ライフサイエンスコーパス: extracranial

1 iple stenotic segments (8 intracranial and 8 extracranial).

2 ere 24 (53.33%) intracranial and 21 (46.67%) extracranial.

3 ies, 71% male) underwent stent placement for extracranial (91%) and intracranial (9%) VAS from 1995 t

4 a-arterial treatment (IAT) in the setting of extracranial and intracranial lesions is considered chal

5 the skull) can help differentiate lesions of extracranial and intracranial origins.

6 anial aneurysms, angioplasty and stenting of extracranial and intracranial stenosis, as well as local

7 tematically investigate, capture, and record extracranial and intracranial venous drainage.

8 ding to previously reported criteria for the extracranial and transcranial US techniques.

9 ts with MS and 42 control subjects underwent extracranial and transcranial venous echo-color Doppler

10 le stenotic segments (16 intracranial and 13 extracranial) and 16 were multiple stenotic segments (8

11 ation in the coronary arteries, aortic arch, extracranial, and intracranial internal carotid arteries

12 Extracranial applications of diffusion-weighted (DW) mag

13 ) atherosclerosis of the aorta and the large extracranial arteries--the internal carotid arteries (IC

14 Extracranial arteriovenous malformations (AVMs) are rare

15 Asymptomatic extracranial artery stenosis (ECAS) is a well-known risk

16 ients with stroke compared with an excess of extracranial atherosclerosis and cardioembolic stroke in

17 tic stroke was 5.00 (95% CI, 1.69 to 14.76); extracranial atherosclerotic stroke, 1.71 (95% CI, 0.80

18 tic stroke was 5.85 (95% CI, 1.82 to 18.73); extracranial atherosclerotic stroke, 3.18 (95% CI, 1.42

19 We present unique case of a giant extracranial atretic occipital lipoencephalocele in an a

20 ic mutations in MAP2K1 are a common cause of extracranial AVM.

21 tial therapeutic agents for individuals with extracranial AVM.

22 We studied extracranial AVMs in order to identify their biological

23 expression of MMP-9 and MMP-2 in aggressive extracranial AVMs.

24 The main safety outcomes were major extracranial bleeding and ischemic or hemorrhagic stroke

25 Extracranial bleeding occurred in 32 patients (6.3%) in

26 %], respectively; p=0.55); the rate of major extracranial bleeding was higher with enoxaparin than wi

27 Major extracranial bleeding was increased by warfarin therapy

28 all stroke (ischemic and hemorrhagic), major extracranial bleeding, and death were extracted independ

29 es of ischemic and hemorrhagic stroke, major extracranial bleeding, and death.

30 Ischemic stroke, intracranial hemorrhage, extracranial bleeding, and myocardial infarction identif

31 ocation increased major gastrointestinal and extracranial bleeds (0.10%vs 0.07% per year, p<0.0001),

32 Case-fatality from major extracranial bleeds was also lower on aspirin than on co

33 mortality and the decrease in risk of major extracranial bleeds with extended use, and their low cas

34 ent cancer, major vascular events, and major extracranial bleeds, with stratification by age, sex, an

35 is a panarteritis that chiefly involves the extracranial branches of the carotid artery.

36 iscussed include hypertension, dyslipidemia, extracranial carotid and intracranial atherosclerotic di

37 n of the supraaortic arteries, including the extracranial carotid and vertebral arteries and intracra

38 Intimal medial thickness of the extracranial carotid arteries (IMT) is related to corona

39 ntified in the renal artery in 294 patients, extracranial carotid arteries in 251 patients, and verte

40 onsecutive patients who underwent CAS of 343 extracranial carotid arteries.

41 thickness and the degree of stenosis in the extracranial carotid arteries.

42 underwent elective (primary) stenting of 271 extracranial carotid arteries.

43 raphic examination of the left ventricle and extracranial carotid arteries.

44 or endovascular carotid interventions on the extracranial carotid artery between 2009 and 2014.

