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1  vehicle control immediately before starting extracorporeal circulation.
2 heir effects on the inflammatory response to extracorporeal circulation.
3  IL-10 (P=0.02) and peak IL-6 (P=0.02) after extracorporeal circulation.
4 eukin-10 (IL-10)] cytokines during and after extracorporeal circulation.
5 d at 5 time points before, during, and after extracorporeal circulation.
6 systemic heparinization in a rabbit model of extracorporeal circulation.
7 en cannula which was connected to an HRD via extracorporeal circulation.
8 with an exaggerated inflammatory response to extracorporeal circulation.
9 rombin formation or activity during in vitro extracorporeal circulation.
10 ombin formation and activity during in vitro extracorporeal circulation.
11 nosed in 2012 in 2 heart surgery patients on extracorporeal circulation.
12 ransfusion, sickle cell disease, sepsis, and extracorporeal circulation.
13 n which coronary perfusion was controlled by extracorporeal circulation.
14 L-10 rose significantly after institution of extracorporeal circulation and remained elevated through
15 ragile infants, extended the capabilities of extracorporeal circulation, and have brought new and inn
16 thrombosis, to inhibit contact activation in extracorporeal circulation, and to treat the swelling di
17 -hour hemodialysis session, during which the extracorporeal circulation blood flow was maintained bet
18          Surgical closure was performed with extracorporeal circulation by either direct suture (n=82
19  10) or an oscillation group (anesthesia and extracorporeal circulation for 20 hr with artificial PaO
20 andomized to a control group (anesthesia and extracorporeal circulation for 20 hr with constant PaO2,
21 n reducing fibrinolysis and blood loss after extracorporeal circulation; however, the effects of anti
22 e care unit) after cardiac surgery involving extracorporeal circulation in a series of 187 consecutiv
23                                              Extracorporeal circulation induces a systemic inflammato
24               High microbubble volume in the extracorporeal circulation loop and a high number of mic
25 moral artery and vein were cannulated and an extracorporeal circulation loop with 2 ultrasonic bubble
26 cine model of pulmonary vein ablation and an extracorporeal circulation loop.
27 re closely mimics the in vivo situation, but extracorporeal circulation may alter the physiologic int
28  as an index of the inflammatory response to extracorporeal circulation, may be useful in identifying
29                             After 10 mins of extracorporeal circulation, restoration of spontaneous c
30 but not monocyte activation during simulated extracorporeal circulation (SECC).
31 nts, these animals were placed on venovenous extracorporeal circulation through a 1-m control circuit
32 d and left untreated for 8 mins, after which extracorporeal circulation was started and its flow adju
33 ergoing coronary artery bypass grafting with extracorporeal circulation were randomly assigned in a d

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