1 ncy and heparin administration as a cause of
extraskeletal 99mTc bone tracer accumulation.
2 There were no
extraskeletal abnormalities and no relationship between
3 epiction, location, and characterization and
extraskeletal abnormalities were evaluated.
4 Pathologic
extraskeletal bone formation, or heterotopic ossificatio
5 (FOP) is a rare but devastating disorder of
extraskeletal bone formation.
6 pamycin significantly decreased or inhibited
extraskeletal bone formation.
7 Extraskeletal bone forms through an endochondral process
8 ic ossification, the pathologic formation of
extraskeletal bone, occurs as a common complication of t
9 ising target to prevent and treat pathologic
extraskeletal bone.
10 Secondary hyperparathyroidism contributes to
extraskeletal calcification and is associated with all-c
11 mic patients are prone to widespread ectopic
extraskeletal calcification resulting from an imbalance
12 nisms are etiological agents of pathological
extraskeletal calcification.
13 moral (0% to 12%; P=0.13) artery plaques and
extraskeletal calcifications (34.4% to 65.6%; P=0.006) i
14 vitamin D deficiency is associated with many
extraskeletal chronic disorders, including the autoimmun
15 Secondary hyperparathyroidism contributes to
extraskeletal complications in chronic kidney disease.
16 at is essential for modeling of skeletal and
extraskeletal connective tissues.
17 ntegrate therapies for both the skeletal and
extraskeletal consequences with a view toward improving
18 ical setting and result in both skeletal and
extraskeletal consequences.
19 oid CS is pathogenetically distinct from its
extraskeletal counterpart.
20 (18)F-FDG PET/CT and whole-body MRI detected
extraskeletal disease that may change the management of
21 develop hypocalcemia, rickets, and possibly
extraskeletal disorders (e.g., type 1 diabetes).
22 ssiva, a disorder characterized by extensive
extraskeletal endochondral bone formation, share a recur
23 rimary renal primitive neuroectodermal tumor/
extraskeletal Ewing's sarcoma (PNET/EES) is a very rare
24 matopoietic abnormalities do not result from
extraskeletal expression of endogenous collagen X or the
25 provided evidence for a role of vitamin D in
extraskeletal health.
26 rimary tumor was bone in 57% of patients and
extraskeletal in 43% of patients.
27 parathyroidism, are thought to contribute to
extraskeletal (
including vascular) calcification among p
28 The (18)F(-)/(18)F-FDG uptake in
extraskeletal lesions showed no significant difference w
29 to-background ratio of (18)F(-)/(18)F-FDG in
extraskeletal lesions showed no significant difference w
30 For
extraskeletal lesions, (18)F-NaF/(18)F-FDG PET/CT and wh
31 Extraskeletal manifestations have not been reported in P
32 haracterized by brittle bones, fractures and
extraskeletal manifestations.
33 fic antigen or radiological), baseline pain,
extraskeletal metastases, and bisphosphonate use, were r
34 ny metastasis, even in the presence of other
extraskeletal metastases, is not recommended.
35 Molecular studies have established that
extraskeletal myxoid chondrosarcoma is a unique entity d
36 A proportion of
extraskeletal myxoid chondrosarcomas (EMC) have been sho
37 c and molecular genetic findings, except for
extraskeletal myxoid chondrosarcomas.
38 molecular genetic changes are diagnostic of
extraskeletal myxoid chondrosarcomas.
39 Notably, 2 cases of
extraskeletal myxoid CS showed neither an EWS/CHN fusion
40 Extraskeletal myxoid CS thus represents yet another sarc
41 ent, suggesting genetic heterogeneity within
extraskeletal myxoid CS.
42 , has been recognized in CS, specifically in
extraskeletal myxoid CS.
43 en characterized in only a limited number of
extraskeletal myxoid CSs and its presence in other types
44 he EWS/CHN gene fusion was present in 6 of 8
extraskeletal myxoid CSs and was not detected in any of
45 We studied 46 cases of CS (8
extraskeletal myxoid, 4 skeletal myxoid, 4 mesenchymal,
46 Extraskeletal new bone formation, as seen in myositis os
47 er of skeletal malformations and progressive
extraskeletal ossification.
48 ormation, as seen in myositis ossificans and
extraskeletal osteosarcoma, represents an exception wher
49 It remains unclear whether skeletal or
extraskeletal problems will develop postnatally from exp
50 ites during development, suggesting possible
extraskeletal roles for these proteins.
51 s to prevent or treat development of bone in
extraskeletal sites.
52 eletal health, and might also have a role in
extraskeletal tissues.
53 Mice bearing
extraskeletal tumors show significantly elevated levels
54 There was, however,
extraskeletal uptake of the 99mTc bone tracer in the ant