45 d to 75% of all strokes; for aortic arch and extracranial carotid artery calcification this incidence

46 Extracranial carotid artery disease accounts for approxi

47 Extracranial carotid artery pseudoaneurysms are rare cas

48 ess invasive method of revascularization for extracranial carotid artery stenosis.

49 hemodynamically significant stenoses of the extracranial carotid artery.

50 contemporary approaches to the management of extracranial carotid atherosclerotic occlusive disease a

51 The presence of extracranial carotid disease (ECD) is associated with le

52 Extracranial carotid disease was defined as cervical int

53 of both endovascular and open techniques for extracranial carotid revascularization.

54 arterectomy remains the standard of care for extracranial carotid stenosis except in specific clinica

55 d symptomatic and asymptomatic patients with extracranial carotid stenosis.

56 for Carotid Endarterectomy With Significant Extracranial Carotid Stenotic Disease).

57 e evaluation of the progression of volume of extracranial carotid vessel walls is feasible with 1.5-T

58 The extracranial carotid, renal, and intracranial arteries w

59 urysm; dissection most often occurred in the extracranial carotid, vertebral, renal, and coronary art

60 0.14 [95% confidence interval, 0.10-0.18] in extracranial carotids, and 0.11 [95% confidence interval

61 No major abnormalities were seen in extracranial cerebral vasculature in 15 patients.

62 It most commonly presents in the renal and extracranial cerebrovascular arteries, either manifestin

63 ion pressure (CPP) can worsen outcome due to extracranial complications of therapy.

64 CPP augmentation protocols that avoid these extracranial complications.

65 om the RVM for the expression of cranial and extracranial cutaneous allodynia, and are consistent wit

66 ough this shift has translated into improved extracranial disease control and patient outcomes, progr

67 h stratification factors of age, duration of extracranial disease control, number of brain metastases

68 Although extracranial disease is controlled with HER2 inhibitors

69 tage IV NSCLC with no more than six sites of extracranial disease who failed early systemic chemother

70 tified by number of metastases and status of extracranial disease.

71 ledge gaps in prevention of stroke caused by extracranial disease.

72 ,215 pediatric tumors representing sarcomas, extracranial embryonal tumors, brain tumors, hematologic

73 nal integrity and synapse recovery following extracranial facial nerve transection in mice.

74 anial aneurysm did not vary with location of extracranial FMD involvement.

75 cy, mutiplicity of intracranial lesions, and extracranial foci or sources of disease.

76 Management of paediatric extracranial germ-cell tumours carries a unique set of c

77 Yearly risk of extracranial haemorrhage was 1.4% (warfarin) versus 1.6%

78 posite of symptomatic intracranial and major extracranial haemorrhage was small and closely similar b

79 symptomatic intracranial haemorrhage, major extracranial haemorrhage, and all-cause mortality.

80 ratio [HR], 0.92 [95% CI, 0.65 to 1.28]) or extracranial hemorrhage (2.12 vs. 2.63 events per 100 pe

81 Absolute increases in major extracranial hemorrhage associated with antithrombotic t

82 Compared with warfarin, major extracranial hemorrhage associated with apixaban led to

83 Absolute increases in major extracranial hemorrhage were small (< or =0.3% per year)

84 rrhages, less adverse consequences following extracranial hemorrhage, and a 50% reduction in fatal co

85 -three children (median age, 7.8 years) with extracranial immature teratomas were enrolled on study.

86 nts (symptomatic intracranial or significant extracranial) in the first 14 days after stroke.

87 al principles have recently been extended to extracranial indications such as lung, spine, and liver

88 isk factors for infection were a concomitant extracranial infection (odds ratio, 2.34; 95% CI, 1.01-5

89 Patients with severe extracranial injuries (AIS >/= 3), death within 72 hours

90 re TBI (n = 10) with extra-cranial injury or extracranial injury only (EC) (n = 10), 92 inflammation-

91 thin film of subdural haemorrhage, but lack extracranial injury.

