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1 d increased expression of HLA-G, a marker of extravillous trophoblast.
2 lly, these cells differentiate into invasive extravillous trophoblast.
3 decreases with differentiation into HLA-G(+) extravillous trophoblast.
4 cental bed but only TGF-beta2 was present in extravillous trophoblast.
5 n preeclamptic placentas, in first-trimester extravillous trophoblasts.
6 families decreased the invasion capacity of extravillous trophoblasts.
7 s in response to HCMV-infected primary fetal extravillous trophoblasts.
9 ii) the maternal decidua, where mononuclear, extravillous trophoblasts anchor the villous region to t
10 mors are thought to arise from intermediate (extravillous) trophoblasts based on histopathological st
11 not HO-1 was detected on all populations of extravillous trophoblast, but expression of HO-2 or HO-1
13 that primary human trophoblast cells and an extravillous trophoblast cell line (HTR8), from first an
15 temporally expressed only in the distal-most extravillous trophoblast cells, which represent a migrat
18 iological expression restricted to placental extravillous trophoblasts contributes to maternal tolera
20 of first-trimester chorionic villi enhanced extravillous trophoblast differentiation and invasive ac
21 ytiotrophoblast (STB) in chorionic villi and extravillous trophoblast (EVT) at the implantation site.
22 and preeclampsia, is the failure of invading extravillous trophoblast (EVT) cells to remodel the mate
26 Invading human leukocyte antigen-G+ (HLA-G+) extravillous trophoblasts (EVT) are rare cells that are
27 oxic decidual natural killer cells (dNK) and extravillous trophoblasts (EVT) at the maternal-fetal in
29 otrophoblasts in floating chorionic villi or extravillous trophoblasts (EVTs) at the anchoring villi.
30 d remodeling of maternal uterine arteries by extravillous trophoblasts (EVTs) in the first trimester
31 hat activation of PAR1 expressed by invasive extravillous trophoblasts (EVTs) influences human placen
33 o initiate remodeling before colonization by extravillous trophoblasts (EVTs); however, the trigger f
37 enta and placental bed but not by villous or extravillous trophoblasts in normal or pathological samp
38 l immune and endothelial cells juxtaposed to extravillous trophoblasts in the uterine implantation si
39 expression profiles of term intravillous and extravillous trophoblasts, including the transcriptome o
40 n mother and fetus, progenitors develop into extravillous trophoblasts invading the maternal uterus a
41 K cells have been demonstrated to facilitate extravillous trophoblast invasion into maternal decidua
45 In the pregnancy complication preeclampsia, extravillous trophoblasts invasion and vessel remodeling
46 n of MIF expression in syncytiotrophoblasts, extravillous trophoblasts, IVB mononuclear cells, and am
47 an MHC class Ib protein that is expressed on extravillous trophoblasts), LILRB1 on CD14(+) macrophage
48 promoting development of progenitors of the extravillous trophoblast lineage in the human placenta.
49 is exclusively detected in precursors of the extravillous trophoblast lineage, forming cell columns a
50 ted in the cytotrophoblast and intermediate (extravillous) trophoblast of normal and molar placentas,
54 d the expression of CD1d on both villous and extravillous trophoblasts, the fetal cells that invade t
56 l studies to determine the susceptibility of extravillous trophoblast to other viruses, and the mecha
57 2 bases with (peri)nuclear expression in the extravillous trophoblast using strand-specific RT-PCR co
58 ctivin/nodal signaling leads to formation of extravillous trophoblast, whereas loss of activin/nodal
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