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4 s and sAC is required for the acute phase of extracellular signal regulated kinase 1/2 activation tri
5 way and that c-Cbl regulates the duration of extracellular signal regulated kinase 1/2 mitogen-activa
6 renin treatment increased phosphorylation of extracellular signal-regulated kinase (1/2) in neurons f
7 olling, distinct cytoskeletal rearrangement, extracellular signal-regulated kinase (1/2) phosphorylat
9 nstrated that sphingosine kinase 1 (SK1) and extracellular signal-regulated kinase 1/2 (ERK-1/2) inte
10 nearby cells and together with HGF promotes extracellular signal-regulated kinase 1/2 (ERK1/2) activ
11 ses both microRNA 21 (miR-21) expression and extracellular signal-regulated kinase 1/2 (ERK1/2) activ
12 ke receptor 4 (TLR4) ligand, by blocking the extracellular signal-regulated kinase 1/2 (ERK1/2) activ
13 d transient IkappaBalpha phosphorylation and extracellular signal-regulated kinase 1/2 (ERK1/2) activ
14 in-A ligands in neurons led to inhibition of extracellular signal-regulated kinase 1/2 (ERK1/2) activ
15 5 and SKI-606 blocked Chk1-inhibitor-induced extracellular signal-regulated kinase 1/2 (ERK1/2) activ
16 ted alpha2B-AR-mediated signaling, including extracellular signal-regulated kinase 1/2 (ERK1/2) activ
17 we previously discovered abnormally elevated extracellular signal-regulated kinase 1/2 (ERK1/2) activ
18 unds, across four pathways [cAMP production, extracellular signal-regulated kinase 1/2 (ERK1/2) activ
19 yos coincided with an enhanced activation of extracellular signal-regulated kinase 1/2 (ERK1/2) and a
20 inin were associated with down-regulation of extracellular signal-regulated kinase 1/2 (ERK1/2) and A
21 In an apparent paradox, STEP(61) regulates extracellular signal-regulated kinase 1/2 (ERK1/2) and p
22 that NPNT stimulates the phosphorylation of extracellular signal-regulated kinase 1/2 (ERK1/2) and p
23 kinase A (PKA)-dependent phosphorylation of extracellular signal-regulated kinase 1/2 (ERK1/2) are a
25 ell proliferation and persistently activated extracellular signal-regulated kinase 1/2 (ERK1/2) durin
27 horylation of p21-activated kinase (PAK) and extracellular signal-regulated kinase 1/2 (ERK1/2) in cu
29 g tumor angiogenesis and directly activating extracellular signal-regulated kinase 1/2 (Erk1/2) in GB
30 ole for the mitogen-activated protein kinase extracellular signal-regulated kinase 1/2 (ERK1/2) in th
31 factor receptor tyrosine kinase (EGFRtk) and extracellular signal-regulated kinase 1/2 (ERK1/2) inhib
32 Recent work suggested that the activity of extracellular signal-regulated kinase 1/2 (ERK1/2) is in
33 d by the MEK1 inhibitor U0126, implying that extracellular signal-regulated kinase 1/2 (ERK1/2) is th
34 tivated by inducible disruption of PTEN, and extracellular signal-regulated kinase 1/2 (ERK1/2) MAPK
35 ical role in controlling the activity of the extracellular signal-regulated kinase 1/2 (ERK1/2) pathw
36 over, we demonstrate that Six1 regulates the extracellular signal-regulated kinase 1/2 (ERK1/2) pathw
37 n target of rapamycin complex 1 (mTORC1) and extracellular signal-regulated kinase 1/2 (ERK1/2) pathw
38 by the cyclic adenosine monophosphate (cAMP)/extracellular signal-regulated kinase 1/2 (ERK1/2) pathw
39 a1 signals through cross-talking Smad2/3 and extracellular signal-regulated kinase 1/2 (ERK1/2) pathw
