ライフサイエンスコーパス: extracerebral

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1 egionally-specific lens protein aggregation, extracerebral amyloid formation, and supranuclear catara

2 aturation are based on the discrimination of extracerebral and cerebral tissue layers, and they can e

3 The cranial circulation, both extracerebral and cerebral, is innervated by fibers from

4 Extracerebral and intraventricular CSF was readily quant

5 ranial SCG targets, such as pineal gland and extracerebral blood vessels (bv).

6 he sympathetic innervation of representative extracerebral blood vessels [internal carotid artery (IC

7 TH protein associated with the extracerebral blood vessels was also significantly decre

8 TH levels in the SCG and extracerebral blood vessels were determined by Western b

9 the heart, external carotid artery, and the extracerebral blood vessels, as well as estrogen recepto

10 gets, the external carotid artery, heart and extracerebral blood vessels.

11 Unlike pineal gland and extracerebral bv, the external carotid artery, an extrac

12 findings provide a neural mechanism by which extracerebral cephalic blood flow couples to brain event

13 ability in a brain preparation isolated from extracerebral compartments.

14 We prospectively captured all extracerebral components of the Sequential Organ Failure

15 nt arms and was more closely associated with extracerebral distant metastases (P = .016) than with is

16 Cyclosporin A (CsA) is well known, for its extracerebral effect, as an immunosuppressant in organ t

17 nd Janeway lesions were associated with more extracerebral emboli (75.0% vs 31.8%, P = .02).

18 Seizure induction in the absence of extracerebral factors promoted the release of IL-1beta f

19 s induce brain inflammation independently on extracerebral factors.

20 However, the prevalence of extracerebral multiple organ dysfunction in postcardiac

21 all 12 dogs survived to 96 hrs without gross extracerebral organ damage (p < .0001).

22 and total brain histologic damage scores and extracerebral organ damage were assessed at 96 hrs.In no

23 diac arrest patients; 96% had some degree of extracerebral organ dysfunction and 66% had severe dysfu

24 ts of this study support the hypothesis that extracerebral organ dysfunction is common and associated

25 The most common extracerebral organ failures were cardiovascular (i.e.,

26 nd 66% had severe dysfunction in two or more extracerebral organ systems.

27 rolonged CPCR in dogs preserves viability of extracerebral organs and improves outcome.

28 Attenuation of water content of extracerebral organs with hypertonic saline treatment ma

29 0, severe damage), and morphologic damage of extracerebral organs.

30 nd the only blood in the jugular bulb was of extracerebral origin.

31 re prion diseases are usually transmitted by extracerebral prion infection, but clinical disease resu

32 progression-free survival, and freedom from extracerebral progression in univariable and multivariab

33 study was to explore the applicability of an extracerebral reference region for the quantification of

34 The highest extracerebral Sequential Organ Failure Assessment score

35 le organ dysfunction (defined as the highest extracerebral Sequential Organ Failure Assessment score)

36 nts in the MLPT group (all P </= .01), while extracerebral space was larger (P < .0001).

37 bral tissue (82+/-14 microM) with respect to extracerebral tissue (30+/-7 microM).

38 measured in cerebral tissue (56%+/-10%) and extracerebral tissue (62%+/-6%).

39 ts, where the top layer (layer 1) represents extracerebral tissue (scalp, skull, dura mater, subarach

40 concentration and saturation in cerebral and extracerebral tissue of adult human subjects, where the

41 on of images was performed by normalizing to extracerebral tissue.

42 e efficiently reducing lipid disturbances in extracerebral tissues.

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