ライフサイエンスコーパス: extraneural

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1 transgenic model is the first to demonstrate extraneural accumulation of PrPres in spleen and intesti

2 Extraneural accumulation of PrPres was also noted in spl

3 ent that CaMKII is also expressed in various extraneural cells.

4 Here we demonstrate direct extraneural effects of leptin to deplete fat content of

5 d with recombinant leptin, indicating direct extraneural effects.

6 on of LRS tissues in neuroinvasion following extraneural inoculation with the HY and DY strains of th

7 We investigated extraneural manifestations in scrapie-infected transgeni

8 rearranged and archetype forms in brain and extraneural organs in all three study groups.

9 In other extraneural organs, the virus was generally associated w

10 ential mechanism for virus dissemination and extraneural pathogenesis.

11 In the early extraneural phase, there was primary amplifying replicat

12 f these patients concurrently had documented extraneural (pulmonary) infection as a result of the sam

13 279 Lys virus was restricted with respect to extraneural replication in monkeys, as viremia and antib

14 pleen and lymph nodes are important sites of extraneural replication, from which infection is likely

15 ted action limits initial DEN replication in extraneural sites and controls subsequent viral spread i

16 CNS metastases from extraneural sites are also briefly covered.

17 persistence in the kidney and possibly other extraneural sites.

18 arise from direct effects of the hormone on extraneural tissues and what intracellular mechanisms ar

19 o diabetes of human CB1 receptor isoforms in extraneural tissues involved in glucose metabolism.

20 ct replication efficiency in both neural and extraneural tissues of the mouse and confer some limited

21 kinase (Trk) receptors also are expressed in extraneural tissues.

22 ributed to the virus, is not associated with extraneural virus dissemination or replication and does

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