40 protein kinase A-dependent activation of the extracellular signal-regulated kinase 1/2 (ERK1/2) pathw
41 g transcription factors or activation of the extracellular signal-regulated kinase 1/2 (Erk1/2) pathw
42 signal transduction pathways such as Akt and extracellular signal-regulated kinase 1/2 (ERK1/2) phosp
43 sphorylated mitogen-activated protein kinase/extracellular signal-regulated kinase 1/2 (Erk1/2) prote
44 ental model of a self-sustained and bistable extracellular signal-regulated kinase 1/2 (ERK1/2) signa
45 s found to be dependent on activation of the extracellular signal-regulated kinase 1/2 (ERK1/2) signa
46 ibitors of Ang II type 1 receptor (AT1R) and extracellular signal-regulated kinase 1/2 (ERK1/2) suppr
48 to test whether synaptic levels of activated extracellular signal-regulated kinase 1/2 (ERK1/2), a pr
49 ogen-activated protein (MAP) kinases p38 and extracellular signal-regulated kinase 1/2 (ERK1/2), and
52 MAP3Ks selectively control the activation of extracellular signal-regulated kinase 1/2 (ERK1/2), Jun
53 ed a cascade of survival signaling involving extracellular signal-regulated kinase 1/2 (Erk1/2), p90R
54 f NgR1 leads to increased phosphorylation of extracellular signal-regulated kinase 1/2 (ERK1/2), sign
55 the eIF4E upstream kinase) or inhibitors of extracellular signal-regulated kinase 1/2 (ERK1/2), the
56 iptolide binds to and activates p38alpha and extracellular signal-regulated kinase 1/2 (ERK1/2), whic
57 ellular signal-regulated kinase 1/2 (Mek1/2)-extracellular signal-regulated kinase 1/2 (Erk1/2)-activ
58 ination, and degradation of EPLIN through an extracellular signal-regulated kinase 1/2 (ERK1/2)-depen
59 ithelial growth regulation by inhibiting the extracellular signal-regulated kinase 1/2 (Erk1/2)-mitog
60 asmin generation, but instead is mediated by extracellular signal-regulated kinase 1/2 (ERK1/2)-regul
64 the mitogen-activated protein kinase kinase/extracellular signal-regulated kinase 1/2 (MEK/ERK1/2) a
65 only combined treatment of mitogen-activated extracellular signal-regulated kinase 1/2 (MEK1/2) and S
66 of the Ras-mitogen-activated protein kinase/extracellular signal-regulated kinase 1/2 (Mek1/2)-extra
67 ell degranulation, TRAIL, and phosphorylated extracellular signal-regulated kinase 1/2 (pERK1/2) expr
68 nducible factor (HIF)-1alpha, phosphorylated extracellular signal-regulated kinase 1/2 (pERK1/2), and
69 or inhibition of cAMP and phosphorylation of extracellular signal-regulated kinase 1/2 (pERK1/2), N-a
70 CREB), and the pro-apoptotic protein kinases extracellular signal-regulated kinase 1/2 (phospho-ERK)
72 racts with mitogen-activated protein kinase (extracellular signal-regulated kinase 1/2 [ERK1/2]) to s
73 GRI977143 was an effective stimulator of extracellular signal-regulated kinase 1/2 activation and
74 , we revealed that protein kinase A-mediated extracellular signal-regulated kinase 1/2 activation fun
75 feration and invasion and inducing Tie-2 and extracellular signal-regulated kinase 1/2 activation in
77 nduced reactive oxygen species and sustained extracellular signal-regulated kinase 1/2 activation, wh
78 C cells and resulted in potent inhibition of extracellular signal-regulated kinase 1/2 activation.
80 activated protein kinase pathway, leading to extracellular signal-regulated kinase 1/2 activation.
