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1  the forebrain at E32-E34 and settled in the extrahypothalamic area.
2  results do not exclude the possibility that extrahypothalamic areas are also modulating the effects
3                                              Extrahypothalamic areas known to be important for HPA ac
4 vity in the rat hypothalamus and a number of extrahypothalamic areas.
5 when injected into multiple hypothalamic and extrahypothalamic areas.
6                              Transmitters of extrahypothalamic arousal systems, including norepinephr
7                                       Within extrahypothalamic autonomic control sites, MC4-R-specifi
8 ith both local intrahypothalamic and distant extrahypothalamic axonal projection sites.
9 nections with multiple intrahypothalamic and extrahypothalamic brain areas.
10 n GAD(65) protein levels in hypothalamic and extrahypothalamic brain regions known to be sexually dim
11 nding of estrogen action in hypothalamic and extrahypothalamic brain regions.
12 rrent study examined the induction of CRF in extrahypothalamic brain sites following generalized clon
13 ed amygdala, which is a key substrate of the extrahypothalamic brain stress system.
14                                              Extrahypothalamic cell groups activated in response to a
15 nd lateral hypothalamic (LHA) areas; and the extrahypothalamic central nucleus of the amygdala (CeA).
16             Faulty regulation of the central extrahypothalamic corticotropin-releasing factor (CRF) e
17 ation of brain stress systems, including the extrahypothalamic corticotropin-releasing factor (CRF) s
18                                              Extrahypothalamic corticotropin-releasing hormone (CRH)
19   We propose that recruitment of anti-reward extrahypothalamic CRF-CRF(1) systems during withdrawal f
20 axis is proposed for acquisition, whereas an extrahypothalamic CRF/CRF1 participation is suggested fo
21 lt in long-term changes in the expression of extrahypothalamic CRH.
22                              A major site of extrahypothalamic expression of corticotropin-releasing
23 n, Y1-R hybridization was evident in several extrahypothalamic forebrain and hindbrain sites involved
24 ir body fat declined 36%, suggesting that an extrahypothalamic mechanism was responsible.
25                                        These extrahypothalamic neurons contain LHRH fragments, rather
26 ransporter immunoreactivity predominantly in extrahypothalamic POMC terminals.
27                                  The densest extrahypothalamic projection was found in the locus coer
28  implanted with stereotaxic cannulae into an extrahypothalamic region termed the dorsal vagal complex
29 eurons, and is ultimately distributed in the extrahypothalamic region.
30 rs and neuronal elements are present in many extrahypothalamic regions of the brain.
31 rred throughout the hypothalamus and in some extrahypothalamic regions, consistent with the known dis
32 lar activation responses in the PVH and most extrahypothalamic regions.
33  has shown increased CRH immunoreactivity in extrahypothalamic sites after kainic-acid (KA)-induced s
34 mRNA and EYFP expression in hypothalamic and extrahypothalamic sites described before, including the
35 roinjected into a number of hypothalamic and extrahypothalamic sites in rats.
36  The localization of leptin receptor mRNA in extrahypothalamic sites in the thalamus and cerebellum s
37       Leptin receptors are also expressed in extrahypothalamic sites including the ventral tegmental
38 ucleus of the hypothalamus (PVN) and several extrahypothalamic sites where expression is activity-dep
39 tagogue receptor (GHSR)] are also present in extrahypothalamic sites where they promote circuit activ
40 n of leptin, both in the hypothalamus and in extrahypothalamic sites within the CNS, and shows our cu
41 pression, regulation, and function of CRF at extrahypothalamic sites.
42 after abstinence, through recruitment of the extrahypothalamic stress peptide corticotropin-releasing
43     Emphasis is placed on the role of CRF in extrahypothalamic systems in the extended amygdala, incl
44 releasing factor (CRF) and norepinephrine in extrahypothalamic systems in the extended amygdala, incl
45 asis on the neuropharmacological function of extrahypothalamic systems in the extended amygdala.
46 ocannabinoid signaling from hypothalamic and extrahypothalamic target neurons.
47  ghrelin also activates GHS-R1A receptors on extrahypothalamic targets that mediate alcohol reward.
48 e been identified in the hypothalamus and in extrahypothalamic tissues.

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