ライフサイエンスコーパス: extrastriate

1 recent suggestions, stress amplified earlier extrastriate activity in a manner consistent with vigila

2 models propose that conscious experience of extrastriate activity requires the integrity of primary

3 Although change blindness resulted in some extrastriate activity, the dorsal activations were clear

4 he exuberant functional connectivity between extrastriate and 'core' semantic retrieval regions might

5 ast and local responses in V1, as well as in extrastriate and contralateral cortical areas.

6 dependent responses in functionally affected extrastriate and frontoparietal regions in AD, while per

7 emapping, we recorded from single neurons in extrastriate and striate cortex while monkeys performed

8 been shown to affect neural activity in both extrastriate and striate visual cortex.

9 lucinations, the forms of which suggest that extrastriate and temporal lobe involvement contributes t

10 ual cortex (V1) with the higher lateromedial extrastriate area (LM) are pyramidal cells (Pyr) and par

11 ion that converts the population response in extrastriate area MT into estimates of target motion to

12 y reported sample of neuronal responses from extrastriate area MT.

13 nd the motion processing neurons residing in extrastriate area MT.

14 The responses of extrastriate area V4 neurons to flashed visual stimuli w

15 lates the visual responses of neurons within extrastriate area V4, where the responses to targets are

16 how that observers' awareness of activity in extrastriate area V5 depends on the amount of activity i

17 how that both primary visual cortex (V1) and extrastriate area V5/MT are causally involved in encodin

18 ve field (ARF) of the labeled region in each extrastriate area with that of the injection site.

19 stream areas MT and V3, but not the earliest extrastriate area, V2, nor ventral stream area V4.

20 primary visual area and feedback activity in extrastriate areas (C1 and N1 reduction).

21 Thus, these extrastriate areas contain neurons that are sensitive to

22 LM is homologous to V2 and that the lateral extrastriate areas do not represent modules within a sin

23 All extrastriate areas except LI increase orientation select

24 Architectural features of striate and extrastriate areas in prosimian galagos are similar to s

25 ed neurons were also found in other proposed extrastriate areas such as the dorsomedial visual area (

26 In mice, several extrastriate areas surrounding V1 have been described.

27 trates a lateralized disturbance in the left extrastriate areas that extract visual motion informatio

28 ed retinotopically and contains at least ten extrastriate areas that likely integrate more complex vi

29 how neurons located along the progression of extrastriate areas that, in the rat brain, run laterally

30 visual cortex are routed to and processed by extrastriate areas to mediate the diverse capacities of

31 [2, 3] has shown that back projections from extrastriate areas to the primary visual area (V1) deter

32 mispheric correlation of resting activity in extrastriate areas was reduced in anophthalmia to the le

33 he source area: outputs from medial/anterior extrastriate areas were more strongly linked to parietal

34 motor, and limbic cortices, whereas lateral extrastriate areas were preferentially connected to temp

35 pulvinar induced fast and local responses in extrastriate areas, followed by weak and diffuse activat

36 ulvinar activation to be different in V1 and extrastriate areas, reflecting the different connectivit

37 ue primary visual cortex (V1) project to two extrastriate areas, the second visual area (V2) and the

38 ment progressively increased from striate to extrastriate areas.

39 s striate cortex, it fans out to a number of extrastriate areas.

40 stimuli produced normal responses in V1 and extrastriate areas.

41 later changes in visual perception depend on extrastriate areas.

42 n of thalamocortical pathways in striate and extrastriate areas.

43 rior midline, caudal posterior parietal, and extrastriate areas.

44 dominantly to layer I in V1, and layer IV in extrastriate areas.

45 ailable for further processing in downstream extrastriate areas.

46 nificant feedback to V1 from layer IV of all extrastriate areas.

47 effect of V1-bypassing geniculate input into extrastriate areas.

48 eld of stimulus presentation but have intact extrastriate attention activity.

