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2 d expression of stage-specific antigens that extravillous CTBs normally upregulate as they exit the p
3 s cytotrophoblast cells and chorion membrane extravillous cytotrophoblast cells contained mRNAs encod
4 NA and protein were expressed in villous and extravillous cytotrophoblast cells up to week 35 of gest
7 a disorder of pregnancy associated with poor extravillous cytotrophoblast invasion and above-normal r
8 placenta as well as syncytiotrophoblast and extravillous cytotrophoblast of normal third-trimester a
10 lass I MHC molecule selectively expressed on extravillous cytotrophoblast; this cell type does not ex
12 blast, neither eNOS or iNOS was expressed by extravillous cytotrophoblasts at any time during invasio
17 F did not alter proliferation, but initiated extravillous differentiation, with decreased alpha6 inte
18 ) is a gestational neoplasm derived from the extravillous (intermediate) trophoblast of the implantat
19 om a transporting epithelium to an invasive, extravillous phenotype that expresses a distinct reperto
20 ytiotrophoblast (STB) in chorionic villi and extravillous trophoblast (EVT) at the implantation site.
21 and preeclampsia, is the failure of invading extravillous trophoblast (EVT) cells to remodel the mate
27 that primary human trophoblast cells and an extravillous trophoblast cell line (HTR8), from first an
29 temporally expressed only in the distal-most extravillous trophoblast cells, which represent a migrat
31 of first-trimester chorionic villi enhanced extravillous trophoblast differentiation and invasive ac
33 K cells have been demonstrated to facilitate extravillous trophoblast invasion into maternal decidua
37 promoting development of progenitors of the extravillous trophoblast lineage in the human placenta.
38 is exclusively detected in precursors of the extravillous trophoblast lineage, forming cell columns a
41 l studies to determine the susceptibility of extravillous trophoblast to other viruses, and the mecha
42 2 bases with (peri)nuclear expression in the extravillous trophoblast using strand-specific RT-PCR co
43 not HO-1 was detected on all populations of extravillous trophoblast, but expression of HO-2 or HO-1
44 ctivin/nodal signaling leads to formation of extravillous trophoblast, whereas loss of activin/nodal
49 ted in the cytotrophoblast and intermediate (extravillous) trophoblast of normal and molar placentas,
53 Invading human leukocyte antigen-G+ (HLA-G+) extravillous trophoblasts (EVT) are rare cells that are
54 oxic decidual natural killer cells (dNK) and extravillous trophoblasts (EVT) at the maternal-fetal in
56 otrophoblasts in floating chorionic villi or extravillous trophoblasts (EVTs) at the anchoring villi.
57 d remodeling of maternal uterine arteries by extravillous trophoblasts (EVTs) in the first trimester
58 hat activation of PAR1 expressed by invasive extravillous trophoblasts (EVTs) influences human placen
60 o initiate remodeling before colonization by extravillous trophoblasts (EVTs); however, the trigger f
62 ii) the maternal decidua, where mononuclear, extravillous trophoblasts anchor the villous region to t
64 iological expression restricted to placental extravillous trophoblasts contributes to maternal tolera
66 enta and placental bed but not by villous or extravillous trophoblasts in normal or pathological samp
67 l immune and endothelial cells juxtaposed to extravillous trophoblasts in the uterine implantation si
68 n mother and fetus, progenitors develop into extravillous trophoblasts invading the maternal uterus a
69 In the pregnancy complication preeclampsia, extravillous trophoblasts invasion and vessel remodeling
72 an MHC class Ib protein that is expressed on extravillous trophoblasts), LILRB1 on CD14(+) macrophage
74 expression profiles of term intravillous and extravillous trophoblasts, including the transcriptome o
75 n of MIF expression in syncytiotrophoblasts, extravillous trophoblasts, IVB mononuclear cells, and am
76 d the expression of CD1d on both villous and extravillous trophoblasts, the fetal cells that invade t
81 mors are thought to arise from intermediate (extravillous) trophoblasts based on histopathological st
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