81 All genetic lesions were associated with extracellular signal-regulated kinase 1/2 activation; ho
82 I-null fibroblasts and this was dependent on extracellular signal-regulated kinase 1/2 activity in th
84 Sa-2 cell lines decreased the phosphorylated extracellular signal-regulated kinase 1/2 and AKT and re
85 asal and insulin-mediated phosphorylation of extracellular signal-regulated kinase 1/2 and Akt in the
86 A and IgM PCs led to Ca(2+) mobilization and extracellular signal-regulated kinase 1/2 and AKT phosph
87 dies suggest that SDF-1alpha/CXCR4-dependent extracellular signal-regulated kinase 1/2 and Akt phosph
88 und that SMCs respond to Shh by upregulating extracellular signal-regulated kinase 1/2 and Akt phosph
89 e the maximal inhibition of their downstream extracellular signal-regulated kinase 1/2 and AKT signal
92 at AR augments G-CSF signaling by activating extracellular signal-regulated kinase 1/2 and also by su
93 -mediated signalling pathway, including p38, extracellular signal-regulated kinase 1/2 and c-Jun N-te
94 ulated PgLPS-mediated phosphorylation of the extracellular signal-regulated kinase 1/2 and c-Jun N-te
97 cKL + FGF23 increased the phosphorylation of extracellular signal-regulated kinase 1/2 and induced Fg
98 A/C gene (LMNA) mutation, and found that the extracellular signal-regulated kinase 1/2 and Jun N-term
99 hosphates) in some signaling events, such as extracellular signal-regulated kinase 1/2 and label free
100 n of wild-type (WT) AKT or protein kinase B, extracellular signal-regulated kinase 1/2 and mitogen-ac
102 active oxygen species-mediated activation of extracellular signal-regulated kinase 1/2 and p38 mitoge
104 activated protein kinases, we show that both extracellular signal-regulated kinase 1/2 and p38, but n
105 processes are dependent on activation of the extracellular signal-regulated kinase 1/2 and phosphatid
107 The stage- and time-specific activations of extracellular signal-regulated kinase 1/2 and protein ki
108 /Galpha(11) deficiency blocked activation of extracellular signal-regulated kinase 1/2 and the TCF El
109 NET25 depletion causes hyperactivation of extracellular signal-regulated kinase 1/2 at the onset o
111 also increased the levels of phosphorylated extracellular signal-regulated kinase 1/2 in apoE(-/-)/b
112 agonist induced phosphorylation of FGFR1 and extracellular signal-regulated kinase 1/2 in rat hippoca
114 ix metalloproteinase 2 activity, but phospho-extracellular signal-regulated kinase 1/2 is blocked by
115 provided evidence that elevated activity of extracellular signal-regulated kinase 1/2 is required fo
116 nsforming growth factor-beta (TGF-beta), and extracellular signal-regulated kinase 1/2 kinase activit
117 s in Cyclin D1, CDK4, phosphorylated Rb, and extracellular signal-regulated kinase 1/2 levels compare
118 MAP2KI (MEK1) expression and phosphorylated-extracellular signal-regulated kinase 1/2 levels in RAMP
119 sociated with elevated expression of Wnt and extracellular signal-regulated kinase 1/2 mitogen-activa
120 stromal-derived factor-1 (SDF1), through the extracellular signal-regulated kinase 1/2 pathway, invol
122 ted signaling, including the AKT, c-myc, and extracellular signal-regulated kinase 1/2 pathways, was
123 nds activated CB(2) receptors as measured by extracellular signal-regulated kinase 1/2 phosphorylatio
124 red mGlu(5)-mediated Ca(2+) mobilization and extracellular signal-regulated kinase 1/2 phosphorylatio
125 (35)S]guanosine 5'-O-(3-thio)triphosphate or extracellular signal-regulated kinase 1/2 phosphorylatio
126 Four functions [adenylate cyclase (AC), extracellular signal-regulated kinase 1/2 phosphorylatio
128 bition of RasGRP3 expression reduced AKT and extracellular signal-regulated kinase 1/2 phosphorylatio
129 hosphorylation of IR/IGF-IR and Akt, whereas extracellular signal-regulated kinase 1/2 phosphorylatio
130 Portal hypertensive gastropathy impairs extracellular signal-regulated kinase 1/2 phosphorylatio