49 ogical deficits manifested by attenuation of extrastriate attention and VWM-related neural activity o

50 the distribution of dLGN cells projecting to extrastriate bears a striking similarity to that of neur

51 etwork in human visual cortex comprising the extrastriate body area (EBA) and the fusiform body area

52 ulation (TMS) to investigate the role of the extrastriate body area (EBA) in the detection of people

53 Multivoxel fMRI activity patterns in the extrastriate body area (EBA), but not in the posterior s

54 l occipital complex (LO), and body-selective extrastriate body area (EBA).

55 engaged the PPC, PMv, and the body-selective extrastriate body area (EBA); activity in the PMv moreov

56 t occipital face area (rOFA) [12], the right extrastriate body area (rEBA) [13], or the right lateral

57 ed right occipital face area (rOFA) or right extrastriate body area (rEBA) at different latencies, up

58 We separately inhibited the extrastriate body area and dorsal premotor cortex in 11

59 In posterior temporal cortex, the extrastriate body area is associated with perceiving the

60 These findings suggest that the right extrastriate body area plays a compensatory role in PD b

61 gion in human lateral occipital cortex (the 'extrastriate body area' or EBA) has been implicated in t

62 orm pathway (e.g., fusiform regions, ventral extrastriate body area) are not critical for biological

63 Following inhibition of the right extrastriate body area, the posture congruency effect wa

64 on in the human occipito-temporal cortex-the extrastriate body area-compensates for altered dorsal pr

65 N1 responses as correlates of activation in extrastriate body area.

66 wning that is anatomically distinct from the extrastriate body area.

67 alized neurons that are clustered within the extrastriate brain.

68 rties, object identity could be decoded from extrastriate, but not prefrontal, cortex, whereas the op

69 he eyes influence the development of striate-extrastriate, but not the size of striate cortex, ends b

70 e are few quantitative measurements of human extrastriate color specializations.

71 ys on the development of ipsilateral striate-extrastriate connections and the interplay that might ex

72 y reflecting vasodilation), developed within extrastriate cortex (area V3A).

73 produced fMRI activity in the right or left extrastriate cortex (BA18), respectively.

74 In extrastriate cortex (from V2 to LO), on the other hand,

75 level information but this diminished in the extrastriate cortex (LO-1/LO-2/LOC), in which the abstra

76 , areas at intermediate processing stages in extrastriate cortex (V4, V3A, MT and V7) showed object-s

77 results of less left posterior cingulate and extrastriate cortex activation in alcoholics than contro

78 at cholinesterase inhibition enhances visual extrastriate cortex activity during stimulus encoding, e

79 significance of overlapping fMRI activity in extrastriate cortex and, by extension, elsewhere in the

80 asurements suggest that regions within human extrastriate cortex are specialized for different percep

81 tivity increased from early visual cortex to extrastriate cortex but then decreased in anterior regio

82 g that the overall category-selective map in extrastriate cortex develops independently from visual e

83 s visual attention by increasing activity in extrastriate cortex generally, it accomplishes this in a

84 It is controversial whether mouse extrastriate cortex has a "simple" organization in which

85 monstrate that direct LGN projections to the extrastriate cortex have a critical functional contribut

86 led studies of the thalamic relationships of extrastriate cortex in apes and humans.

87 -mm voxels), we identified a small region of extrastriate cortex in most participants that responds s

88 rst extensive study of how amblyopia affects extrastriate cortex in nonhuman primates.

89 pathway by which visual information reaches extrastriate cortex in the absence of V1.

90 e differing proposals on the organization of extrastriate cortex in three species of New World monkey

91 Activation in extrastriate cortex increased after a shift of attention

92 effects, we infer that back-projections from extrastriate cortex influence information content in V1,

93 The most direct pathway identified to the extrastriate cortex is a disynaptic one that provides ro

94 of visual-selective processing in posterior extrastriate cortex is disrupted in schizophrenia.

95 lternative explanation, which is that intact extrastriate cortex is required for mediating voluntary

96 re altered, the functional specialization of extrastriate cortex is retained regardless of visual exp

97 al cortex, but its contribution to coding in extrastriate cortex is unexplored.

