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1 consistent with its role in angiogenesis and extracellular matrix remodeling.
2 sive maspin inhibits tumor cell invasion and extracellular matrix remodeling.
3 me course, consistent with a net increase in extracellular matrix remodeling.
4 nal transduction, metabolism, transport, and extracellular matrix remodeling.
5 angiogenesis, immune cell infiltration, and extracellular matrix remodeling.
6 response, cell cycle arrest, apoptosis, and extracellular matrix remodeling.
7 rix turnover is a novel mechanism regulating extracellular matrix remodeling.
8 in regulating physiological and pathological extracellular matrix remodeling.
9 ntegrin, that are required for smooth muscle extracellular matrix remodeling.
10 n that IR can also induce growth factors and extracellular matrix remodeling.
11 e base of the follicle, differentiation, and extracellular matrix remodeling.
12 cal and pathological events characterized by extracellular matrix remodeling.
13 icated in both intracellular proteolysis and extracellular matrix remodeling.
14 in convertase-MT1-MMP axis that can regulate extracellular matrix remodeling.
15 sm whereby cytokines or Ox-LDL may influence extracellular matrix remodeling.
16 ents during the menstrual cycle that require extracellular matrix remodeling.
17 etalloproteinases (MMPs) both participate in extracellular matrix remodeling.
18 OXL2 independent of its conventional role in extracellular matrix remodeling.
19 P-10 may be implicated in H. pylori-mediated extracellular matrix remodeling.
20 ression of genes related to inflammation and extracellular matrix remodeling.
21 th G-protein-mediated muscle contraction and extracellular matrix remodeling.
22 in part, from ongoing inflammation and poor extracellular matrix remodeling.
23 to regulate cytoskeleton, cell adhesion, and extracellular matrix remodeling.
24 Wnt signaling, cell adhesion, migration, and extracellular matrix remodeling.
25 ar cells, cytokine/chemokine expression, and extracellular matrix remodeling.
26 13, which are critical modulators of stromal extracellular matrix remodeling.
27 anges are likely to be the result of scleral extracellular matrix remodeling.
28 by this enzyme family is critical for normal extracellular matrix remodeling.
29 d whether ONOO- and its metabolites modulate extracellular matrix remodeling.
30 s involved in oncogenesis, angiogenesis, and extracellular matrix remodeling.
31 e internal elastic lamina fragmentation, and extracellular matrix remodeling.
32 gulation in RCC and a potential regulator of extracellular matrix remodeling.
33 tes cell proliferation, differentiation, and extracellular matrix remodeling.
34 x metalloproteinases, critical regulators of extracellular matrix remodeling.
35 PARC on beta-catenin accumulation as well as extracellular matrix remodeling.
36 -encoded mitochondrial genes, and increasing extracellular matrix remodeling.
37 icular (ESLV) wall stress is associated with extracellular matrix remodeling activity after myocardia
38 ere was an enrichment in those implicated in extracellular matrix remodeling activity, cytoskeletal n
39 zymogen, MT1-MMP plays an essential role in extracellular matrix remodeling after an undefined proce
41 RATIONALE: Cardiac fibroblasts (CFs) drive extracellular matrix remodeling after pressure overload,
42 inhibited the gene program for fibrosis and extracellular matrix remodeling, although deletion of Tg
43 cerbating local inflammation, and increasing extracellular matrix remodeling, an environment conduciv
44 ort growing evidence that TGFbeta(2) induces extracellular matrix remodeling and abnormal cytoskeleta
46 matrix, and a second phase, characterized by extracellular matrix remodeling and altered mesenchymal-
47 MP-9), which has critical roles in promoting extracellular matrix remodeling and angiogenesis during
48 filtrating the onplants were associated with extracellular matrix remodeling and angiogenesis, in par
49 scriptional regulation of genes critical for extracellular matrix remodeling and cell cycle progressi
50 s) have been shown to be key players in both extracellular matrix remodeling and cell migration durin
51 red in collagen-rich mesenchymal tissues for extracellular matrix remodeling and cell proliferation d
52 re cytoskeletal-based structures involved in extracellular matrix remodeling and cellular motility.
53 rix metalloproteinases (MMPs) participate in extracellular matrix remodeling and degradation and have
54 cell and cell-matrix interactions, affecting extracellular matrix remodeling and endothelial cell mig
55 oteomics from WS revealed an upregulation of extracellular matrix remodeling and focal adhesion proce
56 atrix metalloprotease-9, a known mediator of extracellular matrix remodeling and growth factor activa
57 oproteinases (TIMPs) are involved in scleral extracellular matrix remodeling and have shown different
59 at3 regulates angiogenesis, axon growth, and extracellular matrix remodeling and is essential for lon
60 cellular adhesion, proliferation, branching, extracellular matrix remodeling and organ development.