92 syndromes or acute anemic events (AAE), and extracranial internal carotid artery (eICA) stenoses, de

93 d the rate of acute anemic events (AAEs) and extracranial internal carotid artery (ICA) stenosis as r

94 (V(MCA)) to flow velocity in the ipsilateral extracranial internal carotid artery (V(ICA)) was calcul

95 Extracranial internal carotid artery atherosclerotic occ

96 tio of flow in the middle cerebral artery to extracranial internal carotid artery more than or equal

97 sisting of an in situ interposition graft or extracranial-intracranial bypass, is indicated only for

98 Recent studies suggest that extracranial involvement of giant cell arteritis (GCA) m

99 t 35 years, survival rates for children with extracranial malignant germ cell tumors (GCTs) have incr

100 ced stages of neuroblastoma, the most common extracranial malignant solid tumor of the central nervou

101 Neuroblastoma (NB) is the most common extracranial malignant solid tumor seen in children and

102 ectly, via either cerebrovascular changes or extracranial measurements of electrical/magnetic signals

103 tatus, EGFR exon 19 mutation, and absence of extracranial metastases were associated with improved OS

104 arnofsky performance score, age, presence of extracranial metastases, and number of brain metastases,

105 : patient age, Karnofsky Performance Status, extracranial metastases, and number of brain metastases.

106 Absence of extracranial metastases, Karnofsky performance score >/=

107 ER2-positive or triple-negative subtypes and extracranial metastases.

108 in HER2-positive breast cancer patients with extracranial metastases.

109 eatment with curative intent, three or fewer extracranial metastatic lesions on choline positron emis

110 ancer Study Group centers with biopsy-proven extracranial MT and IT and no prior chemotherapy.

111 Neuroblastoma, the most common solid extracranial neoplasm in children, is remarkable for its

112 inal primary afferent neurons than analogous extracranial neurons, making them potentially productive

113 f placebo in pediatric patients with primary extracranial, nonhematopoietic solid malignant tumors th

114 in children aged 5 to 18 years with primary extracranial, nonhematopoietic solid malignant tumors th

115 Patients with isolated extracranial occlusions were not included.

116 of imaging before treatment in patients with extracranial oligometastases.

117 n, intracranial stenosis is more common than extracranial one, anterior circulation stenosis is more

118 ssors, acute neurosurgical intervention, and extracranial operation.

119 ute brain infarction, 23.2% had at least one extracranial or intracranial stenosis of 50% or more, an

120 s emerged between MS and non-MS subjects for extracranial or intracranial venous flow rates.

121 econdary event related to atherosclerosis of extracranial or intracranial vessels.

122 rior cerebral artery involvement) or tandem (extracranial or intracranial) ICA and M1 occlusion subgr

123 igeminal primary afferent fibers innervating extracranial orofacial structures (such as the cornea, n

124 In most cases the extracranial part of the carotid artery is affected; the

125 over 1 year are more likely to suffer severe extracranial, particularly abdominal, injuries.

126 t duplex ultrasonographic examination of the extracranial parts of the carotid arteries.

127 his study was to determine whether there are extracranial pathophysiologies in these headaches.

128 ides the first set of evidence for localized extracranial pathophysiology in CM.

129 It is the most common extracranial pediatric solid tumor and the most common n

130 Neuroblastoma (NB) is the most common extracranial pediatric solid tumor with an undifferentia

131 Neuroblastoma, the most common extracranial pediatric solid tumor, is responsible for 1

132 To determine whether binding to an extracranial pool of 5-HT transporters contributes to th

133 It also identified 82% of extracranial primary tumor sites, of which 55% were foun

134 with placebo, among pediatric patients with extracranial progressive solid malignant tumors .

135 cerebral bv, the external carotid artery, an extracranial proximal SCG target, showed no change in NG

136 therapy compared with those without previous extracranial radiotherapy (0.59 [95% CI 0.36-0.96], p=0.

