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1 tissues and areas that displayed neutrophil extracellular traps.
2 d can also block the formation of neutrophil extracellular traps.
3 of their nuclear contents to form neutrophil extracellular traps.
4 irulence factors and formation of neutrophil extracellular traps.
5 g by human neutrophils and within neutrophil extracellular traps.
6 eactive oxygen species (ROS), and neutrophil extracellular traps.
7 phagocytosis, degranulation, and neutrophil extracellular traps.
8 gery, which generated luminal DNA neutrophil extracellular traps.
9 ith proteases, which are known as neutrophil extracellular traps.
10 ess of forming specialized structures called extracellular traps.
11 d citrullinated H3, features consistent with extracellular traps.
12 intertwined with the formation of neutrophil extracellular traps.
13 required to trigger formation of neutrophil extracellular traps.
14 ed impairment in the formation of neutrophil extracellular traps, a bactericidal mechanism that opera
15 as revealed with the discovery of neutrophil extracellular traps, a specialized cell death process in
16 itro, with release of extensive histone-rich extracellular traps, an event unsuppressed by dexamethas
18 DNase I to root tips eliminates border cell extracellular traps and abolishes root tip resistance to
19 e the major protein components of neutrophil extracellular traps and are known to have cytotoxic effe
21 killing by neutrophils and within neutrophil extracellular traps and neutralizes LL-37 chemotactic pr
22 ized' neutrophils, as well as the neutrophil extracellular traps and other products made by neutrophi
23 cytoid dendritic cells (PDCs) and release of extracellular traps and proinflammatory cytokines by neu
24 , released from dying cells or in neutrophil extracellular traps) and an increased expression of the
25 trate of PAD4, localize H1 within neutrophil extracellular traps, and detect autoantibodies to citrul
26 is or necrosis, degranulation, or release of extracellular traps, and it decreases the oxidative resp
27 pture exogenous material, extrude neutrophil extracellular traps, and kill bacteria via cathelicidin
28 w players, such as polyphosphate, neutrophil extracellular traps, and microparticles, which have been
29 n, decreases bacterial killing by neutrophil extracellular traps, and modulates S. pyogenes virulence
30 d reactive oxygen species, escape neutrophil extracellular traps, and promote and accelerate phagocyt
31 d lysis, engulfment, formation of neutrophil extracellular traps, and release of antimicrobial peptid
32 nomic DNA, mitochondrial DNA, and neutrophil extracellular traps, and shuttles them in the cytosol of
33 vivo, that these nodules have properties of extracellular traps, and the nodules are not required fo
34 o interfere with the formation of neutrophil extracellular traps, appears to prolong lysis time at a
36 FN by pDCs, which were induced by neutrophil extracellular traps arising from the endocytosis of the
37 an increase in the deployment of neutrophil extracellular traps associated with hyperglycemia slows
41 tivity and enhanced production of neutrophil extracellular traps compared with wild-type neutrophils
42 A released from dying cells or in neutrophil extracellular traps complexed to the antimicrobial pepti
43 MCs with Leishmania led to generation of MC extracellular traps comprising of DNA, histones and tryp
44 trategy for evasion of the host's neutrophil extracellular traps, digesting the DNA scaffold structur
48 ntal model of asthma would induce eosinophil extracellular traps (EETs) in bronchoalveolar lavage flu
50 DNases degrade the chromatin in neutrophil extracellular traps, enabling the bacterium to evade neu
52 e mechanism is reminiscent of the neutrophil extracellular traps (ETs) recently described in vertebra
53 o their unique ability to release neutrophil extracellular traps even in the absence of pathogens.