131 tor-1-stimulated proliferation and increased extracellular signal-regulated kinase 1/2 phosphorylatio
132 s down-regulated, followed by an increase in extracellular signal-regulated kinase 1/2 phosphorylatio
133 ells, Ptges expression was regulated through extracellular signal-regulated kinase 1/2 phosphorylatio
134 eased p38 mitogen-activated protein kinases, extracellular signal-regulated kinase 1/2 phosphorylatio
135 y neurons IL-31 triggered Ca(2+) release and extracellular signal-regulated kinase 1/2 phosphorylatio
136 AF, mitogen-activated protein kinase 1/2, or extracellular signal-regulated kinase 1/2 protein levels
137 Simultaneous inhibition of K-ras downstream extracellular signal-regulated kinase 1/2 signaling in p
138 ediate the effects of GDF15, whereas Src and extracellular signal-regulated kinase 1/2 signaling path
140 independent growth through activation of the extracellular signal-regulated kinase 1/2 signaling path
141 inhibiting cAMP accumulation and activating extracellular signal-regulated kinase 1/2 signaling whil
142 protein tyrosine phosphatase 1B (PTP1B) and extracellular signal-regulated kinase 1/2 signaling.
143 f p-Src and mitogen-activated protein kinase-extracellular signal-regulated kinase 1/2 that were supp
144 nase C levels and loss of phosphorylation of extracellular signal-regulated kinase 1/2 were prevented
145 pathways (phosphatidylinositol 3 kinase and extracellular signal-regulated kinase 1/2) are involved
146 is the MAP 3-kinase component of an ERK-1/2 (extracellular signal-regulated kinase 1/2) MAPK pathway
147 of epidermal growth factor receptor-Erk1/2 (extracellular signal-regulated kinase 1/2) signaling, th
149 ayed rapid increases in activated STAT1/3/5, extracellular signal-regulated kinase 1/2, AKT, CREB, an
150 proliferation associated with increased AKT, extracellular signal-regulated kinase 1/2, and endotheli
151 ncreased phosphorylation of EGF receptor and extracellular signal-regulated kinase 1/2, and increased
152 lated protein-1, triggers phosphorylation of extracellular signal-regulated kinase 1/2, and induces d
153 in 11, p120 isoform switching, activation of extracellular signal-regulated kinase 1/2, and matrix me
154 hreonine protein kinase B, protein kinase A, extracellular signal-regulated kinase 1/2, and p38 mitog
156 es and were associated with an activation of extracellular signal-regulated kinase 1/2, and ribosomal
157 lian target of rapamycin complex 1 (mTORC1), extracellular signal-regulated kinase 1/2, and signal tr
158 t had no significant effect on activation of extracellular signal-regulated kinase 1/2, another impor
159 s VEGF-induced phosphorylation of VEGFR2 and extracellular signal-regulated kinase 1/2, as well as EC
160 ory effects, increased inhibition of phospho-extracellular signal-regulated kinase 1/2, increased mit
161 azonensis amastigotes, through activation of extracellular signal-regulated kinase 1/2, inhibit the a
162 ion of the mitogen activated protein kinase, extracellular signal-regulated kinase 1/2, observed with
163 eated with inhibitors of tyrosine kinase, of extracellular signal-regulated kinase 1/2, or of mRNA pr
164 ng, the robust responses of several kinases (extracellular signal-regulated kinase 1/2, p38, and Akt)
165 ng MMP-9 secretion through the activation of extracellular signal-regulated kinase 1/2, p38, phosphoi
166 down resulted in decreased levels of phospho-extracellular signal-regulated kinase 1/2, phospho-c-Jun
167 ncluding retrovirus-associated DNA sequences/extracellular signal-regulated kinase 1/2, phosphoinosit
168 d PKC (mu, theta, epsilon) but not mTOR, Ras-extracellular signal-regulated kinase 1/2, protein kinas
170 -mediated increase in the phosphorylation of extracellular signal-regulated kinase 1/2, recruitment o
172 two major transformation-promoting pathways: extracellular signal-regulated kinase 1/2-activator prot
