166 disposal mediators of misfolded proteins in extracellular fluids, thereby normally protecting us fro

167 duced alterations in the uptake of 5-HT from extracellular fluid through the action of corticosterone

168 oendopeptidases degrade neuropeptides in the extracellular fluid to terminate signaling, the function

169 ranging from cell motility and propulsion of extracellular fluids to sensory physiology.

170 which is submitted to intense variations in extracellular fluid tonicity generated by the corticopap

171 Finally, GlyR tone varied inversely with extracellular fluid tonicity to mediate the osmotic cont

172 Here, we report how altering extracellular fluid tonicity with admixtures of clinical

173 Further, it is unknown how altering extracellular fluid tonicity with IVFs affects sRBC biom

174 e rates of approach of aqueous cytosolic and extracellular fluids toward the cell membrane.

175 This selective penetration of the tumor extracellular fluid translated into a strong anti-tumor

176 contribute to the clearance of 5-HT from the extracellular fluid under both baseline conditions and m

177 Calcium concentrations in extracellular fluids vary markedly, and are particularly

178 p8b1 bound and internalized cardiolipin from extracellular fluid via a basic residue-enriched motif.

179 Calcium enters the cell from the extracellular fluid via the voltage-dependent L-type Ca(

180 ography along with measurement of total body extracellular fluid volume (ECFV), and ECFV of the neck,

181 excretory function due to contraction of the extracellular fluid volume (vAKI) or due to intrinsic ki

182 The hormone aldosterone increases extracellular fluid volume and blood pressure by activat

183 l (ENaC) has an important role in regulating extracellular fluid volume and blood pressure, as well a

184 aC) have a crucial role in the regulation of extracellular fluid volume and blood pressure.

185 tem is a major regulatory system controlling extracellular fluid volume and blood pressure.

186 l (ENaC) has a key role in the regulation of extracellular fluid volume and blood pressure.

187 e promotes an early and uniform expansion of extracellular fluid volume and increased cardiac output.

188 epithelia and have a key role in regulating extracellular fluid volume and the volume of airway surf

189 Tumor extracellular fluid volume as measured with (99m)Tc DTPA

190 m 25.4+/-2 to 27.9+/-1 mmol/L, P < 0.05) and extracellular fluid volume bicarbonate by an estimated 3

191 ich contained 27 mmol acid, did not increase extracellular fluid volume bicarbonate over the 90-min p

192 mg/kg body wt) was ingested and the gain in extracellular fluid volume bicarbonate was compared with

193 II (AngII) contributes to the maintenance of extracellular fluid volume by regulating sodium transpor

194 The regulation of extracellular fluid volume by renal sodium excretion lie

195 mended when these conditions coexist because extracellular fluid volume depletion is often treated ra

196 The pathophysiology of both dehydration and extracellular fluid volume depletion must be understood

197 As with extracellular fluid volume depletion, arterial underfill

198 n in intravascular volume and is best termed extracellular fluid volume depletion.

199 Extracellular fluid volume expansion occurs in clinicall

200 nt factors, likely related to the effects of extracellular fluid volume expansion, also regulate kidn

201 s, these results show that in the setting of extracellular fluid volume expansion, excessive water re

202 adverse events, including several related to extracellular fluid volume expansion, increased in the i

203 II (ang II), and the natriuresis produced by extracellular fluid volume expansion.

204 are commonly used to treat hypertension and extracellular fluid volume expansion.

205 may be associated with an increased risk of extracellular fluid volume expansion.

206 l reabsorption has important implications in extracellular fluid volume homeostasis and airway fluid

207 nels (ENaC) participate in the regulation of extracellular fluid volume homeostasis and blood pressur

208 Natriuretic regulation of extracellular fluid volume homeostasis includes suppress

209 network of physiological pathways involving extracellular fluid volume homeostasis, cardiac contract

210 reases in plasma volume, and no increases in extracellular fluid volume in vivo.

211 nterval [CI] -3.10 to -2.63%; P < 0.001) and extracellular fluid volume increased by 0.62 L/1.73 m(2)

212 It is known that extracellular fluid volume is expanded (12% to 15%) in H

213 Extracellular fluid volume is expanded (12%) in clinical

214 0.88% (95% CI -0.12 to 1.87%; P = 0.095) and extracellular fluid volume reduction of -0.75 L/1.73 m(2

215 ring biochemical signaling but by regulating extracellular fluid volume to modulate ligand-receptor i

216 Tumor extracellular fluid volume was determined in a separate

217 confidence interval, 5 to 15 /1 to 6 mm Hg), extracellular fluid volume, albuminuria, and proteinuria

218 Blood volume, extracellular fluid volume, and regional blood flow were

219 of Na+ absorption and thus in the control of extracellular fluid volume, blood pressure, and sodium h

220 sma volume, the intravascular portion of the extracellular fluid volume, can be measured using standa

221 levated in the setting of normal or expanded extracellular fluid volume, e.g., in primary aldosteroni

222 inflammatory-like response with expansion of extracellular fluid volume, increased vascular permeabil

223 ception, spermatogenesis, and the control of extracellular fluid volume.

224 e is contracted despite an increase in total extracellular fluid volume.

225 ssure responses to alterations in sodium and extracellular fluid volume.

226 thout changes in body weight, hematocrit, or extracellular fluid volume.

227 and water despite expanded blood, plasma and extracellular fluid volumes.

228 ynthesized probe peroxyxanthone, H(2)O(2) in extracellular fluid was detected only after parenteral a

229 parenteral dosing, Asc(*-) concentrations in extracellular fluid were 4- to 12-fold higher than those

230 lactate, pyruvate, and glutamate in striatal extracellular fluid were collected and measured by a mic

231 In accordance, TGFbeta levels in the BM extracellular fluid were elevated and mesenchymal stroma

232 ration of activity between plasma and marrow extracellular fluid) were measured ex vivo from a periph

233 hat mediates apoptosis, GzmB can be found in extracellular fluids where it is hypothesized to regulat

234 compensated influx into, or efflux from, the extracellular fluid, will result in hypercalcaemia or hy

235 The endolymph is an extracellular fluid with an atypical composition that re