98 extran amine (BDA) injections into the SG or extrastriate cortex labeled inputs terminating primarily

99 , the large-scale organization of high-level extrastriate cortex likely reflects the need for both sp

100 pport the notion that population activity in extrastriate cortex limits the precision of both visual

101 -related activation during imagery in visual extrastriate cortex may be implemented by "top-down" mec

102 ns in the middle temporal area (MT or V5) of extrastriate cortex of alert macaque monkeys.

103 of intrinsic responses to visual stimuli in extrastriate cortex of owl monkeys provided evidence for

104 cal task that is performed by neurons in the extrastriate cortex of the primate brain.

105 PFC disruption decreased the tuning of extrastriate cortex responses, coinciding with decrement

106 nhibitors can improve memory is by enhancing extrastriate cortex stimulus selectivity at encoding, in

107 and intraparietal clusters with frontal and extrastriate cortex suggested correspondences with areas

108 Large islands of extrastriate cortex that are enriched for color-tuned ne

109 initial gain enhancement in anterior ventral extrastriate cortex that is coarsely selective for the t

110 the topography of feedback projections from extrastriate cortex to macaque area 17.

111 The spatial scale of feedback circuits from extrastriate cortex to V1 is, instead, commensurate with

112 patches and the topographic organization of extrastriate cortex using biologically relevant, phase-e

113 owed that modulation of neural processing in extrastriate cortex was significantly enhanced by attent

114 of brain activation in the left frontal and extrastriate cortex were made in adults and children (ag

115 greater connectivity between this region and extrastriate cortex were the most resistant to PFC disru

116 ons among multiple stimuli are eliminated in extrastriate cortex when they are presented in the conte

117 cal terminals are densely distributed in the extrastriate cortex where they form synaptic connections

118 d to its pallial target, the entopallium (E, extrastriate cortex).

119 s to posterior and ventral areas of temporal extrastriate cortex, areas TP and TPI.

120 content-related activation during imagery in extrastriate cortex, but this activity was restricted to

121 hat global motion sensitivity, a property of extrastriate cortex, can be altered by early visual depr

122 sidered the preserve of "category-selective" extrastriate cortex, can nevertheless emerge in retinoto

123 ite matter most prominently in right ventral extrastriate cortex, close to an area previously implica

124 object enhances its neural representation in extrastriate cortex, compared with those of unattended o

125 tinct areas: the striate cortex (V1) and the extrastriate cortex, consisting of V2 and numerous highe

126 to develop robust visual function in primate extrastriate cortex, highlighting a likely mechanism for

127 area (FFA), a face-selective region in human extrastriate cortex, is a matter of active debate.

128 f participation of the left posterior insula/extrastriate cortex, left superior frontal and right ant

129 hey receive from the retina to virtually all extrastriate cortex, parsing this information into dorsa

130 In the macaque extrastriate cortex, robust correlations between percept

131 , and individual patches receive inputs from extrastriate cortex, the medial temporal lobe, and three

132 ecting an attentional selection mechanism in extrastriate cortex, was reduced in amplitude with advan

133 plex" in having a string of areas in lateral extrastriate cortex, which receive direct V1 input.

134 However, one additional region, extrastriate cortex, which was highly active when watchi

135 ct varied substantially across subregions of extrastriate cortex, with some showing a twofold increas

136 matching was found to originate from ventral extrastriate cortex, with the former being generated in

137 ely confined to acallosal regions throughout extrastriate cortex.

138 ree adjacent functionally localized areas in extrastriate cortex.

139 ns; the latter likely involves feedback from extrastriate cortex.

140 ift attention influence sensory responses in extrastriate cortex.

141 " (odd-symmetric) tuning becomes dominant in extrastriate cortex.

142 down biasing effects on pooling processes in extrastriate cortex.

143 n fused (versus flicker) trials in occipital extrastriate cortex.

144 in disparity processing) between striate and extrastriate cortex.

145 "matching" computation possibly performed in extrastriate cortex.

146 etition in favor of the attended stimulus in extrastriate cortex.

147 rpatches differ in the output they convey to extrastriate cortex.

148 istribution of retrogradely labeled cells in extrastriate cortex.

149 uces expectation-driven selective biasing of extrastriate cortex.

150 related with increased volume in the lateral extrastriate cortex.