173 imarily an AMP-activated protein kinase- and extracellular signal-regulated kinase 1/2-dependent even
174 et unidentified ATP-releasing channels in an extracellular signal-regulated kinase 1/2-dependent fash
175 STAT3 was phosphorylated on serine 727 in an extracellular signal-regulated kinase 1/2-dependent mann
176 STAT3, which is significantly enhanced by an extracellular signal-regulated kinase 1/2-dependent mTOR
177 uced AAA formation in mice by phosphorylated extracellular signal-regulated kinase 1/2-mediated cyclo
179 els of phosphorylated IkappaBalpha, Akt, and extracellular signal-regulated kinase 1/2; nuclear trans
180 as signaling and sorting platforms, such as extracellular signal-regulated kinase-1/2 (ERK1/2) and h
181 wn of N-cadherin decreased the activation of extracellular signal-regulated kinase-1/2 (ERK1/2), AKT
183 oteins but can recruit arrestins and promote extracellular signal-regulated kinase-1/2 phosphorylatio
184 but independent of Jun N-terminal kinase-1, extracellular signal-regulated kinase-1/2, or p38 mitoge
185 nic ability, and invasiveness by suppressing extracellular signal-regulated kinases 1/2 (ERK1/2) and
186 ation of phospholipase-Cgamma (PLCgamma) and extracellular signal-regulated kinases 1/2 (ERK1/2) by a
187 er distention are associated with changes in extracellular signal-regulated kinases 1/2 (ERK1/2) phos
190 EGFR, phosphoinositide 3-kinase (PI3K), and extracellular signal-regulated kinases 1/2 (ERK1/2) was
191 ttenuate alpha(2B)-AR-mediated activation of extracellular signal-regulated kinases 1/2 (ERK1/2) with
192 component Exo70 is a direct substrate of the extracellular signal-regulated kinases 1/2 (ERK1/2).
193 treated with necrotic bone showed increased extracellular signal-regulated kinases 1/2 and Ikappa ki
195 USP8 controls basal and acute stress-induced extracellular signal-regulated kinases 1/2 signaling in
198 en-activated protein kinases (MAPKs; p38 and extracellular signal-regulated kinases 1/2) and cyclooxy
200 s also manifested reduced phosphorylation of extracellular signal-regulated kinases 1/2, c-Jun N-term
201 upregulation of renal cortical phospho-p38, extracellular signal-regulated kinases 1/2, COX-2, and r
202 rescued the RES-mediated downregulation of p-extracellular signal-regulated kinases 1/2, p-Smad3, and
203 e mitogen-activated protein kinases (MAPKs), extracellular signal-regulated kinases 1/2, p38 MAPK, an
204 ation of mammalian target of rapamycin, AKT, extracellular signal-regulated kinases 1/2, phosphatase
205 osphorylated focal adhesion kinase (FAK) and extracellular signal-regulated kinases 1/2, whereas Ln-3
206 LG3 increased the migration of VSMC through extracellular signal-regulated kinases 1/2-dependent pat
207 ith the tyrosine kinase Src, which activates extracellular signal-regulated kinases(1/2), to increase
208 RPV1 affecting enzymes such as lipoxygenase, extracellular signal-regulated kinases-1/2, sarcoma, or
210 over, we demonstrate that phosphorylation of extracellular-signal regulated kinase 1/2 (ERK1/2) is do
212 2) (COT, MAP3K8) kinase activates the MEK1/2-extracellular-signal regulated kinase 1/2 MAP kinase sig
213 led to a decrease in phosphorylated Erk1/2 (extracellular-signal regulated kinases 1/2) in sprouting
214 AS silencing in amplified cell lines reduced extracellular-signal-regulated kinase 1/2 (ERK1/2) phosp
215 ng alpha-MSH production via CB1R-induced and extracellular-signal-regulated kinase 1/2 activation- an
217 bitor reduced the phosphorylation of p38 and extracellular-signal-regulated kinases 1/2 (p44/42 MAPK)
218 ory effects, a reduced inhibition of phospho-extracellular-signal-regulated kinases 1/2, a less sever
219 mitogen-activated protein kinase (p38MAPK), extracellular signal-regulated kinase-1/-2 (ERK-1/2), ph
220 odendroglial processes requires signaling by extracellular signal regulated kinase-1 and -2 (Erk1/2),
221 cardiovascular disease, is known to activate extracellular signal regulated kinases 1 and 2 (ERK1/2).
