ライフサイエンスコーパス: extrapancreatic

1 reatic solid tumours and history of previous extrapancreatic cancer underwent EUS-FNA from January/19

2 Patients with BD-IPMN are not at risk of extrapancreatic carcinogenesis.

3 ions (DNA sequencing) and 11 were tested for extrapancreatic CFTR function (clinical and physiologic

4 tes with having 2 CFTR mutations and reduced extrapancreatic CFTR function.

5 , serum alkaline phosphatase, and absence of extrapancreatic cysts predict patients likely to progres

6 (85%) of 86 patients were taken to surgery, extrapancreatic disease was found in nine, and 64 (74%)

7 riteria for resectability: 1) the absence of extrapancreatic disease, 2) no tumor encasement of the s

8 50 hpf, a stage when the ventral bud-derived extrapancreatic duct is the main source of new endocrine

9 ntly increased beta-cell neogenesis near the extrapancreatic duct, demonstrating the feasibility of p

10 olypeptide (GIP) is an incretin hormone with extrapancreatic effects beyond glycemic control.

11 m of trypsin that is co-expressed in several extrapancreatic epithelial cells with ENaC, can activate

12 one marrow of diabetic rats, indicating that extrapancreatic, extrathymic insulin production occurs i

13 Extrapancreatic features were observed in all 24 proband

14 s frequently part of a complex syndrome with extrapancreatic features: 18 genes causing syndromic neo

15 In this analysis, a cluster of extrapancreatic foregut markers, including pepsinogen C,

16 We investigated whether extrapancreatic forms of trypsin are PAR agonists.

17 e developed independently for pancreatic and extrapancreatic gastrointestinal NETs, with novel therap

18 These findings emphasize the existence of extrapancreatic glucagon (perhaps originating from the g

19 ing gastric emptying rather than by altering extrapancreatic glucose metabolism.

20 dipocytes and macrophages is an evidence for extrapancreatic insulin-producing cells.

21 Extrapancreatic invasion occurred in nine cases (75%), a

22 that absence of an autoantigen in syngeneic extrapancreatic islet grafts in diabetic hosts renders t

23 In type 2 diabetic subjects, extrapancreatic KATP channel activation with diazoxide u

24 ug-binding channel-modulating subunit of the extrapancreatic KATP channel.

25 reatic lymphocytes and was not detectable at extrapancreatic lymphoid sites.

26 inous neoplasm (IPMN) is infrequent and that extrapancreatic malignancies (EPMs) occur with unusual f

27 oma which are distinct from its influence in extrapancreatic malignancies and from the mechanistic ef

28 sis assessed the incidence of pancreatic and extrapancreatic malignancies in BD-IPMN patients.

29 sease in patients with a previous history of extrapancreatic malignancies.

30 escribed the clinical profile of AIP and its extrapancreatic manifestations.

31 The extrapancreatic necrosis AUC was the highest for all sys

32 e criterion such as a threshold of 100 mL of extrapancreatic necrosis provides more reliable informat

33 significant relationships were found between extrapancreatic necrosis volume and organ failure, infec

34 Extrapancreatic necrosis volume, Balthazar score, and CT

35 nd intraobserver agreement in the grading of extrapancreatic necrosis was assessed by using kappa sta

36 crosis of more than 50% of the pancreas, and extrapancreatic necrosis.

37 The proportion of IPMN patients having any extrapancreatic neoplasm diagnosed before or coincident

38 as used to assess the risk of a diagnosis of extrapancreatic neoplasms among cases versus controls.

39 l studies have reported an increased risk of extrapancreatic neoplasms in patients with IPMN, but the

40 s with IPMN have increased risk of harboring extrapancreatic neoplasms.

41 nic inflammation, and tumor metastases along extrapancreatic nerves are key features of pancreatic ma

42 in molecular genetics between pancreatic and extrapancreatic NETs, and studies are evaluating whether

43 e pancreas, intrapancreatic nerves, and some extrapancreatic neural pathways, with or without mediati

44 on histology, imaging, endoscopy, serology, extrapancreatic organ involvement, and response to stero

45 P are based on histology, imaging, serology, extrapancreatic organ involvement, and response to stero

46 1Rs) are also widely distributed in multiple extrapancreatic organs, providing a mechanistic explanat

47 le disease but have preoperative findings of extrapancreatic perineural invasion (EPNI) and/or duoden

48 ients, mesenteric vascular encasement in 14, extrapancreatic/peritoneal involvement in 16, and celiac

49 antation experiments showed that most of the extrapancreatic proinsulin-producing cells originated fr

50 The most commonly involved extrapancreatic sites are bile duct, kidney, and retrope

51 biochemical phenotype that protects against extrapancreatic tissue injury to the lung, kidney and li

52 pancreatic adenocarcinomas, and other human extrapancreatic tissues and malignancies was examined, u

53 imens, 22 pancreatic adenocarcinomas, and 58 extrapancreatic tissues and tumors was subjected to RT-P

54 ll tissues arising from pancreas and in most extrapancreatic tissues and tumors.

55 did not observe bioluminescent signals from extrapancreatic tissues of diabetic MIP-TF mice.

56 1R signaling, which has diverse functions in extrapancreatic tissues, remains elusive.

57 erglycemia inducing proinsulin expression in extrapancreatic tissues, we did not observe bioluminesce

58 imilar to protease, serine (PRSS) 3, a major extrapancreatic trypsinogen, was optimum at pH 8.0, and