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1 vimetric method and by direct measurement of extravascular lung water.
2 ting a 169 +/- 166 mL increase in nonindexed extravascular lung water.
3 was performed to record the value of indexed extravascular lung water.
4 iac index, pulmonary blood volume index, and extravascular lung water.
5 ema, which can be assessed by measurement of extravascular lung water.
6 tual extravascular lung water, and predicted extravascular lung water.
7 tment with intravenous beta-agonists reduces extravascular lung water.
8 ased alveolar permeability and contralateral extravascular lung water.
9 erval, 0.65-0.94) was larger than for actual extravascular lung water (0.72; confidence interval, 0.5
10 hough the area under the curve for predicted extravascular lung water (0.8; confidence interval, 0.65
11 for lung edema and has been shown to reduce extravascular lung water and improve lung function in mo
14 drome, to determine the relationship between extravascular lung water and other markers of lung injur
15 respiratory distress syndrome (ARDS) reduced extravascular lung water and plateau airway pressure.
17 , PaO2/Fio2 ratio, oxygenation index, actual extravascular lung water, and predicted extravascular lu
18 iple logistic regression analysis, predicted extravascular lung water but not actual extravascular lu
20 l showed a pattern consistent with increased extravascular lung water (diffuse, bilateral, symmetrica
21 predicted or actual body weight for indexing extravascular lung water does not lead to independence o
22 nd we quantified lung injury in terms of the extravascular lung water (EVLW) content, filtration coef
24 hypothesized that it could be improved using extravascular lung water (EVLWi) and plasma biomarkers o
25 o abrogate significantly the accumulation of extravascular lung water evoked by 6-hour exposure to en
26 ody weight, improves the predictive value of extravascular lung water for survival and correlation wi
27 atio were lower in patients with a change in extravascular lung water >/= 10% than in patients with a
30 lly contributed to prediction of a change in extravascular lung water >/= 10%, independent of the pre
33 lowing endotoxin-induced lung injury reduced extravascular lung water, improved lung endothelial barr
35 the low tidal volume group, as was decreased extravascular lung water in the uninstilled lung in the
40 was 70% in patients with a maximum value of extravascular lung water index >21 mL/kg and 43% in the
42 distress syndrome episode (maximum value of extravascular lung water index and maximum value of pulm
43 ght, p < 0.001 [t-test] for maximum value of extravascular lung water index and median [interquartile
44 rome might be associated with an increase in extravascular lung water index and pulmonary vascular pe
54 In multivariate analyses, maximum value of extravascular lung water index or maximum value of pulmo
60 taneous breathing trial-induced increases in extravascular lung water indexed for ideal body weight,
61 nary edema were 0.89 (95% CI, 0.78-0.99) for extravascular lung water indexed for ideal body weight,
62 ded pulmonary artery occlusion pressure, the extravascular lung water indexed for ideal body weight,
71 50 eligible patients, 132 patients (88%) had extravascular lung water indexed to predicted body weigh
77 e compared between patients with a change in extravascular lung water <10% and patients with a change
79 ding upper limits for fluid resuscitation of extravascular lung water (<10 mL/kg) and global end-dias
88 to predicted body weight, females had a mean extravascular lung water of 9.1 (SD=3.1, range: 5-23) mL
89 tion, intrapulmonary shunting, and increased extravascular lung water (p < .05 compared with baseline
90 ve analysis indicated that EVLWp, Vd/Vt, and extravascular lung water (p = .0005, .009, and .013, res
92 o detect small short-term changes of indexed extravascular lung water secondary to bronchoalveolar la
93 iately after bronchoalveolar lavage, indexed extravascular lung water significantly increased from 12
95 lung water is a better predictor than actual extravascular lung water to identify patients at risk fo
99 ribe and assess the clinical significance of extravascular lung water variations after pulmonary enda
100 cted extravascular lung water but not actual extravascular lung water was a predictor of mortality wi
103 bronchoalveolar lavage, the value of indexed extravascular lung water was significantly different fro
104 s of lung injury, and to examine if indexing extravascular lung water with predicted body weight (EVL
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