151 principle appears to lose prominence in the extrastriate cortex.

152 xtual cueing in V1, V2 and other portions of extrastriate cortex.

153 egregated M-P streams in four areas of human extrastriate cortex.

154 he functional organization in this region of extrastriate cortex.

155 istribution of retrogradely labeled cells in extrastriate cortex.

156 stellate cells, and interneurons within the extrastriate cortex.

157 ons and organization of topographic areas in extrastriate cortex.

158 also distinguish drivers from modulators in extrastriate cortex.

159 ific columns in early/middle stages of human extrastriate cortex.SIGNIFICANCE STATEMENT The magnocell

160 do category-selective regions arise in human extrastriate cortex?

161 The attenuated extrastriate cortical activation at baseline was increas

162 lateral geniculate nucleus directly with the extrastriate cortical area MT.

163 rns to measure the selectivity of neurons in extrastriate cortical area V2 of the macaque (Macaca nem

164 Extrastriate cortical areas are frequently composed of s

165 Studies in a wide range of extrastriate cortical areas have shown that single neuro

166 uperior temporal (MST) area: two neighboring extrastriate cortical areas of the monkey brain housing

167 ficant V1-independent fMRI activation in the extrastriate cortical areas V2, V3, V4, V5/middle tempor

168 forms extensive connections with striate and extrastriate cortical areas, but the impact of these con

169 the pulvinar nucleus can strongly influence extrastriate cortical circuits and exerts a particularly

170 Here we tested whether M-P streams exist in extrastriate cortical columns, in 8 human subjects (4 fe

171 cal function in ASDs may be characterized by extrastriate cortical hyperexcitability or differential

172 oposed existence of a subcortical pathway to extrastriate cortical motion areas (such as areas MT and

173 It has been suggested that a group of extrastriate cortical regions responding more strongly t

174 up and treatment effects was observed in two extrastriate cortical regions that showed physostigmine-

175 tory effects of attention on the strength of extrastriate cortical representations, and the control o

176 d territories indicated that identity of the extrastriate cortical target may be systematically relat

177 d regions of the visual field in striate and extrastriate cortices and cover anisotropic parts of vis

178 Since evidence suggests that extrastriate cortices do have such neurons, we discuss t

179 neurons, we discuss the possibility that the extrastriate cortices play no role in guiding exogenous

180 ron's optimal stimulus size at low contrast; extrastriate feedback connections to V1, instead, are lo

181 d with a decreased neuronal response in left extrastriate, left middle frontal, and left inferior fro

182 eld maps: compared to primary visual cortex, extrastriate maps generally have larger receptive field

183 visual cortex (simple and complex), and the extrastriate motion area MT.

184 ns in the primary visual cortex (V1) and the extrastriate motion area MT/V5 constitute a critical cha

185 es, suggesting more advanced development for extrastriate motion areas than form.

186 ure (second order Wiener-like kernels) in an extrastriate motion processing area (MT) of alert monkey

187 possibility of substantial reorganization of extrastriate networks between infancy and adulthood.

188 The results suggest that the degree to which extrastriate neurons can maintain functional connections

189 articularly strong impact on the activity of extrastriate neurons that project to the striatum and am

190 In sighted controls, most of extrastriate OC, including the MOG, was deactivated duri

191 asks within primary visual cortex, increased extrastriate occipital cortex activation selectively dur

192 nonemotional scenes approximately 1 s before extrastriate occipital cortex, whereas primary occipital

193 t rely on an alternative pathway directly to extrastriate occipital regions.

194 All effects localized to extrastriate occipitotemporal cortex.

195 A sequence of activation from V1/V2 to extrastriate, parietal, and frontal regions occurred wit

196 ulo-thalamo-amygdala and the retino-geniculo-extrastriate pathways, we propose that aberrant function

197 n retinotopic maps were identified in dorsal extrastriate, posterior parietal, and frontal cortex as

198 ng fed forward from primary visual cortex to extrastriate processing areas and to the motor output.

199 ndicating that only a subset of the geniculo-extrastriate projection falls within the K pathway as de

200 cally related to Cal content in the geniculo-extrastriate projection.

