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1 flammation and fibrosis of the intra- and/or extrahepatic bile ducts.
2 pithelial malignant neoplasm of the liver or extrahepatic bile ducts.
3 mechanistically linked to obstruction of the extrahepatic bile ducts.
4 Sar among blood, hepatocytes, and intra- and extrahepatic bile ducts.
5 he inflammatory and fibrosing obstruction of extrahepatic bile ducts.
6 ter than 90% mortality due to obstruction of extrahepatic bile ducts.
7 by age at surgery and anatomy of the atretic extrahepatic bile ducts.
8 sulting in dramatic and rapid enlargement of extrahepatic bile ducts.
10 SC were managed with either resection of the extrahepatic bile ducts and long-term transhepatic stent
11 h biliary tract cancer (237 gallbladder, 127 extrahepatic bile duct, and 47 ampulla of Vater), 895 wi
13 gkin lymphoma (P < .001) and gallbladder and extrahepatic bile duct cancer (P = .01) was observed.
14 higher: 100% for gallbladder cancer, 97% of extrahepatic bile duct cancer, 91% of ampula of Vater ca
15 the body and tail of pancreas, cancer of the extrahepatic bile duct, cancer of the gallbladder, and c
16 d cholangiocytes, that line intrahepatic and extrahepatic bile ducts, contribute substantially to bil
20 of birth, microdissected the gallbladder and extrahepatic bile ducts en bloc 3, 7, and 14 days later,
21 and active caspase-3 staining in livers and extrahepatic bile ducts from Balb/c mice infected with R
22 epatic bile duct in 25 (100%), the recipient extrahepatic bile duct in 17 of 18 (94%), and the anasto
23 c bile ducts in 23 (92%) patients, the donor extrahepatic bile duct in 25 (100%), the recipient extra
24 m an association between age and size of the extrahepatic bile duct in an asymptomatic adult populati
27 inflammatory process that affects intra- and extrahepatic bile ducts, leading to fibrosis and obliter
28 tal stents compared with patients with other extrahepatic bile duct malignant diseases and patients t
29 cholangiocarcinoma (n = 11) or by secondary extrahepatic bile duct malignant tumors (n = 11) were tr
30 ered or partially covered) for palliation of extrahepatic bile duct obstruction initially is more exp
31 related to total bilirubin, indicating that extrahepatic bile duct obstruction leads to down-regulat
33 primed CD8(+) cells preferentially homed to extrahepatic bile ducts of neonatal mice and invaded the
34 role of T lymphocytes in the destruction of extrahepatic bile ducts of neonatal mice using an experi
36 f CD4(+) and CD8(+) T cells within liver and extrahepatic bile duct remnant tissues, indicating the p
37 Vbeta repertoire of T cells from the liver, extrahepatic bile duct remnants, and peripheral blood of
40 progressive fibroinflammatory obstruction of extrahepatic bile ducts that presents as neonatal choles
41 truction of segments or the entire length of extrahepatic bile ducts, the timely pursuit of hepatopor
42 sverse and anteroposterior dimensions of the extrahepatic bile duct were measured proximally at the p
43 epithelial and subepithelial compartments of extrahepatic bile ducts, with onset within 3 days and pe
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