54 We previously demonstrated that neutrophil extracellular traps exacerbate pulmonary injury during i
56 yeloid cell recruitment, and more neutrophil extracellular trap formation (NETs) in WT compared to pc
57 that IL-23 and IL-1beta can induce mast cell extracellular trap formation and degranulation of human
59 ted cytokine release, and induced neutrophil extracellular trap formation and myeloperoxidase release
60 mechanism that is associated with neutrophil extracellular trap formation and selective autophagy in
62 in transgenic mice with impaired neutrophil extracellular trap formation and/or neutrophils with con
63 exhibited an elevated capacity in neutrophil extracellular trap formation at baseline and upon microb
64 showed a significant increase in neutrophil extracellular trap formation but were unable to compensa
65 Thus, initial characterization of macrophage extracellular trap formation due to M. tuberculosis infe
66 yeloid cells, but did not require neutrophil extracellular trap formation involving peptidyl arginine
68 phaMbeta2 integrin activation and neutrophil extracellular trap formation under inflammatory conditio
69 lling and were fully competent in neutrophil extracellular trap formation, a recently identified extr
70 severe pulmonary edema, increased neutrophil extracellular trap formation, and elevated concentration
72 ne-induced neutrophil congestion, neutrophil extracellular trap formation, and thrombosis in the pulm
73 ing, oxidative burst, chemotaxis, neutrophil extracellular trap formation, bacterial killing, and ind
74 nce of ESX-1, IFN-gamma does not restore any extracellular trap formation, mycobacterial aggregation,
81 eveloped computational pipelines to identify extracellular traps from an in vitro human samples visua
83 ate cell activation that included neutrophil extracellular trap generation and elevated surface expre
84 ponse, and we present evidence of neutrophil extracellular trap generation during experimental urinar
88 fungal filaments, suggesting that neutrophil extracellular traps help to protect the epithelial barri
90 nt findings of the involvement of neutrophil extracellular traps in atherogenesis and atherothrombosi
92 d by limited tools to quantify occurrence of extracellular traps in experimental models and human sam
93 ng/mL; p </= 0.05) and identified neutrophil extracellular traps in kidney and liver tissues from unt
94 that macrophages, like neutrophils, can form extracellular traps in response to bacterial pathogens a
96 , possibly through degradation of neutrophil extracellular traps, innate immune structures composed o
97 at model, we identified layers of neutrophil extracellular traps interconnecting and entrapping bacte
98 es to its advantage by converting neutrophil extracellular traps into a bacterial weapon against macr
99 ation and membrane pore formation, and (iii) extracellular trapping mediated by membrane-proximal hep
100 to counteract histone as well as neutrophil extracellular trap-mediated cytotoxicity against host ce
102 ate proteoglycan(s) is present in neutrophil extracellular traps, modulates histone affinity, and mod
103 uses extracellular DNA (eDNA) and neutrophil extracellular trap (NET) accumulation in the precorneal
106 t evidence suggests that enhanced neutrophil extracellular trap (NET) formation activates plasmacytoi
107 o immobilized neutrophils induced neutrophil extracellular trap (NET) formation in response to infect
110 active oxygen species production, neutrophil extracellular trap (NET) formation, and neutrophil elast
111 h neutrophil functions, including neutrophil extracellular trap (NET) formation, are involved in the
116 is, oxidative burst capacity, and neutrophil extracellular trap (NET) generation (NETosis) were measu
117 granule proteins with subsequent neutrophil extracellular trap (NET) release independent of elastase
118 and host components that included neutrophil extracellular trap (NET) structures and that the bacteri
123 trophil complexes, a signature of neutrophil extracellular traps (NET), in the kidneys of tumor-beari
126 tosis and cell death by releasing neutrophil extracellular traps (NETs) (NETosis), which were more ob
128 These chromatin traps are termed neutrophil extracellular traps (NETs) and are decorated with granul
129 o contributes to the formation of neutrophil extracellular traps (NETS) and impacts on the immune res
130 -1 mediates bacterial survival in neutrophil extracellular traps (NETs) and protects GAS from antimic
144 eir anti-microbial defense, neutrophils form extracellular traps (NETs) by releasing decondensed chro
146 The recent discovery of secreted neutrophil extracellular traps (NETs) composed of DNA and histones
153 We also studied the production of neutrophil extracellular traps (NETs) from single neutrophils isola
158 e particles induce the release of neutrophil extracellular traps (NETs) in a size-dependent manner by
159 reactive oxygen species (ROS) and neutrophil extracellular traps (NETs) in mouse and human neutrophil
161 obe size and selectively released neutrophil extracellular traps (NETs) in response to large pathogen
162 tient were incapable of producing neutrophil extracellular traps (NETs) in response to ROS and were u
163 trophils release large amounts of neutrophil extracellular traps (NETs) in the presence of P. aerugin