222 extracellular signal-regulated kinase kinase/extracellular signal regulated kinases 1 and 2 (MEK/ERK1
223 tinct compartmentalization of phosphorylated extracellular signal-regulated kinase 1 and 2 ([ERK1/2]
224 or Toll-like receptor 4 (TLR4) activation of extracellular signal-regulated kinase 1 and 2 (ERK-1/2)
225 n is heavily dependent on both NF-kappaB and extracellular signal-regulated kinase 1 and 2 (ERK-1/2)
226 her decreased by DMF-mediated suppression of extracellular signal-regulated kinase 1 and 2 (ERK1/2) a
229 l as phosphatidylinositol 3-kinase (PI3K) or extracellular signal-regulated kinase 1 and 2 (Erk1/2) s
230 l as phosphatidylinositol 3-kinase (PI3K) or extracellular signal-regulated kinase 1 and 2 (Erk1/2) s
231 in, mitogen-activated protein (MAP) kinases, extracellular signal-regulated kinase 1 and 2 (ERK1/2),
234 regulating cellular asymmetry, also promotes extracellular signal-regulated kinase 1 and 2 (ERK1/2)-d
235 revented by mitogen activated protein kinase/extracellular signal-regulated kinase 1 and 2 (MAPK/ERK1
236 show that SHP2 positively modulates the Ras-extracellular signal-regulated kinase 1 and 2 and the ph
237 anosine triphosphatase activity, and Akt and extracellular signal-regulated kinase 1 and 2 phosphoryl
238 eric modulation of dopamine signaling in the extracellular signal-regulated kinase 1 and 2 phosphoryl
239 f G proteins, inhibition of cAMP production, extracellular signal-regulated kinase 1 and 2 phosphoryl
241 e to PDGF-BB, and reduced phosphorylation of extracellular signal-regulated kinase 1 and 2, Elk-1, p3
242 e, we demonstrate that activated Ras induces extracellular signal-regulated kinase 1 and 2-dependent
243 cell activation, as well as inflammasome and extracellular signal-regulated kinase 1 and 2-mediated n
244 ression and reduced levels of phosphorylated extracellular signal-regulated kinase 1 and active nucle
247 R-gamma ligands correlated with decreases in extracellular signal-regulated kinase-1 and -2 activatio
248 and ErbB2 transactivation leads to prolonged extracellular signal-regulated kinase-1 and -2 signaling
249 e C3a receptor results in phosphorylation of extracellular signal-regulated kinase-1 and 2 (ERK-1/2),
250 d expression of c-MET and phosphorylation of extracellular signal-regulated kinases 1 and 2 (downstre
251 F-1-induced cell migration and activation of extracellular signal-regulated kinases 1 and 2 (ERK) MAP
253 carcinoma (HCT116) cells treated with H2O2, extracellular signal-regulated kinases 1 and 2 (ERK1/2)
255 l assay of A(1)R mediated phosphorylation of extracellular signal-regulated kinases 1 and 2 (ERK1/2)
256 or 9 min stimulated rapid phosphorylation of extracellular signal-regulated kinases 1 and 2 (ERK1/2)
257 bition of CHK1 can promote the activation of extracellular signal-regulated kinases 1 and 2 (ERK1/2)
258 Here, we show that sustained activation of extracellular signal-regulated kinases 1 and 2 (ERK1/2)
259 ses 1 and 2 (MNK1 and MNK2) are activated by extracellular signal-regulated kinases 1 and 2 (ERK1/2)
260 re, we show that sustained activation of the extracellular signal-regulated kinases 1 and 2 (ERK1/2)