201 As in normal rats, striate-extrastriate projections in rats enucleated at birth con

202 t different ages on both the distribution of extrastriate projections originating from restricted loc

203 Anomalies in patterns of striate-extrastriate projections were not observed in rats enucl

204 visual information to V4 via direct thalamo-extrastriate projections.

205 ity in a network of regions that included an extrastriate region associated with object processing.

206 In the autism group this extrastriate region showed reduced functional connectivi

207 Nevertheless, an extrastriate region, such as the shape-selective lateral

208 hibited reduced functional connectivity with extrastriate regions during painful stimulation relative

209 ngs have identified functional properties of extrastriate regions in the ventral visual pathway that

210 elation between CT and drive for thinness in extrastriate regions involved in body perception.

211 similar to those caused by damage to monkey extrastriate regions V4 andor TEO, which are thought to

212 In most extrastriate regions, perifoveal pRFs were larger in the

213 emantics, blind subjects activate additional extrastriate regions, which are coupled with frontal and

214 ater overall capacity in the hippocampal and extrastriate regions.

215 nces were found in circumscribed frontal and extrastriate regions.

216 In an event-related analysis, extrastriate responses to PM trials were enhanced by nic

217 The findings suggest that contributions from extrastriate sources are greater with the cVEP paradigm

218 In this report, we demonstrate that extrastriate ventral area V4 contains a retinotopic sali

219 the activity of amygdala, MDN, pulvinar, and extrastriate ventral visual regions with fMRI as a group

220 connecting adjacent gyri; (iii) it arises in extrastriate visual 'association' areas; and (iv) it pro

221 properties of neurons in V1 and an adjacent extrastriate visual area (V2L) of anesthetized mice with

222 In extrastriate visual area MT of the rhesus macaque, for e

223 We used the responses of neurons in extrastriate visual area MT to determine how well neural

224 tic simulation of the population response in extrastriate visual area MT, the suboptimal decoder has

225 e sensory representation of visual motion in extrastriate visual area MT.

226 the LGN because disynaptic transport from an extrastriate visual area should require a relay through

227 cortex (V1) to beta oscillation dynamics in extrastriate visual area V4 of behaving monkeys.

228 nterestingly, other brain activities in both extrastriate visual areas (the P1 component) and in the

229 rtex (V1), a small number project instead to extrastriate visual areas and have been suggested to pla

230 ases neural responses to attended stimuli in extrastriate visual areas and, to a lesser degree, in ea

231 These results indicate that extrastriate visual areas are involved in the process of

232 can alter the baseline or gain of neurons in extrastriate visual areas but that it cannot change tuni

233 and low-level features increases activity in extrastriate visual areas even in the absence of a stimu

234 It seems likely that extrastriate visual areas further along the visual pathw

235 est that amplified cortical projections from extrastriate visual areas involved in visual motion proc

236 have been few studies of the development of extrastriate visual areas that integrate outputs from V1

237 primary visual cortex (V1) as well as higher extrastriate visual areas V2-V4, and moreover, reliably

238 microstimulation that LPP is connected with extrastriate visual areas V4V and DP and a scene-selecti

239 stic than probabilistic stimuli; conversely, extrastriate visual areas were more active for probabili

240 such filtering depends on top-down inputs to extrastriate visual areas, originating in structures imp

241 reased feedforward interactions with FEF and extrastriate visual areas, whereas identical stimulation

242 n V3 and V3A are similar to those from other extrastriate visual areas.

243 ected transynaptic rabies virus into several extrastriate visual areas.

244 were most strongly connected with early and extrastriate visual areas.

245 to gaze-centered coordinates in parietal and extrastriate visual areas.

246 asis of figure-ground segmentation, in early extrastriate visual cortex (area V2).