166 platelets induce the formation of neutrophil extracellular traps (NETs) in transfusion-related acute
168 cies in the phagosome and release neutrophil extracellular traps (NETs) into their surroundings to im
170 tes and the possible formation of neutrophil extracellular traps (NETs) may result in chromatin relea
172 itates formation of prothrombotic neutrophil extracellular traps (NETs) mediated by RAGE, exposing ad
173 iew, we examine the evidence that neutrophil extracellular traps (NETs) play a critical role in innat
174 There is emerging evidence that neutrophil extracellular traps (NETs) play important roles in infla
175 crystals became enmeshed in the neutrophilic extracellular traps (NETs) produced from host cells in r
181 , we analyzed the contribution of neutrophil extracellular traps (NETs) to the mediation of protectio
182 ial invasion, neutrophils release neutrophil extracellular traps (NETs) to trap and kill extracellula
183 Here we report that ANCA induces neutrophil extracellular traps (NETs) via receptor-interacting prot
184 morhonuclear granulocytes to form neutrophil extracellular traps (NETs) was determined using fluoresc
185 Moreover, increased release of neutrophil extracellular traps (NETs) was observed, which was most
188 nic bacteria and an impaired ability to form extracellular traps (NETs), an important neutrophil func
190 y aimed to explore the release of neutrophil extracellular traps (NETs), associated antimicrobial pro
191 ned the relationships between CLS neutrophil extracellular traps (NETs), bacterial components as trig
192 eliminate pathogens they release neutrophil extracellular traps (NETs), being chromatin fibers decor
193 depends on their ability to form neutrophil extracellular traps (NETs), but the underlying mechanism
194 ve oxygen species, and release of neutrophil extracellular traps (NETs), can result in severe patholo
195 he yeast and subsequently release neutrophil extracellular traps (NETs), complexes of DNA, histones,
197 is, characterized by formation of neutrophil extracellular traps (NETs), decondensed chromatin thread
199 blood, through the generation of neutrophil extracellular traps (NETs), is procoagulant and prothrom
201 lysozyme and PGRP-S colocalize in neutrophil extracellular traps (NETs), suggesting that these granul
202 tinal lumen, which appeared to be neutrophil extracellular traps (NETs), suggesting that V. cholerae
203 nsed chromatin structures, termed neutrophil extracellular traps (NETs), that have been implicated in
204 lacking ACT induces formation of neutrophil extracellular traps (NETs), whereas wild-type B. pertuss
206 an and bovine neutrophils release neutrophil extracellular traps (NETs), which are protein-studded DN
207 ed that human neutrophils release neutrophil extracellular traps (NETs), which are protein-studded DN
208 , and granule proteins to produce neutrophil extracellular traps (NETs), which can trap microbes.
209 stimuli and pathogens, they form neutrophil extracellular traps (NETs), which capture and kill extra
210 eightened capacity to synthesize neutrophils extracellular traps (NETs), which display increased exte
211 erminants of NTHI survival within neutrophil extracellular traps (NETs), which we have shown to be an
212 ly, LukGH promoted the release of neutrophil extracellular traps (NETs), which, in turn, ensnared but
233 uclear leukocytes to release DNA [neutrophil extracellular traps (NETs)], thereby immobilizing microb
235 ) is released into the blood from neutrophil extracellular traps(NETs) in response to severe infectio
236 tion of granular constituents and neutrophil extracellular traps, neutrophils target microbes and pre
237 ation and membrane pore formation, and (iii) extracellular trapping of FGF2 mediated by heparan sulfa
238 nt an additional role for macrophages in the extracellular trapping of lipoproteins in atherosclerosi
240 tion, reactive oxygen species and neutrophil extracellular trap production, and endolysosomal signali
241 leukocyte phagocytosis, oxidative burst, and extracellular trap production, promoting bacterial survi
245 ur work documents and provides details about extracellular trap release in human neutrophils activate
246 mination of linker histones links neutrophil extracellular trap release with autoantibodies in system
248 of DNA fibrils with colocalized histones in extracellular traps released from bovine macrophages.
249 anulocyte activation causes the formation of extracellular traps, releasing web-like structures of DN
250 ave recently been shown to release DNA-based extracellular traps that contribute to microbicidal kill
251 echanisms, including formation of neutrophil extracellular traps through a recently described distinc
252 quired to induce the formation of neutrophil extracellular traps through multiple activation pathways
253 on of reactive oxygen species and neutrophil extracellular traps, two mechanisms utilized by neutroph
255 E in cancer patients by releasing neutrophil extracellular traps whereas monocytes may express TF.
256 us to initiate DNA extrusion into neutrophil extracellular traps, which bind NE and cathepsin G.
258 ils displayed a greater tendency to protrude extracellular traps, which were more strongly incorporat
259 neutrophils affected formation of neutrophil extracellular traps while not influencing phagocytosis,
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