261 c1 is associated with enhanced activities of extracellular signal-regulated kinases 1 and 2 (ERK1/2)
263 response through activation of NF-kappaB via extracellular signal-regulated kinases 1 and 2 (ERK1/2)
264 uble tyrosine/threonine phosphorylation, the extracellular signal-regulated kinases 1 and 2 (ERK1/2)
265 Inactivation of PldD inhibited activation of extracellular signal-regulated kinases 1 and 2 (ERK1/2),
266 ly enhanced carbachol-mediated activation of extracellular signal-regulated kinases 1 and 2 (ERK1/2),
267 n skeletal muscle cells is the activation of extracellular signal-regulated kinases 1 and 2 (ERK1/2).
268 kinase (JNK) but decreased levels of phospho-extracellular signal-regulated kinases 1 and 2 (ERK1/2).
269 2+)(i)) mobilization, and phosphorylation of extracellular signal-regulated kinases 1 and 2 (pERK1/2)
270 2+)(i)) mobilization, and phosphorylation of extracellular signal-regulated kinases 1 and 2 (pERK1/2)
271 to activation of nuclear factor kappa B and extracellular signal-regulated kinases 1 and 2 and subse
273 P-p38 MAPK, P-c-jun-N-terminal kinase, and P-extracellular signal-regulated kinases 1 and 2 in the tr
275 ation with IL-13, IL-13Ralpha2 increased the extracellular signal-regulated kinases 1 and 2 phosphory
276 ause the signaling molecules RAS and ERK1/2 (extracellular signal-regulated kinases 1 and 2) are acti
277 neurons, we found that inhibition of Erk1/2 (extracellular signal-regulated kinases 1 and 2) enhanced
278 ing Janus-like kinase (JAK)-1, JAK-2, c-Src, extracellular signal-regulated kinases 1 and 2, AKT, and
279 ated kinase 1 (MSK1), a downstream target of extracellular signal-regulated kinases 1 and 2, and an i
280 ion of p38 mitogen-activated protein kinase, extracellular signal-regulated kinases 1 and 2, and Jun
281 reased the phosphorylation (= activation) of extracellular signal-regulated kinases 1 and 2, c-Jun N-
282 ethyl-D-aspartate receptors or activation of extracellular signal-regulated kinases 1 and 2, or block
283 had higher concentrations of phosphorylated extracellular signal-regulated kinases 1 and 2, Trx1, Tr
287 strated a decrease in the phosphorylation of extracellular-signal-regulated kinases 1 and 2, confirmi
288 Abl kinase stimulates Cdk6 expression via an extracellular signal-regulated kinase 1-dependent pathwa
290 lly interacts with the NR2A subunits and the extracellular signal-regulated kinase 1 (ERK1) and ERK2
291 this bias is lost in mutants with increased extracellular signal-regulated kinase 1 (ERK1) and ERK2
295 OPK kinase activity but had little effect on extracellular signal-regulated kinase 1 (ERK1), c-jun-NH
296 , phosphoinositide 3-kinase (PI3K) p85alpha, extracellular signal-regulated kinase 1 (ERK1), or nucle
297 mitogen-activated protein kinase (MAPK) and extracellular signal-regulated kinase 1 (ERK1)/2 abrogat
298 neurin, and mitogen-activated protein kinase-extracellular signal-regulated kinase 1-extracellular si
300 pposing effects on phosphorylation of CBP by extracellular signal-regulated kinase 1 that correlated
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