247 as well as category-selective regions of the extrastriate visual cortex (for example, fusiform gyrus

248 the primary auditory cortex from nonprimary extrastriate visual cortex (V2M, V2L) and from the multi

249 We find that in extrastriate visual cortex (V4), modulations related to

250 rons in the middle temporal (MT) area of the extrastriate visual cortex and are used to drive smooth

251 ically, and connectionally distinct areas of extrastriate visual cortex and that they are gateways fo

252 assessments of magnification factors in the extrastriate visual cortex and used these measures to co

253 li can reduce the firing rates of neurons in extrastriate visual cortex below the rate elicited by a

254 Extrastriate visual cortex both mediates the perception

255 cleation on the surface areas of striate and extrastriate visual cortex by using magnetic resonance i

256 anization of the two kinds of information in extrastriate visual cortex in humans.

257 different states that influence activity in extrastriate visual cortex in opposite directions: where

258 ity-dependent expression of Arc in the mouse extrastriate visual cortex in response to a structured v

259 otion-sensitive middle temporal area (MT) of extrastriate visual cortex instructs learning in smooth

260 Neuronal activity in area MT of the extrastriate visual cortex is correlated with the choice

261 medial superior temporal area (MSTd) in the extrastriate visual cortex is thought to play an importa

262 p that was localized to neural generators in extrastriate visual cortex of the ventral occipital lobe

263 th unilateral damage to different regions of extrastriate visual cortex on a series of visual discrim

264 onkeys found that inactivating feedback from extrastriate visual cortex produced effects in striate c

265 we show that traveling waves in the primate extrastriate visual cortex provide a means of integratin

266 cesses within the superior parietal lobe and extrastriate visual cortex that in turn modulate the rea

267 ural measure of processing within the dorsal extrastriate visual cortex that is thought to be particu

268 iously shown to be innervated by a region of extrastriate visual cortex thought to be concerned prima

269 l superior temporal area (MSTd), a region of extrastriate visual cortex thought to be involved in sel

270 anomalous and the sizes of both striate and extrastriate visual cortex were significantly reduced.

271 e time-varying signals that originate in the extrastriate visual cortex, accumulating evidence for or

272 efrontal cortex modulates sensory signals in extrastriate visual cortex, in part via its direct proje

273 ence of hierarchically ordered operations in extrastriate visual cortex, in which the selection based

274 l learning must operate quite effectively in extrastriate visual cortex, providing new hope and direc

275 n this article, we consider the test case of extrastriate visual cortex, where a highly systematic fu

276 superior temporal area (MSTd) of the macaque extrastriate visual cortex, which is thought to be invol

277 teral geniculate nucleus (LGN) indirectly to extrastriate visual cortex.

278 otopic modulation of preparatory activity in extrastriate visual cortex.

279 aused a reduction in the size of striate and extrastriate visual cortex.

280 were observed in the fusiform face area and extrastriate visual cortex.

281 techniques to map out and record from mouse extrastriate visual cortex.

282 top-down influences that can be measured in extrastriate visual cortex.

283 subcortical relay of retinal information to extrastriate visual cortex.

284 of visual processing, including striate and extrastriate visual cortex.

285 r regions of left hemispheric prefrontal and extrastriate visual cortex.

286 ing its effect on the activity of neurons in extrastriate visual cortex.

287 hypothesized to be due to network effects on extrastriate visual cortex.

288 ked P1 component, reflecting activity in the extrastriate visual cortex.

289 f 23 lesions were negatively correlated with extrastriate visual cortex.

290 c retinotopic atlas of occipital and ventral extrastriate visual cortex.

291 dynamics of neurons in both monkey and human extrastriate visual cortex.

292 light on the mechanisms of beta activity in extrastriate visual cortex: The preserved spontaneous os

293 cortex, the deep layers receive inputs from extrastriate visual cortical areas and from auditory, so

294 zheimer's disease is linked to a disorder of extrastriate visual cortical motion processing reflected

295 ng and late- and early-deafened cats from an extrastriate visual cortical region known to be involved

296 specific enhanced activation in striate and extrastriate visual cortical representations of the two

297 in left medial prefrontal cortex, bilateral extrastriate visual cortices, and cerebellum.

298 PMLS is also an extrastriate visual motion processing area and is widely

299 source of top-down signals that can modulate extrastriate visual processing in accordance with behavi

300 range of response in limbic-subcortical and extrastriate visual regions was evident in the depressed