1 olk sac abnormalities due to a deficiency in extraembryonic mesoderm.

2 in embryonic-derived lineages, including the extraembryonic mesoderm.

3 o signals from the primitive endoderm or the extraembryonic mesoderm [1,2].

4        Slugh expression is first detected in extraembryonic mesoderm and is later detected in many me

5 onsistent with the expression of Bmp2 in the extraembryonic mesoderm cells and promyocardium.

6      Depleting Zfp568 affects the ability of extraembryonic mesoderm cells to migrate.

7 l-1, rbtn2, GATA2, and GATA-1 in a subset of extraembryonic mesoderm cells.

8 c ectoderm (ExE) and in the epiblast-derived extraembryonic mesoderm (ExM), in which the PGCs, allant

9         Each of these genes was expressed in extraembryonic mesoderm, from which blood islands are de

10 zygote-derived tetraploid tissues implicates extraembryonic mesoderm in manifesting the effects of ge

11  support previously undescribed roles of the extraembryonic mesoderm in yolk sac morphogenesis and in

12 These results show that BMP4 produced in the extraembryonic mesoderm is directly influencing the SMAD

13 embryonic mesoderm is produced; in contrast, extraembryonic mesoderm is relatively abundant.

14 ruffling of the yolk sac membrane, defective extraembryonic mesoderm morphogenesis and vasculogenesis

15 y non-cardiogenic posterior mesoderm and the extraembryonic mesoderm of the amnion.

16                 It is first expressed in the extraembryonic mesoderm of the yolk sac within the morph

17 y migrate from their initial location in the extraembryonic mesoderm to the genital ridge, the gonada

18 ives, including the primitive heart, gut and extraembryonic mesoderm, whereas it is nonessential in t