61 data support a role for Tbx20 in repressing extracellular matrix remodeling and promoting cell proli
62 role and high energy requirements of hGFs in extracellular matrix remodeling and response to inflamma
64 human polymorphisms in proteins involved in extracellular matrix remodeling and the immune response
65 ions into therapeutics of diseases involving extracellular matrix remodeling and the immune response.
66 ed atherosclerotic plaques may contribute to extracellular matrix remodeling and the onset of acute t
68 in increasing trabecular meshwork outflow by extracellular matrix remodeling and/or by modulation of
69 ycle re-entry, loss of contractile elements, extracellular matrix remodeling, and altered signaling b
70 ed in cell signaling, immune system/defense, extracellular matrix remodeling, and cell cycle regulati
71 tivation, M2 macrophages, adaptive immunity, extracellular matrix remodeling, and cell proliferation.
72 encompass inflammation, hormonal signaling, extracellular matrix remodeling, and compensatory adapta
74 intercellular interactions, cytoskeletal and extracellular matrix remodeling, and gradients of agonis
75 ure, improved re-epithelialization, enhanced extracellular matrix remodeling, and greater nerve reinn
76 tion of hematopoietic stem cell development, extracellular matrix remodeling, and inflammatory cytoki
77 g-GLI signaling and of proteases involved in extracellular matrix remodeling, and matricellular prote
79 angiogenesis, blood coagulation, apoptosis, extracellular matrix remodeling, and regulation of infla
80 n the pathogenesis of collagen accumulation, extracellular matrix remodeling, and renal and cardiac f
81 motes tumor progression, including fibrosis, extracellular matrix remodeling, and the metabolic/catab
82 d in pathways that regulate inflammation and extracellular matrix remodeling, and they include matrix
83 enes essential for mesenchyme proliferation, extracellular matrix remodeling, and vasculogenesis.
84 variety of growth factor, immunomodulatory, extracellular matrix-remodeling, and stress response gen
85 nsight into the effects of hydroxytyrosol on extracellular matrix remodeling; and ii) test whether hy
86 of chondrocyte death, chondroclast function, extracellular matrix remodeling, angiogenesis and bone f
87 on of mesenchymal transition, cell adhesion, extracellular matrix remodeling, angiogenesis, and wound
89 s" AMR, which guides membrane deposition and extracellular matrix remodeling at the division site.
90 differences suggest disparate mechanisms of extracellular matrix remodeling between these 2 groups o
91 th and survival of cardiomyocytes as well as extracellular matrix remodeling by cardiac fibroblasts.
92 hat CTGF is a crucial regulator of cartilage extracellular matrix remodeling by generating Ctgf(-/-)
94 moved under perfusion and that inhibition of extracellular matrix remodeling causes mESCs to differen
95 actin-positive blood vessels, and 5) of key extracellular matrix remodeling (CD44, Col1a1, integrins
96 he tumor microenvironment and is involved in extracellular matrix remodeling, cell proliferation and
97 sociated matrix metalloproteinase (MMP)-2 in extracellular matrix remodeling compared with that of th
98 nvolved in vascular permeability, secretion, extracellular matrix remodeling, cytoskeleton reorganiza
99 d in MR-null myoblasts, suggesting a role in extracellular matrix remodeling during cell motility.
