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1 function, and three of them developed subtle extrapyramidal signs.
2 ough a subgroup developed moderate to severe extrapyramidal signs.
3                       All subjects developed extrapyramidal signs.
4 ding frontotemporal dementia, pyramidal, and extrapyramidal signs.
5 duced, although there was a mild increase in extrapyramidal signs; 112 patients met the criteria for
6 ogeneous group of disorders with progressive extrapyramidal signs and neurological deterioration, cha
7 nce of myoclonus, seizures, pyramidal signs, extrapyramidal signs, and cerebellar signs) from all ind
8  as well as the development of myoclonus and extrapyramidal signs are consistent manifestations of di
9 eimer's disease (AD) is often accompanied by extrapyramidal signs attributed to nigrostriatal dysfunc
10 ertension, diabetes mellitus, heart disease, extrapyramidal signs, depression, psychosis, aggression,
11            Patients with AD (with or without extrapyramidal signs) did not show neuronal loss in the
12 ulative antipsychotic doses, and presence of extrapyramidal signs early in treatment.
13                                              Extrapyramidal signs frequently accompany Alzheimer's di
14 e of frontal lobe release signs, presence of extrapyramidal signs, gait disturbance, history of falls
15  typical cases, svPPA-tau showed significant extrapyramidal signs, greater executive impairment, and
16                          Age and severity of extrapyramidal signs have been consistently associated w
17                                        Thus, extrapyramidal signs in AD correlate best with tangle pa
18 the basal ganglia or subthalamic nucleus and extrapyramidal signs in AD.
19  neuropil threads were positively related to extrapyramidal signs in AD.
20  disease associated with age and severity of extrapyramidal signs is related primarily to their combi
21 threads was increased in AD (with or without extrapyramidal signs) nigra compared to control tissue,
22  and combined effects of age and severity of extrapyramidal signs on the risk of incident dementia in
23 llitus, heart disease, incident strokes, and extrapyramidal signs, only conventional antipsychotic us
24 (mMMS) score, and the presence or absence of extrapyramidal signs or psychotic features.
25 nts included abnormal involuntary movements, extrapyramidal signs, psychiatric symptoms, and medical
26                                  In phase A, extrapyramidal signs tended to be greater with the stand
27 amination scores was slower, the presence of extrapyramidal signs was decreased, and the development
28                              The presence of extrapyramidal signs was determined using the Unified Pa

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