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1                              Using the human extravillous CTB cell line SGHPL-4, we examined the effe
2 d expression of stage-specific antigens that extravillous CTBs normally upregulate as they exit the p
3 s cytotrophoblast cells and chorion membrane extravillous cytotrophoblast cells contained mRNAs encod
4 NA and protein were expressed in villous and extravillous cytotrophoblast cells up to week 35 of gest
5 ost-mitotic syncytiotrophoblast and invasive extravillous cytotrophoblast cells.
6 eas m/sG2 protein was present exclusively in extravillous cytotrophoblast cells.
7 a disorder of pregnancy associated with poor extravillous cytotrophoblast invasion and above-normal r
8  placenta as well as syncytiotrophoblast and extravillous cytotrophoblast of normal third-trimester a
9 (NK) cell inhibitory ligand expressed on the extravillous cytotrophoblast of the human placenta.
10 lass I MHC molecule selectively expressed on extravillous cytotrophoblast; this cell type does not ex
11                                              Extravillous cytotrophoblasts (CTBs) fail to differentia
12 blast, neither eNOS or iNOS was expressed by extravillous cytotrophoblasts at any time during invasio
13                 During early human pregnancy extravillous cytotrophoblasts invade the uterus and also
14                 During early human pregnancy extravillous cytotrophoblasts invade the uterus and spir
15                     The expression of NOS on extravillous cytotrophoblasts was studied in placental b
16  the syncytiotrophoblast and the villous and extravillous cytotrophoblasts.
17 F did not alter proliferation, but initiated extravillous differentiation, with decreased alpha6 inte
18 ) is a gestational neoplasm derived from the extravillous (intermediate) trophoblast of the implantat
19 om a transporting epithelium to an invasive, extravillous phenotype that expresses a distinct reperto
20 ytiotrophoblast (STB) in chorionic villi and extravillous trophoblast (EVT) at the implantation site.
21 and preeclampsia, is the failure of invading extravillous trophoblast (EVT) cells to remodel the mate
22                                uNK cells and extravillous trophoblast (EVT) cells were isolated from
23 ost and replaced by fibrinoid, incorporating extravillous trophoblast (EVT) cells.
24                                        Human extravillous trophoblast (EVT) invades the decidua via i
25               We determined that transformed extravillous trophoblast (HTR-8/SVneo) cells were suscep
26                                              Extravillous trophoblast cell (EVT) invasion of decidua
27  that primary human trophoblast cells and an extravillous trophoblast cell line (HTR8), from first an
28                          Shallow invasion by extravillous trophoblast cells into the uterine wall red
29 temporally expressed only in the distal-most extravillous trophoblast cells, which represent a migrat
30 B, FOS-like (FOSL) 1, and FOSL2) proteins in extravillous trophoblast cells.
31  of first-trimester chorionic villi enhanced extravillous trophoblast differentiation and invasive ac
32                                              Extravillous trophoblast expression was determined by im
33 K cells have been demonstrated to facilitate extravillous trophoblast invasion into maternal decidua
34                                              Extravillous trophoblast invasion is a fundamental compo
35                It is associated with shallow extravillous trophoblast invasion of the decidua, leadin
36 f decidual macrophages implicated in shallow extravillous trophoblast invasion of the decidua.
37  promoting development of progenitors of the extravillous trophoblast lineage in the human placenta.
38 is exclusively detected in precursors of the extravillous trophoblast lineage, forming cell columns a
39 eptor promotes proliferation and survival of extravillous trophoblast progenitors.
40 trast, B7-H2 and B7-H3 were prominent on the extravillous trophoblast throughout gestation.
41 l studies to determine the susceptibility of extravillous trophoblast to other viruses, and the mecha
42 2 bases with (peri)nuclear expression in the extravillous trophoblast using strand-specific RT-PCR co
43  not HO-1 was detected on all populations of extravillous trophoblast, but expression of HO-2 or HO-1
44 ctivin/nodal signaling leads to formation of extravillous trophoblast, whereas loss of activin/nodal
45 d increased expression of HLA-G, a marker of extravillous trophoblast.
46 lly, these cells differentiate into invasive extravillous trophoblast.
47 decreases with differentiation into HLA-G(+) extravillous trophoblast.
48 cental bed but only TGF-beta2 was present in extravillous trophoblast.
49 ted in the cytotrophoblast and intermediate (extravillous) trophoblast of normal and molar placentas,
50 e (MAPK) in 84% of cases, whereas the normal extravillous trophoblastic cells did not.
51 t, neither compound had any effect on normal extravillous trophoblastic cells or JEG-3 cells.
52 ed to choriocarcinoma JEG-3 cells and normal extravillous trophoblastic cells.
53 Invading human leukocyte antigen-G+ (HLA-G+) extravillous trophoblasts (EVT) are rare cells that are
54 oxic decidual natural killer cells (dNK) and extravillous trophoblasts (EVT) at the maternal-fetal in
55       During pregnancy, semiallogeneic fetal extravillous trophoblasts (EVT) invade the uterine mucos
56 otrophoblasts in floating chorionic villi or extravillous trophoblasts (EVTs) at the anchoring villi.
57 d remodeling of maternal uterine arteries by extravillous trophoblasts (EVTs) in the first trimester
58 hat activation of PAR1 expressed by invasive extravillous trophoblasts (EVTs) influences human placen
59                                           In extravillous trophoblasts (EVTs), B. abortus and B. suis
60 o initiate remodeling before colonization by extravillous trophoblasts (EVTs); however, the trigger f
61                                   Developing extravillous trophoblasts accumulate heparin-binding EGF
62 ii) the maternal decidua, where mononuclear, extravillous trophoblasts anchor the villous region to t
63                                Fetal-derived extravillous trophoblasts come in direct contact with ma
64 iological expression restricted to placental extravillous trophoblasts contributes to maternal tolera
65                                     Invasive extravillous trophoblasts derived from cytotrophoblast p
66 enta and placental bed but not by villous or extravillous trophoblasts in normal or pathological samp
67 l immune and endothelial cells juxtaposed to extravillous trophoblasts in the uterine implantation si
68 n mother and fetus, progenitors develop into extravillous trophoblasts invading the maternal uterus a
69  In the pregnancy complication preeclampsia, extravillous trophoblasts invasion and vessel remodeling
70 lasts can terminally differentiate to either extravillous trophoblasts or syncytiotrophoblasts.
71 ne protease highly expressed in the invasive extravillous trophoblasts that invade decidua.
72 an MHC class Ib protein that is expressed on extravillous trophoblasts), LILRB1 on CD14(+) macrophage
73 d invasion and maternal vascular remodeling (extravillous trophoblasts, EVTs).
74 expression profiles of term intravillous and extravillous trophoblasts, including the transcriptome o
75 n of MIF expression in syncytiotrophoblasts, extravillous trophoblasts, IVB mononuclear cells, and am
76 d the expression of CD1d on both villous and extravillous trophoblasts, the fetal cells that invade t
77                            JEG-3 cells model extravillous trophoblasts, which predominate during the
78 n preeclamptic placentas, in first-trimester extravillous trophoblasts.
79  families decreased the invasion capacity of extravillous trophoblasts.
80 s in response to HCMV-infected primary fetal extravillous trophoblasts.
81 mors are thought to arise from intermediate (extravillous) trophoblasts based on histopathological st

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