100 ch biological processes as wound closure and extracellular matrix remodeling during tissue developmen
101 portance has been ascribed to this enzyme in extracellular matrix remodeling during tumoral, inflamma
102 osomes and metalloproteases is essential for extracellular matrix remodeling, enabling migration thro
103 d that WNT2-mediated fibroblast motility and extracellular matrix remodeling enhanced cancer cell inv
104 ulation in lysyl oxidase (LOX) expression,an extracellular matrix remodeling enzyme, in a highly inva
105 othelial cells, with decreased expression of extracellular matrix remodeling-enzyme coding genes and
106 ased lactate secretion, enhanced activity of extracellular matrix remodeling enzymes, and impaired cl
107 te to angiogenesis, metabolic reprogramming, extracellular matrix remodeling, epithelial-mesenchymal
108 pathways, principally cell-cycle regulation, extracellular matrix remodeling, epithelial-to-mesenchym
109 cal injury processes of the immune response, extracellular matrix remodeling, epithelial-to-mesenchym
110 metalloproteinases (MMPs) play a key role in extracellular matrix remodeling events associated with h
111 ersatile MT-MMP playing an important role in extracellular matrix remodeling events in the brain and
112 tokines, yet their role in the regulation of extracellular matrix remodeling genes remains largely un
113 Micu2(-/-) mice had increased expression of extracellular matrix remodeling genes, while single-cell
115 arrest; but, it induced transcription of the extracellular matrix-remodeling genes MMP1 and PAI2, whi
116 Focal brain ischemia induces inflammation, extracellular matrix remodeling, gliosis, and neovascula
117 ne metabolism, cell adhesion and chemotaxis, extracellular matrix remodeling, hypoxia and oxidative s
118 immune responses, prohormone processing, and extracellular matrix remodeling important to bone develo
119 i by proinflammatory molecules may influence extracellular matrix remodeling in atherosclerosis by re
120 ental processes, valvular cell behavior, and extracellular matrix remodeling in congenital and acquir
121 GF-beta immunoreactivity in conjunction with extracellular matrix remodeling in gamma-irradiated mous
122 as well as myofibroblast differentiation and extracellular matrix remodeling in obstructed kidneys, t
123 d on previous studies implicating changes in extracellular matrix remodeling in other, related optic
124 tion in TM cells and offer new insights into extracellular matrix remodeling in the aqueous outflow p
125 ing, respectively, suggesting a new role for extracellular matrix remodeling in the establishment of
126 Ps) is responsible for multiple processes of extracellular matrix remodeling in the healthy body but
127 y be a quantitative biomarker of subclinical extracellular matrix remodeling in the lungs of communit
128 g of the ocular globe and is associated with extracellular matrix remodeling in the posterior sclera.
129 suggest that changes in a key determinant of extracellular matrix remodeling, in addition to changes
130 sponses to insults such as oxidative stress, extracellular matrix remodeling, inflammation, or cell d
131 ic and failing hearts that are indicative of extracellular matrix remodeling, insulin resistance and
133 d gene expression in the heart that included extracellular matrix remodeling, ion handling, and metab
135 erative bronchiolitis: neutrophil influx and extracellular matrix remodeling leading to luminal obstr
136 P signaling promotes hypertrophic growth and extracellular matrix remodeling, leading to the developm
138 xtracellular matrix components, mediators of extracellular matrix remodeling, matricellular proteins,
139 Collectively, these data uncover a novel extracellular matrix remodeling mechanism required for p
140 -C); 2) inflammation (interleukin-1beta); 3) extracellular matrix remodeling (MMP1, MMP3, TIMP1, uPA,
141 VEGF), inflammatory response (IL-1beta), and extracellular matrix remodeling (MMP3 and 9) were measur
142 t the potential modulation of cell adhesion, extracellular matrix remodeling, motility, metabolism, s
143 fined cell culture system recapitulating the extracellular matrix remodeling observed in vivo, we sho
146 tion of certain anti-glaucoma drugs involves extracellular matrix remodeling of ocular ciliary muscle
147 be required for all MMP functions including extracellular matrix remodeling of the tracheal system,
150 enzymes is comprised of critically important extracellular matrix remodeling proteases whose activity
151 (TAAs) develop secondary to abnormal aortic extracellular matrix remodeling, resulting in a weakened
152 ring cell necrosis and molecules involved in extracellular matrix remodeling such as hyaluronan, bigl
153 -MMP as a master regulator of the pathologic extracellular matrix remodeling that characterizes rheum
154 may be a marker for an initial phase of the extracellular matrix remodeling that occurs during the d
155 several disease markers for hypertrophy and extracellular matrix remodeling that were upregulated in
156 In addition to the well-recognized role in extracellular matrix remodeling, the tissue inhibitor of
157 nase (MT-MMPs) family are dual regulators of extracellular matrix remodeling through direct degradati
158 of numerous genes including those regulating extracellular matrix remodeling (Timp3, Adamts9) and cel
159 ndrogenesis, and is a key regulator coupling extracellular matrix remodeling to angiogenesis at the g
160 njectable biomaterials can induce beneficial extracellular matrix remodeling to stimulate tissue repa
161 omes release signaling molecules that induce extracellular matrix remodeling to support long-lasting
162 pithelial cancers and has been implicated in extracellular matrix remodeling, tumor growth, and metas
163 teinase 1 (MT1-MMP) plays a critical role in extracellular matrix remodeling under both physiological
164 al a novel mechanism of myocyte-orchestrated extracellular matrix remodeling via PDE5/cyclic guanosin
165 nhibitor of matrix metalloproteinase-1; this extracellular matrix remodeling was also significantly a
167 th factor and cytokine processing as well as extracellular matrix remodeling, we evaluated the role o
169 nstitutes a unique mechanism, independent of extracellular matrix remodeling, whereby a proteolytical
170 o the greatest extent were those involved in extracellular matrix remodeling with both UVA1 (P=5.5e-7
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