137 therapy compared with those without previous extracranial radiotherapy (HR 0.50 [0.30-0.84], p=0.0084

138 38 (39%) of 97 patients received extracranial radiotherapy and 24 (25%) of 97 patients re

139 3]) and for patients who previously received extracranial radiotherapy compared with those without pr

140 7]) and for patients who previously received extracranial radiotherapy compared with those without pr

141 exhibit unusual distributions compared with extracranial regions.

142 Extracranial rhabdoid tumours are rare, and often occur

143 ent was ICA, present in 14 (66.6%) out of 21 extracranial segments.

144 s delivery of ablative doses of radiation to extracranial sites.

145 scans for identifying NB in soft tissue and extracranial skeletal structures, for revealing small le

146 aging metric with excellent repeatability in extracranial soft tissues across a wide range of tumor s

147 ent diffusion coefficient (ADC) estimates in extracranial soft-tissue diffusion-weighted magnetic res

148 es are a frequent challenge in patients with extracranial solid cancers.

149 Neuroblastoma, the most frequent extracranial solid tumor in children, harbors the amplif

150 mpathetic nervous system, is the most common extracranial solid tumor in children.

151 Neuroblastoma is the most common extracranial solid tumor of childhood, and survival rema

152 Neuroblastoma (NB), the most common extracranial solid tumor of childhood, is responsible fo

153 children with neuroblastoma, the most common extracranial solid tumor of childhood.

154 l crest progenitor cells, is the most common extracranial solid tumor of childhood.

155 Neuroblastoma is the most common extracranial solid tumor of childhood.

156 Neuroblastoma, the most common extracranial solid tumor that occurs in early childhood,

157 Neuroblastoma is the most common childhood extracranial solid tumor.

158 otherapy dose of cediranib for children with extracranial solid tumors is 12 mg/m(2)/d administered o

159 ) recommendations in pediatric patients with extracranial solid tumors.

160 ger with high-risk, recurrent, or refractory extracranial solid tumors.

161 ma (HR-NB) is the most frequent, aggressive, extracranial solid tumour in childhood.

162 population in terms of age and frequency of extracranial sources of cerebrovascular disease.

163 mmunication of the subarachnoid space to the extracranial space, usually a paranasal sinus.

164 Most commonly involved extracranial stenosis segment was ICA, present in 14 (66

165 holesterolemia are the major risk factors of extracranial stenosis.

166 a greater side effect profile compared with extracranial stimulation, though all forms of stimulatio

167 son with atrial fibrillation-related stroke, extracranial systemic embolic events (SEEs) remain poorl

168 xy to deliver a high dose of radiation to an extracranial target in the body in a single dose or a fe

169 High doses of AZ10419369 exerted an extracranial tissue vasoconstriction that was comparable

170 ine headache triggered by pathophysiology of extracranial tissues, such as muscle tenderness and mild

171 iated pain but also nociceptive responses in extracranial tissues.

172 Low levels of accumulation were measured in extracranial tissues.

173 Consequently, a trial of extracranial-to-intracranial (EC/IC) arterial bypass for

174 s of < or =15 microg/dL (25th percentile for extracranial trauma patients) or one cortisol of < 5 mic

175 Neuroblastoma is a pediatric extracranial tumor and a major cause of death in childre

176 tumor stem cell markers for many intra- and extracranial tumor entities.

177 Neuroblastoma (NB) is the most common extracranial tumor in children.

178 meningioma patients but one with an atypical extracranial tumor showed high uptake of DOTATOC.

179 ession in the neovasculature of the primary, extracranial tumor.

180 f adult cancer, the reported experience with extracranial tumors of childhood is limited.

181 No significant differences in the extracranial venous systems between MS patients and HC s

182 The commonest occlusive sites were: extracranial vertebral artery (52 patients, 15 bilateral

183 d by thromboembolism from an intracranial or extracranial vessel, the heart, or the placenta.

184 ne differences between cerebral arteries and extracranial vessels and partly explain the technical ch

185 seen in larger cerebral vessel walls nor in extracranial vessels.

186 und among patients undergoing imaging of the extracranial vessels; however, there are no large studie